ライフサイエンスコーパス: cerebrospinal

1 aims to characterize the evidence regarding cerebrospinal fluid (1,3)-beta-d-glucan measurement to d

2 Cerebrospinal fluid (CSF) (1,3)-beta-d-glucan measuremen

3 lyzed associations between PiB retention and cerebrospinal fluid (CSF) Abeta concentrations in 17 M(+

4 Participants were enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis cond

5 Participants were enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis that

6 Individuals enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis were

7 ricles, is considered a disease of excessive cerebrospinal fluid (CSF) accumulation and thereby treat

8 xchange between interstitial fluid (ISF) and cerebrospinal fluid (CSF) along the paravascular spaces

9 is a source of secreted signaling factors in cerebrospinal fluid (CSF) and a key barrier between bloo

10 use brain that manifests as pro-inflammatory cerebrospinal fluid (CSF) and accumulation of ChP macrop

11 quencing (scRNA-Seq) was performed on paired cerebrospinal fluid (CSF) and blood from subjects with r

12 as to evaluate the diagnostic performance of cerebrospinal fluid (CSF) and blood KLK8 for AD and mild

13 We examined immune activation markers in cerebrospinal fluid (CSF) and blood specimens collected

14 s for visualizing CAA and assessing Abeta in cerebrospinal fluid (CSF) and brain.

15 use brain interstitial fluid (ISF) and human cerebrospinal fluid (CSF) and chronic sleep deprivation

16 1 (CHI3L1), markers of glial activation, in cerebrospinal fluid (CSF) and plasma and determined the

17 Novel cerebrospinal fluid (CSF) and plasma biomarkers are amon

18 Cerebrospinal fluid (CSF) and plasma from 35 patients wi

19 n individual HIV-1 polymerase genomes in the cerebrospinal fluid (CSF) and plasma in individuals with

20 HIV-1 evolution in the cerebrospinal fluid (CSF) and plasma may result in disco

21 tigated a broad set of protein biomarkers in cerebrospinal fluid (CSF) and plasma using a highly sens

22 Whilst cerebrospinal fluid (CSF) and positron emission tomograp

23 We studied the cerebrospinal fluid (CSF) and serum biochemical inflamma

24 Sera, cerebrospinal fluid (CSF) and tissue samples from 100 fa

25 or identifying Mycobacterium tuberculosis in cerebrospinal fluid (CSF) are inadequate.

26 d for distribution and antiviral activity in cerebrospinal fluid (CSF) as well as neurocognitive (NC)

27 d for distribution and antiviral activity in cerebrospinal fluid (CSF) as well as neurocognitive (NC)

28 alised brain structure which forms the blood-cerebrospinal fluid (CSF) barrier.

29 Associations with cerebrospinal fluid (CSF) biomarkers (amyloid beta1-42,

30 ortality, and B cell phenotypes in blood and cerebrospinal fluid (CSF) by flow cytometry in HIV-infec

31 alitis, diagnosed by detection of VZV DNA in cerebrospinal fluid (CSF) by PCR and concomitant symptom

32 contribute to memory consolidation, whereas cerebrospinal fluid (CSF) clears metabolic waste product

33 However, we recently found that cerebrospinal fluid (CSF) concentration of the "social"

34 dy were to determine longitudinal changes in cerebrospinal fluid (CSF) concentrations of MMPs after a

35 The cerebrospinal fluid (CSF) contains an extracellular thre

36 Circulation of the cerebrospinal fluid (CSF) contributes to body axis forma

37 Extravasated erythrocytes in cerebrospinal fluid (CSF) critically contribute to the p

38 gate whether neurodegenerative biomarkers in cerebrospinal fluid (CSF) differentiate patients with su

39 les approach in order to investigate how the cerebrospinal fluid (CSF) enters the brain through a per

40 The incidence of abnormal cerebrospinal fluid (CSF) flow dynamics in children with

41 Lymphocyte activity in cerebrospinal fluid (CSF) from 4 cohorts of HTT gene exp

42 e intent of this study was to investigate if cerebrospinal fluid (CSF) from autoimmune encephalitis (

43 ed rat hippocampal neurons were treated with cerebrospinal fluid (CSF) from patients with anti-NMDAR

44 with higher cryptococcal antigen levels, the cerebrospinal fluid (CSF) fungal burden by quantitative

45 agenomic next-generation sequencing (NGS) of cerebrospinal fluid (CSF) has the potential to identify

46 Cerebrospinal fluid (CSF) hcrt-1 measurement has been we

47 uitary morphologic structure, and aqueductal cerebrospinal fluid (CSF) hydrodynamics relative to spac

