ライフサイエンスコーパス: cerebral spinal fluid

1 e coronavirus 2 from nasopharyngeal swab and cerebral spinal fluid.

2 mission tomography and glucose monitoring in cerebral spinal fluid.

3 aqueduct to the canalostomy due to influx of cerebral spinal fluid.

4 9 areas of normal appearing white matter and cerebral spinal fluid.

5 al canal where they block circulation of the cerebral spinal fluid.

6 infected hamsters using 2-microl aliquots of cerebral spinal fluid.

7 mixture of peptides dissolved in artificial cerebral spinal fluid.

8 ular space where they are transported to the cerebral spinal fluid.

9 leotide delivered by a single injection into cerebral spinal fluid.

10 Surprisingly, cerebral spinal fluid Abeta levels transiently rise, and

11 jority of interneurons (21/25) in artificial cerebral spinal fluid (aCSF) exhibited spontaneous tonic

12 Artificial cerebral spinal fluid (aCSF) or 100 microM bumetanide in

13 ted context, BLA was infused with artificial cerebral spinal fluid (ACSF) or ACSF containing an N-met

14 plasma, synthetic urine (SU), and artificial cerebral spinal fluid (aCSF) using ethyl acetate as the

15 duced by stimulation with high-K+ artificial cerebral spinal fluid (aCSF) were observed.

16 Exposure to low bicarbonate artificial cerebral spinal fluid (aCSF), pH 6.70 for 30 min at 32 d

17 is followed by microdialysis with artificial cerebral spinal fluid allowed isolation of 14C-glutamine

18 nclusion were absence of correlation between cerebral spinal fluid amyloid-beta1-42 and Boston naming

19 nal fluid t-tau, brain amyloid-beta load via cerebral spinal fluid amyloid-beta1-42 and vascular dise

20 rebral spinal fluid t-tau) and amyloid load (cerebral spinal fluid amyloid-beta1-42) all independentl

21 Cerebral spinal fluid analysis revealed elevated levels

22 Immunoglobulin in cerebral spinal fluid and antibody secreting cells (ASC)

23 Notably, ALA and PBG concentrations in the cerebral spinal fluid and CNS regions were markedly elev

24 concentrations of allopregnanolone in human cerebral spinal fluid and in rat brain sections.

25 Reports of rotavirus RNA in the cerebral spinal fluid and serum of children infected wit

26 ulin forms in human neocortex, white matter, cerebral spinal fluid, and serum by immunostaining and m

27 Serum or cerebral spinal fluid antigen titers did not correlate w

28 Cerebral spinal fluid antigen titers were higher with me

29 S induces nuclear ASPP2 in vivo at the blood-cerebral spinal fluid barrier (the brain's barrier to in

30 ionships between pulse pressure and distinct cerebral spinal fluid biomarker profiles.

31 isolated primary viruses from autopsy brain, cerebral spinal fluid, blood, spleen, and lymph node sam

32 observed to correlate with viral RNA in the cerebral spinal fluid but not with plasma viral load, co

33 ntrathecal (IT) delivery of AAV vectors into cerebral spinal fluid can avoid many issues, although di

34 Analysis of imatinib mesylate cerebral spinal fluid concentrations revealed levels 155

35 Solutions of artificial cerebral spinal fluid containing 50 muM DA, 50 muM NE, a

36 ic modulator (NAM) that achieves exposure in cerebral spinal fluid (CSF) 2.5x above the in vitro IC(5

37 l Alzheimer's disease (AD), as classified by cerebral spinal fluid (CSF) AB(42)/Tau ratio.

38 Severe cerebral spinal fluid (CSF) abnormalities were present i

39 er analysis of neurofilament light chain and cerebral spinal fluid (CSF) alpha-synuclein oligomer loa

40 ciates investigated the status of HIV in the cerebral spinal fluid (CSF) and revealed ongoing presenc

41 rotein, with a similar half-life in both the cerebral spinal fluid (CSF) and the CNS.

