ライフサイエンスコーパス: cerebellar Purkinje cell

1 nd most prominent in hippocampal neurons and cerebellar Purkinje cells.

2 mately yield a long-term depression (LTD) of cerebellar Purkinje cells.

3 of the sample, such as occurs when counting cerebellar Purkinje cells.

4 icroscopy to measure dendritic spines in rat cerebellar Purkinje cells.

5 holinergic neurons, dopaminergic neurons and cerebellar Purkinje cells.

6 uman Atxn1 with an expanded polyglutamine in cerebellar Purkinje cells.

7 nels that are expressed at high densities in cerebellar Purkinje cells.

8 ), is important for postnatal development of cerebellar Purkinje cells.

9 lize molecular diffusion within dendrites of cerebellar Purkinje cells.

10 tive exon results in abnormal development of cerebellar Purkinje cells.

11 tory synapses on the somata and dendrites of cerebellar Purkinje cells.

12 lays a dramatic phenotype of degeneration of cerebellar Purkinje cells.

13 udied the electrophysiological properties of cerebellar Purkinje cells.

14 of the ovary, cerebral cortical neurons, and cerebellar Purkinje cells.

15 triking abnormalities other than the loss of cerebellar Purkinje cells.

16 e inhibition at three types of synapses onto cerebellar Purkinje cells.

17 cell counts reveal no significant absence of cerebellar Purkinje cells.

18 topoiesis, and lack of early degeneration of cerebellar Purkinje cells.

19 nd dendrites of whole-cell patch-clamped rat cerebellar Purkinje cells.

20 several cranial nerves nuclei, as well as in cerebellar Purkinje cells.

21 reveals abundant CYP2J9 mRNA and protein in cerebellar Purkinje cells.

22 neocortex and within the dendritic arbors of cerebellar Purkinje cells.

23 in dendrites of cortical pyramidal cells and cerebellar Purkinje cells.

24 ipheral locations or different modalities to cerebellar Purkinje cells.

25 eticulum vesicles in the dendritic spines of cerebellar Purkinje cells.

26 rt tremor phenotypes associated with loss of cerebellar Purkinje cells.

27 ed by long-term synaptic depression (LTD) in cerebellar Purkinje cells.

28 a pronounced abnormality in the location of cerebellar Purkinje cells.

29 er, and neurodegeneration, especially of the cerebellar Purkinje cells.

30 progressive motor deterioration, and loss of cerebellar Purkinje cells.

31 anocytes and smooth endoplasmic reticulum in cerebellar Purkinje cells.

32 nductance levels (9, 13-14, and 17-18 pS) in cerebellar Purkinje cells.

33 unique persistent and resurgent currents in cerebellar Purkinje cells.

34 progressive motor deterioration, and loss of cerebellar Purkinje cells.

35 n were also detected in cortical neurons and cerebellar Purkinje cells.

36 progressive motor deterioration, and loss of cerebellar Purkinje cells.

37 und that ataxin-1 localizes to the nuclei of cerebellar Purkinje cells.

38 age-gated currents during somatic spiking in cerebellar Purkinje cells.

39 behaviors such as thalamic relay neurons and cerebellar Purkinje cells.

40 eus (DCN) that bear considerable homology to cerebellar Purkinje cells.

41 e in which Scn1b was specifically deleted in cerebellar Purkinje cells.

42 ning and the NF1 gene is highly expressed in cerebellar Purkinje cells.

43 ke mice during two-photon calcium imaging of cerebellar Purkinje cells.

44 kainate receptor subtype, are coexpressed in cerebellar Purkinje cells.

45 e and show that IP6K3 is highly expressed in cerebellar Purkinje cells.

46 IP6K3 is highly concentrated in the brain in cerebellar Purkinje cells.

47 of cytoskeletal disposition and function of cerebellar Purkinje cells.

48 b5 is expressed in brain capillaries, and by cerebellar Purkinje cells.

49 ing in neurons and labeled large speckles in cerebellar Purkinje cells.

