ライフサイエンスコーパス: cerebellar

1 nt control while setting the global level of cerebellar activity through inhibition via rate coding m

2 g results about the role and the location of cerebellar activity, and the lack of behavioral measures

3 DTBM revealed marked hypoplasia of cerebellar afferent systems in DS, including fronto-pont

4 deficits in SCA1 arise from a combination of cerebellar and extra-cerebellar dysfunctions.

5 postnatal days 2-9, adult (postnatal day 70) cerebellar and hippocampal endocannabinoids, related lip

6 (g) There are no adult cerebellar and tectal shh-GFP cells confirming their exc

7 ses their pre-existing ataxia and diminishes cerebellar and thalamic vacuolation and Purkinje cell de

8 ations for the understanding of ET and other cerebellar and tremor disorders.

9 ates a critical role for GPI biosynthesis in cerebellar and white matter development.

10 nd quantify axons from thalamocortical, deep cerebellar, and cortical projection neurons, validating

11 In contrast, some basal ganglia, cerebellar, and limited cortical areas showed enhanced v

12 crease in the size of the cerebral cortical, cerebellar, and pontine regions.

13 rguing against a motor explanation, no other cerebellar area exhibited stimulus specificity, includin

14 of vascular loops was the anterior inferior cerebellar artery (right: 66.67%, left: 65.00%).

15 ein (Cacna2d2), the deletion of which causes cerebellar ataxia and epilepsy in mice and humans.

16 syndromes, including stiff-person syndrome, cerebellar ataxia and epilepsy.

17 th early-onset movement disorders comprising cerebellar ataxia and/or extrapyramidal symptoms.

18 show that the feedback gain in patients with cerebellar ataxia matches that of healthy subjects, but

19 Here, we identify the cerebellar ataxia spinocerebellar ataxia, autosomal rece

20 Cerebellar ataxia with neuropathy and bilateral vestibul

21 ubsequently expanded to the upper limbs, (2) cerebellar ataxia, (3) psychosis and/or severe mood diso

22 memories, and locomotion in mouse models for cerebellar ataxia, Alzheimer's disease, and spinal cord

23 y and/or intellectual disability, hypotonia, cerebellar ataxia, cerebellar atrophy, and facial dysmor

24 However, for cerebellar ataxia, epilepsy and other syndromes with dif

25 and seizures and later psychiatric symptoms, cerebellar ataxia, extrapyramidal signs, and extensive c

26 In addition to cerebellar ataxia, motor neuron disease is often seen in

27 Cerebellar ataxia, neuropathy and vestibular areflexia s

28 tor complex subunit 1 (RFC1) as the cause of cerebellar ataxia, neuropathy, vestibular areflexia synd

29 a slow-progressing syndrome characterized by cerebellar ataxia, psychotic episodes, and obsessive beh

30 ented as variable multisystemic, early-onset cerebellar ataxia, with complicating features ranging fr

31 se (MJD), the most common autosomal dominant cerebellar ataxia.

32 s Sirt1 dysfunction in SCA7, indicating that cerebellar ataxias exhibit altered calcium homeostasis b

33 Brain MRI demonstrated cerebellar atrophy and leukoencephalopathy.

34 c quadriparesis and progressive cortical and cerebellar atrophy in an effort to determine the genetic

35 Given the fact that cerebellar atrophy is seen in other IGDs, flow cytometry

36 Progressive cerebellar atrophy was also noted.

37 Cerebellar atrophy was universal on MRI (100%), with cer

38 velopmental syndrome with sleep disturbance, cerebellar atrophy, and facial dysmorphisms, and suggest

39 al disability, hypotonia, cerebellar ataxia, cerebellar atrophy, and facial dysmorphisms.

40 Cerebellar-based learning is thought to rely on synaptic

41 sed on circuit features he exemplified using cerebellar basket cell projections.

42 Here we probed CB-M1 interactions using cerebellar brain inhibition (CBI) in young healthy femal

43 be more sensitive to online manipulation of cerebellar (CB) activity using transcranial direct curre

44 These distinct cerebellar-cerebral interactions respond differently to

45 Here, we show evidence for two distinct cerebellar-cerebral interactions using cerebellar stimul

46 that directional TMS can probe two distinct cerebellar-cerebral pathways that likely contribute to i

47 These plastic cerebellar changes were complemented by changes in corti

48 ism linking heterochromatin dysregulation to cerebellar circuit dysfunction and behavioral abnormalit

49 ghlight both current evidence for predictive cerebellar circuit function that extends beyond the clas

50 must be addressed to unify our understanding cerebellar circuit function.

