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1 e that arises from the head-tail symmetry of cell-to-cell interaction).
2 a process that facilitates immune evasion or cell to cell interaction.
3 that ECs can directly activate basophils via cell-to-cell interaction.
4 ors, while the CD8+ T cell response required cell-to-cell interaction.
5 hat influence survival, cell morphology, and cell-to-cell interaction.
6 des present on adjacent cells, thus creating cell to cell interactions.
7 ation during PMN vascular as well as mucosal cell to cell interactions.
8 ass of biological systems exhibiting typical cell to cell interactions.
9 se to the theoretically calculated limit and cell-to-cell interactions.
10 tumor microenvironmental patterns, including cell-to-cell interactions.
11 numerous cellular activities, processes, and cell-to-cell interactions.
12 ems that collectively make decisions through cell-to-cell interactions.
13 population structure and closer interspecies cell-to-cell interactions.
14 ear cells (PBMCs) in order to study in vitro cell-to-cell interactions.
15 feration and differentiation, through direct cell-to-cell interactions.
16 ion via stimulating both cell-to-surface and cell-to-cell interactions.
17            These changes implicate extensive cell-to-cell interactions.
18 release and CD4+ T cell proliferation during cell-to-cell interactions.
19 tracellular matrix components, bacteria tune cell-to-cell interactions.
20 hibit decreased migration speed and impaired cell-to-cell interactions.
21 nformation about cellular subpopulations and cell-to-cell interactions.
22 of cell surface receptors as well as general cell-to-cell interactions.
23 mpassing proliferation, differentiation, and cell-to-cell interactions.
24 supported by various types of spatiotemporal cell-to-cell interactions.
25 is, including cytokines, growth factors, and cell-to-cell interactions.
26 ned influence of hydrodynamic and mechanical cell-to-cell interactions.
27 s a complex network of soluble mediators and cell-to-cell interactions allowing human classically act
28                Interestingly, alterations of cell-to-cell interactions among the immune sub-populatio
29                                 An in silico cell-to-cell interaction analysis highlights the CXCL9/C
30  cell cultures do not resemble intercellular cell-to-cell interaction and communication; however, thr
31 on as a unique signaling system triggered by cell-to-cell interaction and have been shown to mediate
32  a necessity when considering characterizing cell-to-cell interaction and rolling dynamics during cel
33 ecies that colonize human host via extensive cell-to-cell interactions and biofilm formation.
34 ts: DE due to pathology, anatomical regions, cell-to-cell interactions and cellular microenvironment.
35 trafficking of cells that affect the dynamic cell-to-cell interactions and determine the outcome of i
36 lates endothelial cytoskeletal architecture, cell-to-cell interactions and lumen formation.
37 nd the tumor microenvironment is mediated by cell-to-cell interactions and soluble factors.
38                This might influence directly cell-to-cell interactions and the community effects with
39 t regulate growth, transcription regulation, cell to cell interactions, and morphology.
40  chemokines, CCL5 and CCL6, was dependent on cell-to-cell interaction, and was observed only with fib
41 ned our understanding of tissue composition, cell-to-cell interactions, and cell signaling.
42 ell covariance and conditional distribution, cell-to-cell interactions, and short/long-scale arrangem
43 chanisms of inter-leukemic communication and cell-to-cell interactions are proposed to be important f
44 hich fail to recapitulate spatial aspects of cell-to-cell interactions as well as tissue gradients pr
45 detect and define, and make inferences about cell-to-cell interactions at single-cell resolution.
46                    Microenvironment cues and cell-to-cell interactions balance stem cell quiescence w
47 ed a computational method to infer potential cell-to-cell interactions based on transcriptome data of
48 mily of proteins that are thought to mediate cell to cell interactions between neurons.
