ライフサイエンスコーパス: cerebral amyloid angiopathy

1 farcts, Circle of Willis atherosclerosis, or cerebral amyloid angiopathy.

2 ts, displayed a prominent Abeta38-containing cerebral amyloid angiopathy.

3 fect of HJ6.3 on total plasma cholesterol or cerebral amyloid angiopathy.

4 with the immune system and the importance of cerebral amyloid angiopathy.

5 ial activation and enhancing astrocytosis or cerebral amyloid angiopathy.

6 ients with multiple lobar CMBs suggestive of cerebral amyloid angiopathy.

7 he effects of these gene variants on risk of cerebral amyloid angiopathy.

8 lihood of developing Alzheimer's disease and cerebral amyloid angiopathy.

9 mice in parallel to significantly increased cerebral amyloid angiopathy.

10 partly by attenuating neuroinflammation and cerebral amyloid angiopathy.

11 plicated in other amyloid diseases including cerebral amyloid angiopathy.

12 mutations associated with familial forms of cerebral amyloid angiopathy.

13 tide (Abeta) in Alzheimer's disease (AD) and cerebral amyloid angiopathy.

14 th a hereditary amyloid disease known as PrP cerebral amyloid angiopathy.

15 that forms amyloid deposits associated with cerebral amyloid angiopathy.

16 d Asp-23 that cause familial forms of AD and cerebral amyloid angiopathy.

17 nd the walls of meningoencephalic vessels as cerebral amyloid angiopathy.

18 tions at Glu-22 with Alzheimer's disease and cerebral amyloid angiopathy.

19 neuronal and vascular degeneration in AD and cerebral amyloid angiopathy.

20 and Abeta peptides causing familial forms of cerebral amyloid angiopathy.

21 , which are pathological hallmarks of AD and cerebral amyloid angiopathy.

22 intracerebral hemorrhage is the hallmark of cerebral amyloid angiopathy.

23 e and that this contributes significantly to cerebral amyloid angiopathy.

24 aged cerebral blood vessels of patients with cerebral amyloid angiopathy.

25 ogical features with Alzheimer's disease and cerebral amyloid angiopathy.

26 nces in response to amyloid beta plaques and cerebral amyloid angiopathy.

27 and Abeta3pE-40, an Abeta-pattern similar to cerebral amyloid angiopathy.

28 ition and 80-90% AD subjects also experience cerebral amyloid angiopathy.

29 ological hallmark of Alzheimer's disease and cerebral amyloid angiopathy.

30 issue after both ICH and ICH associated with cerebral amyloid angiopathy.

31 myloid plaques, neurofibrillary tangles, and cerebral amyloid angiopathy.

32 at two sites near APOC2 was associated with cerebral amyloid angiopathy.

33 its in cortical blood vessels reminiscent of cerebral amyloid angiopathy.

34 r younger patients presenting with suspected cerebral amyloid angiopathy.

35 sporadic AD and two pathways associated with cerebral amyloid angiopathy.

36 des excellent contrast for Abeta plaques and cerebral amyloid angiopathy.

37 ablished haemorrhagic neuroimaging marker of cerebral amyloid angiopathy.

38 t risk of iatrogenic Alzheimer's disease and cerebral amyloid angiopathy.

39 ound B values, but not Braak tangle stage or cerebral amyloid angiopathy.

40 ial siderosis, with a particular emphasis on cerebral amyloid angiopathy.

41 2%) with arteriolosclerosis, 23 (20.4%) with cerebral amyloid angiopathy, 18 (15.9%) with mixed SVD,

42 ive response DNA-binding protein 43, 8.1% to cerebral amyloid angiopathy, 6.0% to atherosclerosis, an

43 ng in 38 non-demented patients with probable cerebral amyloid angiopathy (69 +/- 10 years) and 29 sim

44 -97; I(2)=0%), with common co-pathologies of cerebral amyloid angiopathy (71%, 54-88; I(2)=89%), Lewy

45 th early and late onset Alzheimer's disease (cerebral amyloid angiopathy: 86% versus 79%, Fisher exac

46 om animal models point to a central role for cerebral amyloid angiopathy - a condition characterized

47 ic CSF mobility alterations in patients with cerebral amyloid angiopathy, a brain disorder associated

48 e causes, it is emerging as a key feature of cerebral amyloid angiopathy, a common and important age-

49 early manifestations of Iowa-type hereditary cerebral amyloid angiopathy, a form of the disorder with

50 redominantly of hyperphosphorylated tau; and cerebral amyloid angiopathy, a microangiopathy affecting

51 s were conducted adjusting for ICH location, cerebral amyloid angiopathy according to Boston 2.0 or s

52 blood vessel walls, potentially resulting in cerebral amyloid angiopathy after several decades.

