ライフサイエンスコーパス: cerebellar granule cell

1 of cerebellar Purkinje cells and atrophy of cerebellar granule cells.

2 of excitatory AMPA receptors in adult murine cerebellar granule cells.

3 s of [Ca(2+)](i) than other cells, including cerebellar granule cells.

4 inst Tat-induced apoptosis in PC12 cells and cerebellar granule cells.

5 ay an inductive role in the embryogenesis of cerebellar granule cells.

6 e extrasynaptic GABA(A) receptor currents in cerebellar granule cells.

7 , alpha6beta3delta, expressed exclusively in cerebellar granule cells.

8 for the slower decay of spontaneous IPSCs in cerebellar granule cells.

9 nd mature electrophysiological properties of cerebellar granule cells.

10 pmental speeding of miniature EPSCs in mouse cerebellar granule cells.

11 equired for proper migration and survival of cerebellar granule cells.

12 ABAergic synaptic activity in cultured mouse cerebellar granule cells.

13 This hypothesis was also tested in cultured cerebellar granule cells.

14 d stage-specific effects on the migration of cerebellar granule cells.

15 edgehog promotes proliferation of developing cerebellar granule cells.

16 -CAM have overlapping expression patterns in cerebellar granule cells.

17 f tetrodotoxin on ATX-induced Ca2+ influx in cerebellar granule cells.

18 nce of function of adult sensory neurons and cerebellar granule cells.

19 osely the time course of the native I(SA) in cerebellar granule cells.

20 5-methyl-4-isoxazolepropionate) receptors on cerebellar granule cells.

21 overexpress wild-type and mutant SNAP-25 in cerebellar granule cells.

22 tion and glutamate release from cultured rat cerebellar granule cells.

23 sion proteins in PC12 cells and cultured rat cerebellar granule cells.

24 resting intracellular Ca2+ levels in weaver cerebellar granule cells.

25 o control the development of early postnatal cerebellar granule cells.

26 ate excitotoxicity and lithium protection in cerebellar granule cells.

27 tamate-induced excitotoxicity in cultures of cerebellar granule cells.

28 on results in the extensive death of midline cerebellar granule cells.

29 ad, and viability of primary cultures of rat cerebellar granule cells.

30 not polyadenylated, and highly expressed in cerebellar granule cells.

31 the high voltage-activated R-type current in cerebellar granule cells.

32 firmed this close interaction in cultures of cerebellar granule cells.

33 s and endocytosis in synaptic boutons of rat cerebellar granule cells.

34 to a shift in viral tropism from the glia to cerebellar granule cells.

35 st currents through extrasynaptic GABAARs in cerebellar granule cells.

36 ays a critical role in promoting survival of cerebellar granule cells.

37 h Atoh1 and NeuroD1, a marker of postmitotic cerebellar granule cells.

38 d increase neurite outgrowth from developing cerebellar granule cells.

39 l thalamus, dentate gyrus granule cells, and cerebellar granule cells.

40 can participate in integration and firing of cerebellar granule cells.

41 -1 in primary cortical neurons and postnatal cerebellar granule cells.

42 the cerebellum and increases the survival of cerebellar granule cells.

43 thereby regulating excitability in cultured cerebellar granule cells.

44 lium-derived factor (PEDF) protects immature cerebellar granule cells (1-3 days in vitro) against ind

45 the inhibition of cAMP formation in cultured cerebellar granule cells; 2) protection against excitoto

46 elease can activate GABAA receptors in adult cerebellar granule cells: action potential-evoked exocyt

47 y, we suggest that BDNF and FGF2 may protect cerebellar granule cells against excitotoxicity by alter

48 selective ablation of Abl family kinases in cerebellar granule cells alone does not cause any abnorm

49 In cerebellar granule cells, although the frequency of inhi

50 synaptic vesicles in a subset of boutons on cerebellar granule cells, an effect that was reversed by

51 By targeting Pten in cerebellar granule cells and activating the AKT1-mTOR pa

52 ly impaired, leading to massive apoptosis of cerebellar granule cells and degeneration of Purkinje ce

53 ypes and in phenotypic maintenance of mature cerebellar granule cells and dorsal root ganglia.

