ライフサイエンスコーパス: cerebellar cortex

1 encephalon and metencephalon (aside from the cerebellar cortex).

2 el fibre (PF)-Purkinje cell (PC) synapses in cerebellar cortex.

3 undantly and differentially expressed in the cerebellar cortex.

4 y control over the integrated outflow of the cerebellar cortex.

5 branches terminating as mossy fibers in the cerebellar cortex.

6 sors and subsequent massive apoptosis of the cerebellar cortex.

7 ating the flow of sensory information in the cerebellar cortex.

8 sequently, to the processing of input to the cerebellar cortex.

9 at the terminals of basket cell axons in the cerebellar cortex.

10 lators of presynaptic differentiation in the cerebellar cortex.

11 thin both the deep cerebellar nuclei and the cerebellar cortex.

12 the adult cortex, hippocampus, thalamus, and cerebellar cortex.

13 resent time intervals in a robust way in the cerebellar cortex.

14 spiny inhibitory neurons of neostriatum and cerebellar cortex.

15 s, which form one of two major inputs to the cerebellar cortex.

16 itory input from Purkinje cells (PCs) of the cerebellar cortex.

17 f instruction, and last in sites outside the cerebellar cortex.

18 ceiving" parts of the C1 and C3 zones in the cerebellar cortex.

19 GBC was found in the right putamen and left cerebellar cortex.

20 ral tasks is in part computed locally in the cerebellar cortex.

21 e spontaneous firing rate of GoCs in the rat cerebellar cortex.

22 ate and sustain oscillatory processes in the cerebellar cortex.

23 depending on the investigated region of the cerebellar cortex.

24 ons: Brodmann area 19 (occipital cortex) and cerebellar cortex.

25 somatosensory cortices, anterior insula, and cerebellar cortex.

26 e of the most extensively studied regions of cerebellar cortex.

27 ule cells and mapped this convergence across cerebellar cortex.

28 h deficient Ca(2)(+) channel function in the cerebellar cortex.

29 PCs), which represent the sole output of the cerebellar cortex.

30 cy MF signals through the input layer of the cerebellar cortex.

31 evises the inhibitory circuit diagram of the cerebellar cortex.

32 the source for all motor coordination in the cerebellar cortex.

33 transmission through the input layer of the cerebellar cortex.

34 Purkinje cells (PC), the sole output of the cerebellar cortex.

35 types in OPCs from rat optic nerve and mouse cerebellar cortex.

36 between Golgi and granule cells in the mouse cerebellar cortex.

37 paired dendritic claw differentiation in the cerebellar cortex.

38 d protein was significantly increased in the cerebellar cortex.

39 xcessive migration of granule neurons in the cerebellar cortex.

40 s a substantial disynaptic projection to the cerebellar cortex.

41 ant VGLUT in the neocortex, hippocampus, and cerebellar cortex.

42 n granule neuron parallel fiber axons in the cerebellar cortex.

43 n two tangential but orthogonal paths in the cerebellar cortex.

44 oscillations, requiring amplification by the cerebellar cortex.

45 a ubiquitous neurotransmitter throughout the cerebellar cortex.

46 ant VGLUT in the neocortex, hippocampus, and cerebellar cortex.

47 ontact in primary granule neurons of the rat cerebellar cortex.

48 activation of the left prefrontal and right cerebellar cortex.

49 postmitotic mammalian neurons and in the rat cerebellar cortex.

50 ired long-term depression (LTD) induction in cerebellar cortex.

51 nt stimulation (tDCS) applied over the right cerebellar cortex.

52 ion of granule neuron dendrite arbors in the cerebellar cortex.

53 y mossy fibers, thereby gating inputs to the cerebellar cortex.

54 axonal projections (climbing fibers) in the cerebellar cortex.

55 rimary granule neurons of the developing rat cerebellar cortex.

56 anule neurons in dissociated cultures and in cerebellar cortex.

57 naptic granule neuron dendritic claws in the cerebellar cortex.

58 ion of granule neuron dendritic claws in the cerebellar cortex.

59 suggest a novel form of neural coding in the cerebellar cortex.

60 ry inputs and provide the sole output of the cerebellar cortex.

61 nd external granular layer of the developing cerebellar cortex.

