ライフサイエンスコーパス: cerebral cortex

1 alog the vast diversity of cell types in the cerebral cortex.

2 ume abnormalities in multiple regions of the cerebral cortex.

3 , ventral midbrain, periaqueductal gray, and cerebral cortex.

4 d with increased FAN1 mRNA expression in the cerebral cortex.

5 here gadolinium is retained in rat and human cerebral cortex.

6 formation ("birthdate") in postmortem human cerebral cortex.

7 ly on the activity of specialized neurons in cerebral cortex.

8 vation of pyramidal neurons in the mammalian cerebral cortex.

9 kes apparent the modular organization of the cerebral cortex.

10 ocused on the role of regions located within cerebral cortex.

11 r influence on information processing in the cerebral cortex.

12 t reshape the structural architecture of the cerebral cortex.

13 ple imposed by the physical embedding of the cerebral cortex.

14 uropathology of mainly the basal ganglia and cerebral cortex.

15 activity as well as its synchronization with cerebral cortex.

16 unidentified ligands in the membranes of the cerebral cortex.

17 nterneurons are frequently myelinated in the cerebral cortex.

18 choroid plexus epithelium as well as in the cerebral cortex.

19 their functional relations with areas in the cerebral cortex.

20 sic functional connectivity within the human cerebral cortex.

21 ht be present in membrane fractions of mouse cerebral cortex.

22 sity of cell types appropriate for the human cerebral cortex.

23 into functional ensembles in the developing cerebral cortex.

24 F receptor synthesis and neurogenesis in the cerebral cortex.

25 m higher cognitive centers occurs within the cerebral cortex.

26 eguard mechanisms for the development of the cerebral cortex.

27 which is repeated in parallel throughout the cerebral cortex.

28 ters orchestrate embryonic patterning of the cerebral cortex.

29 facilitate the higher order functions of the cerebral cortex.

30 1 expressed in neurons, here focusing on the cerebral cortex.

31 oward the less neuronally dense areas of the cerebral cortex.

32 with the notion of lateralized functions in cerebral cortex.

33 pses, about 1 per microm(3) in the mammalian cerebral cortex.

34 the hippocampus and 22.1 +/- 4.9 muM in the cerebral cortex.

35 represents the folding characteristic of the cerebral cortex.

36 mpaired neuronal migration in the developing cerebral cortex.

37 a nigra pars compacta (SNpc) and, later, the cerebral cortex.

38 listic microvascular networks from the mouse cerebral cortex.

39 expression of anti-oxidative enzymes in the cerebral cortex.

40 en excitatory and inhibitory synapses in the cerebral cortex.

41 s (the striatum and cerebellum), but not the cerebral cortex.

42 al swollen morphology in the hippocampus and cerebral cortex.

43 Memories of experiences are stored in the cerebral cortex.

44 mpaired, resulting in disorganization of the cerebral cortex.

45 cture that sits between the striatum and the cerebral cortex.

46 ogenitors and the resultant formation of the cerebral cortex.

47 iate into functional neurons in the neonatal cerebral cortex.

48 ions in the basal ganglia, nucleus gyrus and cerebral cortex.

49 brillary lesions and neuritic plaques in the cerebral cortex.

50 ndritic spine density in the hippocampus and cerebral cortex.

51 ny developmental features with the mammalian cerebral cortex.

52 f P450arom, but also 17beta-estradiol in the cerebral cortex.

53 metric spatial neurovascular coupling in the cerebral cortex.

54 n against ischemia/reperfusion injury of the cerebral cortex.

55 detailing circuit mapping strategies in the cerebral cortex.

56 ions are a prominent activity pattern in the cerebral cortex.

57 cortex is much more tightly folded than the cerebral cortex.

58 neuron identity demarcates each layer of the cerebral cortex.

59 ons that were synchronized across the BG and cerebral cortex.

60 granting a reservoir of young cells for the cerebral cortex.

61 rarchical spatial gradients throughout human cerebral cortex.

62 spheres of rats with ischemic lesions in the cerebral cortex.

63 hibitory circuit formation in the developing cerebral cortex.