49 bel-free, and noninvasive probing of dynamic cell-to-cell interactions between adherent and nonadhere
50                     These findings show that cell-to-cell interactions between intraepithelial gammad
51                                              Cell-to-cell interactions between intraepithelial lympho
52  local non-MHC stimulatory factors or direct cell-to-cell interactions between the T cells themselves
53 plethora of signalling processes influencing cell-to-cell interactions between the vascular endotheli
54 istance, are required for citrate-stimulated cell-to-cell interactions, but the GraRS regulatory syst
55 s and can alter the microenvironment through cell-to-cell interactions by fusion with the plasma memb
56 s establishing the theoretical basis for how cell-to-cell interactions can influence population level
57 scriptomic analyses revealed that sequential cell-to-cell interactions direct functional maturation o
58 ng a unique insight into the dynamics of how cell-to-cell interaction drives intratumor heterogeneity
59  stem-cell behavior during its migration and cell-to-cell interaction during the process of homing.
60 propose that alpha-hemolysin is required for cell-to-cell interactions during biofilm formation.
61 f secreted signaling molecules that regulate cell-to-cell interactions during development.
62 ss, the implications of matrix plasticity on cell-to-cell interactions during EC outgrowth, along wit
63 ller incorporates both short- and long-range cell-to-cell interactions (e.g. via paracrine and endocr
64 odel predicts three possible benefits of the cell-to-cell interactions: First, the asymmetric interac
65 s and coupled analytical methods can recover cell-to-cell interactions, groups of spatially covarying
66                     Furthermore, analyses of cell-to-cell interactions highlight different networks o
67 is important in establishing and maintaining cell to cell interactions in epithelial cells, thereby p
68 active eicosanoids that can be formed during cell to cell interactions in human tissues to self limit
69 e characterization of immune populations and cell-to-cell interactions in granular detail.
70  has become a widely used technique to study cell-to-cell interactions in living organisms.
71 spatiotemporal gene expression landscape and cell-to-cell interactions in their native microenvironme
72 identified that NOTCH signaling drives these cell-to-cell interactions in tumors, with pericytes and
73 , however, bypasses the normal mechanisms of cell-to-cell interactions in which Notch-1 participates.
74      Mechanical forces are also essential in cell-to-cell interactions, including the widespread inte
75 raded when derived from a model that ignored cell-to-cell interactions, indicating the importance of
76  expression analyses, pathway enrichment and cell-to-cell interaction inferences are more accurate.
77 function and that this may be largely due to cell-to-cell interactions mediated by P-selectin.
78                      We propose that besides cell-to-cell interaction-mediated Fas triggering, stimul
79                                              Cell-to-cell interaction modeling and assays in vitro de
80 tic Elucidation and Assessment of Regulatory Cell-to-cell Interaction Networks (SEARCHIN) to identify
81            Treatment with ZD1839 reduced the cell-to-cell interaction of HUVECs, resulting in disrupt
82 e three-dimensional spatial organization and cell-to-cell interactions of brain tissue that are presu
83 ine how alterations in cellular function and cell-to-cell interactions of pulmonary resident cells an
84 to better understand the composition and the cell-to-cell interactions of the alloreactive inflammato
85               For the first time, the direct cell-to-cell interactions of tumor cells with bead-activ
86 ollowed by a relatively slower step in which cell-to-cell interactions predominate (the aggregation p
87                                 By analyzing cell-to-cell interaction profiles of the mouse developin
88 ), through deregulation of the expression of cell-to-cell interaction proteins and protein secretion.
89 gth and whether pilus length is critical for cell-to-cell interactions remain unknown.
90 d of the macroscopic patterns resulting from cell-to-cell interactions remains largely qualitative.
91 n that the bystander phenomenon must involve cell-to-cell interactions, the relevance of such single-
92 omposition, transcriptional cell states, and cell-to-cell interactions, thereby providing insight int
93 rs cell proliferation, death, migration, and cell-to-cell interaction through contact inhibition.
94    This antiapoptotic effect required direct cell-to-cell interactions, was associated with phosphory
95 bind avidly to C. albicans hyphae via direct cell-to-cell interaction, while the cariogenic pathogen
96 hat their morphology is sculpted by specific cell to cell interactions with neurons and each other.
97                        OC formation requires cell-to-cell interactions with osteoblasts and can be ac
98 evidence that migratory DCs execute targeted cell-to-cell interactions with stationary MCs before lea