62 naptic granule neuron dendritic claws in the cerebellar cortex.

63 lasticity in target nuclei downstream of the cerebellar cortex.

64 g down of neuron production in the postnatal cerebellar cortex.

65 rkinje cells are the only output cell of the cerebellar cortex.

66 reduced postnatal growth but throughout the cerebellar cortex.

67 m group had decreased volume in thalamus and cerebellar cortex.

68 CN) leads to reduced postnatal growth of the cerebellar cortex.

69 n at both the input and output layers of the cerebellar cortex.

70 inje cells, or for CRF2 in any aspect of the cerebellar cortex.

71 d negative stripes of PCs across most of the cerebellar cortex.

72 ion because they form the sole output of the cerebellar cortex.

73 als to generate climbing fibre inputs to the cerebellar cortex.

74 kably selective for the IN compared with the cerebellar cortex.

75 ated by plastic changes occurring within the cerebellar cortex.

76 cells (PCs) provide the sole output from the cerebellar cortex.

77 Purkinje cells in the molecular layer of the cerebellar cortex.

78 n the C1 zone in the copula pyramidis of the cerebellar cortex.

79 thway from the cerebellar nuclei back to the cerebellar cortex.

80 als to generate climbing fibre inputs to the cerebellar cortex.

81 ed by Purkinje cells, the sole output of the cerebellar cortex.

82 to forward signals to climbing fibers in the cerebellar cortex.

83 lele of rs10937625 and reduced expression in cerebellar cortex.

84 stinct areas of the midbrain, brainstem, and cerebellar cortex.

85 laterals and their target neurons within the cerebellar cortex.

86 ssy fibers relaying vestibular inputs to the cerebellar cortex.

87 ion because they form the sole output of the cerebellar cortex.

88 f GABAergic inhibitory synapses in the mouse cerebellar cortex.

89 pment, showing some layer specificity in the cerebellar cortex.

90 erlying communication between modules in the cerebellar cortex.

91 urkinje cells as the major output neurons of cerebellar cortex.

92 For cerebellar cortex, a cerebellar multilayer sandwich (CMS

93 At the input layer of the cerebellar cortex, a single type of interneuron, the Gol

94 eyelid conditioning, where disconnecting the cerebellar cortex abolishes one component of learning, r

95 mbalance of excitation and inhibition in the cerebellar cortex affecting Purkinje cell output may und

96 rkinje cells, the sole output neurons of the cerebellar cortex, also directly inhibit granule cells v

97 In the cerebellar cortex, anatomical and functional evidence in

98 The important roles of both cerebellar cortex and AIP nucleus in eyeblink conditioni

99 e GABAergic input from Purkinje cells of the cerebellar cortex and are thought to contribute to the a

100 sensorimotor cortex, thalamus, contralateral cerebellar cortex and deep cerebellar nuclei (FDR q < 0.

101 Experiments focusing on the role of cerebellar cortex and deep nuclei in delay versus trace

102 comprehensive functional parcellation of the cerebellar cortex and evaluated it by predicting functio

103 hippocampus, we stimulated two sites of the cerebellar cortex and examined hippocampal function at m

104 S: Purkinje cells are the sole output of the cerebellar cortex and fire two distinct types of action

105 Purkinje cells are the sole output of the cerebellar cortex and fire two distinct types of action

106 Purkinje neurons are the output cells of the cerebellar cortex and generate spikes in two distinct mo

107 g fiber conveys an all-or-none signal to the cerebellar cortex and help to link learning and timing t

108 , ventral midbrain, frontal cerebral cortex, cerebellar cortex and hippocampus in Ube3a deficient and

109 rning-related activation in the left lateral cerebellar cortex and in the right premotor and inferior

110 pse as an important target of ethanol in the cerebellar cortex and indicate that ethanol significantl

111 current pharmacological disconnection of the cerebellar cortex and intense sensory stimulation in the

112 nal magnetic resonance imaging (fMRI) of the cerebellar cortex and interposed cerebellar nuclei simul

113 egulate numerous circuit elements within the cerebellar cortex and is well suited to contribute to pr

114 tion available about the organization of the cerebellar cortex and its synaptic inputs, relatively li

115 fMRI signals increased concomitantly in the cerebellar cortex and nuclei during early acquisition of