64 ntial development of face and place areas in cerebral cortex.

65 (acute or chronic) and decompression of the cerebral cortex.

66 r the development of topographic maps in the cerebral cortex.

67 cent ADHD is determined by plasticity of the cerebral cortex.

68 ter is linked to functional variation of the cerebral cortex.

69 n quantitative 3D laminar atlas of the human cerebral cortex.

70 example of such internal monitoring reaching cerebral cortex.

71 processes traversing multiple layers of the cerebral cortex.

72 the size and configuration of the mammalian cerebral cortex.

73 that model features of the developing human cerebral cortex(1,2).

74 neurons and glia in the developing mammalian cerebral cortex(1-4).

75 pical surface of the ventricular zone of the cerebral cortex(5-8).

76 ing an ectopic neuronal layer that resembles cerebral cortex abnormalities in humans.

77 ared with neuronal cell densities within the cerebral cortex, across brain structures, across species

78 ion induced by Abeta applied directly to the cerebral cortex, administered intravascularly, or overpr

79 Dyslexia has been frequently linked to cerebral cortex alterations; however, recent evidence al

80 stributed in the cat brain, including in the cerebral cortex, an important target since mental retard

81 l the distinctive radial organization of the cerebral cortex and allow for the study of neuronal migr

82 st types of interneurons to migrate into the cerebral cortex and become incorporated into functional

83 sive connections with various regions of the cerebral cortex and by hypotheses surrounding its possib

84 ied with increases in apoptosis, whereas the cerebral cortex and cerebellum are unaffected.

85 The cerebral cortex and cerebellum both play important roles

86 vels of overlap with the hand regions of the cerebral cortex and cerebellum, and do not necessarily n

87 less pronounced than variability between the cerebral cortex and cerebellum.

88 is processed in the hand regions of both the cerebral cortex and cerebellum.

89 omplex tissues by applying it to mouse adult cerebral cortex and fetal forebrain.

90 ome of the earliest generated neurons of the cerebral cortex and has important developmental function

91 DNF, Akt, mTOR, p70S6K, ERK and CREB) in the cerebral cortex and hippocampal formation throughout of

92 of specific forebrain regions, including the cerebral cortex and hippocampus.

93 postmitotic excitatory neurons in the mouse cerebral cortex and hippocampus.

94 mplex I and complex II (OXPHOSCI+CII) in the cerebral cortex and hippocampus.

95 Nrg3 was abundantly expressed throughout the cerebral cortex and hippocampus.

96 significantly reversed by i-Extract in mouse cerebral cortex and hippocampus.

97 associated with epilepsy originating in the cerebral cortex and hippocampus.

98 ning was inspired by the architecture of the cerebral cortex and insights into autonomy and general i

99 additional insight into the structure of the cerebral cortex and its hierarchical organization.

100 th human Neuroligin-4 primarily expressed in cerebral cortex and localized to excitatory synapses.

101 s, which may receive synaptic input from the cerebral cortex and other brain regions beyond the core

102 eptors are highly abundant in the developing cerebral cortex and play major roles in early developmen

103 method to frozen postmortem samples of human cerebral cortex and retina and were able to identify tra

104 ntified EZH2 in the cerebellum, NR3C1 in the cerebral cortex and SRF in the basal forebrain.

105 s from tissue to the synaptic compartment of cerebral cortex and striatum strongly supports our appro

106 rimotor, cognitive and limbic regions of the cerebral cortex and subcortex.

107 aggerated beta frequency oscillations in the cerebral cortex and subthalamic nucleus (STN).

108 istribution of amyloid-beta removal from the cerebral cortex and tau pathology, cerebrovascular terri

109 The susceptible cell types in the cerebral cortex and the molecular mechanisms underlying

110 eatures (the directed network of the macaque cerebral cortex and the swim central pattern generator o

111 NIFICANCE STATEMENT Sensory signals from the cerebral cortex and the thalamus converge onto the stria

112 ing astrocytes from spinal cord, cerebellum, cerebral cortex, and hippocampus.