53 frequent diffuse plaques and moderate/severe cerebral amyloid angiopathy (all P < 0.05).

54 a patient with dementia can be secondary to cerebral amyloid angiopathy alone.

55 rly-phase trials of candidate treatments for cerebral amyloid angiopathy, an untreatable cause of hae

56 in the brains of wild-type mice, and formed cerebral amyloid angiopathy and Abeta plaques after a 12

57 -beta and phospho-tau pathology, severity of cerebral amyloid angiopathy and cortical microhaemorrhag

58 microinfarcts are linked to pathologies like cerebral amyloid angiopathy and dementia.

59 the blood vessel walls was characteristic of cerebral amyloid angiopathy and did not co-localize with

60 a with Lewy bodies, multiple system atrophy, cerebral amyloid angiopathy and elderly controls free of

61 CA1 also considerably increased the level of cerebral amyloid angiopathy and exacerbated cerebral amy

62 In summary, more severe levels of cerebral amyloid angiopathy and higher parenchymal beta-

63 ctors may play a role in the pathogenesis of cerebral amyloid angiopathy and in the accumulation of A

64 The severity of cerebral amyloid angiopathy and microhaemorrhages did no

65 an age 85 +/- 6 years) with pathology-proven cerebral amyloid angiopathy and multiple microbleeds on

66 Cerebral amyloid angiopathy and neurofibrillary tangles

67 ht mediate the relationship between advanced cerebral amyloid angiopathy and neurologic dysfunction a

68 of cerebral small vessel disease (lobar for cerebral amyloid angiopathy and non-lobar for arteriolos

69 in vivo observed interaction effects between cerebral amyloid angiopathy and plaque density (beta = 0

70 rfamily are involved in an inherited form of cerebral amyloid angiopathy and readily form amyloid fib

71 ce of the grey matter and in artery walls as cerebral amyloid angiopathy and tau protein accumulates

72 ral haemorrhage (39 with clinically probable cerebral amyloid angiopathy) and 47 age-matched controls

73 as vascular dementia, small vessel disease, cerebral amyloid angiopathy, and Alzheimer's disease.

74 arteriolosclerotic small vessel disease and cerebral amyloid angiopathy, and determined the Braak ta

75 mice accelerated amyloid loading, increased cerebral amyloid angiopathy, and reduced insulin-signali

76 ia induced by Abeta and in the mechanisms of cerebral amyloid angiopathy, and support the possibility

77 e form of frequent cortical amyloid plaques, cerebral amyloid angiopathy, and tauopathy.

78 maging are specific for microhaemorrhages in cerebral amyloid angiopathy, and that increasing the res

79 ing-defined microbleeds and microinfarcts in cerebral amyloid angiopathy, and to explore the patholog

80 genotype to risk of Alzheimer's disease and cerebral amyloid angiopathy, APOE has been studied in ot

81 clear that, at the pathophysiological level, cerebral amyloid angiopathy appears to be in part a prot

82 Neuroimaging markers of severe cerebral amyloid angiopathy are cortical microbleeds and

83 Although chronic hypertension and cerebral amyloid angiopathy are the underlying cerebral

84 We show that both TDP-43 proteinopathy and cerebral amyloid angiopathy associate with cognitive imp

85 ratio [aOR], 2.08; 95% CI, 1.32 to 3.27) and cerebral amyloid angiopathy at index ICH (aOR, 2.21; 95%

86 tomography can noninvasively detect isolated cerebral amyloid angiopathy before overt signs of tissue

87 mentia (FBD), previously designated familial cerebral amyloid angiopathy-British type, is an autosoma

88 a significant reduction of Abeta plaques and cerebral amyloid angiopathy burden and a reduction of th

89 ab, even in vessels where there was a severe cerebral amyloid angiopathy burden.

