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1 ted by lyso-PA, sphingosine 1-phosphate, and ceramide 1-phosphate.
2 d that it can phosphorylate ceramide to form ceramide 1-phosphate.
3 lysophosphatidic acid, phosphatidic acid, or ceramide-1-phosphate.
5 ified and characterized interactions between ceramide 1-phosphate and the annexin a2-p11 heterotetram
6 d derivative, capnine, are sulfur analogs of ceramide-1-phosphate and sphingosine-1-phosphate, respec
7 id classes such as lysophosphatidylinositol, ceramide 1-phosphate, and lysophosphatidylethanolamine a
8 elated sphingolipids, including dihydro-S1P, ceramide 1-phosphates, and sphingosine phosphocholine as
9 exposed phosphate group: phosphatidic acid, ceramide-1-phosphate, and dihydroceramide-1-phosphate.
11 sphoinositides, diacylglycerolpyrophosphate, ceramide-1-phosphate, and phosphatidic acid belong to a
15 ssibility that ceramide metabolites, such as ceramide-1-phosphate (C-1-P), may explain these discrepa
18 ceramides, sphingosine 1-phosphate (S1P) and ceramide 1-phosphate (C1P) has been known for decades.
19 n to this accepted mechanism of two decades, ceramide 1-phosphate (C1P) has been shown to increase th
22 at TTYH1 mediates endolysosomal clearance of ceramide 1-phosphate (C1P), a sphingolipid that dampens
23 osphatidic acid (PA), lysophosphatidic acid, ceramide 1-phosphate (C1P), and sphingosine 1-phosphate
24 ated the potential for another sphingolipid, ceramide 1-phosphate (C1P), to modulate efflux pumps at
28 active sphingolipids resulting in decreased ceramide-1-phosphate (C1P) content as observed in human
31 Cellular distributions of the sphingolipid ceramide-1-phosphate (C1P) impact essential biological p
32 expression following ER stress, blockade of ceramide-1-phosphate (C1P) suppressed Tg-induced hBD2/hB
34 se (CERK) phosphorylates ceramide to produce ceramide-1-phosphate (C1P), which is involved in the dev
35 rated that ceramide kinase, and its product, ceramide 1-phosphate (Cer-1-P), were mediators of arachi
36 (N,N-diMe-Sph) were weak inhibitors whereas ceramide-1-phosphate (Cer-1-P) and sphingosine-1-phospha
39 r human ceramide kinase (hCERK) activity and ceramide 1-phosphate formation could lead to enhanced ph
40 sphingolipids, including phosphatidylserine, ceramide-1-phosphate, glucosylceramide, and sulfatide, v
42 ritical event in phagocytosis, the effect of ceramide-1-phosphate in promoting the fusion of liposome
43 evels in biological samples by conversion to ceramide-1-phosphate, in a manner analogous to that of t
44 not C(8)-glucosylceramides, sphingosine, or ceramide 1-phosphate, increases ABCA12 mRNA expression i
48 d product by thin-layer chromatography since ceramide-1-phosphate is the only radiolabeled lipid in o
49 vector control and simultaneously increased ceramide 1-phosphate levels 2-fold compared with vector
55 idic acid nor other sphingolipids, ceramide, ceramide-1-phosphate, or sphingosylphosphorylcholine, in
57 lucosylceramide, sulfatide, ganglioside GM1, ceramide 1-phosphate, sphingosine 1-phosphate, dihydroce
59 The C1P binding site in ACD11 and in human ceramide-1-phosphate transfer protein (CPTP) is surround
60 C1P levels are spatiotemporally regulated by ceramide-1-phosphate transfer protein (CPTP), which effi