113 studying the anatomical organization of the cerebral cortex, and potentially its functional organiza

114 e associated with the expansion of the human cerebral cortex, and showed that these are related to th

115 cted information network architecture in the cerebral cortex, and suggest that features of the inform

116 actors to predict connections of the primate cerebral cortex: architectonic similarity (structural mo

117 all of episodic memories, nerve cells in the cerebral cortex are activated in precisely timed sequenc

118 Ipsilateral motor areas of cerebral cortex are active during arm movements and even

119 Visual responses in the cerebral cortex are believed to rely on the geniculate i

120 Excitatory neurons of the mammalian cerebral cortex are organized into six functional layers

121 E STATEMENT Projections from the ipsilateral cerebral cortex are the major source of input to the str

122 Which regions of the cerebral cortex are the origin of descending commands th

123 eceptors (FGFRs) in early development of the cerebral cortex are well established.

124 Among 33 regions, cerebral cortex areas, hypothalamus, and cerebellar gray

125 cographic (ECoG) activity across the lateral cerebral cortex as people heard and then mentally rehear

126 e information being processed throughout the cerebral cortex as well as maintain the modular structur

127 learning in the cerebellum, hippocampus, and cerebral cortex, as well as in artificial systems.

128 we perform single-cell RNA-seq on the mouse cerebral cortex at a progenitor driven phase (embryonic

129 ed with the mouse and rat brain flatmaps are cerebral cortex atlas level contours based on the refere

130 synapses (type As), which originate from the cerebral cortex, (b) GABAergic terminals forming single

131 on project to a wide variety of sites in the cerebral cortex, basal forebrain, bed nucleus of the str

132 the medial temporal lobe, the diencephalon, cerebral cortex, basal ganglia, and cerebellum.

133 eduction in neurons throughout the postnatal cerebral cortex, but with a more prominent impact on tho

134 e and neurogenic divisions of the developing cerebral cortex by combining conditional genetic deletio

135 ow that microelectrodes implanted in the rat cerebral cortex can detect neuronal activity with remark

136 this way the proximal tissues, including the cerebral cortex, can be spared.

137 bition, two opposing forces in the mammalian cerebral cortex, can dynamically affect the output of th

138 ease in excitation-inhibition (E-I) ratio in cerebral cortex, causing hyperexcitability and excess sp

139 we quantify the neuronal composition of the cerebral cortex, cerebellum and remaining brain structur

140 y shows that each division of the forebrain (cerebral cortex, cerebral nuclei, thalamus, hypothalamus

141 Neural responses in the cerebral cortex change dramatically between the 'synchro

142 on had greater than two microinfarcts in the cerebral cortex, compared with 10% of those without (chi

143 ives extensive connections from areas of the cerebral cortex concerned with vision, touch, movement,

144 Neurons in the cerebral cortex connect through descending pathways to h

145 tathione, glutamate, and/or glutamine in the cerebral cortex, consistent with a post-inflammatory res

146 The cerebral cortex contains "immature" neurons, which are g

147 Because the white matter of the cerebral cortex contains axons that connect distant neur

148 The human cerebral cortex contains many cell types that likely und

149 The cerebral cortex contains multiple areas with distinctive

150 The mouse cerebral cortex contains neurons that express choline ac

151 tivity in visual, auditory, or somatosensory cerebral cortex, depending on task requirements.

152 The development of the mammalian cerebral cortex depends on careful orchestration of prol

153 s with Huntington disease whose striatum and cerebral cortex develop inclusions associated with exten

154 line availability modulates neurogenesis and cerebral cortex development through the regulation of ne

155 rns that yield important insights into human cerebral cortex development.

156 Areas of the cerebral cortex differ from each other not only in their

157 In the mammalian cerebral cortex, different classes of GABAergic interneu

158 on of large-scale functional networks on the cerebral cortex differs between individuals and is parti

159 d attention-related fMRI activity across the cerebral cortex during experimental induction of neglect

160 ghly enriched in the ventricular zone of the cerebral cortex, EGFP is mostly expressed in developing

161 nt mouse model of RTT that the border of the cerebral cortex exhibits increased number of inflammator

162 t ARHGAP11B, a human-specific gene, augments cerebral cortex expansion by regulating metabolic pathwa

163 Cerebral cortex expansion is a hallmark of mammalian bra

164 In the cerebral cortex, female mice had two-fold more genes res

165 nvestigators have paid much attention to the cerebral cortex, few studies have detailed the basal gan

166 The mechanisms by which the human cerebral cortex folds into its final form remain poorly

167 i and to compute the distance to the nearest cerebral cortex for 9626 contacts.