90 in microglia and is associated with greater cerebral amyloid angiopathy but not Alzheimer's disease

91 mice, effects associated with a reduction of cerebral amyloid angiopathy but not amyloid plaques.

92 olipoprotein E4 genotype was associated with cerebral amyloid angiopathy, but not HS or arteriosclero

93 hind the observed effects in TgSwDI mouse, a cerebral amyloid angiopathy (CAA) and AD model.

94 d role of amyloid-beta (Abeta) deposition in cerebral amyloid angiopathy (CAA) and Alzheimer disease

95 In cerebral amyloid angiopathy (CAA) and Alzheimer's diseas

96 uding old and recent infarcts, haemorrhages, cerebral amyloid angiopathy (CAA) and arteriosclerosis,

97 Cerebral amyloid angiopathy (CAA) and beta-amyloid (Abet

98 of parenchymal amyloid-beta (Abeta) plaques, cerebral amyloid angiopathy (CAA) and neurofibrillary ta

99 s and subgroups of 190 AD subjects exhibited cerebral amyloid angiopathy (CAA) and other frequently a

100 Furthermore, all the oligomers induce cerebral amyloid angiopathy (CAA) and plaque burden in t

101 nile plaques and along cerebrovasculature as cerebral amyloid angiopathy (CAA) are hallmarks of AD.

102 farcts and leukoencephalopathy (CADASIL) and cerebral amyloid angiopathy (CAA) are two distinct vascu

103 c or primary angiitis of the CNS (PACNS) and cerebral amyloid angiopathy (CAA) are unusual vasculopat

104 Cerebral Amyloid Angiopathy (CAA) consists of cerebrovas

105 utive patients with pathologically diagnosed cerebral amyloid angiopathy (CAA) for evidence of an inf

106 Although cerebral amyloid angiopathy (CAA) has important clinical

107 Cerebral amyloid angiopathy (CAA) has never been more re

108 Targeting Abeta to prevent cerebral amyloid angiopathy (CAA) has not been rigorousl

109 between intracerebral haemorrhage (ICH) and cerebral amyloid angiopathy (CAA) in a systematic review

110 oligomeric strain selectively induces acute cerebral amyloid angiopathy (CAA) in neonatally-injected

111 Sporadic cerebral amyloid angiopathy (CAA) is a common age relate

112 Cerebral amyloid angiopathy (CAA) is a common age-relate

113 Cerebral amyloid angiopathy (CAA) is a common cause of b

114 Cerebral amyloid angiopathy (CAA) is a common cause of i

115 Cerebral amyloid angiopathy (CAA) is a common cause of s

116 Cerebral amyloid angiopathy (CAA) is a common cause of s

117 he brain in the form of neuritic plaques and cerebral amyloid angiopathy (CAA) is a key feature of Al

118 Cerebral amyloid angiopathy (CAA) is a prevalent vascula

119 to its role in hemorrhagic stroke, advanced cerebral amyloid angiopathy (CAA) is also associated wit

120 Cerebral amyloid angiopathy (CAA) is an age-associated c

121 Cerebral amyloid angiopathy (CAA) is an age-related smal

122 Cerebral amyloid angiopathy (CAA) is an important cerebr

123 Cerebral amyloid angiopathy (CAA) is associated with lob

124 Cerebral amyloid angiopathy (CAA) is characteristically

125 Cerebral amyloid angiopathy (CAA) is characterized by de

126 Cerebral amyloid angiopathy (CAA) is characterized by th

127 Cerebral amyloid angiopathy (CAA) is characterized by th

128 Cerebral amyloid angiopathy (CAA) is common in Alzheimer

129 Cerebral amyloid angiopathy (CAA) is common in the agein

130 Cerebral amyloid angiopathy (CAA) is the accumulation of

131 Cerebral amyloid angiopathy (CAA) may play a key role in

132 arch output and major trends in the field of cerebral amyloid angiopathy (CAA) over six decades, from