50 man ataxia-telangiectasia and Atm(-/-) mouse cerebellar Purkinje cells.

51 y identified presynaptic partners, including cerebellar Purkinje cells.

52 for dendritic growth and branching of mouse cerebellar Purkinje cells.

53 ataxia and concomitant axon degeneration of cerebellar Purkinje cells.

54 t display age-dependent neurodegeneration of cerebellar Purkinje cells.

55 retinal starburst amacrine cells (SACs) and cerebellar Purkinje cells.

56 mplitude of the inhibitory synaptic input in cerebellar Purkinje cells.

57 cal symptoms in mice and the degeneration of cerebellar Purkinje cells.

58 -6 is known to cause significant loss of the cerebellar Purkinje cells.

59 Polysynaptic pathways were also labeled from cerebellar Purkinje cells.

60 4-AP do not increase the inhibitory drive of cerebellar Purkinje cells.

61 ations is loss of precision of pacemaking in cerebellar Purkinje cells.

62 rallel fiber segments of the granule axon on cerebellar Purkinje cells.

63 ularly within brain motor systems, including cerebellar Purkinje cells.

64 the slow excitatory postsynaptic current in cerebellar Purkinje cells.

65 ne transporters (DATs) were all expressed in cerebellar Purkinje cells.

66 types involved in motor function, including cerebellar Purkinje cells.

67 Only 5-10% of cerebellar Purkinje cells, a less vulnerable population,

68 We show that, in cerebellar Purkinje cells, a single activation of the cl

69 in selected regions of the cerebral cortex, cerebellar Purkinje cells, a subset of striatal neurons,

70 hallmark of A-T is fulminant degeneration of cerebellar Purkinje cells accompanied by a progressive a

71 x has been shown to reduce the regularity of cerebellar Purkinje cell activity and to induce episodic

72 oreactive neurons appear to be innervated by cerebellar Purkinje cell afferents.

73 Cerebellar Purkinje cells also showed progressive degene

74 d to double the simple spike activity of the cerebellar Purkinje cell and eliminates complex spike ac

75 nts, including decreased firing frequency of cerebellar Purkinje cells and a decline in motor functio

76 tmortem brain tissue reveals degeneration of cerebellar Purkinje cells and a reduced level of cerebel

77 ias (SCAs) are hereditary diseases affecting cerebellar Purkinje cells and are a one of neurodegenera

78 are known to be expressed preferentially in cerebellar Purkinje cells and are involved in triggering

79 cript is associated with progressive loss of cerebellar Purkinje cells and ataxia.

80 t ganglion and there was progressive loss of cerebellar Purkinje cells and atrophy of cerebellar gran

81 R)alpha, is essential for the development of cerebellar Purkinje cells and bone tissue.

82 otor coordination due to the degeneration of cerebellar Purkinje cells and brain stem neurons.

83 However, in fast-spiking GABAergic neurons (cerebellar Purkinje cells and cortical interneurons), tw

84 Cerebellar Purkinje cells and granule cells have been st

85 In contrast, in GABAergic neurons such as cerebellar Purkinje cells and hippocampal pyramidal bask

86 Cerebellar Purkinje cells and hippocampal pyramidal cell

87 elta2 receptor is predominantly expressed in cerebellar Purkinje cells and in the heterozygous Lurche

88 We found that both cerebellar Purkinje cells and inhibitory thalamic reticu

89 rize the pH-dependent activation of ASICs in cerebellar Purkinje cells and investigate how they are m

90 ives dynamic gene expression networks within cerebellar Purkinje cells and is indispensable for neona

91 el subunit Kv3.3 is prominently expressed in cerebellar Purkinje cells and is known to be important f

92 that localizes to the soma and dendrites of cerebellar Purkinje cells and is required for dendritic

93 t throughout the elaborate dendritic tree of cerebellar Purkinje cells and is required for normal neu

94 ion of a feedback inhibitory circuit between cerebellar Purkinje cells and molecular layer interneuro