54 ads to a dramatic reduction in the number of cerebellar granule cells and ectopic location of many of

55 with neuronostatin promoted the migration of cerebellar granule cells and elicited direct depolarizin

56 hIP1 and Kv4.2 are concentrated in somata of cerebellar granule cells and in synaptic glomeruli that

57 BSN colocalized with one another in cultured cerebellar granule cells and in synaptic junction membra

58 Here we show that in both cerebellar granule cells and in Xenopus laevis oocytes e

59 antly up-regulated during differentiation of cerebellar granule cells and is absent from astrocytes i

60 s system prevented apoptosis of cultured rat cerebellar granule cells and macrophages, and the preven

61 nt roles of Smarc proteins and argue against cerebellar granule cells and other progeny of hGFAP-posi

62 atergic parallel fiber (PF) terminals of the cerebellar granule cells and participates in synaptic pl

63 hough the NR2C subunit is highly enriched in cerebellar granule cells and plays a unique role in cere

64 ential role in the migration and survival of cerebellar granule cells and precerebellar neurons and f

65 Its expression is later restricted to cerebellar granule cells and precerebellar neurons exten

66 cations within the hindbrain, giving rise to cerebellar granule cells and precerebellar nuclei.

67 HGF is neuroprotective for cerebellar granule cells and promotes growth of human me

68 reeminence of Ptch1 as a tumor suppressor in cerebellar granule cells and reveal other genomic events

69 ch KCl withdrawal induces apoptotic death of cerebellar granule cells and suggest that additional ele

70 cerebellar granule cells; they produce both cerebellar granule cells and ventral derivatives, some o

71 th SRSF2, hnRNP H1/F, ALYREF and hnRNP A1 in cerebellar granule cells and with SRSF2, hnRNP H1/F and

72 articipates in the AraC-induced apoptosis of cerebellar granule cells and, if so, the relationship be

73 r theory predicts that the dimensions of the cerebellar granule-cell and Drosophila Kenyon-cell repre

74 ATX is neurotoxic in primary cultures of rat cerebellar granule cells, and this neuronal death is pre

75 pontaneously develop ataxia, degeneration of cerebellar granule cells, and vacuolation of white matte

76 T-20 cells, pituitary gonadotropes, cultured cerebellar granule cells, and yeast with high concentrat

77 es to afferent mixing.SIGNIFICANCE STATEMENT Cerebellar granule cells are among the simplest neurons,

78 Cerebellar granule cells are an attractive model system

79 e, we show that multiple synapses of waggler cerebellar granule cells are arrested at an immature sta

80 Cerebellar granule cells are inhibited phasically by GAB

81 aptic alpha6-containing GABA(A) receptors in cerebellar granule cells are selectively regulated by AM

82 Mutant cerebellar granule cells are susceptible to exogenous an

83 Cerebellar granule cells are the most abundant neurons i

84 Cerebellar granule cells are the most abundant neurons i

85 Cerebellar granule cells are the most abundant type of n

86 RRL clones comprised the EGL and cerebellar granule cells, as expected.

87 ternalization of GABAA receptors in cultured cerebellar granule cells, as reflected by diminished imm

88 In vitro and in vivo cerebellar granule cell assays show a decrease in neuron

89 nsients in individual presynaptic boutons of cerebellar granule cells at near-physiological temperatu

90 port that activation of GABA(A) receptors on cerebellar granule cell axons modulates both transmitter

91 e have shown that exogenous GABA depolarizes cerebellar granule cell axons through local activation o

92 The growth of cerebellar granule cell axons was examined by placing fo

93 t of clinical illness and slows apoptosis of cerebellar granule cells but has no effect on white matt

94 rtant roles in the apoptotic death of weaver cerebellar granule cells, but the downstream events asso

95 n the regulation of kainate neurotoxicity in cerebellar granule cells by calcium entry through voltag

96 it is possible that inhibition of IK(SO) in cerebellar granule cells by synaptically released zinc m

97 VPA treatment of rat cerebellar granule cells caused a robust dose- and time-

98 Whereas human cerebellar granule cells (CGC) express only MT1 receptor

99 otoxins produce neuronal injury and death in cerebellar granule cells (CGC) following acute exposure.

100 rahydrofolate (5-FTHF) into primary cultured cerebellar granule cells (CGC) was studied.

101 on primary neuronal cultures, consisting of cerebellar granule cells (CGC).