51 Ns are critical players in the regulation of cerebellar circuitry and function.

52 activity of PNs and thus the output from the cerebellar circuitry.

53 er, such evidence points to a broad role for cerebellar circuits in generating and testing prediction

54 well as the innervation of Purkinje cells by cerebellar climbing fibers.

55 data offer a basis for comparing aspects of cerebellar coding that are conserved and that diverge ac

56 estigations have shed light on the nature of cerebellar cognitive processing and information exchange

57 gative associations between progesterone and cerebellar coherence.

58 from the cerebellar nuclei that communicate cerebellar computation to other brain areas.

59 nts is associated with both motor cortex and cerebellar connectivity.

60 ers (ASDs); however, the circuits underlying cerebellar contributions to ASD-relevant behaviors remai

61 We tested the hypothesis that cerebellar contributions to cognition are guided by neur

62 Nevertheless, cerebellar contributions to cognitive control are poorly

63 , applied to the cerebellum, could elucidate cerebellar contributions to cognitive control.

64 These findings suggest that cerebellar contributions to cognitive networks are selec

65 rimotor integration, raising the question of cerebellar contributions to freezing.

66 stitutes evidence for double dissociation of cerebellar contributions to semantic prediction versus e

67 der SPL7, somatosensory PSC, ventral LOC and cerebellar control.SIGNIFICANCE STATEMENT Clumsy disaste

68 We determined that, in the developing rodent cerebellar cortex (of both sexes), there is a transient

69 hippocampus, we stimulated two sites of the cerebellar cortex and examined hippocampal function at m

70 ate high-frequency information from both the cerebellar cortex and the two main excitatory inputs of

71 ferent time points after injection, with the cerebellar cortex as the reference region.

72 n recapitulate the compressive forces on the cerebellar cortex from primary (e.g., glioblastoma) and

73 thological findings reveal severe atrophy of cerebellar cortex in SCA1 patients.

74 The surface of the human cerebellar cortex is much more tightly folded than the c

75 Recently, it has been reported that the cerebellar cortex is required for consolidation of condi

76 gulate translation information processing in cerebellar cortex output neurons.

77 and on-demand optogenetic modulation of the cerebellar cortex was shown to be highly effective at at

78 etations of the connectivity and function of cerebellar cortex within the full motor system.

79 encephalon and metencephalon (aside from the cerebellar cortex).

80 For cerebellar cortex, a cerebellar multilayer sandwich (CMS

81 In the cerebellar cortex, molecular layer interneurons use chem

82 Focusing on the cerebellar cortex, thinner collagen fibers and disorgani

83 By focusing on the neocortex and the cerebellar cortex, we demonstrate that reductions of inh

84 Purkinje cells in the molecular layer of the cerebellar cortex.

85 ed by Purkinje cells, the sole output of the cerebellar cortex.

86 undantly and differentially expressed in the cerebellar cortex.

87 s, which form one of two major inputs to the cerebellar cortex.

88 depending on the investigated region of the cerebellar cortex.

89 el fibre (PF)-Purkinje cell (PC) synapses in cerebellar cortex.

90 rkinje cells are the only output cell of the cerebellar cortex.

91 We delineated a circuit from cerebellar cortical areas Right crus 1 (Rcrus1) and post

92 ames Albus proposed a computational model of cerebellar cortical function based on the pioneering cir

93 ses to restore cerebellar GABAergic tone and cerebellar cortical inhibitory efficacy.

94 oth optogenetic excitation and inhibition of cerebellar cortical output neurons, Purkinje cells, atte

95 Here, we investigate the influence of the cerebellar cortical output, the Purkinje cells, on ident

96 d 2-arachidonoyl glycerol in females only in cerebellar Crus I; and (3) increased dorsal hippocampus

97 to the distinct ages of onset and timing of cerebellar degeneration in infant- and adult-onset SCA13

98 axis of CDR1as and its antisense transcript, cerebellar degeneration related protein 1 (CDR1).

99 57,048 novel regulatory elements regulating cerebellar development, synapse assembly, and hindbrain

100 ogenesis, and caused rapid cell death during cerebellar development.

101 d viability of Purkinje cells in vivo during cerebellar development.