95 hat caused the degeneration of virtually all cerebellar Purkinje cells and most olivary neurons and g

96 Structural changes in cerebellar Purkinje cells and neighbouring neuronal popu

97 rms alpha, gamma, and delta are expressed by cerebellar Purkinje cells and neurons in the cerebellar

98 ation revealed elevated superoxide levels in cerebellar Purkinje cells and nigral dopaminergic neuron

99 me areas where neurons did not express NeuN: cerebellar Purkinje cells and olfactory bulb mitral cell

100 orphology, number and length, is affected on cerebellar Purkinje cells and on pyramidal neurons in th

101 sulted in a marked decrease in the number of cerebellar Purkinje cells and parvalbumin-positive inter

102 Pcp2 is expressed in cerebellar Purkinje cells and retinal bipolar neurons, t

103 ganglion cells in mouse and human, including cerebellar Purkinje cells and retinal ganglion cells.

104 containing family, is present exclusively in cerebellar Purkinje cells and retinal ON bipolar cells.

105 g the activity of NMDA receptors (NMDARs) in cerebellar Purkinje cells and their possible functions.

106 P450 that is abundant in brain, localized to cerebellar Purkinje cells, and active in the biosynthesi

107 olf) is highly expressed in the striatum and cerebellar Purkinje cells, and co-localized with cortico

108 in three principal neurons of the mouse CNS: cerebellar Purkinje cells, and cortical and hippocampal

109 which receive direct inhibitory inputs from cerebellar Purkinje cells, and excitatory collaterals fr

110 riatum, CA1 subfield of the hippocampus, and cerebellar Purkinje cells, and high densities of Kv beta

111 cholinergic neurons in the basal forebrain, cerebellar Purkinje cells, and substantia gelatinosa of

112 e lysosomal-endosomal compartment, a loss of cerebellar Purkinje cells, and widespread axonal degener

113 Our data indicates that cerebellar Purkinje cells are an important node for dysf

114 Climbing fiber responses in cerebellar Purkinje cells are described as being invaria

115 pite lacking N-methyl-D-aspartate receptors, cerebellar Purkinje cells are highly vulnerable to ischa

116 ce are targets of oxidative damage, and that cerebellar Purkinje cells are particularly affected.

117 During locomotion, cerebellar Purkinje cells are rhythmically active, shapi

118 n stratum oriens and piriform cortex, and in cerebellar Purkinje cell areas.

119 normal mice, CAR8 is abundantly expressed in cerebellar Purkinje cells as well as in several other ce

120 Anti-PDE4D antibody distinctly labeled cerebellar Purkinje cells as well as neurons in the medi

121 cked the late phase of LTD in mouse cultured cerebellar Purkinje cells, as did deletion of the immedi

122 ating bone marrow cells with, in particular, cerebellar Purkinje cells, as well as the subsequent for

123 In the brain, there were reduced numbers of cerebellar Purkinje cells, atrophic vestibular sense org

124 Here we use recordings from visualized rat cerebellar Purkinje cell axons to localize the site of i

125 mutation alters calcium channel currents in cerebellar Purkinje cells, both because these cells are

126 , striatum, septal nuclei, substantia nigra, cerebellar Purkinje cells, brainstem and spinal motor ne

127 y dispersed synapses are activated on rodent cerebellar Purkinje cells but that it reduces presynapti

128 xamined the phosphorylation of PDE5 in mouse cerebellar Purkinje cells by immunocytochemistry and Wes

129 We propose that in the absence of ATM, cerebellar Purkinje cells cannot respond adequately to t

130 t conditional deletion of all neuroligins in cerebellar Purkinje cells caused loss of distal climbing

131 ermine if the properties of Ca2+ channels in cerebellar Purkinje cells change during postnatal develo

132 In cerebellar Purkinje cells, climbing fiber synapses provi

133 Although cerebellar Purkinje cell complex spikes occur at low fre

134 Loss of CCP1 in mice causes cerebellar Purkinje cell degeneration.

135 ainst a wide range of experimental data from cerebellar Purkinje cells demonstrates that a kinetic sc

136 The molecular pathways controlling cerebellar Purkinje cell dendrite formation and maturati

137 ynaptic and intrinsic membrane properties of cerebellar Purkinje cell dendrites, and a 4-aminopyridin

138 croinjected into molluscan neurons or rabbit cerebellar Purkinje cell dendrites, calexcitin was highl

139 In cerebellar Purkinje cells, dendritic spikes can trigger

140 t controls expression of genes important for cerebellar Purkinje cell development and excitability.