42 one of two delivery devices showed improved cerebral spinal fluid (CSF) biomarker profiles and slowe

43 A cerebral spinal fluid (CSF) cavity within the conus medu

44 ng were reviewed to identify the presence of cerebral spinal fluid (CSF) clefts around the tumors.

45 Cerebral spinal fluid (CSF) examination showed marked ab

46 concentrations (measured by immunoassay) in cerebral spinal fluid (CSF) from tuberculous meningitis

47 Proteomic analysis of cerebral spinal fluid (CSF) from young males with active

48 Our objective was to measure the levels cerebral spinal fluid (CSF) GNMPB in cALD patients to de

49 gtin for the first time in the regulation of cerebral spinal fluid (CSF) homeostasis.

50 Cerebral spinal fluid (CSF) is a promising biospecimen f

51 n on the pathophysiologic changes within the cerebral spinal fluid (CSF) of these patients.

52 Lumbar puncture revealed high cerebral spinal fluid (CSF) opening pressure (32 cm H(2)

53 Cerebral spinal fluid (CSF) opening pressure was 155.0 +

54 eatments for HD only alleviate symptoms, but cerebral spinal fluid (CSF) or central nervous system (C

55 Hydrocephalus is a common disorder of cerebral spinal fluid (CSF) physiology resulting in abno

56 During postherpetic neuralgia (PHN), the cerebral spinal fluid (CSF) possesses the capability to

57 We tested 46 plasma samples and 36 cerebral spinal fluid (CSF) samples taken from patients

58 Plasma and cerebral spinal fluid (CSF) samples were collected perio

59 omatic cryptococcal antigenemia but negative cerebral spinal fluid (CSF) studies.

60 Biotin is likely transported into cerebral spinal fluid (CSF) via one or more specific tra

61 v compartmentalization between the blood and cerebral spinal fluid (CSF) was apparent with all neurol

62 MRAs with arteries, veins, brain parenchyma, cerebral spinal fluid (CSF), and embolized vessels.

63 of the three constituent tissue types (i.e., cerebral spinal fluid (CSF), gray matter, white matter)

64 the presence of antiviral antibodies in the cerebral spinal fluid (CSF), indicating local maintenanc

65 orectal and lymph node tissues, bone marrow, cerebral spinal fluid (CSF), plasma, and very large numb

66 nvolved in the production and circulation of cerebral spinal fluid (CSF), suggesting that loss of mFu

67 ectivity between brain parenchymal fluid and cerebral spinal fluid (CSF), we reasoned enhancing drug

68 lament light (NfL) was reduced in plasma and cerebral spinal fluid (CSF).

69 ventricles and spinal cord to circulate the cerebral spinal fluid (CSF).

70 increase the concentrations of Abeta1-42 in cerebral spinal fluid (CSF).

71 age fluid (n = 152), pleural fluid (n = 76), cerebral spinal fluid (CSF; n = 152), pericardial fluid

72 ment include the use of brain and spine MRI, cerebral spinal fluid cytology, neurological examination

73 a macrophage-tropic virus isolated from the cerebral spinal fluid, did not use CCR5 or CXCR4.

74 rt of folates across the choroid plexus into cerebral spinal fluid due to inactivating mutations in t

75 0 expands functionality to urine, blood, and cerebral spinal fluid, enhancing the realism of blood sp

76 lar tonsillar crowding and restore posterior cerebral spinal fluid flow, but regional tissue biomecha

77 ells to invade the spinal canal and obstruct cerebral spinal fluid flow.

78 dividuals; and protein QTLs were measured in cerebral spinal fluid from 544 individuals and plasma fr

79 tify a proteolytic fragment of cystatin C in cerebral spinal fluids from patients diagnosed with mult

80 s characterized in vitro using an artificial cerebral spinal fluid gelatin.

