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1 ctions of PV are with S2 and PV of the other cerebral hemisphere.
2 mm min(-1), respectively; n = 5) across the cerebral hemisphere.
3 f the laminar architecture of the developing cerebral hemisphere.
4 zed by epilepsy and degeneration of a single cerebral hemisphere.
5 at extend over nearly the full length of the cerebral hemisphere.
6 induced by controlled cortical impact on one cerebral hemisphere.
7 gion near the temporal pole of the mammalian cerebral hemisphere.
8 stinguish it from the medial boundary of the cerebral hemisphere.
9 activity of the right (language nondominant) cerebral hemisphere.
10 atory fronto-temporal projection in the left cerebral hemisphere.
11 from the typical lateralization in the left cerebral hemisphere.
12 ied in clinical severity throughout the left cerebral hemisphere.
13 y movable microelectrodes spanning an entire cerebral hemisphere.
14 and 11 perfusion parameters were derived per cerebral hemisphere.
15 ning across the lateral surface of the right cerebral hemisphere.
16 zations can spread widely across the injured cerebral hemisphere.
17 died in a patient who was born with only one cerebral hemisphere.
18 r degeneration primarily affecting the right cerebral hemisphere.
19 r patients with lesions situated in the left cerebral hemisphere.
20 tions by co-opting the capacities of the two cerebral hemispheres.
21 hic affecting the spine, cranial nerves, and cerebral hemispheres.
22 e neuroendocrine system from any part of the cerebral hemispheres.
23 losum is the major commissure connecting the cerebral hemispheres.
24 ions between the ectoderm and the developing cerebral hemispheres.
25 in and incorporate into the neocortex of the cerebral hemispheres.
26 nsive growth and cleaves into right and left cerebral hemispheres.
27 onal transfer of information between the two cerebral hemispheres.
28 nvolved receive their motor output from both cerebral hemispheres.
29 in, and biological features of, normal human cerebral hemispheres.
30 n the mammalian brain, linking the bilateral cerebral hemispheres.
31 fiber tract that connects the left and right cerebral hemispheres.
32 tial failure of the brain to divide into the cerebral hemispheres.
33 along midline structures as compared to the cerebral hemispheres.
34 iented and densely-packed axons bridging the cerebral hemispheres.
35 ined as the incomplete separation of the two cerebral hemispheres.
36 ording to least and most clinically affected cerebral hemispheres.
37 and astrogliosis in dorsal striatum in both cerebral hemispheres.
38 predominantly in the posterior region of the cerebral hemispheres.
39 ural and functional connectivity between the cerebral hemispheres.
40 rved postmortem chimpanzee (Pan troglodytes) cerebral hemispheres.
41 rized by overgrowth of either one of the two cerebral hemispheres.
42 of the leptomeninges over large areas of the cerebral hemispheres.
43 he morphophysiological independence of their cerebral hemispheres.
44 olves incomplete or failed separation of the cerebral hemispheres.
45 acterized by an incomplete separation of the cerebral hemispheres.
46 n of axonal projections that connect the two cerebral hemispheres.
47 cerebral arteries serving the left and right cerebral hemispheres.
48 non-motor cortical regions within and across cerebral hemispheres.
49 needed for the expansion and bifurcation of cerebral hemispheres.