141 The relationship of cerebellar Purkinje cell discharge to the control of arm

142 d NBQX inhibited kainate-induced currents in cerebellar Purkinje cells, DRG neurons, and human GluR5-

143 tive, cell-autonomous and apoptotic death of cerebellar Purkinje cells during postnatal development.

144 cal, suggesting that they are coregulated in cerebellar Purkinje cells during this early compensatory

145 of plasticity in the simple-spike firing of cerebellar Purkinje cells during trial-over-trial learni

146 Conversely, Gq-DREADD activation of cerebellar Purkinje cells enhanced endocannabinoid signa

147 In particular, fz4 is expressed in cerebellar Purkinje cells, esophageal skeletal muscle, a

148 We now report that cerebellar Purkinje cells express KCa3.1 channels, as ev

149 llar BDNF expression and improved ataxia and cerebellar Purkinje cell firing rate deficits.

150 urons that send climbing fibers to innervate cerebellar Purkinje cells for the control of motor learn

151 LTD was absent in cultured cerebellar Purkinje cells from mutant mice lacking the A

152 The cortical pyramidal neurons and cerebellar Purkinje cells from the BK-D434G mice show hy

153 railed-2 (En-2) was ectopically expressed in cerebellar Purkinje cells from the late embryonic stage

154 dination caused primarily by a disruption of cerebellar Purkinje cell function.

155 armacological profile of acutely dissociated cerebellar Purkinje cell GABAARs from untreated, artific

156 terminal PDZ binding motif, were perfused in cerebellar Purkinje cells grown in culture.

157 expression of the mutant SCA1 allele within cerebellar Purkinje cells has divergent effects on the m

158 Cerebellar Purkinje cells have one of the most elaborate

159 Cerebellar Purkinje cells have two distinct action poten

160 We show here that a cerebellar Purkinje cell in a ferret can learn to respon

161 ions of a biophysically realistic model of a cerebellar Purkinje cell in a pattern recognition task s

162 lesions of the CBF and, to a lesser extent, cerebellar Purkinje cells in a dose-dependent fashion.

163 bellum of control mice, are expressed within cerebellar Purkinje cells in a mouse model of MS, chroni

164 )1.8 mRNA and protein are upregulated within cerebellar Purkinje cells in animal models of multiple s

165 We made electrophysiological recordings from cerebellar Purkinje cells in both V408A/+ mice and their

166 the structure and plasticity of synapses on cerebellar Purkinje cells in Fmr1 knockout mice, which a

167 Cerebellar Purkinje cells in GABA(A) receptor alpha1-/-

168 that results in the cell-autonomous death of cerebellar Purkinje cells in heterozygous lurcher (+/Lc)

169 Death of cerebellar Purkinje cells in lurcher animals is due to a

170 assessed the chronic toxicity of ibogaine on cerebellar Purkinje cells in male Fischer 344 rats.

171 In newborn mice sema-D is expressed by cerebellar Purkinje cells in parasagittal bands located

172 We demonstrate the presence of alpha-TTP in cerebellar Purkinje cells in patients having vitamin E d

173 showed that ibogaine causes degeneration of cerebellar Purkinje cells in rats.

174 temporally protracted heredodegeneration of cerebellar Purkinje cells in shaker mutant rats can be m

175 of the corpus callosum, and degeneration of cerebellar Purkinje cells in the NPC mouse.