133 Cerebral amyloid angiopathy (CAA) results from depositio

134 euritic plaques, but has no direct effect on cerebral amyloid angiopathy (CAA) severity, whereas APOE

135 d densities of tangles, neuropil threads and cerebral amyloid angiopathy (CAA) similar to unimmunized

136 scans for hematoma location and presence of cerebral amyloid angiopathy (CAA) using computed tomogra

137 on in brain parenchyma and blood vessels (as cerebral amyloid angiopathy (CAA)) and by neurofibrillar

138 isease (mainly hypertensive arteriopathy and cerebral amyloid angiopathy (CAA)) is an important cause

139 ovascular function is an early biomarker for cerebral amyloid angiopathy (CAA), a neurovascular disea

140 st AD patients have cerebrovascular amyloid (cerebral amyloid angiopathy (CAA), and cardiovascular ri

141 We postulated that cerebral amyloid angiopathy (CAA), characterised by cort

142 Cerebral amyloid angiopathy (CAA), characterized by cere

143 A definite diagnosis of cerebral amyloid angiopathy (CAA), characterized by the

144 d beta-protein (Abeta), a condition known as cerebral amyloid angiopathy (CAA), is a common pathologi

145 cerebral blood vessels, a condition known as cerebral amyloid angiopathy (CAA), is a key pathological

146 d beta-protein (Abeta), a condition known as cerebral amyloid angiopathy (CAA), is a key pathological

147 eptides in the walls of brain blood vessels, cerebral amyloid angiopathy (CAA), is common in patients

148 otein E (apoE) genotype in hemorrhage due to cerebral amyloid angiopathy (CAA), it is unclear whether

149 Cerebral amyloid angiopathy (CAA), limbic-predominant ag

150 -beta (Abeta) in cerebral arteries, known as cerebral amyloid angiopathy (CAA), occurs both in the se

151 18-month-old APPsw (Tg2576) mice, a model of cerebral amyloid angiopathy (CAA), suggesting a pivotal

152 Cerebral amyloid angiopathy (CAA), the deposition of cer

153 Cerebral amyloid angiopathy (CAA), where beta-amyloid (A

154 ain vasculature lead to a condition known as cerebral amyloid angiopathy (CAA), which impairs blood-b

155 There is increasing evidence that cerebral amyloid angiopathy (CAA), which itself can caus

156 l confirmation, clinical cases of iatrogenic cerebral amyloid angiopathy (CAA)-attributed to the tran

157 proteinase-9 (MMP-9), in the pathogenesis of cerebral amyloid angiopathy (CAA)-induced spontaneous he

158 ta) has recently been reported to exacerbate cerebral amyloid angiopathy (CAA)-related microhemorrhag

159 VD subtypes of hypertensive arteriopathy and cerebral amyloid angiopathy (CAA).

160 the form of parenchymal amyloid plaques and cerebral amyloid angiopathy (CAA).

161 disease (AD), are primarily associated with cerebral amyloid angiopathy (CAA).

162 n the cerebral cortex and in blood vessel as cerebral amyloid angiopathy (CAA).

163 s the development of Alzheimer's disease and cerebral amyloid angiopathy (CAA).

164 ving elevated levels of soluble Abeta but no cerebral amyloid angiopathy (CAA).

165 lated disorders, including familial forms of cerebral amyloid angiopathy (CAA).

166 ngophilic amyloid in blood vessels is called cerebral amyloid angiopathy (CAA).

167 ques and in the walls of cerebral vessels as cerebral amyloid angiopathy (CAA).

168 implicate similar inflammatory processes in cerebral amyloid angiopathy (CAA).

169 iseases such as Alzheimer's disease (AD) and cerebral amyloid angiopathy (CAA).

170 mited by poor clearance of a core AD lesion, cerebral amyloid angiopathy (CAA).

171 osis (CSS) are the characteristic markers of cerebral amyloid angiopathy (CAA).

172 flow or reactivity might be early markers of cerebral amyloid angiopathy (CAA).

173 ferential occipital distribution in sporadic cerebral amyloid angiopathy (CAA).

174 as plaques and in cerebral blood vessels as cerebral amyloid angiopathy (CAA).

175 orrhage (cSAH) is increasingly recognised in cerebral amyloid angiopathy (CAA).