102 and zeta are present in rat primary cultured cerebellar granule cells (CGCs) 6-14 days in vitro (DIV)

103 y expressed in mouse cerebellum and cultured cerebellar granule cells (CGCs) and modulates P/Q-type c

104 ncreasing the expression of NMDA subunits in cerebellar granule cells (CGCs) by transfection was stud

105 excitatory synaptic currents in cultures of cerebellar granule cells (CGCs) from NR2A knockout (NR2A

106 relatively simple and compact morphology of cerebellar granule cells (CGCs) has led to the view that

107 One of these is a model of cerebellar granule cells (CGCs) in which apoptosis is in

108 Cerebellar granule cells (CGCs) need NMDAR stimulation t

109 ncrease of tonic current was observed in the cerebellar granule cells (CGCs) of alpha1-/- compared wi

110 -tagged PSD-95 (PSD-95gfp) into cultured rat cerebellar granule cells (CGCs) to investigate the role

111 A miniature IPSCs (mIPSCs) from cultured rat cerebellar granule cells (CGCs) while varying the extrac

112 To further examine Srg1 expression in cerebellar granule cells (CGCs), we used an in vitro cel

113 ors (GlyRs) have been repeatedly detected in cerebellar granule cells (CGCs), where they deliver excl

114 apoptotic and necrotic death of EtOH-exposed cerebellar granule cells (CGCs).

115 for assessing the effects of disturbances in cerebellar granule cell circuitry on gait and other aspe

116 P endonuclease, were significantly higher in cerebellar granule cells compared with astrocytes.

117 Cerebellar granule cells constitute the majority of neur

118 weaver cerebellar granule cells could be rescued from cell deat

119 ssive mouse mutation that causes progressive cerebellar granule cell death and peripheral motor axon

120 t in a well characterized in vitro paradigm, cerebellar granule cell death induced by withdrawal of d

121 In cerebellar granule cells, delta subunit-containing GABA(

122 Comparatively, mitochondria isolated from cerebellar granule cells demonstrated a higher Ca2+ upta

123 Cerebellar granule cells demonstrated high TUNEL stainin

124 role for a CaMKK2/CaMKIV cascade involved in cerebellar granule cell development and show specificall

125 , Ig-NRG, plays a crucial role in aspects of cerebellar granule cell development in vitro.

126 s indicate that Nr-CAM and L1 play a role in cerebellar granule cell development, and suggest that cl

127 g developmental apoptosis in early postnatal cerebellar granule cells, dorsal root ganglia, embryonic

128 excitatory postsynaptic currents (EPSCs) in cerebellar granule cells during a time-varying, quantifi

129 A expression was rapidly induced in cultured cerebellar granule cells during apoptosis induced by a l

130 e examined how MeHg affects the migration of cerebellar granule cells during early postnatal developm

131 FGF22 is expressed by cerebellar granule cells during the period when they rec

132 Cerebellar granule cells exhibit distinct modes of migra

133 Primary XRCC1 heterozygous mouse cerebellar granule cells exhibit increased strand break

134 Many neurons, including cerebellar granule cells, exhibit a tonic GABA current m

135 xin-sensitive stimulation of 22Na+ influx in cerebellar granule cells exposed to ATX.

136 torage; and viability of primary cultures of cerebellar granule cells exposed to NMDA receptor agonis

137 Apparently, the PM of cerebellar granule cells exposed to NMDA was more depola

138 Unlike mammals, only few cerebellar granule cells expressed AptCB1R transcripts a

139 Nr-CAM-deficient cerebellar granule cells failed to extend neurites in vi

140 Cerebellar granule cells fire in bursts, and their paral

141 ced life span, ataxic gait with apoptosis of cerebellar granule cells followed by Purkinje cell deple

142 Mossy fiber afferents to cerebellar granule cells form the primary synaptic relay

143 In cultured rat cerebellar granule cells, FRET between CFP-SNAP-25 and Y

144 Immunohistochemistry of primary cerebellar granule cells from ataxia3 mice demonstrated

145 sistant phosphomimetic MEF2D mutant protects cerebellar granule cells from cell death after DNA damag

146 and extrasynaptic GABA-mediated currents in cerebellar granule cells from Gabra6(100R/100R) and Gabr

147 rescued excitatory synaptic transmission in cerebellar granule cells from stargazer mice, the decay

148 Cerebellar granule cell GABAA receptor responses to alco

149 e we demonstrate that genetic differences in cerebellar granule cell GABAA receptor responses to recr

150 vel and implications of such interactions at cerebellar granule cell GABAA receptors are discussed.

151 Here, we show that strengthening of mouse cerebellar granule cell GABAergic synapses occurs by a d

152 Dissociated cultures of cerebellar granule cells (GCs) demonstrate improved long

153 ple reactomes of epigenetic pathway genes in cerebellar granule cells (GCs) during circuit formation.