176 ed in the neurons of cerebral cortex and the cerebellar purkinje cells, in a pattern similar to that

177 subunit Kv3.3 is expressed at high levels in cerebellar Purkinje cells, in auditory brainstem nuclei

178 ate the role of cartwheel cells, homologs of cerebellar Purkinje cells, in producing this inhibition.

179 and the intensity of Trio immunostaining in cerebellar Purkinje cells increase from P1 to P30.

180 n mice, the initial formation of synapses on cerebellar Purkinje cells involves a presynaptic protein

181 receptor (mGluR) second messenger system in cerebellar Purkinje cells is constructed.

182 Mice lacking Fmr1 gene expression in cerebellar Purkinje cells (L7-Fmr1 KO) were selectively

183 campus (0 vs. 3.5), subiculum (0 vs. 4), and cerebellar Purkinje cell layer (2 vs. 4) (all p < .05).

184 mEAAT1 mRNA was present predominantly in the cerebellar Purkinje cell layer and at a much lower abund

185 -II mRNA exhibits peak expression within the cerebellar Purkinje cell layer and the hippocampus, as w

186 RGS8 mRNA is highly expressed in the cerebellar Purkinje cell layer as well as in several mid

187 18.2% of wild-type L-SMase activity, but the cerebellar Purkinje cell layer, which is lost by 4 month

188 Deletion of Npc1 in mature cerebellar Purkinje cells leads to an age-dependent impa

189 that the mouse sticky mutation, which causes cerebellar Purkinje cell loss and ataxia, is a missense

190 Here, we demonstrate that cerebellar Purkinje cell loss in aged mice is not spatia

191 se brains also exhibited cortical neuron and cerebellar Purkinje cell loss, astrogliosis, and decreas

192 or coordination abnormalities, prevention of cerebellar Purkinje cell loss, correction of the ultrast

193 clodextrins (HPbetaCD) significantly delayed cerebellar Purkinje cell loss, slowed progression of neu

194 wz) mutation develop adult-onset ataxia with cerebellar Purkinje cell loss.

195 These phenomena suggest that cerebellar Purkinje cells may be a uniquely vulnerable n

196 (type I) devoid of cellular organelles, and cerebellar Purkinje cells, nearly all of which accumulat

197 the ATM gene and is characterized by loss of cerebellar Purkinje cells, neurons with high physiologic

198 with ataxin-1 localized to aggregates within cerebellar Purkinje cells nuclei.

199 Furthermore, we show that cerebellar Purkinje cell number and dendritic branching

200 Degeneration of cerebellar Purkinje cells occurs at an earlier stage and

201 Inactivation of the Nfasc gene in cerebellar Purkinje cells of adult mice causes rapid los

202 rents were recorded from acutely dissociated cerebellar Purkinje cells of homozygous leaner (tgla/tgl

203 wild-type littermates, while the staining of cerebellar Purkinje cells, oligodendrocytes, microglial

204 Using synapses formed by cerebellar Purkinje cells onto deep cerebellar nuclei as

205 he limbic forebrain, the locus coeruleus and cerebellar Purkinje cells, or for CRF2 in any aspect of

206 omplex spike synchrony across populations of cerebellar Purkinje cells oriented in the parasagittal a

207 Here, we found that genetically silencing cerebellar Purkinje cell output blocked tremor in mice t

208 ith the transcriptional repressor CIC drives cerebellar Purkinje cell pathogenesis; however, the impo

209 ing phase and the prolonged plateau phase of cerebellar Purkinje cell (PC) action potentials.

210 ly demonstrated that Hx-induced reduction of cerebellar Purkinje cell (PC) activity results in locomo

211 shed that the climbing fiber (CF) input to a cerebellar Purkinje cell (PC) can exert a controlling in

212 are characterized by loss of balance due to cerebellar Purkinje cell (PC) degeneration.