116 malization methods to assess function of the cerebellar cortex and nuclei during simple hand movement

117 urther investigate the relative roles of the cerebellar cortex and nuclei in eyeblink conditioning, a

118 e and Golgi cells, is the first stage of the cerebellar cortex and processes spatiotemporal informati

119 mate the total number of Purkinje neurons in cerebellar cortex and pyramidal neurons in the hippocamp

120 synaptic dendritic claw morphogenesis in the cerebellar cortex and suggest novel functions for SUMO E

121 ate high-frequency information from both the cerebellar cortex and the two main excitatory inputs of

122 to control the flow of information into the cerebellar cortex and understanding their responses duri

123 The model, comprising both the cerebellar cortex and vestibular nuclei, reproduces beha

124 tory synapses on Purkinje cells (PCs) in the cerebellar cortex, and long-term potentiation and depres

125 n the cerebral cortex, Purkinje cells in the cerebellar cortex, and motor neurons of the somatic moto

126 rkinje cells, the only output neurons of the cerebellar cortex, and their postsynaptic target neurons

127 tal, cingulate, insular, temporoparietal and cerebellar cortex, and with a more localized increase in

128 Although the wiring of the cerebellar cortex appears to be uniform, the neurons in

129 Instead, upstream interneurons within the cerebellar cortex are also characterized by similar prop

130 creasingly posterior lobules of the anterior cerebellar cortex are associated with increasingly compl

131 The neuronal circuits of the cerebellar cortex are essential for motor and sensory le

132 ons (MLIs, stellate and basket cells) of the cerebellar cortex are linked together by chemical and el

133 intraparietal sulcus, and posterior superior cerebellar cortex are modulated by atypically high phasi

134 Different patterns of atrophy of the cerebellar cortex are well known.

135 BPND and SUVR were calculated using the cerebellar cortex as a reference region and were compare

136 rdized uptake value ratios (SUVRs) using the cerebellar cortex as a reference region were calculated

137 ution volume ratio were determined using the cerebellar cortex as a reference region.

138 -SSP values were computed using the pons and cerebellar cortex as reference regions.

139 With the cerebellar cortex as the reference region, the optimal z

140 ferent time points after injection, with the cerebellar cortex as the reference region.

141 t time points post-injection (p.i.) with the cerebellar cortex as the reference region.

142 model estimating DVR (DVR [MRTM]) using the cerebellar cortex as the reference tissue.

143 s widely available at the input stage of the cerebellar cortex, as required by forward models of cere

144 e relevant neural circuits downstream of the cerebellar cortex, as well as the timing requirements of

145 Picrotoxin or muscimol was applied to the cerebellar cortex at the borders of the recording array.

146 modeling with plasma- and reference-region (cerebellar cortex)-based methods was performed.

147 e results suggest that functional input from cerebellar cortex becomes increasingly important for the

148 ing with impaired dendrite patterning in the cerebellar cortex, behavioral analyses reveal that TRPC5

149 type 6, pathology was not restricted to the cerebellar cortex but also involved the cerebellar nucle

150 tribute to the functional recruitment of the cerebellar cortex by decreasing Golgi cell inhibition on

151 stonia via pharmacological excitation of the cerebellar cortex by local application of kainic acid in

152 serve these roles at the input layer of the cerebellar cortex by releasing GABA to inhibit granule c

153 The cerebellar cortex can be used as a reference region for

154 rkinje cells, the sole output neurons of the cerebellar cortex, can also drive motor learning in mice

155 ward inhibition, such as that present in the cerebellar cortex, can contribute to temporal coding.