168 des evidence of common mechanisms across the cerebral cortex for generating global percepts from sepa

169 The basal ganglia, thalamus, and cerebral cortex form an interconnected network implicate

170 We examined cerebral cortex from 46 species that encompassed most or

171 regarding the structural development of the cerebral cortex from childhood to adulthood, and provide

172 ogenitors (RGPs) in the developing mammalian cerebral cortex generates deep and superficial layer neu

173 n that predicts the degree of folding of the cerebral cortex, given the clade-specific scaling of cor

174 In the hypoxic cerebral cortex, glial transcriptome responses most clos

175 low and oxygenation disturbances that impair cerebral cortex growth and cause life-long cognitive and

176 optimized to image a vertical column of the cerebral cortex > 1 mm in depth in awake mice with low (

177 However, gadolinium retention in the cerebral cortex has not been systematically investigated

178 acroscopic anatomical characteristics of the cerebral cortex have been identified in individuals affe

179 in distinct areas of the CNS, primarily the cerebral cortex, hippocampus, amygdala, suprachiasmatic

180 ized by progressive neurodegeneration in the cerebral cortex, histopathologically hallmarked by amylo

181 nced spatiotemporal dynamics observed in the cerebral cortex; however, evidence of a causal relations

182 as Egr-1, Arc1, and BDNF specifically in the cerebral cortex, impacting behavioral functions only mar

183 an important role in the development of the cerebral cortex in humans and mice.

184 remapping in the deprived hand region of the cerebral cortex in one-handers.

185 Materials and Methods The cerebral cortex in Sprague-Dawley rats treated with gado

186 ys) optical access to 45 mm(2) of the dorsal cerebral cortex in the mouse.

187 istry demonstrated knockdown of GLT-1 in the cerebral cortex in the synGLT-1 KO mice.

188 ar resolution recordings from cerebellum and cerebral cortex in unrestrained mice, demonstrate its op

189 that result from abnormal development of the cerebral cortex in utero.

190 , i.e. the distance of each contact from the cerebral cortex, in order to discriminate between white

191 ndantly expressed in cells of the developing cerebral cortex, including neural progenitor cells and d

192 ce for this hypothesis mainly comes from the cerebral cortex, including the observation that cortical

193 mation transfer across multiple nodes in the cerebral cortex, including visual, posterior parietal, a

194 The oxidation in the cerebral cortex induced by pentylenetetrazole was signif

195 in a variety of brain regions, including the cerebral cortex (insular and infralimbic areas), bed nuc

196 Sensory areas of the cerebral cortex integrate the sensory inputs with the on

197 sly innervate motor and sensory areas of the cerebral cortex involved in whisker movement control.

198 mitotic neuronal migration in the developing cerebral cortex involves Nesprin-2, which recruits cytop

199 The laminar organization of the cerebral cortex is a fundamental characteristic of the b

200 of reproducible folding in the gyrencephalic cerebral cortex is a topic of great interest to neurosci

201 ating the cellular architecture of the human cerebral cortex is central to understanding our cognitiv

202 usion anisotropy within the developing fetal cerebral cortex is longitudinally characterized in the r

203 contrast, the number of inputs per neuron in cerebral cortex is more uniform and large.

204 Although the cerebral cortex is organized into six excitatory neurona

205 The cerebral cortex is organized into specialized sensory ar

206 ether new neurons are added in the postnatal cerebral cortex is still debated.