176 of amyloid in the cerebrovasculature [i.e., cerebral amyloid angiopathy (CAA)] and a heightened risk

177 erized by Abeta in the cerebral vasculature [cerebral amyloid angiopathy (CAA)] in the majority of pa

178 roscopic lesions (n=9), microinfarcts (n=8), cerebral amyloid angiopathy (CAA, n=7), lacunes (n=6), w

179 ients with high vascular amyloid deposition (cerebral amyloid angiopathy [CAA]) but not in patients w

180 Cerebrovascular deposition of beta-amyloid (cerebral amyloid angiopathy [CAA]) is a major cause of h

181 ced cerebrovascular beta-amyloid deposition (cerebral amyloid angiopathy, CAA) is associated with cer

182 A range of potential outcome markers for cerebral amyloid angiopathy can be measured against the

183 Cerebral amyloid angiopathy can cause life-threatening b

184 eta peptide in the wall of cerebral vessels (cerebral amyloid angiopathy), can lead to weakness and r

185 targeted and untargeted sampling combined in cerebral amyloid angiopathy cases.

186 erebral microinfarct, cerebral macroinfarct, cerebral amyloid angiopathy, cerebral atherosclerosis, h

187 In cerebral amyloid angiopathy cohorts, cortical superficia

188 Cerebral amyloid angiopathy commonly co-occurs with Alzh

189 anges in markers potentially associated with cerebral amyloid angiopathy compared to ADAD.

190 h lobar cerebral microbleeds suggesting that cerebral amyloid angiopathy contributes to the relations

191 5%) of 320 patients in the high-risk CT-APOE cerebral amyloid angiopathy criteria group versus 14 (8%

192 cerebral haemorrhage in the low-risk CT-only cerebral amyloid angiopathy criteria group was 45 (12%)

193 al hemorrhage, apolipoprotein E subtypes and cerebral amyloid angiopathy; deep intracerebral hemorrha

194 t Abeta1-40 deposition is characteristic for cerebral amyloid angiopathy deposition and maturation in

195 b administration has on reducing the rate of cerebral amyloid angiopathy deposition and restoring cer

196 mpairment in Alzheimer's disease and related cerebral amyloid angiopathy disorders.

197 in Alzheimer's disease and related familial cerebral amyloid angiopathy disorders.

198 Additionally, several familial forms of cerebral amyloid angiopathy exist including the Dutch (E

199 ts with early onset symptomatic amyloid-beta cerebral amyloid angiopathy following childhood exposure

200 ramework and timeline for the progression of cerebral amyloid angiopathy from subclinical pathology t

201 sy diagnoses of AD, Lewy body disease (LBD), cerebral amyloid angiopathy, frontotemporal lobar degene

202 A PrP cerebral amyloid angiopathy has not been reported in dis

203 ucoencephalopathy (CADASIL)and some forms of cerebral amyloid angiopathy have a genetic basis.

204 individuals with CMBs related to symptomatic cerebral amyloid angiopathy have abnormal vascular react

205 Hereditary cerebral amyloid angiopathy (HCAA) is a rare familial fo

206 hological findings include severe widespread cerebral amyloid angiopathy, hippocampal plaques, and ne

207 ereditary, sporadic, and iatrogenic forms of cerebral amyloid angiopathy, histopathological analyses

208 Restriction of analysis to definite/probable cerebral amyloid angiopathy ICH uncovered a stronger eff

209 Decreased biosynthesis of NO contributes to cerebral amyloid angiopathy in Alzheimer's disease (AD)

210 Cerebral amyloid angiopathy in Alzheimer's disease is ch

211 f the brain and contributes significantly to cerebral amyloid angiopathy in Alzheimer's disease.

212 sited early in diffuse and focal plaques and cerebral amyloid angiopathy in humans and nonhuman prima

213 Autopsy revealed cerebral amyloid angiopathy in the complete absence of a

214 on in TgCRND8 mice and ADan (Danish Amyloid) cerebral amyloid angiopathy in the mouse model of FDD.