154 Cerebellar granule cells (GCs) make up the majority of a

155 not KChIP2) were expressed at high levels in cerebellar granule cells (GCs).

156 esicle priming that is strongly expressed in cerebellar granule cells (GCs).

157 Ethanol enhanced function of cerebellar granule cell GIRKs coupled to GABAB receptors

158 at GABAergic neuronal progenitors as well as cerebellar granule cells give rise to MBG3 with their di

159 In cultured cerebellar granule cells, GlnT is expressed only on neur

160 Golgi cells (GoCs) of the cerebellum inhibit cerebellar granule cells (GrCs) and are driven both by f

161 Cerebellar granule cells (GrCs) constitute over half of

162 lls project via the pons to a vast number of cerebellar granule cells (GrCs), forming a fundamental p

163 ted neurite outgrowth of rat postnatal day 6 cerebellar granule cells grown on a substratum of NgCAM,

164 HEK293 cells, and native GABAA receptors of cerebellar granule cells, hippocampal neurons, and thala

165 This resulted in early cerebellar granule cell hyper-proliferation and a 48% in

166 )-alpha6 subunit mRNA, which is expressed in cerebellar granule cells in an activity-independent mann

167 We show here that Cbln1 is secreted from cerebellar granule cells in complex with a related prote

168 We used rat cerebellar granule cells in culture because these neuron

169 The results demonstrate that cerebellar granule cells in culture express functional k

170 these observations, presynaptic terminals of cerebellar granule cells in dilute-lethal mice showed in

171 ion of 5-bromo-2'-deoxyuridine-labeled mouse cerebellar granule cells in slice cultures, underscoring

172 s on oligodendrocytes, striatal neurons, and cerebellar granule cells in the context of altered micro

173 or CaMKIV disrupts the ability of developing cerebellar granule cells in the external granule cell la

174 programs regulate the autonomous turning of cerebellar granule cells in vitro.

175 its role in the cell cycle-related death of cerebellar granule cells in vivo.

176 dent, p53-independent cell death response in cerebellar granule cells in vivo.

177 sults suggest that AraC-induced apoptosis of cerebellar granule cells involves the expression of both

178 The cerebellar granule cell is the most numerous neuron in t

179 nic conductance through glycine receptors of cerebellar granule cells is a yet undiscovered element o

180 t growth factor (FGF) receptor activation in cerebellar granule cells is associated with increased GA

181 gions to MeHg exposure, and profound loss of cerebellar granule cells is detected in the brains of pa

182 between proliferation and differentiation in cerebellar granule cells is poorly understood.

183 he molecular correlate of R-type currents in cerebellar granule cells is the alpha1E subunit, which y

184 this phenomenon are beginning to emerge: in cerebellar granule cells, it alters the gain of transmis

185 with this idea, we detected UPR induction in cerebellar granule cells lacking stargazin.

186 Combinatorial expansion by the cerebellar granule cell layer (GCL) is fundamental to th

187 ic and nontransgenic mice was highest in the cerebellar granule cell layer and hippocampal neuronal l

188 idence suggests that local activation of the cerebellar granule cell layer produces a much more restr

189 The localization of apoptosis to the cerebellar granule cell layer, identified these cells as

190 bution of TWIK-1, with highest levels in the cerebellar granule cell layer, thalamic reticular nucleu

191 c loss of GABA(A) receptor expression in the cerebellar granule cell layer.

192 mGlu1b receptor mRNA levels increased in the cerebellar granule cell layer.

193 ule cell layer and throughout the cortex and cerebellar granule cell layer.

194 on of TREK-2 was primarily restricted to the cerebellar granule cell layer.

195 ughout all forebrain areas as well as in the cerebellar granule cell layer.

196 rmal granule cells and the transformed human cerebellar granule cell line DAOY, OGR1 promoted express

197 en receptors (ERs) expressed in the immature cerebellar granule cell line E(t)C.1 by transfecting suc

198 rcity, tonically active glycine receptors of cerebellar granule cells make a significant impact on ac

199 fects on integration of excitatory inputs by cerebellar granule cells might result from different mod

200 ein (GRIP) and the physiologic regulation of cerebellar granule cell migration by SR.

201 ed on our studies, we propose that defective cerebellar granule cell migration secondary to disorgani

202 Ca(2+) elevations triggers the completion of cerebellar granule cell migration.

203 ally inhibited PC12 cell differentiation and cerebellar granule cell migration.

