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1 gh multiple mechanisms, including effects on cell matrix interaction.
2 lar mechanism and function of Ambn in enamel cell-matrix interaction.
3 anisms for its role in tissue remodeling and cell-matrix interaction.
4 oproteins in vertebrates that broadly affect cell-matrix interaction.
5 uggest a role of PS1 in cell adhesion and/or cell-matrix interaction.
6 t of its phosphorylation is regulated by the cell-matrix interaction.
7 ss after liver injury, involves considerable cell-matrix interaction.
8 duced by anticancer agents via disruption of cell-matrix interaction.
9 d other genes involved in axon outgrowth and cell matrix interactions.
10 n alone, which is indicative of nanoparticle/cell matrix interactions.
11  matrix proteins and interference with renal cell-matrix interactions.
12 s in cell spreading and the tension state of cell-matrix interactions.
13 aminin at the cell surface, affecting normal cell-matrix interactions.
14 elial integrity when cells are denied proper cell-matrix interactions.
15 rane, particularly at sites of cell-cell and cell-matrix interactions.
16 nced proliferation and altered cell-cell and cell-matrix interactions.
17  result of competition between cell-cell and cell-matrix interactions.
18  help to dissect the role of this protein in cell-matrix interactions.
19 es a physiologically relevant model to study cell-matrix interactions.
20 ent that leads to dysregulated cell-cell and cell-matrix interactions.
21 ese features suggest a role in cell-cell, or cell-matrix interactions.
22  genetic programs and orchestrated cell-cell/cell-matrix interactions.
23  FAK was due to ECM remodeling and increased cell-matrix interactions.
24 me depends on the magnitude of cell-cell and cell-matrix interactions.
25 cellular sensing as well as in cell-cell and cell-matrix interactions.
26  cell movements depend on both cell-cell and cell-matrix interactions.
27 ly significant functions in cell-cell and/or cell-matrix interactions.
28 ar differentiation through its modulation of cell-matrix interactions.
29 , which is required for normal cell-cell and cell-matrix interactions.
30 ial cells, events that require regulation of cell-matrix interactions.
31  and subsequent impact on potential aberrant cell-matrix interactions.
32  tumor metastasis, leading to alterations in cell-matrix interactions.
33 ers of three major gene families involved in cell-matrix interactions.
34 rombospondin 2 (TSP2) regulates a variety of cell-matrix interactions.
35 mbrane proteins, which mediate cell-cell and cell-matrix interactions.
36  are dependent on dysregulated cell-cell and cell-matrix interactions.
37  does not depend on its ability to stabilize cell-matrix interactions.
38 oteinase expression, cell proliferation, and cell-matrix interactions.
39 hat must be accounted for when interrogating cell-matrix interactions.
40 CAM has been shown to modulate cell-cell and cell-matrix interactions.
41 t it may play an important role in cell-cell/cell-matrix interactions.
42  extracellular cues, including cell-cell and cell-matrix interactions.
43 ne-spanning molecule that may play a role in cell-matrix interactions.
44 predicted to be involved in cell-cell and/or cell-matrix interactions.
45 to have suppressing effects on cell-cell and cell-matrix interactions.
46  believed to play key roles in cell-cell and cell-matrix interactions.
47 nsformation of adjacent keratinocytes and on cell-matrix interactions.
48 oducts, has been implicated in cell-cell and cell-matrix interactions.
49 pillary morphogenesis involves cell-cell and cell-matrix interactions.
50 licating these proteins in the regulation of cell-matrix interactions.
51 or the beta3C integrin subunit in modulating cell-matrix interactions.
52 kine not previously known to be regulated by cell-matrix interactions.
53 esis has been a long-standing model to study cell-matrix interactions.
54 nd development, including mammary epithelial cell-matrix interactions.
55 ptors appear to have functional roles in the cell-matrix interactions.
56 by exogenous mitogens and both cell-cell and cell-matrix interactions.
57 egulation of actin cytoskeleton dynamics and cell-matrix interactions.
58  as CDH2, thus affecting cell morphology and cell-matrix interactions.
59 nrichment of glycolysis, focal adhesion, and cell-matrix interactions.
60 lly isotropic matrix via locally reinforcing cell-matrix interactions.
61 ating physiologically relevant cell-cell and cell-matrix interactions.