207 The cerebral cortex is the main striatal afferent, and progr

208 The cerebral cortex is the source of our most complex cognit

209 y behaviors.SIGNIFICANCE STATEMENT While the cerebral cortex is widely viewed as playing an essential

210 ocused on pathomechanisms of dyslexia at the cerebral cortex level, several lines of evidence suggest

211 noncoding exon 1 of GPR56 (e1m) leads to the cerebral cortex malformation and epilepsy.

212 d defective ion channels in individuals with cerebral cortex malformations, which reflect abnormaliti

213 that applying different TMS currents to the cerebral cortex may reveal cerebellar inputs to both the

214 Malformations of the cerebral cortex (MCCs) are devastating developmental dis

215 Excitatory neurons of the cerebral cortex migrate radially from their place of bir

216 This receptor has been purified from porcine cerebral cortex mitochondria by a new Atx-affinity-based

217 Neurons and networks in the cerebral cortex must operate reliably despite multiple s

218 years with acute ischaemic stroke involving cerebral cortex (National Institute of Health Stroke Sca

219 niscent of processes in the developing human cerebral cortex necessary for generating expanded neuron

220 axonal regeneration of mechanically injured cerebral cortex neurons from mice.

221 During the development of the cerebral cortex, neurons are generated directly from rad

222 -weighted signal intensities, we studied the cerebral cortex of a large cohort of patients in early s

223 ypical mammalian astrocyte architecture, the cerebral cortex of humans exhibits a radial distribution

224 roinflammatory factors was also found in the cerebral cortex of Malat1 KO mice after ischemic stroke

225 oking structural changes in the meninges and cerebral cortex of male and female mice.

226 glia regulate NPC function in the developing cerebral cortex of mammalian species.

227 The cerebral cortex of mammals exhibits intricate interareal

228 Some immature neurons in the cerebral cortex of mammals might wait for years before t

229 xpression and chromatin accessibility in the cerebral cortex of mice over a 3-d period, including a 6

230 n Ndp (KO) retinas with those in the hypoxic cerebral cortex of mice that were housed for 1 week in a

231 hed astrocytes and oligodendrocytes from the cerebral cortex of neonatal wild-type (WT), Heph KO and

232 , extracellular concentrations of DMT in the cerebral cortex of normal behaving rats, with or without

233 pathogenic role for GABAergic neuron in the cerebral cortex of patients with GPR56 mutations.

234 rtical extracts as well as synaptosomes from cerebral cortex of synGLT-1 KO compared with control lit

235 Building a cerebral cortex of the proper size involves balancing ra

236 teristics similar to a phenotype seen in the cerebral cortex of Vldlr(null) mice.

237 lest module of information processing of the cerebral cortex; one which Vernon Mountcastle called the

238 The Dp140 isoform was expressed in the cerebral cortex only in foetal life stages, while in the

239 Here, we derive organoids resembling the cerebral cortex or the hindbrain/spinal cord and assembl

240 regates (neurofibrillary tangles) across the cerebral cortex parallels symptom severity(2,3).

241 direct mapping between terms for describing cerebral cortex parcellation according to six schemes in

242 The activity of the cerebral cortex patterns into recurring dynamic motifs.

243 Gadolinium retention was detected in the cerebral cortex, pia mater, and pia-ensheathed leptomeni

244 F-brain barrier pathology is also evident at cerebral cortex pial and ventricular ependymal surfaces

245 INMT transcripts were identified in the cerebral cortex, pineal gland, and choroid plexus of bot

246 Interlaminar astrocytes (ILA) in the cerebral cortex possess a soma in layer I and extend an

247 physical, and cytological dimensions of the cerebral cortex, possibly reflecting variations and pers

248 The frontal area of the cerebral cortex provides long-range inputs to sensory ar

249 show that conditional loss of BubR1 in mouse cerebral cortex recapitulates microcephaly.

250 ateral geniculate nucleus, LGN) and V5/MT, a cerebral cortex region involved in visual movement proce

251 n on neuronal function, in particular in the cerebral cortex, remain little studied.

252 NCE STATEMENT Inhibitory interneurons in the cerebral cortex represent a heterogenous group of cells

253 citatory inputs of interneurons in the mouse cerebral cortex, respectively.