50 had tumors that were localized to either the cerebral hemispheres (133 of 349 [38.1%]) or midline str
51 oss after seizures and (b) with a more focal cerebral hemisphere abnormality usually due to vascular
55 most of the external morphology of the right cerebral hemisphere and a fragment of fossilized face th
56 bral nuclei form the ventral division of the cerebral hemisphere and are thought to play an important
58 cessing was strongly lateralized to the left cerebral hemisphere and involved a network of regions in
59 ade to demonstrate the medial surface of the cerebral hemisphere and the medial wall of the lateral v
60 omotopic and other locations in the opposite cerebral hemisphere and to a surrounding array of eight
61 g on the neurons and neuroglial cells in the cerebral hemispheres and associated fiber tracts of the
63 fferent and afferent connections between the cerebral hemispheres and between the cortex and thalamus
64 ificant age-related differences in volume of cerebral hemispheres and caudate nuclei (controls, but n
65 ray of cerebral cortical layering, fusion of cerebral hemispheres and cerebellar folia, and aberrant
69 s; in mice lacking Foxg1, development of the cerebral hemispheres and olfactory epithelium (OE) is se
70 ereby limits neuronal trafficking across the cerebral hemispheres and reduces input to cortical inhib
71 is a reduction in white matter volume in the cerebral hemispheres and structural abnormalities in mye
72 rectomy (i.e. surgical removal of one entire cerebral hemisphere) and 10 age- and sex-matched control
73 l cells (sensory ganglia and outer aspect of cerebral hemispheres) and glial cells (radial glia, sate
74 izures, progressive degeneration of a single cerebral hemisphere, and autoimmunity directed against g
75 ch-motor regions of the non-dominant (right) cerebral hemisphere, and extend this theory to include t
76 cerebrovascular accidents affected the right cerebral hemisphere, and the most commonly affected vess
77 mors on the right (n = 41) and left (n = 48) cerebral hemispheres, and evaluated on a hold-out cohort
78 t of marginal zone components, fusion of the cerebral hemispheres, and scalloping of the dentate gyru
79 fibre tract in the brain, connecting the two cerebral hemispheres, and thereby facilitating the integ
81 s originating from the contralesional intact cerebral hemisphere are capable of sprouting into the de
85 the rest of the brain (and, conversely, the cerebral hemispheres are smaller with respect to the cer
88 amining the connectional organization of the cerebral hemispheres as a whole (right and left cerebral
89 ction in the white matter connecting the two cerebral hemispheres, as well as in the correlated activ
90 n the perisylvian area of the left and right cerebral hemispheres, as well as in the frontal and occi
91 developed marked infarcts in the ipsilateral cerebral hemisphere at 24 hr and showed significant loss
93 ight ratios of ipsilateral and contralateral cerebral hemispheres at 12 hrs, 24 hrs, or 2, 3, or 7 da
96 re consistent with the known predominance of cerebral hemisphere axonal injury in cardiac arrest and
97 usion, fibrin accumulates in the ipsilateral cerebral hemisphere, based upon immunoblotting, and loca
98 generated an average representation of each cerebral hemisphere between 14 and 31 weeks' gestation w
99 hyperintensities in the white matter of the cerebral hemispheres, brainstem, cerebellum, and cervica
100 ntral nervous system (CNS) that were high in cerebral hemispheres but progressively decreased toward
101 ive of edema in the posterior regions of the cerebral hemispheres, but the changes often involved oth
103 sed by about 50% the weight loss of the left cerebral hemisphere caused by hypoxia-ischemia in neonat
105 Brain structures investigated included the cerebral hemispheres, cerebral cortex, diencephalon, cau
107 microhemorrhage was observed in the treated cerebral hemisphere compared with the contralateral side
108 alized and specialized capacities of the two cerebral hemispheres constitutes a hallmark feature of h
109 Nearly every tissue section from bilateral cerebral hemispheres contained either macroscopic or mic
113 of 50 fetuses, and the medial surface of the cerebral hemisphere could be traced posteriorly around t
115 the corpus callosum, cortical axons from one cerebral hemisphere cross the midline to reach their tar
116 that resulted in holoprosencephaly (a single cerebral hemisphere), cyclopia (a single midline eye), a
120 coarse coding hypothesis postulates that the cerebral hemispheres differ in their breadth of semantic