62 th the integrity of extracellular matrix and cell-matrix interactions.
63 ember of the proteoglycan family involved in cell-matrix interactions.
64 that early and late amelogenesis depend upon cell-matrix interactions.
65 sion-related genes, disrupting cell-cell and cell-matrix interactions.
66 n domains capable of mediating cell-cell and cell-matrix interactions.
67  time of cuticle secretion and remodeling of cell-matrix interactions.
68 une system functioning through cell-cell and cell-matrix interactions.
69 ar differentiation through its modulation of cell-matrix interactions.
70  of uPA overexpression on cell migration and cell-matrix interactions.
71 sion molecules that direct key cell-cell and cell-matrix interactions.
72  as key regulators of cellular functions and cell-matrix interactions.
73 the cells it carries, promoting advantageous cell-matrix interactions.
74  of adherens junctions, tight junctions, and cell-matrix interactions.
75 cell proliferation, and altered cell-cell or cell-matrix interactions.
76 in growth factor signaling and cell-cell and cell-matrix interactions.
77 sion/growth-regulatory galectins within cell-cell/matrix interactions.
78 reduced motility and altered cell - cell and cell - matrix interactions.
79 ation of most cells is strictly dependent on cell-matrix interactions, a phenomenon called anchorage
80 athogenic viral strains alters cell-cell and cell-matrix interactions, affecting extracellular matrix
81  of how context, in particular cell-cell and cell-matrix interactions, affects endothelial cell respo
82 ell survival signals caused by disruption of cell--matrix interactions and contributes to anoikis res
83 MAGP-2 interacts with Jagged-1 that controls cell-matrix interaction and cell motility, two key facto
84                                Cell-cell and cell-matrix interaction and cell spreading are reduced w
85 ation, lineage determination, cell adhesion, cell-matrix interaction and cytoskeleton remodeling.
86 kinase (ILK) has been implicated in podocyte cell-matrix interaction and is induced in proteinuria.
87  in the development of colitis by modulating cell-matrix interaction and wound healing.
88  a variety of biological processes including cell-matrix interactions and activation of chemokines, e
89 f collagen fibrils play an important role in cell-matrix interactions and are a manifestation of thei
90 ein 15 (LRRC15) is involved in cell-cell and cell-matrix interactions and came into focus as a promis
91 ndins (TSPs) are extracellular regulators of cell-matrix interactions and cell phenotype.
92 serine/threonine kinase that is important in cell-matrix interactions and cell signaling.
93 ted deregulation of epithelial cell-cell and cell-matrix interactions and cohesion of epithelial stru
94 es (ECMs), providing new pathways to explore cell-matrix interactions and direct cell fate under phys
95 s and the extracellular matrix (ECM) through cell-matrix interactions and ECM remodelling.
96 cell morphology, cell-cell interactions, and cell-matrix interactions and eventually result in the fo
97 dhesion receptors mediate both cell-cell and cell-matrix interactions and have been shown to play vit
98   These findings have provided insights into cell-matrix interactions and how these interactions diff
99 iochemistry of matrix components affects the cell-matrix interactions and how these interactions vary
100 ein that has been implicated in cell-cell or cell-matrix interactions and in the proteolysis of molec
101 etween tumor and normal tissue will decrease cell-matrix interactions and inhibit matrix cross-linkin
102  and myocardial dysfunction, but the role of cell-matrix interactions and integrins in this process h
103 nctions, including maintenance of epithelial cell-matrix interactions and intestinal homeostasis.
104 that matrix glycation interferes with normal cell-matrix interactions and intracellular signaling tha
105 paran sulfate proteoglycan that can regulate cell-matrix interactions and is enriched in focal adhesi
106 he inhibition of the AGE-RAGE axis to resume cell-matrix interactions and maintain tissue integrity.
107         Furthermore, SAM68 depletion impacts cell-matrix interactions and motility through induction
108 ole of the newly deposited matrix protein in cell-matrix interactions and remodeling.
109 e other transglutaminases, it is involved in cell-matrix interactions and serves as an adhesion co-re
110 xL, Bak, and Bax) or mediators of epithelial cell-matrix interactions and survival (e.g., alpha5beta1
111 imulates the localization of ERK to sites of cell-matrix interactions and that this is mediated by th
112 nto banded fibers is essential for mediating cell-matrix interactions and tissue mechanics.