102 ith severe ataxia consistent with defects in cerebellar development.

103 tured normally and did not degenerate during cerebellar development.

104 that important eye movement abnormalities in cerebellar disorders (i.e., ataxias) could be captured f

105 al domains, i.e. a cortical, subcortical and cerebellar domain.

106 t of several gait parameters consistent with cerebellar dysfunction from 40- to 80-d-old mice.

107 Cerebellar dysfunction has been demonstrated in autism s

108 light complex mechanisms converging onto the cerebellar dysfunction in the phenotypic model and provi

109 c neuropathy, particularly, but not only, if cerebellar dysfunction, vestibular involvement and cough

110 s, to the later appearance of vestibular and cerebellar dysfunction.

111 e from a combination of cerebellar and extra-cerebellar dysfunctions.

112 Accordingly, the cerebellar endocannabinoid system exhibits robust sex-sp

113 g strategies may favor temporal precision of cerebellar excitatory outputs associated with specific f

114 The most intriguing finding was high OPRM1 cerebellar expression in humans and chimpanzees and low

115 Theories of cerebellar functions posit that the cerebellum implement

116 , could be compensatory responses to restore cerebellar GABAergic tone and cerebellar cortical inhibi

117 ed human-unique expansion and maintenance of cerebellar germinal zones, reminiscent of processes in t

118 hat formed synapses when cultured with mouse cerebellar glia and granule cells and fired large calciu

119 ditionally, a significant loss occurs in the cerebellar granular neurons and striatal neurons in Tubb

120 pac is involved in long term potentiation at cerebellar granule cell-to-Purkinje cell synapses.

121 Cerebellar granule cells (GCs) make up the majority of a

122 s on oligodendrocytes, striatal neurons, and cerebellar granule cells in the context of altered micro

123 rs with endogenous AMPAR currents from mouse cerebellar granule cells, we have determined a likely pr

124 firmed this close interaction in cultures of cerebellar granule cells.

125 We show that SMB55 cells, and the primary cerebellar granule neuron precursors (GNPs) from which t

126 ogen and the extracellular matrix to control cerebellar granule neurons (CGN) GZ occupancy.

127 polarization and maturation of post-mitotic cerebellar granule neurons (CGNs).

128 d ATP release using HEK-293 cells and murine cerebellar granule neurons, along with bioluminescence,

129 Finally, cerebellar granule-cell-specific CB1KOs exhibit normal e

130 ns, cerebral cortex areas, hypothalamus, and cerebellar gray and white matter evolved rapidly in huma

131 tural brain measures, including cerebral and cerebellar gray matter (GM) and white matter (WM) volume

132 8)F-AZD4694 values were normalized using the cerebellar gray matter as a reference region.

133 ent role for marijuana in alcohol effects on cerebellar gray matter trajectories.

134 Decreased right cerebellar gray matter was the only abnormality common t

135 Using an inferior cerebellar grey matter reference, 80-100-min 18F-flortau

136 prospectively tracked deviations from normal cerebellar growth trajectories in adolescents before and

137 Alcohol use-related cerebellar growth trajectory differences from normal inv

138 ritical switch for neuronal polarization and cerebellar GZ exit.

139 in IUGR foetuses were significantly lower in cerebellar hemispheres (CH) (1.239 vs. 1.280.5 x 10(-3)

140 The cerebellar hemispheres were the major target of resultin

141 mbic system, whereas the occipital lobes and cerebellar hemispheres were unaffected.

142 of the cerebellar vermis with fusion of the cerebellar hemispheres, in 8/10 individuals.

143 Autosomal-recessive cerebellar hypoplasia and ataxia constitute a group of h

144 Brain MRI revealed cerebellar hypoplasia and ventriculomegaly.

145 cts and developmental delay, associated with cerebellar hypoplasia in one case.

146 gical treatment in patients with spontaneous cerebellar ICH are lacking, and the risk of bias in publ

147 based treatment guidelines for patients with cerebellar ICH.

148 2062 patients (40% female) with spontaneous cerebellar ICH.

149 ntine (middle cerebellar peduncle) and olivo-cerebellar (inferior cerebellar peduncle) connections.

150 tor learning tasks that are known to process cerebellar input in different ways.

151 I, indicating that cortical networks process cerebellar inputs in distinct ways.

152 S currents to the cerebral cortex may reveal cerebellar inputs to both the premotor cortex and M1.

153 asks, which suggests they represent separate cerebellar inputs to the premotor cortex and M1.