122 omatic mutations that affect each developing cerebral hemisphere differently with more neurons than e
123 onous activation of cortical loci in the two cerebral hemispheres during development leads to the sta
124 .0 vs 22 seconds +/- 7.8 [P = .004]) in both cerebral hemispheres during low-flow cardiopulmonary byp
125 ient and symmetrically in the cortex of both cerebral hemispheres except for precentral gyri in the s
127 human forebrain, and in its severe form, the cerebral hemispheres fail to completely separate into tw
128 of the human forebrain and midface where the cerebral hemispheres fail to separate into distinct left
129 itudinal changes in network activity in each cerebral hemisphere, focusing specifically on the "presy
130 ls of N434A, but not H435A, decreased in the cerebral hemispheres following bilateral injection into
131 ifference was found only in the volumetry of cerebral hemispheres for both the groups: mean volume of
132 h with defects in neurulation, fusion of the cerebral hemispheres, formation of the great forebrain c
134 A recent study of a child born with one cerebral hemisphere has revealed an extreme developmenta
136 s Sonobiopsy was performed by sonicating the cerebral hemisphere in 2-month-old PS19 and wild-type mi
137 demonstrated along the medial surface of the cerebral hemisphere in 36 (72%) of 50 fetuses, and the m
143 Understanding the role of the unaffected cerebral hemisphere in the motor recovery process has be
146 In addition, defects in development of the cerebral hemispheres in Foxg1(-/-) mice are not rescued
148 us callosum (CC) connects the left and right cerebral hemispheres in mammals and its development requ
149 ispheres for both the groups: mean volume of cerebral hemispheres in patients with severe AS was 884.
150 size and anatomical connectivity across the cerebral hemispheres in persons with Tourette's disorder
151 in coordinating neural activity between the cerebral hemispheres in placental mammals, but the rules
152 genetic determination of the left and right cerebral hemispheres in the nonright-handed twin pairs i
153 nferior frontal and temporal regions of both cerebral hemispheres in the titled and untitled conditio
160 l videos of neuronal calcium flux across the cerebral hemispheres is a key step before mapping featur
161 either the unmyelinated axon of the immature cerebral hemispheres is relatively resistant to traumati
162 ted with progressive destruction of a single cerebral hemisphere, is an autoimmune disease in which o
165 ends in part on multiple regions of the left cerebral hemisphere, located outside the classic languag
166 IFICANCE STATEMENT Focal damage to the right cerebral hemisphere may result in a variety of deficits,
167 inner ear, nerve, brainstem, cerebellum and cerebral hemispheres may all affect vestibular functioni
168 of manually annotated samples from a single cerebral hemisphere, measured from 2D histological secti
169 within the distributed motor system in both cerebral hemispheres, more so in patients with greater i
170 Tumor locations were diencephalon (n = 58), cerebral hemisphere (n = 3), and cerebellum (n = 17).
171 ), and bilateral (n = 12, 63%), occurring in cerebral hemispheres (n = 12, 59%), basal ganglia (n = 1
175 r, were transplanted into the spinal cord or cerebral hemisphere of immunosuppressed normal and myeli
177 surface area and volume of V1 in one or both cerebral hemispheres of 15 neurologically normal human b
180 metry of venous drainage in the pathological cerebral hemisphere on CT and MRI dynamic angiography.
181 ntation between connecting regions, age, and cerebral hemisphere on the strength of effective connect
182 te a differential role of the left and right cerebral hemispheres on immune functions in humans.
183 gliomas, we injected hMSCs directly into the cerebral hemisphere opposite an established human glioma
184 ted changes were greater in magnitude in the cerebral hemisphere opposite the more clinically affecte
185 iencephalon, but also in the olfactory bulbs/cerebral hemispheres, optic tectum/tegmentum, retina, an
186 aced finger tapping studies indicate the two cerebral hemispheres preferentially control different rh
188 occipital-temporal scalp areas of the right cerebral hemisphere regardless of the visual field of st
189 rtals and Cro-Magnon 1, who have the largest cerebral hemispheres relative to cerebellum volume of an
190 e hypothesis that asymmetric routing between cerebral hemispheres represents an important mechanism f
192 ephalon failed to bulge or separate into two cerebral hemispheres, resulting in holoprosencephaly.