113  and rich in cysteine (SPARC) is critical in cell-matrix interactions and tissue remodeling.
114 the immediate microenvironment (cell-cell or cell-matrix interactions) and the extended tumour microe
115 defy anoikis, cell death caused by a lack of cell-matrix interaction, and grow in an anchorage-indepe
116 a membrane protein involved in cell-cell and cell-matrix interactions, and 3' sequences from the huma
117 t a variety of stimuli, including cytokines, cell-matrix interactions, and challenge with foreign mat
118 gnaling critical to intercellular junctions, cell-matrix interactions, and cytoskeletal regulation.
119  breast epithelial-endothelial cell-cell and cell-matrix interactions, and examined the expression of
120 al alterations in keratinocyte cell-cell and cell-matrix interactions, and extensive remodeling of th
121  MMP-9 promotes neuronal death by disrupting cell-matrix interactions, and MMP-9 knockout mice have r
122  events, including cell-cell communications, cell-matrix interactions, and response to environmental
123 of drug and nutrient diffusion, insufficient cell-matrix interactions, and tedious fabrication proced
124 onclude that TSP2 plays an important role in cell-matrix interactions, and that a deficiency in the p
125  motifs indicative of a role in cell-cell or cell-matrix interactions, and the PKD2 encodes a protein
126 on, cytoskeleton organization, cell-cell and cell-matrix interactions, apoptosis, cell cycle, and oxi
127 he engagement of the alphavbeta3 integrin in cell-matrix interactions appears to coordinate an intens
128 ffects of MMAC1 on motility, cell - cell and cell - matrix interactions are due to its tumor suppress
129 's effects on cell motility, cell - cell and cell - matrix interactions are thought to be due to its
130             The less well organized sites of cell-matrix interaction are sufficient for translocating
131 zation of cells into tissues, and defects in cell-matrix interactions are an important element in man
132                   For example, cell-cell and cell-matrix interactions are crucial for leukocyte homin
133                                 Furthermore, cell-matrix interactions are dynamic owing to matrix rem
134 or cells (LEPC) that incorporate the in vivo cell-matrix interactions are essential to enhance LEPC e
135                                              Cell-matrix interactions are frequently mediated by the
136    Together, our data suggest that defective cell-matrix interactions are linked to Wnt signaling and
137                                              Cell-matrix interactions are made possible by integrins,
138                                Cell-cell and cell-matrix interactions are of utmost importance in the
139 CM), yet the pathogenic mechanisms involving cell-matrix interactions are poorly understood.
140       These results show that alterations in cell-matrix interactions are sufficient to initiate and
141                    Although it is known that cell/matrix interactions are essential for directional m
142 esults shed light on the dynamics of diverse cell-matrix interactions as drivers of biofilm developme
143 alloproteinase ADAM9 modulates cell-cell and cell-matrix interactions as well as ectodomain shedding
144 ystic kidney disease (ADPKD), is involved in cell-matrix interactions as well as in ciliary signaling
145  These 3D cultures incorporate cell-cell and cell-matrix interactions, as well as diffusion dynamics
146 l revealed numerous defects in cell-cell and cell-matrix interactions, as well as disruption of cell
147  potential novel regulators of cell-cell and cell-matrix interactions, as well as of matrix degradati
148 ossesses multifaceted roles in modulation of cell-matrix interactions, as well as tumor growth and me
149  factor in preventing CD44- and HA-dependent cell-matrix interactions at the inflammatory site.
150                         The role of integrin/cell matrix interactions between the RPE and the basemen
151 ent-derived cells identify integrin-mediated cell-matrix interactions between MERTK+ macrophages and
152 d in a wide range of cell-cell signaling and cell-matrix interactions, both in vitro and in vivo in i
153 in epithelial cells is generated not only by cell-matrix interaction but also by cell-cell interactio
154 differentiation is likely to involve altered cell-matrix interactions but the mechanism is not known.