154 l motor control theories have suggested that cerebellar internal models predict the somatosensory con

155 idonoyl glycerol and arachidonic acid in the cerebellar interpositus nucleus in males only; (2) decre

156 ations for surgical treatment of spontaneous cerebellar intracerebral haemorrhage (ICH) differ.

157 Brainstem or cerebellar involvement occurred in 62/185 (34%) MOGAD pa

158 t that ACh could play a key role in altering cerebellar learning by modifying how sensorimotor input

159 ontal thickness), suggesting an interplay of cerebellar learning with cortical structures enabled thr

160 pursuit eye movements for four principles of cerebellar learning.

161 ation of the climbing fiber input, underlies cerebellar learning.

162 ole of Purkinje neuron membrane potential in cerebellar learning.

163 ed in the context of AD, renewed interest in cerebellar lesions has recently arisen as they may link

164 om the guinea pig dorsal cochlear nucleus, a cerebellar-like brainstem circuit.

165 nto gray matter volume patterns in prefronto-cerebellar, limbic, and sensory networks.

166 nding multimodal motor training, we compared cerebellar lobular volume and white matter microstructur

167 ICARS (R (2) = 0.3) and reduced GM volume in cerebellar lobule VI (R (2) = 0.35).

168 The present study demonstrates that cerebellar lobule VIIb/VIIIa activity patterns are selec

169 Cerebellar lobule VIIb/VIIIa delay-period activation acc

170 iatum and connected to prefrontal-projecting cerebellar lobules and anterior prefrontal cortex, formi

171 Other cerebellar lobules did not relate to temporal variabilit

172 rchitecture of the PF-PC synapses located in cerebellar lobules that differ in vulnerability to damag

173 iction, whereas beta stimulation of the same cerebellar location increased neural signals of semantic

174 y continue to make important predictions for cerebellar mechanism, and we show that evidence appearin

175 es accompanied by abnormal calcium-dependent cerebellar membrane excitability.

176 orrection of postsynaptic density protein 95 cerebellar misexpression, a major fine cerebellar struct

177 The present study examined the role of cerebellar MLIs in the formation of fear memory using a

178 Within molecularly defined cerebellar modules we found spatial overlap of mossy fib

179 strengthens inhibitory GABAergic synapses of cerebellar molecular layer interneurons.

180 ioural studies, GALNT2-CDG mice demonstrated cerebellar motor deficits, decreased sociability, and im

181 For cerebellar cortex, a cerebellar multilayer sandwich (CMS) model is proposed t

182 fluence of sex steroid hormones on intrinsic cerebellar network dynamics has yet to be established.

183 estradiol and progesterone alter functional cerebellar networks at rest in a woman densely sampled o

184 ighlight the potential importance of cerebro-cerebellar networks in the clinical manifestations of DS

185 It is unclear to what extent cerebellar networks show long-term plasticity and accomp

186 erse cognitive functions, yet to what extent cerebellar neurodegeneration contributes to cognitive al

187 ies known to share the basic architecture of cerebellar neuronal circuitry for their ability to acqui

188 adeno-associated virus, we found that adult cerebellar neurons are particularly vulnerable to mutant

189 differential changes in the excitability of cerebellar neurons contribute to the distinct ages of on

190 cule L1 (L1) in brain tissue and in cultured cerebellar neurons results in the generation and nuclear

191 ents displayed increased poly(ADP-ribose) in cerebellar neurons, supporting poly(ADP-ribose) polymera

192 tations lead to loss-of-function or death of cerebellar neurons.

193 eficits in motor coordination and associated cerebellar neuropathology have been described.

194 we set out to study how specific subtypes of cerebellar nuclear neurons of the medial nucleus are con

195 background activity at the Purkinje to deep cerebellar nuclear neurons synapses (PC_DCNs).

196 second timescale through adaptive control of cerebellar nuclear output.

197 We find that in mice the deep cerebellar nuclei (DCN) and vestibular nuclei (VN) are t

198 inently expressed around neurons of the deep cerebellar nuclei (DCN), but their role in adult cerebel

199 that neurons of the mouse medial (fastigial) cerebellar nuclei (mCbN), which fire spontaneously with

200 1 (Rcrus1) and posterior vermis through the cerebellar nuclei and ventromedial thalamus and culminat

201 g, which alters the activity of their target cerebellar nuclei cells.