48 pituitary deformation, augmented aqueductal cerebrospinal fluid (CSF) hydrodynamics, and expansion o

49 horylation in the choroid plexus and reduces cerebrospinal fluid (CSF) hypersecretion in a model of p

50 bound concentrations of bictegravir (BIC) in cerebrospinal fluid (CSF) in 15 asymptomatic, virologica

51 p-tau) and amyloid-beta (Abeta) 42 levels in cerebrospinal fluid (CSF) in the Amsterdam Dementia Coho

52 t microbiologically confirmed bloodstream or cerebrospinal fluid (CSF) infection between 24 h after r

53 -infection influenced clinical presentation, cerebrospinal fluid (CSF) inflammation, and outcome from

54 (GS) hypothesis states that advective driven cerebrospinal fluid (CSF) influx from the perivascular s

55 erivascular spaces that promotes movement of cerebrospinal fluid (CSF) into the brain and clearance o

56 Tau protein in cerebrospinal fluid (CSF) is a central and relevant biom

57 Magnesium isotopic analysis of cerebrospinal fluid (CSF) is a potentially interesting a

58 r the absence of SARS-CoV-2 particles in the cerebrospinal fluid (CSF) is a topic of debate.

59 Cerebrospinal fluid (CSF) is a vital liquid, providing n

60 As testing for HHV-6 in cerebrospinal fluid (CSF) is more readily available usin

61 Cerebrospinal fluid (CSF) is produced in the cerebral ve

62 that is released from erythrocytes into the cerebrospinal fluid (CSF) is suggested to cause vasocons

63 led perivascular spaces (PVS), through which cerebrospinal fluid (CSF) is transported from the subara

64 ctivity (EFA) of Cryptococcus clearance from cerebrospinal fluid (CSF) is used as a surrogate endpoin

65 neither the relationship between plasma and cerebrospinal fluid (CSF) KP metabolites nor their assoc

66 vel demographic and clinical information and cerebrospinal fluid (CSF) laboratory results.

67 Cerebrospinal fluid (CSF) lactate levels can differentia

68 Cerebrospinal fluid (CSF) leakage is a risk factor for d

69 s within the BACE2 locus are associated with cerebrospinal fluid (CSF) levels of Abeta1-42.

70 Serum and cerebrospinal fluid (CSF) levels of alpha-fetoprotein an

71 volume, cortical surface area and thickness, cerebrospinal fluid (CSF) levels of amyloid-beta (Abeta(

72 ing the A/T/N classification, relying on the cerebrospinal fluid (CSF) levels of beta-amyloid peptide

73 Here we simultaneously measured cerebrospinal fluid (CSF) levels of established amyloid

74 Here, we investigated the potential of cerebrospinal fluid (CSF) lipocalin 2 (LCN2), a secreted

75 ogression from normal aging to early AD, and cerebrospinal fluid (CSF) miR-195 levels of MCI subjects

76 We aimed to investigate the value of cerebrospinal fluid (CSF) neuronal pentraxins (NPTXs), a

77 lobin-haptoglobin complexes were measured in cerebrospinal fluid (CSF) of 44 patients with aSAH and r

78 d examined the immune cell profiles from the cerebrospinal fluid (CSF) of Neuro-COVID patients compar

79 Cerebrospinal fluid (CSF) of patients with anti-NMDAR en

80 e blood as well as on ventricular and lumbar cerebrospinal fluid (CSF) of pediatric patients treated

81 lf-lives and permeability into the brain and cerebrospinal fluid (CSF) of recombinant human (rh) proS

82 Cerebrospinal fluid (CSF) oligoclonal bands were rare in

83 medications, coexisting medical conditions, cerebrospinal fluid (CSF) opening pressure, treatments,

84 ociated with increased NFL concentrations in cerebrospinal fluid (CSF) or plasma in MCI Abeta+ and AD

85 fied two miR-298 SNPs associated with higher cerebrospinal fluid (CSF) p-tau and lower CSF Abeta42 le

86 Cerebrospinal fluid (CSF) p-tau181 (tau phosphorylated a

87 ut not significantly different compared with cerebrospinal fluid (CSF) P-tau217, CSF P-tau181, and ta

88 >=100 cells/muL by clinical characteristics, cerebrospinal fluid (CSF) parameters, and 18-week surviv

89 Cerebrospinal fluid (CSF) physiology is important for th

90 Testing was restricted to patients with cerebrospinal fluid (CSF) pleocytosis.

91 of multiple sclerosis (MS), but whether the cerebrospinal fluid (CSF) profile can help to identify p

92 Cerebrospinal fluid (CSF) protects the central nervous s

93 t genome-wide association study (GWAS) of 59 cerebrospinal fluid (CSF) proteins with a connection to