204 o mediate SDF-1alpha/CXCR4 signal in guiding cerebellar granule cell migration.

205 3 gene product, shown previously to regulate cerebellar granule cell migrations, also controls the gu

206 levation of nuclear CaM also was observed in cerebellar granule cells, neocortical neurons, and denta

207 studies define an essential role for Brd4 in cerebellar granule cell neurogenesis and are critical fo

208 activation is required for proliferation of cerebellar granule cell neuron precursors during develop

209 ical, and histological findings of a case of cerebellar granule cell neuronopathy (GCN), a JCV-associ

210 e have demonstrated that JC virus can infect cerebellar granule cell neurons and cortical pyramidal n

211 ary cell culture revealed that Ifi27l2a(-/-) cerebellar granule cell neurons and macrophages but not

212 grated into the cerebellum, and WNV-infected cerebellar granule cell neurons expressed higher levels

213 d macrophages, dendritic cells, fibroblasts, cerebellar granule cell neurons, and cortical neurons re

214 crophages, dendritic cells, fibroblasts, and cerebellar granule cell neurons, but not cortical neuron

215 ies were investigated in primary cultures of cerebellar granule cell neurons.

216 which is required for normal development of cerebellar granule cell neurons.

217 erived neurotrophic factor (BDNF) protein in cerebellar granule cell neurons.

218 CNS, we demonstrated a dramatic increase in cerebellar granule cell number that appeared to be attri

219 have examined the effects of somatostatin in cerebellar granule cells of early postnatal mice, becaus

220 els of BDNF and higher rates of apoptosis in cerebellar granule cells of neuroD2(-/-) mice indicate t

221 lices rich in OECs enhanced axonal growth of cerebellar granule cells or hippocampal neurons; axons g

222 a has been attributed to loss of AMPARs from cerebellar granule cells, other cerebellar neurons have

223 Here we show that migration of cerebellar granule cells out of their proliferative zone

224 rmalities, we examined the ultrastructure of cerebellar granule cell output synapses and measured the

225 ors and A(1) receptors are both localized on cerebellar granule cell parallel fiber terminals and bas

226 At the synapse between cerebellar granule cell parallel fibers (PFs) and Purkin

227 pendent manner by comparing synapses made by cerebellar granule cell parallel fibers onto Golgi cells

228 Glycine receptors of cerebellar granule cells play their functional role not

229 xpressed by developing inner ear hair cells, cerebellar granule cells, precerebellar neurons, and col

230 Here we show that cerebellar granule cell precursors (GCPs) migrate along

231 Hedgehog signaling controls proliferation of cerebellar granule cell precursors (GCPs), and its aberr

232 ons that express high levels of Ptchd1 mRNA: cerebellar granule cell precursors and dentate granule c

233 These HSPG co-receptors are expressed by cerebellar granule cell precursors and promote Shh bindi

234 facial branchiomotor nucleus, the spread of cerebellar granule cell precursors to form the external

235 nuclei, while later derivatives comprise of cerebellar granule cell precursors, a unique proliferati

236 d, Sonic hedgehog, as a powerful mitogen for cerebellar granule cell precursors.

237 specifically deleted Smarca4 and Smarcb1 in cerebellar granule cell precursors.

238 Cerebellar granule cells prepared from p53 knock-out mic

239 Cerebellar granule cell progenitors (GCP) proliferate ex

240 tic cerebellum in which the proliferation of cerebellar granule cell progenitors (GCPs) in response t

241 ere we have shown that deletion of Chd7 from cerebellar granule cell progenitors (GCps) results in re

242 neuronal cells in the mammalian cerebellum, cerebellar granule cell progenitors (GCPs), we performed

243 illustrate the utility of MADM, we show that cerebellar granule cell progenitors are fated at an earl

244 Cerebellar granule cell progenitors proliferate postnata

245 is abundantly expressed in the proliferating cerebellar granule cell progenitors, regulates the cell

246 og (Shh) signaling, which normally regulates cerebellar granule cell proliferation.

247 tering mice have been shown to have abnormal cerebellar granule cell-Purkinje cell synapses and leane

248 Cerebellar granule cells receive five orders of magnitud

249 Cerebellar granule cells receive mossy fiber inputs that

250 After their final mitosis, cerebellar granule cells remain in the external granular

251 -isoxazolepropionic acid (AMPA) receptors in cerebellar granule cells requires stargazin, a member of

252 s [delayed Ca(2+) deregulation (DCD)] in rat cerebellar granule cells resulting from chronic activati

253 e imaging of Venus-tagged ASTN1 in migrating cerebellar granule cells reveals the intracellular traff

254 cipated passive and active properties of the cerebellar granule cell's unmyelinated axon.