156 e projections from the inferior olive to the cerebellar cortex, carry sensorimotor error and clock si

157 oactivating inhibitory Purkinje cells in the cerebellar cortex caused robust, short-latency suppressi

158 Neurons in the cerebellar cortex, cerebellar nuclei, and inferior olive

159 ke discharge reflects the main output of the cerebellar cortex, changes in simple spike firing likely

160 Golgi cells are important elements of the cerebellar cortex, controlling the flow of mossy fibre i

161 ys multiple modalities of information to the cerebellar cortex, converging on Purkinje cells (PC), th

162 The cerebellar cortex coordinates movements and maintains ba

163 There was functional involvement of the cerebellar cortex despite no or little structural change

164 Purkinje cells, the sole output cells of the cerebellar cortex, differs between cerebellar modules an

165 Recordings from the cerebellar cortex during conditioning have revealed CS-t

166 The siblings presented cerebellar cortex dysplasia characterized by the presenc

167 r architecture is that Purkinje cells in the cerebellar cortex each receive input from a single climb

168 muscle activity, to the hypothesis that the cerebellar cortex embodies complex internal models of li

169 study uncovers biophysical gradients in the cerebellar cortex enabling a Fourier-like transformation

170 d with the Bergmann glia of the adult murine cerebellar cortex, expresses the stem cell markers Sox2

171 n recapitulate the compressive forces on the cerebellar cortex from primary (e.g., glioblastoma) and

172 ns across models were parieto-temporal lobe, cerebellar cortex, frontal lobe, hypothalamus and striat

173 ficits of AS mice do not arise from impaired cerebellar cortex function.

174 Within the cerebellar cortex, functional magnetic resonance imaging

175 es expressed by glutamatergic neurons in the cerebellar cortex (GAP-43, BDNF, and GABA OLE_LINK2>(A)-

176 absence of any overt external stimulus, the cerebellar cortex generates a slow oscillation that is c

177 tively uniform anatomy and physiology of the cerebellar cortex has given rise to the idea that this s

178 The well established anatomy of the cerebellar cortex has led to suggestions that cerebellar

179 s complete and the major output cells of the cerebellar cortex have been specified.

180 g with the pruning of climbing fibres in the cerebellar cortex, implicates the climbing fibre collate

181 RPC5 regulates dendrite morphogenesis in the cerebellar cortex in a cell-autonomous manner.

182 formation (HF) and, to a lesser degree, the cerebellar cortex in AD cases without diabetes exhibit m

183 bies virus (RV) into selected regions of the cerebellar cortex in cebus monkeys and used retrograde t

184 der Purkinje cells were found in ipsilateral cerebellar cortex in cerebellar lobule HVI and in lobule

185 cating a more fundamental involvement of the cerebellar cortex in CR generation.

186 The role of the cerebellar cortex in eyeblink classical conditioning rem

187 cific role of deep cerebellar nuclei and the cerebellar cortex in eyeblink conditioning are not well

188 This study examined the role of cerebellar cortex in eyeblink conditioning under conditi

189 s (9 mm) suppresses transmission through the cerebellar cortex in low, but not high, alcohol consumin

190 he pathway from the cerebellar nuclei to the cerebellar cortex in mice includes collaterals of cerebe

191 only hypothesized as a major function of the cerebellar cortex in motor control.

192 d their downstream targets in the developing cerebellar cortex in postnatal rat.

193 thological findings reveal severe atrophy of cerebellar cortex in SCA1 patients.

194 n primary neurons and importantly in the rat cerebellar cortex in vivo robustly increases the density

195 ursts can be successfully transmitted to the cerebellar cortex in vivo, having a significant impact o

196 bellar granule neurons, including in the rat cerebellar cortex in vivo, reveals a requirement for the

197 utofluorescence optical imaging in the mouse cerebellar cortex in vivo, this study demonstrates that

198 ging and single-cell recordings in the mouse cerebellar cortex in vivo, this study reexamines the bea

199 alyses in primary rat neurons and in the rat cerebellar cortex in vivo, we report that CaMKIIbeta ope

200 l fiber/Purkinje cell synapses in the rodent cerebellar cortex in vivo.

201 airs migration of granule neurons in the rat cerebellar cortex in vivo.

202 anule neuron parallel fiber axons in the rat cerebellar cortex in vivo.

203 raction-like mechanism of temporal coding in cerebellar cortex in which activity in a subset of granu

204 cells engage mechanisms of plasticity in the cerebellar cortex; in turn, changes in the cerebellum de

205 re >/=30% in the deep cerebellar nuclei, the cerebellar cortex, inferior olive, and thalamus relative

206 tion of several brain regions, including the cerebellar cortex, insect mushroom body, and dentate gyr

207 Large lesions of the midline cerebellar cortex involving the OMV cause saccades to be

208 a new method for stereologic sampling of the cerebellar cortex, involving calculating the volume of t

209 The cerebellar cortex is a crystal-like structure consisting

210 The cerebellar cortex is among the brain's most well-studied

211 cerebellum; activation of this system in the cerebellar cortex is associated with deficits in motor c

212 The cerebellar cortex is centrally involved in motor coordin

213 While research on the cerebellar cortex is crystallizing our understanding of

214 f molecular layer interneurons (MLIs) of the cerebellar cortex is enhanced.

