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1 ertebral and basilar arteries (the posterior cerebral circulation).
2 mportant role in the control of systemic and cerebral circulation.
3 remained unaffected in the celiac artery and cerebral circulation.
4 or proximal vessel occlusion in the anterior cerebral circulation.
5 Alzheimer disease (AD) suffer from impaired cerebral circulation.
6 n MS, ET-1 is released from the brain to the cerebral circulation.
7 shRNA lentiviral particles injected into the cerebral circulation.
8 endothelial function in the BH(4)-deficient cerebral circulation.
9 usly expressed in the vasculature, including cerebral circulation.
10 t soluble Abeta can significantly impair the cerebral circulation.
11 sanoid that produces vasoconstriction in the cerebral circulation.
12 embolism to the arterial tree, including the cerebral circulation.
13 channels in terminal arterioles from rabbit cerebral circulation.
14 n venous and arterial input functions in the cerebral circulation.
15 he carotid bed and 37 +/- 6% at 20/s for the cerebral circulation.
16 be related to vasospastic phenomena in human cerebral circulation.
17 m to elicit an NVC response in the posterior cerebral circulation.
18 ic nerve activity are unique features to the cerebral circulation.
19 ears; 75% of AAs were in the arch vessels or cerebral circulation.
20 ssion of destructive pulsatile energy to the cerebral circulation.
21 ecrease in platelet concentration across the cerebral circulation.
22 e for better understanding the mechanisms of cerebral circulation.
24 vascular smooth muscle cells (cVSMCs) of the cerebral circulation, although the identity of their mol
25 imum reduction was 37 +/- 6% at 20/s for the cerebral circulation and 11 +/- 3% at 2/s for bulk carot
26 vel dilatory signaling pathway for CO in the cerebral circulation and appears to be the first demonst
28 th aSAH with high sensitivity independent of cerebral circulation and bleeding severity while produci
29 ongenital vascular variants of the posterior cerebral circulation and cerebral hypoperfusion could pa
30 (CEP) devices may reduce embolization to the cerebral circulation and hence the incidence of stroke.
32 eposition and fibrin accumulation within the cerebral circulation and would be expected to contribute
33 ment a dissociation between the systemic and cerebral circulations and potentially explain earlier cl
34 and autoregulatory responses throughout the cerebral circulation, and interactions with parasympathe
36 e and longer-term effects of hemodialysis on cerebral circulation, cerebral structure, and cognitive
37 t the parasympathetic nerves innervating the cerebral circulation contain NOS and furthermore, since
38 reductions (5-7%) in anterior and posterior cerebral circulations, due in part to the reduced cross-
39 might influence platelet function across the cerebral circulation during exercise at high altitude re
41 versely, clamping of the arch vessels halted cerebral circulation, ensuring the permanent cessation o
42 ted by ET-1, which is likely released in the cerebral circulation from reactive astrocytes in plaques
43 nd stent placement in the carotid artery and cerebral circulation has given rise to concerns about th
46 azard with proven deleterious effects on the cerebral circulation, including a decrease in cerebral b
47 ial blood directly sampled from the ischemic cerebral circulation indicated local shedding of platele
48 ndow of thrombolysis and prolong the time of cerebral circulation intervention for neurosurgical proc
51 eculate that autonomic neural control of the cerebral circulation is tonically active and likely play
52 ts, and that, when measured in the posterior cerebral circulation, it is the higher rather than lower
53 rate hypoxia with lack of expected change in cerebral circulation, leading to hypotension, bradycardi
56 ral venous flow is crucial for understanding cerebral circulation mechanisms and clinical application
57 projection image (25/3.45) of the posterior cerebral circulation obtained with MR angiography of the
60 wn influences of autonomic activation on the cerebral circulation played a smaller but significant ro
61 obstruct brain blood vessels or deteriorate cerebral circulation, resulting in ischemic or hemorrhag
62 proximal arterial occlusion in the anterior cerebral circulation that was confirmed on vessel imagin
63 in II (Ang II) exerts detrimental effects on cerebral circulation, the mechanisms of which have not b
64 is to discuss arterial anomalies of anterior cerebral circulation, their prevalence and demonstrate r
65 ring mechanical thrombectomy in the anterior cerebral circulation, thrombus embolization resulting in
66 lium produces endothelial dysfunction in the cerebral circulation through a mechanism involving oxida
69 tter understand integrated approaches in the cerebral circulation, we also briefly address reactivity
70 Significantly higher normalized BFVs in the cerebral circulation were measured in children with both
71 synthase (eNOS) contributes to regulation of cerebral circulation, whereas that produced by neuronal
72 on of the parasympathetic innervation of the cerebral circulation with emphasis on the vertebral and
73 nt reduction in resistance that involved the cerebral circulation with little effect on bulk carotid