81 -67 exhibited robust in vivo activity in the cerebral spinal fluid glycine biomarker model in both ro

82 The mean cerebral spinal fluid HIV load was 5949 copies/mL.

83 Notice of retraction: the article "Role of Cerebral Spinal Fluid in Space Flight Induced Ocular Cha

84 4 (ST4) was the predominant sequence type of cerebral spinal fluid isolates from cases of meningitis.

85 Age, temporal or hemorrhagic lesions, and cerebral spinal fluid lymphocytosis were significantly a

86 While cerebral spinal fluid neuregulin levels were unaltered i

87 u since elevated tau levels are found in the cerebral spinal fluid of AD patients.

88 rferon (IFN-gamma), which is elevated in the cerebral spinal fluid of HAD patients.

89 ell tolerated in humans and elevates cGMP in cerebral spinal fluid of healthy volunteers, confirming

90 The presence of EBV DNA in the cerebral spinal fluid of HIV patients with focal brain l

91 d to those detected in seminal plasma and/or cerebral spinal fluid of six individuals.

92 Hypothalamic tanycytes contact the cerebral spinal fluid of the third ventricle and send pr

93 95% CI, 1.13-1.56) and ACE protein levels in cerebral spinal fluid (OR per 1-SD decrease, 1.12; 95% C

94 monstrated CNS penetration preclinically and cerebral spinal fluid penetration clinically.

95 ent and lumbar puncture for determination of cerebral spinal fluid phosphorylated tau (P-tau) and bet

96 different specimens: bronchoalveolar lavage, cerebral spinal fluid, plasma, and whole blood.

97 ith neuroinflammatory symptoms, neutrophilic cerebral spinal fluid pleocytosis, and complement consum

98 ient neuroinflammation included neutrophilic cerebral spinal fluid pleocytosis, constitutive compleme

99 inflammatory secretome of AD iNs and patient cerebral spinal fluid revealed a neuronal senescence-ass

100 ed to normalize unknown peak mobilities in a cerebral spinal fluid sample.

101 R analysis was used to genotype parasites in cerebral spinal fluid samples from 8 of 10 human immunod

102 dentify druggable targets in cPIIRS, patient cerebral spinal fluid samples underwent transcriptional

103 was >/=25 parasites/ml in amniotic fluid and cerebral spinal fluid samples.

104 dult respiratory, pediatric stool, and adult cerebral spinal fluid specimens, and detected 64% of cli

105 d not correlate with mortality at 90 days or cerebral spinal fluid sterilization at 2 weeks.

106 ith multiple specimen types including blood, cerebral spinal fluid, stool, and respiratory.

107 olyadenylation sequences, ASO injection into cerebral spinal fluid successfully corrected Stmn2 pre-m

108 y (structural magnetic resonance imaging and cerebral spinal fluid t-tau) and amyloid load (cerebral

109 gnetic resonance images, neuronal injury via cerebral spinal fluid t-tau, brain amyloid-beta load via

110 ow known to be widely distributed in plasma, cerebral spinal fluid, tears, and saliva.

111 plate brain as well as GM, white matter, and cerebral spinal fluid tissue probability maps to facilit

112 n increase in CXCL1 and CXCL10 levels in the cerebral spinal fluid, TNF-alpha expression in the epend

113 oit the interconnectivity of parenchymal and cerebral spinal fluid to enhance the amount of temozolom

114 and perfusing them with hyperoxic artificial cerebral spinal fluid to minimize peripheral chemorecept

115 At the same time that cerebral spinal fluid viral load increased in macaques t

116 Therefore, cerebral spinal fluid volume was decreased (Delta-40ml,

117 For sampling, artificial cerebral spinal fluid was infused in through one channel

118 e it exists in steady-state equilibrium with cerebral spinal fluid where it has the potential to serv

119 regulin activity is also detectable in human cerebral spinal fluid where its expression appears to be