215 At birth, each Purkinje cell (PC) in the cerebellar cortex is innervated by multiple CFs; an acti

216 The cerebellar cortex is involved in the control of diverse

217 eractions among PCs, which suggests that the cerebellar cortex is more functionally diverse than is a

218 The surface of the human cerebellar cortex is much more tightly folded than the c

219 riptional control of circuit assembly in the cerebellar cortex is not well understood.

220 Recently, it has been reported that the cerebellar cortex is required for consolidation of condi

221 An unusual feature of the cerebellar cortex is that its output neurons, Purkinje c

222 The climbing fiber input to the cerebellar cortex is thought to provide instructive sign

223 s), suggesting that the main function of the cerebellar cortex is to shape the timing of CRs.

224 Due to the uniform cyto-architecture of the cerebellar cortex, its overall physiological characteris

225 In cerebellar cortex, Kv3.3 expression was found in Purkinj

226 By using 2-photon microscopy in rodent cerebellar cortex labeled with fluorescent indicator dye

227 e, we provide evidence that SLRs unmasked by cerebellar cortex lesions are mediated by an associative

228 m inhibitory interneurons located within the cerebellar cortex limits the extent of neuronal excitati

229 EMENT Purkinje cells, the sole output of the cerebellar cortex, manifest two fundamentally different

230 It has also been suggested that the cerebellar cortex may be important during early stages o

231 xpression of SLRs depends on both direct and cerebellar cortex-mediated sensory information from the

232 In the cerebellar cortex, molecular layer interneurons use chem

233 ys required for the normal maturation of the cerebellar cortex, notably developmental pathways for gr

234 eta subunit (CTb) into selected areas of the cerebellar cortex of 18 male Wistar rats.

235 osterior cingulate cerebral cortices and the cerebellar cortex of 87 end-of-life patients (64 with AD

236 G methylation in DS and control cerebral and cerebellar cortex of adults and cerebrum of fetuses.

237 Neurophysiological recordings in the cerebellar cortex of awake-behaving animals are revoluti

238 ported by our finding that infusion into the cerebellar cortex of either the Kv1.2 inhibitor tityusto

239 he expression of STK32B was increased in the cerebellar cortex of patients and expression quantitativ

240 tional state of glutamatergic neurons in the cerebellar cortex of patients with schizophrenia.

241 We injected these vectors into the cerebellar cortex of rhesus macaques and tested vector e

242 number of Purkinje and granule cells in the cerebellar cortex of the three strains.

243 We determined that, in the developing rodent cerebellar cortex (of both sexes), there is a transient

244 ior olive or reduce the disinhibition of the cerebellar cortex on the deep cerebellar nuclei could tr

245 ions show that reducing the influence of the cerebellar cortex on the oculomotor pathway reduces the

246 gray matter, controls had stable (thalamus, cerebellar cortex) or decreasing volumes (cortex), where

247 gulate translation information processing in cerebellar cortex output neurons.

248 ues were significantly over-expressed in the cerebellar cortex (P = 1x10(-)(5)) suggesting possible i

249 In the cerebellar cortex, parallel fiber-to-stellate cell (PF-S

250 ule cells, which form the input layer of the cerebellar cortex, permit high-resolution patch-clamp re

251 re under afferent control independent of the cerebellar cortex, potentially diversifying its roles in

252 mpus (CA1), cerebral cortex (layer III), and cerebellar cortex (Purkinje spines), respectively.

253 ciated with deficient CB1R signalling in the cerebellar cortex, putatively linked with compromised Ca

254 The PiB uptake was quantified as a region to cerebellar cortex ratio.

255 dorsal cochlear nucleus (DCN) and vestibular cerebellar cortex receive glutamatergic mossy fiber inpu

256 reater decreases in the occipital cortex and cerebellar cortex, respectively.