254 ucture of live microglia purified from human cerebral cortex samples obtained at autopsy and during n

255 In the developing cerebral cortex, sequential transcriptional programs tak

256 For the cerebral cortex, significant evidence has accumulated on

257 In the cerebral cortex, single-cell RNA sequencing (scRNA-seq)

258 Human cerebral cortex size and complexity has increased greatl

259 principles shared among the multiple maps in cerebral cortex, some properties appear idiosyncratic.

260 rolling the flow of information in the adult cerebral cortex, SST(+) cells play important roles in th

261 Within the cerebral cortex, subunits alpha1, alpha5, beta2, beta3,

262 Histological atlases of the cerebral cortex, such as those made famous by Brodmann a

263 pread extensive connectivity with the entire cerebral cortex, suggesting a prominent role in 'higher

264 o many experimental observations, neurons in cerebral cortex tend to operate in an asynchronous regim

265 KO mouse showed prominent vacuolation in the cerebral cortex, thalamus, and cerebellum and particular

266 specific regions, subregions, and layers of cerebral cortex that are critical for higher cognition,

267 Of eleven isoforms in SCN and cerebral cortex that exhibit exon variation across two p

268 MS) is a noninvasive method to stimulate the cerebral cortex that has applications in psychiatry, suc

269 s in monkeys and rats to identify regions of cerebral cortex that have multisynaptic connections with

270 th a distinct transcriptome signature in the cerebral cortex that is characterized by downregulation

271 of amygdala functional connectivity with the cerebral cortex that related to individual differences i

272 critical circuits for movement control: the cerebral cortex, the cerebellum, and the basal ganglia.

273 ives extensive excitatory afferents from the cerebral cortex, the influence of different cortical are

274 CB1) in the formation of sensory maps in the cerebral cortex, the topographic representation of the w

275 In the cerebral cortex, the tyrosine kinase receptor ErbB4 is c

276 However, within the cerebral cortex these tools are largely limited to broad

277 it carrying this CD extends from midbrain to cerebral cortex through a relay in the thalamus.

278 Visual information reaches the cerebral cortex through parallel ON and OFF pathways tha

279 Here, we use the male and female mouse cerebral cortex to show that a higher percentage of micr

280 arifies neuronal decoding strategies used by cerebral cortex to translate cortical spiking into perce

281 trocyte morphology and contacts in the mouse cerebral cortex, tracing of individual pyramidal neurons

282 The developing cerebral cortex uses a complex developmental plan involv

283 ed changes in functional architecture of the cerebral cortex using a dataset comprising a cross-secti

284 unction and impairs lactate transport in the cerebral cortex using rat models of HE (bile duct ligati

285 s prediction was recently confirmed in mouse cerebral cortex using serial block-face electron microsc

286 having greater than two microinfarcts in the cerebral cortex was 2.12 (95% CI, 1.12-4.02; p = 0.02).

287 cusing on the highly heterogeneous mammalian cerebral cortex, we apply PESCA to find enhancers and ge

288 developing callosal projection of the mouse cerebral cortex, we mapped molecular enrichments in tran

289 rsal thalamus project topographically to the cerebral cortex, whereas the much smaller epithalamus (2

290 Notably, gene sets pertaining to the cerebral cortex (which did not differ in volume between

291 us has extensive connections with the entire cerebral cortex, which can also serve to integrate infor

292 zation of these regions in comparison to the cerebral cortex, which is not conducive to neurogenesis

293 iest generated populations of neurons in the cerebral cortex, which play an important role in the mat

294 c and hypoxia-induced lactate release in the cerebral cortex, which was normalized by OP treatment.

295 represented in multiple regions in the human cerebral cortex, while amodal semantic information appea

296 cal Layers 5 and 6 are the only cells in the cerebral cortex with axons that leave the cortex to infl

297 cation hub of the forebrain and provides the cerebral cortex with inputs from sensory organs, subcort

298 The cerebral cortex, with all its computational power, can o

299 on was stronger for microinfarcts within the cerebral cortex, with those who experienced severe sepsi

300 SCs were topically applied to the surface of cerebral cortex within 1 hour of experimental TBI.