155     Embryogenesis is guided by cell-cell and cell-matrix interactions, but it is unclear how these ph
156 er, our results suggest that Tmem2 regulates cell-matrix interactions by affecting both ECM organizat
157 an Arg-Gly-Asp (RGD) motif needed to mediate cell-matrix interactions by binding to cell-surface inte
158 pe and function in response to cell-cell and cell-matrix interactions by guiding cytoskeletal structu
159 ural plasticity by altering cell-cell and/or cell-matrix interactions by increasing intermolecular sp
160 l migration, and show that PAI-1 can control cell-matrix interactions by regulating the accessibility
161                                   Mechanical cell-matrix interactions can drive the innate immune res
162  is enriched for genes with defined roles in cell-matrix interactions, cell motility, and endocytosis
163  (MMP1, MMP3, TIMP1, uPA, and PAI-1); and 4) cell-matrix interactions (Col1alpha1, Col1alpha2, and in
164 rface association of activated MMP-2, on the cell-matrix interactions controlled by collagen-specific
165 acellular motifs indicative of cell-cell and cell-matrix interactions, coupled through multiple trans
166          Each of these steps is regulated by cell-matrix interactions, cytokine and growth factor sec
167                           ILK is involved in cell-matrix interactions, cytoskeletal organization, and
168 ariety of processes, including cell-cell and cell-matrix interactions, cytoskeletal remodeling, prote
169 hyaluronan is of low affinity, and effective cell/matrix interaction depends on multiple interactions
170  and migration in ADA-GEL with cell-cell and cell-matrix interaction, dissimilarly to Alg/HA/Gel, in
171 es: not only proliferation but cell-cell and cell-matrix interactions, DNA repair, invasion and motil
172 our of mono-dispersed epithelial cells where cell-matrix interactions dominate and hinder formation o
173 a indicate that GRIP1 is required for normal cell-matrix interactions during early embryonic developm
174  AI and further highlights the importance of cell-matrix interactions during enamel formation.
175 t participates in a variety of cell-cell and cell-matrix interactions during fertilization and develo
176  that the invasive GBM cells rely heavily on cell-matrix interactions during invasion and remodeling.
177              Integrins are key regulators of cell-matrix interactions during joint development and jo
178 impact of transmembrane protein 2 (tmem2) on cell-matrix interactions during muscle morphogenesis in
179 n important role in modulating cell-cell and cell-matrix interactions during nervous tissue histogene
180 ry rather than structural role in modulating cell-matrix interactions during normal development and r
181 te Elements for Biomechanics) that simulates cell-matrix interactions during sprouting angiogenesis.
182 dulatory proteins are important effectors of cell-matrix interactions during tissue remodeling and re
183 es associated with glycolysis, angiogenesis, cell-matrix interaction, epithelial to mesenchymal trans
184 Our recent efforts have focused on the tumor cell-matrix interactions essential to tumor cell activat
185 vidence that Nck directs the polarization of cell-matrix interactions for efficient migration in thre
186 to overcome difficulties in tracking stromal cell-matrix interactions for several days in live mice.
187 ithelial cells are dependent on cell-cell or cell-matrix interactions for survival and undergo apopto
188 lycan that co-operates with integrins during cell-matrix interactions for the assembly of focal adhes
189 ion to the well-recognized, force-regulated, cell-matrix interactions, forces also tune the interacti
190 ydrogels to recapitulate the composition and cell-matrix interactions found in the native microenviro
191 ling as biofilms age, shifting the nature of cell-matrix interactions from attractive to repulsive an
192 s achieved, in part, via protection of renal cell-matrix interactions from damage by a variety of RCS
193 e a rare population defined by expression of cell-matrix interaction genes.
194 teases in intact tissues where cell-cell and cell-matrix interactions had been maintained.
195  ventricular myocytes, yet the importance of cell-matrix interactions has not been extensively examin
196 ar protein with a primary role in modulating cell-matrix interactions, has been implicated in tissue
197                                              Cell-matrix interactions have critical roles in regenera
198 d alginate for mechanical support and better cell-matrix interaction; histidine-grafted hyaluronic ac
199 mal tubule epithelial cell and cell-cell and cell-matrix interactions important for this repair funct
200 rful model system to study integrin-mediated cell-matrix interaction in an in vivo context, as it is
201 that IGF signaling is masked by signals from cell-matrix interaction in anchorage-dependent condition
202   We provide evidence that decorin modulates cell-matrix interaction in this context by stimulating c
203                                              Cell-matrix interactions in 3D environments significantl
204  cell fate is contextual and is modulated by cell-matrix interactions in a cell type-specific manner.