193 rsal view with little superimposition by the cerebral hemispheres, short temporal lobe, and long sylv
194 typically bilaterally distributed across the cerebral hemispheres, show similarity to known white mat
195 ured cerebral volume on four contiguous 5 mm cerebral hemisphere slices at each time point, using an
196 o cortical sulci over the convexities of the cerebral hemispheres, sparing the brainstem, cerebellum
197 elencephalon, the embryonic precursor of the cerebral hemispheres, specialized cell types form a midl
200 ex of visual orienting was recorded over the cerebral hemisphere that was ipsilateral to the attentio
201 ducted a cytoarchitectonic analysis by using cerebral hemispheres that were cut at oblique angles and
202 The microprobes were inserted through the cerebral hemisphere, the superior colliculus and the mid
203 The larger units of analysis, including the cerebral hemispheres, the major grey and white matter st
204 aschitic hemisphere-that is, the ipsilateral cerebral hemisphere (THGr(Ce)) and the contralateral cer
205 notypic features such as failure of anterior cerebral hemisphere to divide, hydrocephalus and cleft p
206 s of the entire neural axis ranging from the cerebral hemispheres to the peripheral autonomic nerves
207 ntly better for patients with cerebellar and cerebral hemisphere tumors (n = 75) than those with tumo
208 ction (GTR) was attempted for cerebellar and cerebral hemisphere tumors, with biopsy or less aggressi
210 rder characterized by an enlarged, malformed cerebral hemisphere, typically causing epilepsy that req
211 he cerebellum and contralateral sensorimotor cerebral hemisphere underscore the strong physiological
212 is controlled primarily by the contralateral cerebral hemisphere; unilateral brain lesions typically
213 ose of this study was to examine how the two cerebral hemispheres use perceptual information about wo
215 ns that connect homotopic regions of the two cerebral hemispheres via the corpus callosum and that ar
216 allosal projection neurons (CPN) connect the cerebral hemispheres via the corpus callosum, integratin
218 eter measured with the medial surface of the cerebral hemisphere was 10.7 mm, compared with the true
220 male cats anaesthetized with chloralose, one cerebral hemisphere was exposed and the middle cerebral
221 rotid ligation, perfusion of the ipsilateral cerebral hemisphere was markedly diminished, suggesting
222 r each case, the entire hippocampus from one cerebral hemisphere was sliced into 5mm slabs (5-7 slabs
223 During mammalian evolution, expansion of the cerebral hemispheres was accompanied by expansion of the
225 metries of input from the two eyes into each cerebral hemisphere, we measured ocular dominance column
227 nes and early members of the genus Homo, the cerebral hemispheres were large in proportion to the cer
230 nce between blood flow in the right and left cerebral hemispheres when autoregulation was impaired in
231 f the BF-1 gene results in hypoplasia of the cerebral hemispheres, which is more severe in structures
232 e mean QA of the nigrostriatal tract of each cerebral hemisphere with the striatal (18)F-FEOBV distri
233 wavefronts selectively over the ipsilateral cerebral hemispheres with a velocity of 3.8 +/- 0.70 mm
234 G34R-mutant gliomas are lethal tumors of the cerebral hemispheres with unknown mechanisms of regional
235 zation are present in the left vs. the right cerebral hemisphere, with the left hemisphere showing a
236 n brain is composed of two broadly symmetric cerebral hemispheres, with an abundance of reciprocal an
237 ain origin of this speech rhythm in the left cerebral hemisphere, within or near pre-Rolandic motor a
238 was hypothesized that disconnection between cerebral hemispheres would disrupt mental simulation of
239 between the left and right visual hemifields/cerebral hemispheres, yet somehow WM feels seamless.