213 r olive make strong excitatory synapses onto cerebellar Purkinje cell (PC) dendrites and trigger dist

214 channel Kv1.2 alpha-subunit is expressed in cerebellar Purkinje cell (PC) dendrites where its pharma

215 d separately with the folic acid (FA) on the cerebellar Purkinje cell (PC) firing and histology, and

216 post-mortem studies in ASD patients showing cerebellar Purkinje cell (PC) loss, and isolated cerebel

217 Mossy fiber inputs are transformed into cerebellar Purkinje cell (PC) outputs by granule cell (G

218 ctions in receptor trafficking and modulates cerebellar Purkinje cell (PC) synaptic activity.

219 ynaptic transmission from parallel fibers to cerebellar Purkinje cells (PCs) and from climbing fibers

220 Cerebellar Purkinje cells (PCs) are particularly hyperse

221 Cerebellar Purkinje cells (PCs) are predominantly affect

222 Cerebellar Purkinje cells (PCs) are primarily affected i

223 ding-bundling proteins that are expressed in cerebellar Purkinje cells (PCs) but are not detected in

224 reviously, we demonstrated that Tsc1 loss in cerebellar Purkinje cells (PCs) can cause altered social

225 Cerebellar Purkinje cells (PCs) express two glutamate tr

226 Cerebellar Purkinje cells (PCs) fire spontaneously and d

227 KEY POINTS: Cerebellar Purkinje cells (PCs) generate two types of ac

228 ional postnatal loss of P/Q-type channels in cerebellar Purkinje cells (PCs) in mice (purky) leads to

229 matic representations through the cerebellum.Cerebellar Purkinje cells (PCs) linearly encode whisker

230 y of dendritic spines were identified in the cerebellar Purkinje cells (PCs) of Bmal1 knockout (KO) m

231 Because cerebellar Purkinje cells (PCs) only express this single

232 Because cerebellar Purkinje cells (PCs) predominantly, if not ex

233 We found that suppressing the firing of cerebellar Purkinje cells (PCs) rapidly excites forebrai

234 Cerebellar Purkinje cells (PCs) receive GABAergic input

235 in intrinsic properties and excitability of cerebellar Purkinje cells (PCs) resulting from the leane

236 Conditional deletion of the Pten gene in cerebellar Purkinje cells (PCs) results in cellular hype

237 Cerebellar Purkinje cells (PCs) seem to be primarily aff

238 be a novel ionotropic GABA receptor in mouse cerebellar Purkinje cells (PCs) using agents reported to

239 ons, such as hippocampal pyramidal cells and cerebellar Purkinje cells (PCs), it has not been demonst

240 Cerebellar Purkinje cells (PCs), the sole output neurons

241 ssion of dystrophin in the mouse brain is in cerebellar Purkinje cells (PCs), where it colocalizes wi

242 ss of PI31 in spinal motor neurons (MNs) and cerebellar Purkinje cells (PCs).

243 f neurons, they play a different role in the cerebellar Purkinje cells (PCs).

244 racterized by CT protein aggregates found in cerebellar Purkinje cells (PCs).

245 and its receptor Robo2 in self-avoidance of cerebellar Purkinje cells (PCs).

246 These findings establish how cerebellar Purkinje cells predict sensory outcomes of se

247 Brief strong depolarization of cerebellar Purkinje cells produces a slow inward cation

248 /putamen, globus pallidus, substantia nigra, cerebellar Purkinje cells, pyramidal cells of frontal co

249 sion is limited to neurons, particularly the cerebellar Purkinje cells, pyramidal cells of the hippoc

250 Cerebellar Purkinje cells receive two distinctive types

251 We find that HCN1 channels in cerebellar Purkinje cells reduce the duration of inhibit

252 Depolarization of cerebellar Purkinje cells reduced the firing rate of nea

253 ensory evidence, optogenetic manipulation of cerebellar Purkinje cells reduces the accuracy of subseq

254 expression of long-term depression (LTD) in cerebellar Purkinje cells results from the internalisati

255 Applying iPEEL to developing and mature cerebellar Purkinje cells revealed differential enrichme

256 Recently, the PRC theory applied to cerebellar Purkinje cells revealed that these behave as

257 tants calbindin immunostaining for surviving cerebellar Purkinje cells revealed widespread degenerati

258 nently in short-term synaptic plasticity and cerebellar Purkinje cell rhythmicity.