257 ns, bidirectional tracer injections into the cerebellar cortex retrogradely labeled somata in the cer

258 Histological analysis of the TR4(-/-) cerebellar cortex reveals reductions in granule cell den

259 inferior olive and multiple areas within the cerebellar cortex showed a robust response to time chang

260 ositive FBB (FBB+), defined by a cortical to cerebellar cortex standardised uptake value ratio (SUVR)

261 y fiber axons, whereas Purkinje cells of the cerebellar cortex strongly expressed Kv3.3 subunits in a

262 was correlated with responses in ipsilateral cerebellar cortex, suggesting a novel computational role

263 tiple ischaemic-like lesions occurred in the cerebellar cortex suggestive of vascular smooth muscle c

264 Stereotyped circuitry within the cerebellar cortex suggests that similar computations are

265 ral morphology of the molecular layer of the cerebellar cortex that are the result of the binge ethan

266 ngs in vivo, and are the only neurons in the cerebellar cortex that express the alpha6delta-containin

267 erate on-beam and off-beam inhibition in the cerebellar cortex that is hypothesized to control the ti

268 ations in the interpositus nucleus (IPN) and cerebellar cortex that were time-locked both to hippocam

269 -the primary source of synaptic input to the cerebellar cortex-that sensory stimulation can produce b

270 In cerebellar cortex the GluR1 AMPA receptor subunit is exp

271 k responses primarily takes place within the cerebellar cortex, the interposed nuclei, or both.

272 In the cerebellar cortex, the labeled fibers were found mostly

273 the sensory mossy fiber input to the IN and cerebellar cortex, then blocking the MCP should abolish

274 Focusing on the cerebellar cortex, thinner collagen fibers and disorgani

275 their axons are the only projection from the cerebellar cortex to deeper cerebellar structures.

276 The ability of cerebellar cortex to generate population rhythms within

277 How does the inner ear communicate with the cerebellar cortex to maintain balance and posture?

278 dy to demonstrate beam-like responses in the cerebellar cortex to peripheral, MF, and GC stimulation

279 je cells guide transfer of learning from the cerebellar cortex to the deep cerebellar nucleus, with e

280 ctions of the GABAergic projections from the cerebellar cortex to the dentate nucleus.

281 eural signals are subject to learning in the cerebellar cortex versus the deep cerebellar nuclei; and

282 eurons regulate plasticity and timing in the cerebellar cortex via the climbing fiber pathway, but di

283 The alpha3-immunoreactivity in the cerebellar cortex was relatively weak, but it was abunda

284 and on-demand optogenetic modulation of the cerebellar cortex was shown to be highly effective at at

285 By focusing on the neocortex and the cerebellar cortex, we demonstrate that reductions of inh

286 ng fibres mediate synaptic plasticity in the cerebellar cortex, we have examined the effects of conju

287 multiple molecular layer interneurons in the cerebellar cortex, we reveal specific, nonrandom connect

288 to the red nucleus also collateralize to the cerebellar cortex, we used a Cre-dependent viral approac

289 tory bulb, hippocampus, cerebral cortex, and cerebellar cortex were exceptionally rich in selenoprote

290 which provide the eye-related output of the cerebellar cortex, were found to increase or decrease th

291 ception to this targeting pattern was in the cerebellar cortex, where PMCA2a also concentrates postsy

292 These neurons form the 'input layer' of the cerebellar cortex, where sensorimotor information carrie

293 ellar nuclei (CN) project profusely into the cerebellar cortex, where they make synaptic contacts on

294 are among the first neurons to populate the cerebellar cortex, where they sprout exuberant axon coll

295 rebellar nuclear connections that can bypass cerebellar cortex, whereas a cerebellar cortical long-te

296 placeable binding was largely reduced in the cerebellar cortex, which in mice was spatially indisting

297 ve identified a pathway linking the vlPAG to cerebellar cortex, which terminates as climbing fibres i

298 s pharmacologically disrupted input from the cerebellar cortex while training with an interstimulus i

299 ition driven by climbing fiber inputs to the cerebellar cortex, with poor retention; (2) learned resp

300 etations of the connectivity and function of cerebellar cortex within the full motor system.