202 , called eurydendroid neurons (ENs) in fish (cerebellar nuclei in mammals).

203 Cerebellar nuclei integrate high-frequency information f

204 re, I investigated the Purkinje cell to deep cerebellar nuclei neuron synapses (PC_DCNs), which displ

205 al output, the Purkinje cells, on identified cerebellar nuclei neurons in vivo in male mice.

206 neurotransmitter release in projections from cerebellar nuclei neurons onto gigantocellular reticular

207 kinje cell discharges depends on the type of cerebellar nuclei neurons targeted.SIGNIFICANCE STATEMEN

208 directed to the Purkinje cell output in the cerebellar nuclei reduced tremor in freely moving mice.

209 the permanent discharge of neurons from the cerebellar nuclei that communicate cerebellar computatio

210 m excitatory parvalbumin-positive neurons in cerebellar nuclei was sufficient to generate an action t

211 tively promote firing in neurons in the deep cerebellar nuclei with remarkable speed and precision.

212 ion evolution at cell-type resolution in the cerebellar nuclei, the output structures of the cerebell

213 cerebellar white matter and within the deep cerebellar nuclei, where neuron loss also occurred.

214 synaptic transmission with tetanus toxin in cerebellar nuclei, which also reversed the tremor phenot

215 This set constitutes an archetypal cerebellar nucleus that was repeatedly duplicated to for

216 utput neurons in the mouse fastigial (medial cerebellar) nucleus, we identify five major classes of g

217 Together with previous reports of low cerebellar OPRM1 expression in mice, this suggests an ev

218 We therefore combined cerebellar optogenetic stimulation and CA1 calcium imagi

219 ibition of Purkinje cells, which can entrain cerebellar output for driving temporally precise behavio

220 Suppression of cerebellar output may therefore facilitate freezing.

221 inhibition and excitation are integrated by cerebellar output neurons in association with motor beha

222 es to movements through signals generated by cerebellar output neurons, called eurydendroid neurons (

223 ynaptic phenotypes contribute to the altered cerebellar output.

224 ough Purkinje target neurons, which transmit cerebellar output.

225 ght to test whether it is possible to toggle cerebellar participation in episodic memory versus seman

226 s reinforce the central role for Cic in SCA1 cerebellar pathophysiology and suggest that only modest

227 ing with cortical structures enabled through cerebellar pathways.

228 For example, cerebellar patients present exacerbated temporal variabi

229 Lastly, we improve cerebellar patients' movement control by altering (phase

230 KII regulates the direction of plasticity in cerebellar PCs.

231 s can determine whether LTD or LTP occurs in cerebellar PCs.

232 ts of brain morphology, including the middle cerebellar peduncle (MCP), superior cerebellar peduncle

233 e middle cerebellar peduncle (MCP), superior cerebellar peduncle (SCP), pons, and midbrain, indicatin

234 Diffuse middle cerebellar peduncle MRI-lesions favoured MOGAD (17/37 (4

235 tems in DS, including fronto-pontine (middle cerebellar peduncle) and olivo-cerebellar (inferior cere

236 lar peduncle) and olivo-cerebellar (inferior cerebellar peduncle) connections.

237 and increased mean diffusivity in the middle cerebellar peduncle, right medial lemniscus, bilateral p

238 Ttbk2 in adult mice results in degenerative cerebellar phenotypes that recapitulate aspects of SCA11

239 88 in adult mice results in nearly identical cerebellar phenotypes to those of the Ttbk2 knockout, in

240 bellar nuclei (DCN), but their role in adult cerebellar plasticity and behavior is far from clear.

241 The cerebellar posterior vermis generates an estimation of o

242 on climbing fiber synaptic refinement during cerebellar postnatal development using the Npc1(nmf164)

243 However, this expanded view of cerebellar processing also raises many new questions abo

244 departure from the current understanding of cerebellar processing and have critical implications for

245 itation and inhibition at the first stage of cerebellar processing.