259 we find that deletion of HCN1 channels from cerebellar Purkinje cells selectively impairs late stage

260 inated fibers (serum 2%, CSF 2%); 4) against cerebellar Purkinje cells (serum 0%, CSF 2%); and 5) aga

261 ions of human cortical pyramidal neurons and cerebellar Purkinje cells show significant expression of

262 in which mutant ATXN1 is expressed solely in cerebellar Purkinje cells shows no evidence of olivary h

263 Outside-out patch recordings from cerebellar Purkinje cell somas confirmed these 4-AP-sens

264 The cerebellar Purkinje cell-specific PCP-2 gene is transcri

265 We selectively disrupted Foxp2 in the cerebellar Purkinje cells, striatum or cortex of mice an

266 The signals in cerebellar Purkinje cells sufficient to instruct motor l

267 ous simulations using a realistic model of a cerebellar Purkinje cell suggested that synaptic control

268 heir density in the postsynaptic membrane at cerebellar Purkinje cell synapses.

269 Cerebellar motor coordination and cerebellar Purkinje cell synaptic function require metab

270 s that trigger motor learning by controlling cerebellar Purkinje cell synaptic plasticity and dischar

271 mossy/parallel fibers and climbing fibers to cerebellar Purkinje cells that acquire a precisely timed

272 s a postsynaptic, cell type-specific role in cerebellar Purkinje cells that is essential for normal m

273 n was evident in the deep cerebellar nuclei, cerebellar Purkinje cells, the brainstem and the ventral

274 two types of action potentials generated by cerebellar Purkinje cells, the simple spikes and complex

275 lls that is essential for three processes in cerebellar Purkinje cells: the matching and maintenance

276 e growth factor-differentiated PC12 cells or cerebellar Purkinje cells to cyanide elicits rapid incre

277 which is the peak period of vulnerability of cerebellar Purkinje cells to ethanol.

278 odulate the activity of spatially-delineated cerebellar Purkinje cells to evaluate the impact on aggr

279 nt throughout the soma and dendritic tree of cerebellar Purkinje cells, to be required for the mainte

280 Here, we show that brief depolarization of a cerebellar Purkinje cell triggers a slow inward current.

281 In cerebellar Purkinje cells, TRPC3 channels cause the meta

282 Here, we demonstrate that cerebellar Purkinje cells upregulate excitability in del

283 Here, we investigated these two MAP3Ks in cerebellar Purkinje cells using loss- and gain-of functi

284 Brief depolarization of cerebellar Purkinje cells was found to inhibit parallel

285 Stimulation of a climbing fiber input to cerebellar Purkinje cells was shown to generate an anion

286 ng dendritic patch-clamp recordings from rat cerebellar Purkinje cells, we find that somatic depolari

287 ricted to splenic B-cells, the placenta, and cerebellar Purkinje cells where it colocalized with HNK-

288 ct reduced calcium currents, particularly in cerebellar Purkinje cells, where these channels are most

289 lled by i.c.v. injection of 192-saporin, and cerebellar Purkinje cells which are killed by OX7-sapori

290 neurones, but not in dorsal vagal nucleus or cerebellar Purkinje cells (which express other Kv3 subun

291 We have examined single NMDA channels in cerebellar Purkinje cells (which possess NR1 and 2D), de

292 , nor could we detect loss of noncholinergic cerebellar Purkinje cells, which also express p75.

293 In cerebellar Purkinje cells, which are a key component of

294 Loss of cerebellar Purkinje cells, which are commonly associated

295 ynaptic plasticity at excitatory synapses of cerebellar Purkinje cells, which express the highest lev

296 Some of these terminals are presumably from cerebellar Purkinje cells, which were also labeled by bo

297 s to manipulate and map microRNA function in cerebellar Purkinje cells with temporal precision, revea