94 oimaging Initiative (ADNI) participants with cerebrospinal fluid (CSF) Ptau collected at baseline, di

95 h diagnostic value of a second generation of cerebrospinal fluid (CSF) Real-Time Quaking-Induced Conv

96 We summarize recent work illuminating how cerebrospinal fluid (CSF) regulates brain function.

97 gadoteric acid (Gd-DOTA) administration into cerebrospinal fluid (CSF) requires pre-contrast data for

98 r change in profile and concentration in the cerebrospinal fluid (CSF) resulted from fluctuations in

99 te the diagnostic accuracy of NAA tests with cerebrospinal fluid (CSF) samples against that of cultur

100 ament light (NFL) chain in serum, plasma and cerebrospinal fluid (CSF) samples collected over 12 mont

101 Routine EV/PeV RT-PCR testing of cerebrospinal fluid (CSF) samples in children impacted c

102 erial diseases in The Gambia where blood and cerebrospinal fluid (CSF) samples of hospitalized partic

103 tive viral load assays of plasma, semen, and cerebrospinal fluid (CSF) samples to detect HIV-1 RNA.

104 e, and neurologic examination, with elective cerebrospinal fluid (CSF) sampling, brain diffusion tens

105 re used: (1) regression of white matter (WM)/cerebrospinal fluid (CSF) signals only (BASE); (2) WM/CS

106 outcome was the change from baseline in the cerebrospinal fluid (CSF) SOD1 concentration at day 85.

107 Among suspected cases, 16 885 (92.5%) had a cerebrospinal fluid (CSF) specimen collected, 13 625 (80

108 developing Alzheimer disease (AD) and lower cerebrospinal fluid (CSF) tau biomarker levels.

109 disease (AD) has been suggested in GWASs of cerebrospinal fluid (CSF) tau levels and longitudinal ch

110 Elevated concentrations of cerebrospinal fluid (CSF) tau, a marker of axonal injury

111 We measured the relationship between cerebrospinal fluid (CSF) TB bacillary load with mortali

112 For diagnosis of neurosyphilis, cerebrospinal fluid (CSF) TP-PA has similar performance

113 Molecular testing of cerebrospinal fluid (CSF) using the BioFire FilmArray me

114 Pharmacokinetic data from plasma and cerebrospinal fluid (CSF) were analyzed with nonlinear m

115 l etiology, yet EV RNA is rarely detected in cerebrospinal fluid (CSF)(2).

116 t cohorts to identify protein alterations in cerebrospinal fluid (CSF), a proximal site to pathology.

117 beta-amyloid(42) and tau were quantified in cerebrospinal fluid (CSF), and white matter hyperintensi

118 ed directly from clinical specimens, such as cerebrospinal fluid (CSF), blood, or urine.

119 In blood plasma and cerebrospinal fluid (CSF), most proteins, including cand

120 No studies have examined C4 levels in cerebrospinal fluid (CSF), presumably a better biofluid

121 In cerebrospinal fluid (CSF), we measured 3 macrophage and

122 e and is characterized by enlargement of the cerebrospinal fluid (CSF)-filled brain ventricles result

123 re hydrocephalus (iNPH), an expansion of the cerebrospinal fluid (CSF)-filled brain ventricles.

124 Cancer cells within the cerebrospinal fluid (CSF)-filled leptomeninges face subs

125 n body fluids such as human blood plasma and cerebrospinal fluid (CSF).

126 has not been prospectively studied in human cerebrospinal fluid (CSF).

127 ter, and cortical regions and nonventricular cerebrospinal fluid (CSF).

128 ry end point was HTT(Rx) pharmacokinetics in cerebrospinal fluid (CSF).

129 s spp. were quantified directly from patient cerebrospinal fluid (CSF).

130 istering drugs intrathecally directly to the cerebrospinal fluid (CSF).

131 ce, as revealed by its increased presence in cerebrospinal fluid (CSF).

132 analysis by mass spectrometry in plasma and cerebrospinal fluid (CSF).

133 rylated at threonine 181 (p-tau), and NFL in cerebrospinal fluid (CSF); and one or more of PET with (

134 rkers of neuroinflammation and injury in the cerebrospinal fluid (tumor necrosis factor-alpha, kynure

135 Cerebrospinal fluid Abeta40 was reduced at 6 months and

136 Cerebrospinal fluid analyses and neuroimaging can identi

137 Cerebrospinal fluid analyses during the acute stage were

138 Cerebrospinal fluid analyses were negative for SARS-CoV-

139 ersons who underwent lumbar puncture and had cerebrospinal fluid analysis (January 1, 2008-December 3

140 Cerebrospinal fluid analysis reveals lineages not detect

141 Same-day cerebrospinal fluid analysis was unremarkable, and unenh

142 Same-day cerebrospinal fluid analysis was unremarkable, and unenh

143 y magnetic resonance imaging (MRI) and/or by cerebrospinal fluid analysis.

