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1 sociated glycoprotein (MAG), and neuron-glia cell adhesion molecule (L1).
2 ders (FASD) in part by disrupting the neural cell adhesion molecule L1.
3 motoneuron axons they encounter express the cell adhesion molecule L1.
4 Many of these axons expressed the cell adhesion molecule L1.
5 ion relative to axon tracts that express the cell adhesion molecule L1.
6 cell adhesion mediated by the immunoglobulin cell adhesion molecule L1.
7 children with mutations in the gene for the cell adhesion molecule L1.
8 entifying beneficial functions of the neural cell adhesion molecule L1.
9 lar neurons as novel binding partners of the cell adhesion molecule L1.
10 h two tyrosine kinases, Fyn and Lyn, and the cell adhesion molecule, L1.
11 that the expression of the glycoprotein and cell adhesion molecule L1, a member of the immunoglobuli
13 ntially relevant to skin homeostasis: neural cell adhesion molecule L1 and dipeptidyl peptidase 4.
15 ellular sources in lesion sites produced the cell adhesion molecules L1 and laminin, and these molecu
18 ar levels of Sema3A receptors (neuropilin-1, cell adhesion molecule L1, and plexinA4), but significan
19 ited myelination decreased expression of the cell adhesion molecule L1, and stimulation under conditi
22 alpha v beta 3 can interact with the neural cell adhesion molecule L1-CAM; a member of the immunoglo
23 embly of Nav1.6, betaIV spectrin, and the L1 cell adhesion molecules (L1 CAMs) neurofascin and NrCAM
24 entalization, we focused on two L1 family of cell adhesion molecules (L1-CAMs) [L1/neuron-glia cell a
25 evelopmental spine pruning through L1 family cell adhesion molecules (L1-CAMs) and class 3 Semaphorin
26 that promotes neurite growth on the neuronal cell adhesion molecule L1, did not interact with any NCA
27 expression of polysialic acid (PSA) and the cell adhesion molecule L1 during axonal regeneration and
29 rowth-associated protein 43 (GAP-43) and the cell adhesion molecule L1 has been correlated with CNS a
33 tor, we expressed the regeneration-promoting cell adhesion molecule L1 in both neurons and glia in th
34 Previous reports on the expression of the cell adhesion molecule L1 in pancreatic ductal adenocarc
40 congenital hydrocephalus secondary to neural cell adhesion molecule L1 (L1CAM) gene mutations include
47 munohistochemistry, the distributions of the cell adhesion molecules L1, NCAM, and TAG-1 and the extr
48 d previously that axonal accumulation of the cell adhesion molecule L1/neuron-glia cell adhesion mole
49 NEEP21 in correct trafficking of the axonal cell adhesion molecule L1/neuron-glia cell adhesion mole
50 adhesion molecules (L1-CAMs) [L1/neuron-glia cell adhesion molecule (L1/NgCAM) and neurofascin (NF)]
51 ficking of multiple receptors, including the cell adhesion molecule L1/NgCAM, the neurotransmitter re
53 ctor-2 (FGF2), nerve growth factor (NGF), L1 cell adhesion molecule (L1), or beta-galactosidase (LacZ
54 lucose-6-phosphate dehydrogenase, the neural cell adhesion molecule L1, phenylalanine hydroxylase, pa
57 d using the cytoplasmic domain of the axonal cell adhesion molecule L1 to identify binding partners t
58 cin-C (tenascin) and an antibody (Ab) to the cell adhesion molecule L1 to specifically mimic survival