215 vessel disease (hypertensive arteriopathy or cerebral amyloid angiopathy) in a multicentre, cross-sec

216 arly onset, familial Alzheimer's disease and cerebral amyloid angiopathy, in which patients develop n

217 pecific neuropathologies-amyloid plaques and cerebral amyloid angiopathy-in immunohistochemically-sta

218 ied CERAD score and global AD pathology) and cerebral amyloid angiopathy independent of APOE after ad

219 In the present study, we examined whether cerebral amyloid angiopathy influences tau deposition an

220 Cerebral amyloid angiopathy interacted with neuritic pla

221 Cerebral amyloid angiopathy is a common form of small-ve

222 Cerebral amyloid angiopathy is a common neuropathologica

223 Sporadic cerebral amyloid angiopathy is a common, well-defined sm

224 orrhage; our results suggest that underlying cerebral amyloid angiopathy is a contributing factor to

225 Cerebral amyloid angiopathy is a form of cerebrovascular

226 actor for ICH among persons without AVM, and cerebral amyloid angiopathy is a vasculopathy frequently

227 As cerebral amyloid angiopathy is an almost invariable path

228 also have serious consequences; for example, cerebral amyloid angiopathy is an important cause of hae

229 Cerebral amyloid angiopathy is characterized by accumula

230 The Tg2576 transgenic mouse model of human cerebral amyloid angiopathy is characterized by age-depe

231 From a clinical standpoint, cerebral amyloid angiopathy is characterized by individu

232 Cerebral amyloid angiopathy is common among elderly pati

233 Cerebral amyloid angiopathy is commonly associated with

234 Prominent cerebral amyloid angiopathy is often observed in the bra

235 Determining the presence and burden of cerebral amyloid angiopathy is particularly important wh

236 in mice; however, its involvement in AD and cerebral amyloid angiopathy is poorly understood.

237 Imaging of cerebrovascular beta-amyloid (cerebral amyloid angiopathy) is complicated by the nearl

238 ta-protein (Abeta) deposition, also known as cerebral amyloid angiopathy, is a common pathological fe

239 e cerebral vasculature, a condition known as cerebral amyloid angiopathy, is increasingly recognized

240 nt of leptomeningeal, cortical and capillary cerebral amyloid angiopathy, large infarcts, lacunar inf

241 Seven pathologies-leptomeningeal cerebral amyloid angiopathy, large infarcts, lacunar inf

242 , atherosclerosis, arteriolar sclerosis, and cerebral amyloid angiopathy), Lewy bodies, transactive r

243 ic underlying microvascular abnormalities of cerebral amyloid angiopathy (lobar structures) and hyper

244 with microbleeds in locations suggestive of cerebral amyloid angiopathy (lobar with or without cereb

245 Although severe small vessel disease or cerebral amyloid angiopathy may contribute in some cases

246 Fourteen autopsy cases with cerebral amyloid angiopathy (mean age at death 73 years,

247 rther research is needed to evaluate whether cerebral amyloid angiopathy mediates the observed associ

248 t of treatment), without adversely affecting cerebral amyloid angiopathy, microhemorrhages, myelinati

249 ations provide further in vivo evidence that cerebral amyloid angiopathy might be caused by transmiss

250 defines 5 ICH subtypes: arteriolosclerosis, cerebral amyloid angiopathy, mixed small vessel disease

251 ded moderate/severe occipital leptomeningeal cerebral amyloid angiopathy, moderate/severe arteriolosc

252 l pathologies such as arteriolosclerosis and cerebral amyloid angiopathy modify progression or worsen

253 We hypothesized that in patients with cerebral amyloid angiopathy, multiple small spatially di

254 patients with severe small vessel disease or cerebral amyloid angiopathy, neither VEGF nor MAG:PLP1 c

255 ctures may damage the brain and give rise to cerebral amyloid angiopathy, neuronal dysfunction, and c

256 Cerebral amyloid angiopathy, non-neuritic and perivascul

257 share neurological features including severe cerebral amyloid angiopathy, non-neuritic plaques, and f

258 Neither arteriosclerosis, atherosclerosis, cerebral amyloid angiopathy, nor microinfarcts were asso

259 The term 'cerebral amyloid angiopathy' now encompasses not only a

260 noreactivity (IR) is abundant in plaques and cerebral amyloid angiopathy of AD and Down syndrome pati

261 act possessed either TDP-43 proteinopathy or cerebral amyloid angiopathy of sufficient severity to in

262 s to be the result of predominantly advanced cerebral amyloid angiopathy of the leptomeningeal vessel

263 N) familial forms of Alzheimer's disease and cerebral amyloid angiopathy on the structure of the 21-3

264 al vascular pathology (small vessel disease, cerebral amyloid angiopathy or VWF).