205  (alpha and beta), is involved in regulating cell-matrix interactions in a variety of tissues, includ
206 rins and laminins are important mediators of cell-matrix interactions in both vertebrates and inverte
207 odel that can be used to address the role of cell-matrix interactions in cartilage homeostasis and os
208 ortant role for collagen XVIII/endostatin in cell-matrix interactions in certain tissues that may be
209   These studies underscore the importance of cell-matrix interactions in controlling cardiac gene exp
210 onnective tissues, and the key to unraveling cell-matrix interactions in diseases.
211                       To address the role of cell-matrix interactions in gland development, pellets c
212 ts on cell growth, apoptosis, cell-cell, and cell-matrix interactions in immortalized human thyroid c
213 ensional (3D) configuration of cell-cell and cell-matrix interactions in native tissue can deliver ph
214 osis of NRP1 and a5/B1-integrins to modulate cell-matrix interactions in response to intrinsic and ex
215 proteases, and structural proteins influence cell-matrix interactions in the context of left ventricu
216 beta signaling that influences cell-cell and cell-matrix interactions in the developing nervous syste
217 the Hep II domain of fibronectin could alter cell-matrix interactions in the TM and provides an inter
218                         Here, we investigate cell-matrix interactions in Vibrio cholerae, the causati
219 lts suggest that TGFBIp may modulate scleral cell-matrix interactions in vivo, thereby affecting scle
220   Recent research advances in characterizing cell-matrix interactions include detailed descriptions o
221 ly regulated genes include those involved in cell-matrix interactions including proline 4-hydroxylase
222 last-selective Smo dictates AKI fate through cell-matrix interactions, including nidogen-1, and offer
223  receptor c-met results in the regulation of cell-matrix interactions, including the MAPK-dependent s
224                              Whether and how cell-matrix interactions influence virus particle uptake
225                   Additionally, it modulates cell-matrix interactions, influencing cell adhesion and
226                         Presumably an intact cell-matrix interaction inhibits caspase activation and
227   We provide evidence that integrin-mediated cell-matrix interaction is a key player in pluripotency
228 axillin-dependent ERK activation at sites of cell-matrix interaction is critical for HGF-stimulated e
229 dation, a minimal network model of a dynamic cell-matrix interaction is proposed.
230 ch regulated interplay between cell-cell and cell-matrix interactions is likely to have wide relevanc
231  integrin-linked kinase (ILK), a mediator of cell-matrix interactions, is indispensable for retinal a
232 ecreted protein that regulates cell-cell and cell-matrix interactions, leading to alterations in cell
233 ing their intercellular contacts in favor of cell-matrix interactions, leading to Yap nuclear translo
234 nges from dendritic to stellate/bipolar, and cell-matrix interactions mature from punctate to focal a
235 gest that Jagged regulation of cell-cell and cell-matrix interactions may contribute to the control o
236                     Recent studies show that cell-matrix interactions mediated by integrins can trans
237                                              Cell-matrix interactions, mediated by cellular force and
238                   However, the cell-cell and cell-matrix interactions necessary for re-creating the n
239 faces, these fibrous materials recapitulated cell-matrix interactions observed with collagen matrices
240 tivity and dysregulates the cell-cell and/or cell-matrix interactions of infected cytotrophoblasts an
241 and motility as well as affect cell-cell and cell-matrix interactions of malignant human glioma cells
242   By reproducing physiological cell-cell and cell-matrix interactions of the native niche environment
243 as the regulatory influence of cell-cell and cell-matrix interactions on signaling outcomes.
244 ing of ITGB6 resulting in either compromised cell-matrix interaction or compromised ITGB6 activation
245 g drug studies with quantitative analysis of cell-matrix interactions, our results are able to provid
246 teins are involved in neuronal cell-cell and cell-matrix interactions, particularly in the developing
247 d in known proteins involved in cell-cell or cell-matrix interactions, PKD2 has homology to PKD1 and
248                                Cell-cell and cell-matrix interactions play a critical role in tissue
249  membrane matrix integrity, composition, and cell-matrix interactions play an important role in ancho
250                                Cell-cell and cell-matrix interactions play important regulatory roles
251                            Integrin-mediated cell-matrix interactions play important roles in regulat
252                      Many factors, including cell-matrix interactions, play a role in the development
253      Rationale: Mechanical signaling through cell-matrix interactions plays a major role in progressi
254 cognition, nutrient transport, and cell-cell/cell-matrix interactions plays an absolutely critical ro
255 Cdc42 deficiency leads to a defect in global cell-matrix interactions reflected by a decrease in coll
256 tudies provide a better understanding of how cell-matrix interactions regulate angiogenesis and, ther
257                                              Cell-matrix interactions regulate chondrocyte differenti
258 derstanding the molecular mechanisms whereby cell-matrix interactions regulate liver regeneration may
259 glycoprotein implicated in intercellular and cell matrix interactions, regulation of the complement s
260  sustain oncogenic signals in the absence of cell-matrix interactions remains poorly understood.