255 is concentrated along the plasma membrane of cerebellar granule cell somata and dendrites.

256 nal Ca(2+) channel gamma subunit, exhibits a cerebellar granule cell-specific brain-derived neurotrop

257 on of GABA(A) receptor alpha6, delta (mature cerebellar granule cell-specific proteins), and beta3 su

258 Finally, cerebellar granule-cell-specific CB1KOs exhibit normal e

259 t experiments on hypoglossal motoneurons and cerebellar granule cells suggest that the resting curren

260 vels of genes encoding proteins that support cerebellar granule cell survival, including brain-derive

261 wly, K(+) was retained in the cytoplasm, and cerebellar granule cells survived the challenge; in the

262 ng.In conclusion, this study demonstrates in cerebellar granule cells that external pH can either red

263 w that Cbln1 is a glycoprotein secreted from cerebellar granule cells that is essential for three pro

264 extracellular matrix protein synthesized in cerebellar granule cells that plays an important role in

265 TARP expression in gamma-2-lacking stargazer cerebellar granule cells--the classic model of TARP defi

266 r cells are not restricted to producing only cerebellar granule cells; they produce both cerebellar g

267 ting the derivation of medulloblastomas from cerebellar granule cells through activation of the Sonic

268 ge in intrinsic membrane excitability of rat cerebellar granule cells through modification of Kv4 A-t

269 Cerebellar granule cell to Purkinje cell synapses have b

270 ordings and modeling of synaptic activity at cerebellar granule cell to Purkinje cell synapses of mic

271 For cerebellar granule cell to stellate cell (grc-->SC) syna

272 iating the chemotactic response of migrating cerebellar granule cells to BDNF through its regulation

273 We have used primary cultures of rat cerebellar granule cells to examine mechanisms whereby g

274 Exposure of cerebellar granule cells to glutamate induces a rapid in

275 Furthermore, stimulation of fused knockout cerebellar granule cells to proliferate with Sonic Hh re

276 pac is involved in long term potentiation at cerebellar granule cell-to-Purkinje cell synapses.

277 Likewise, in cerebellar granule cells transfected with vGlut1-pHluori

278 anule cell-Purkinje cell synapses and leaner cerebellar granule cells undergo abnormal apoptosis duri

279 uced currents by external H+ was examined in cerebellar granule cells using whole-cell and single-cha

280 AraC-induced apoptosis of cerebellar granule cells was preceded by an increase in

281 Neurite outgrowth of cerebellar granule cells was stimulated on astrocytes ex

282 rs with endogenous AMPAR currents from mouse cerebellar granule cells, we have determined a likely pr

283 he modulation of GAP-43 expression, cultured cerebellar granule cells were exposed to these transmitt

284 ine kinetics and consequences to morphology, cerebellar granule cells were treated in vitro with d-th

285 Primary cerebellar granule cells were used in this study to dete

286 Cultured cerebellar granule cells were used to examine the protec

287 ate (NMDA) receptors are highly expressed in cerebellar granule cells where they mediate the majority

288 eport here that erbB4 is expressed in mature cerebellar granule cells, where it appears to be concent

289 rongly for glucocorticoid receptors, such as cerebellar granule cells, where PP5 is either absent or

290 ons, such as hippocampal pyramidal cells and cerebellar granule cells, where we have previously also

291 Cerebellar granule cells, which constitute half the brai

292 ncipal cytopathology appears to be a loss of cerebellar granule cells, which frequently display conde

293 uman brain contains approximately 60 billion cerebellar granule cells, which outnumber all other brai

294 f the TrkC ligand neurotrophin-3 (NT-3) from cerebellar granule cells, which provide major afferent i

295 polarized manner facing the leading pole of cerebellar granule cells with a migratory morphology.

296 Pretreatment of cerebellar granule cells with IL-10 (1-50 ng/ml) elicite

297 g term, but not acute, treatment of cultured cerebellar granule cells with LiCl induces a concentrati

298 Pretreatment of aging cerebellar granule cells with lithium or VPA alone provi

299 ceptor, or BDNF, act as chemoattractants for cerebellar granule cells, with SDF-1 action being select

300 ptors increased kainate receptor activity in cerebellar granule cells without changing NMDA receptors