265 Moreover, depletion of fibrinogen lessened cerebral amyloid angiopathy pathology and reduced cognit

266 Cerebral amyloid angiopathy pathology was rated as none,

267 By contrast, atherosclerosis and cerebral amyloid angiopathy pathology were associated wi

268 amyloid phase, but not Braak tangle stage or cerebral amyloid angiopathy predicted (11)C-Pittsburgh c

269 in the PrP gene (PRNP), associated with PrP cerebral amyloid angiopathy (PrP-CAA) and Gerstmann-Stra

270 thies; we propose to name this phenotype PrP cerebral amyloid angiopathy (PrP-CAA).

271 cal filaments tau neurofibrillary tangle and cerebral amyloid angiopathy ratings in the whole sample

272 examined the relationship between markers of cerebral amyloid angiopathy-related brain injury, networ

273 DNA from 94 unrelated patients with sporadic cerebral amyloid angiopathy-related hemorrhage found no

274 However, their contribution to cerebral amyloid angiopathy-related hemorrhage remains u

275 Cerebral amyloid angiopathy-related inflammation (CAA-ri

276 Cerebral amyloid angiopathy-related inflammation (CAA-ri

277 d imaging lesions was 9/39 (23%) in probable cerebral amyloid angiopathy-related intracerebral haemor

278 morrhage, and are three times more common in cerebral amyloid angiopathy-related intracerebral haemor

279 cerebral amyloid angiopathy and exacerbated cerebral amyloid angiopathy-related microhemorrhage in A

280 ting of the vessel wall (an advanced form of cerebral amyloid angiopathy-related vascular damage) in

281 .5 [1.5-3.7]), and they had a higher risk of cerebral amyloid angiopathy (RR 6.6, 1.5-29.6).

282 p = 0.037; OR, 2.48; 95% CI, 1.06-5.82), and cerebral amyloid angiopathy severity (p = 0.032; OR, 4.1

283 omeningeal vessels (P < 0.0001), but reduced cerebral amyloid angiopathy severity in cortical vessels

284 d more cortical amyloid-beta plaque load and cerebral amyloid angiopathy than early disease onset and

285 o at risk of intracerebral hemorrhage due to cerebral amyloid angiopathy, the result of beta-amyloid

286 he structural brain network in patients with cerebral amyloid angiopathy to healthy control subjects

287 We used transgenic mice with prominent cerebral amyloid angiopathy to investigate the ability o

288 Many of the candidate outcomes for cerebral amyloid angiopathy trials are probably applicab

289 osited in blood vessels from subjects having cerebral amyloid angiopathy was 90 degrees out of phase

290 e brains of APP/PS1 and APP/PS1/AI mice, but cerebral amyloid angiopathy was reduced in APP/PS1/AI mi

291 ntly higher levels of hippocampal Abeta, and cerebral amyloid angiopathy was significantly more commo

292 To examine the effect of Apoa-I deletion on cerebral amyloid angiopathy, we measured insoluble Abeta

293 Lewy body disease and cerebral amyloid angiopathy were common in both early an

294 latter could be, at least partly, related to cerebral amyloid angiopathy, which belongs to the contin

295 Cerebral amyloid angiopathy, which is defined by cerebro

296 centrum semi-ovale have been associated with cerebral amyloid angiopathy, while those in the basal ga

297 statin C amyloids cause a hereditary form of cerebral amyloid angiopathy whilst cystatin B aggregates

298 l examination of the proband revealed severe cerebral amyloid angiopathy, widespread neurofibrillary

299 (Abeta) sequence develop severe early-onset cerebral amyloid angiopathy with some of the related var

300 Icelandic young adults that leads to severe cerebral amyloid angiopathy, with no effective therapy.