246 itation and inhibition at the first stage of cerebellar processing.SIGNIFICANCE STATEMENT The cerebel

247 r olive make strong excitatory synapses onto cerebellar Purkinje cell (PC) dendrites and trigger dist

248 Here, we found that genetically silencing cerebellar Purkinje cell output blocked tremor in mice t

249 Because cerebellar Purkinje cells (PCs) only express this single

250 Because cerebellar Purkinje cells (PCs) predominantly, if not ex

251 Conversely, Gq-DREADD activation of cerebellar Purkinje cells enhanced endocannabinoid signa

252 Degeneration of cerebellar Purkinje cells occurs at an earlier stage and

253 ions of human cortical pyramidal neurons and cerebellar Purkinje cells show significant expression of

254 odulate the activity of spatially-delineated cerebellar Purkinje cells to evaluate the impact on aggr

255 Here, we demonstrate that cerebellar Purkinje cells upregulate excitability in del

256 mplitude of the inhibitory synaptic input in cerebellar Purkinje cells.

257 nd most prominent in hippocampal neurons and cerebellar Purkinje cells.

258 In mouse cerebellar Purkinje neurons (PNs), the climbing fiber (C

259 ATXN1 co-repressor Capicua (Cic) in anterior cerebellar Purkinje neurons.

260 Casting caused cortical and cerebellar regions controlling the disused extremity to

261 ospinal fluid volume expanded faster in most cerebellar regions of male youths than female youths.

262 well as connectivity with cortical and with cerebellar regions on a functional level.

263 ssed the evidence that a set of cortical and cerebellar regions were sensitive to grasp configuration

264 s to sexual differentiation of the brain and cerebellar-related dysfunctions.

265 lum provide a roadmap for the next decade of cerebellar research, challenging some old concepts, rein

266 However, it remains unclear whether these cerebellar responses reflect processes specific to VWM o

267 lutamate can provoke paranodal elongation in cerebellar slice cultures, which could be reversed by an

268 tion (120 s) to mimic background activity in cerebellar slices from mature mice of both sexes, I iden

269 terenol potentiated synaptic transmission in cerebellar slices from mice of either sex, an effect tha

270 ordings were obtained from Purkinje cells in cerebellar slices prepared from male mice ~48 h after th

271 Whole-cell recordings of PCs from acute cerebellar slices revealed altered climbing fiber (CF)-e

272 renol-induced increase in vesicle docking in cerebellar slices.

273 tinct cerebellar-cerebral interactions using cerebellar stimulation in combination with directional t

274 ctivity rhythms and could be used to develop cerebellar stimulation interventions for specific neuroc

275 h an object-exploration task, and found that cerebellar stimulation reduced the representation of pla

276 We found that cerebellar stimulation strongly modulates hippocampal ac

277 in 95 cerebellar misexpression, a major fine cerebellar structural abnormality in Cdkl5 knockout mice

278 on an artificially narrow interpretation of cerebellar structure and motor function.

279 pendent adults, raising the possibility that cerebellar structures affected by youthful drinking may

280 vide novel evidence for the participation of cerebellar structures in the persistent storage of visua

281 synaptic profile of brain circuits, such as cerebellar structures, that are thought to engage in pro

282 concrete, proximal actions and motor-distal, cerebellar sub-regions supporting abstract, future proce

283 pports cognitive control with motor-adjacent cerebellar sub-regions supporting control of concrete, p

284 Cerebellar surface recordings showed a functional pertur

285 The patient presented with subacute cerebellar syndrome and myoclonus several days after gen

286 In human subjects of both sexes, cerebellar theta stimulation improved episodic memory en

287 expressed in both hPSC-PCs and native human cerebellar tissue.

288 l gyrus (BA 34) and caudate body, and in the cerebellar tonsils (P < 0.001).

289 SCA2 cerebellar transcriptomes were also determined, and we a

290 Medulloblastoma (MB) is a highly malignant cerebellar tumor predominantly diagnosed during childhoo

291 Further, cerebellar V (T) did not change between baseline and blo

292 al vasculature, mildly stunted growth of the cerebellar vasculature and little or no effect on the va

293 e and female mice, acute perturbation of the cerebellar vermis (lobule 4/5) or simplex produced relia

294 These results indicate that diverse cerebellar vermis functions could be mediated by modular

295 esults suggest Purkinje cell activity in the cerebellar vermis regulates aggression, and further supp

296 activation of the right amygdala and midline cerebellar vermis to nonemotional as opposed to emotiona

297 acterized by partial or complete loss of the cerebellar vermis with fusion of the cerebellar hemisphe

298 pp5 depletion led to microcephaly, decreased cerebellar volume and cortical thickness.

299 ial activation was particularly prominent in cerebellar white matter and within the deep cerebellar n

300 n measures, such that cortical thickness and cerebellar WM volume were primarily influenced by enviro