261 h of which would be predicted to compete for cell-matrix interactions, resulted in a dose-dependent r
262 ing septation may cause loss of cell-cell or cell-matrix interactions, resulting in apoptosis (anoiki
263 ely activated RhoA protein that disrupts the cell-matrix interaction results in dephosphorylation of
264 array analysis were involved in cell-cell or cell-matrix interaction, Rho signaling, calcium homeosta
265 inds calcium and collagens, and can modulate cell-matrix interactions, so altering cell shape, growth
266 lation of ICAP-1alpha phosphorylation by the cell-matrix interaction suggests an important role of IC
267 oenvironment with a balance of cell-cell and cell-matrix interactions supports distinctive phenotypes
268     The defect is associated with an altered cell-matrix interaction that is evident by morphologic c
269                These effects are opposite to cell-matrix interactions that are strengthened by proton
270                  To explore the signals from cell-matrix interactions that control orientation of cel
271 late the spatial conformation, cell-cell and cell-matrix interactions that exist in vivo and in patie
272 ment of physiological cell-cell contacts and cell-matrix interactions that lessen their dependence on
273   Langerin may have an unanticipated role in cell-matrix interactions that modulate LC development, l
274 TG2) and fibronectin (FN) is involved in the cell-matrix interactions that regulate cell signaling, a
275 GF) is a matricellular protein that mediates cell-matrix interaction through various subtypes of inte
276 single-cell migration with matrix fibers and cell-matrix interactions through contact guidance and ma
277                              In these cells, cell-matrix interactions through integrins are required
278 rlooked is mechanical force, which regulates cell-matrix interactions through intracellular focal adh
279 ovium, plays a central role in cell-cell and cell-matrix interactions through ligation of cell surfac
280 s have the potential to be key modulators of cell-matrix interactions through the activities of their
281 anisms of resistance involving cell-cell and cell-matrix interactions, thus accelerating preclinical
282 les, but rather regulate matrix proteins and cell-matrix interactions to influence normal cellular fu
283 ignaling by growth factors and cell-cell and cell-matrix interactions to prevent apoptosis, senescenc
284 l pathways that transmit signals from proper cell-matrix interactions to promote cell survival.
285 ents, we studied formation and maturation of cell-matrix interactions under conditions in which we co
286       Relevance of matrix viscoplasticity to cell-matrix interactions was established through a quant
287 de-binding protein involved in cell-cell and cell-matrix interactions, was recently shown to be a tum
288               To acquire information on this cell-matrix interaction, we developed a technique for co
289 ar mechanisms responsible for these abnormal cell-matrix interactions, we compared the levels of matr
290 e regulation of the paracellular pathway and cell-matrix interactions, we studied whether protein com
291                However, several mediators of cell-matrix interaction were reduced in the Tgfbr2(derma
292                                Cell-cell and cell-matrix interactions were quantified and correlated
293 tein, polycystin-1, involved in cell-cell or cell-matrix interactions, whereas the PKD2 gene product,
294 e embryonic heart, and suggest that specific cell-matrix interactions which facilitate cell migration
295 tated by a precise sequence of cell-cell and cell-matrix interactions, which are mediated in part thr
296 unctions that involve numerous cell-cell and cell-matrix interactions, which ultimately mediate the h
297 ithin the integrin-based adhesome that links cell-matrix interactions with the tissue-specific functi
298 1 is an important regulator of cell-cell and cell-matrix interactions, with important roles in nerve
299 l the cell type, organization, and cell-cell/cell-matrix interactions within these systems.
300 d rich in cysteine) is a protein involved in cell matrix interactions, wound repair, and cell migrati

 
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