144 tracranial air (ICA)], water-filled balloon (cerebrospinal fluid and blood) and agarose gel (brain).

145 uencing (mNGS) of RNA and DNA extracted from cerebrospinal fluid and brain tissue now offers another

146 , soluble TREM2 (sTREM2), is abundant in the cerebrospinal fluid and its levels positively correlate

147 We detected inflammation in the cerebrospinal fluid and leptomeninges before the parench

148 able to monitor the changes in DA levels in cerebrospinal fluid and plasma of a mouse model of PD an

149 effector C3 were present at higher levels in cerebrospinal fluid and plasma(8,9) in men than in women

150 cleavage product of TREM2, is elevated in AD cerebrospinal fluid and positively correlates with cogni

151 We applied cerebrospinal fluid and serum from PCD patients as well

152 Additional imaging, cerebrospinal fluid and serum testing, information from

153 pathogens, immune cells, and cytokines into cerebrospinal fluid and the brain.

154 rate (82% vs. 24%) and allowed detection in cerebrospinal fluid and urine.

155 the recognition of oligoclonal bands in the cerebrospinal fluid as a possible marker of disseminatio

156 She was ultimately found to have sterile cerebrospinal fluid ascites which was treated successful

157 r's disease are accessible only via invasive cerebrospinal fluid assays, and reactive oxygen species

158 In vivo tauopathy has been measured using cerebrospinal fluid assays, but these do not provide inf

159 heir phase II metabolites permeate the blood-cerebrospinal fluid barrier (B-CSF-B) of sheep and to pr

160 The blood-cerebrospinal fluid barrier (BCSFB) is a highly dynamic

161 luding the blood brain barrier and the blood cerebrospinal fluid barrier, protect the CNS from extern

162 Additional analyses included continuous cerebrospinal fluid biomarker levels to examine the effe

163 to investigate the cognitive impairment and cerebrospinal fluid biomarkers in a follow-up study of p

164 I clinical trial and engages TREM2 based on cerebrospinal fluid biomarkers.

165 can result in very low folate levels in the cerebrospinal fluid causing childhood neurodegenerative

166 everal important clinical, neuroimaging, and cerebrospinal fluid characteristics.

167 ated tau concentrations in serial samples of cerebrospinal fluid collected from participants who were

168 Sleep loss increases the cerebrospinal fluid concentration of amyloid-beta and ta

169 Cerebrospinal fluid concentrations of GS and GFAP were m

170 sed of amyloid beta (Abeta) peptides and the cerebrospinal fluid concentrations of those peptides are

171 safe and achieves pharmacologically relevant cerebrospinal fluid concentrations.

172 areas, without interfering with drug plasma/cerebrospinal fluid concentrations.

173 Cerebrospinal fluid contacting neurons (CSF-cNs) represe

174 nifesting in abnormal cerebral perfusion and cerebrospinal fluid convection, present observation pres

175 ics of the alpha-synuclein aggregates in the cerebrospinal fluid could be used to readily distinguish

176 ositive participants, 3 of whom had negative cerebrospinal fluid CrAg tests from lumbar punctures (LP

177 imary endpoint was mean rate of clearance of cerebrospinal fluid cryptococcal infection (EFA).

178 Serum and cerebrospinal fluid cytokine levels were generally simil

179 gh titers of IgG autoantibodies in serum and cerebrospinal fluid directed against the nuclei of Purki

180 Here, we tested whether cerebrospinal fluid entry into the glymphatic system, a

181 in vivo biomarkers of AD, whether by serum, cerebrospinal fluid examination or PET, have transformed

182 atic system, a brain-wide interstitial fluid-cerebrospinal fluid exchange described in rodents, exist

183 This study analyzed the cerebrospinal fluid features of 31 COVID-19 patients wit

184 athological, clinical, molecular imaging and cerebrospinal fluid features of the most common neurodeg

185 s to irregularities in motile cilia-mediated cerebrospinal fluid flow [7-9].

186 Age, positive cerebrospinal fluid for oligoclonal bands, infratentoria

187 suffered from errors due to heterogeneity of cerebrospinal fluid fraction and partial volume.

188 CA assay can discriminate between samples of cerebrospinal fluid from patients diagnosed with Parkins

189 Nearer to the site of pathology, cerebrospinal fluid from patients with NSAb-mediated dis

190 sis secreted higher levels of glutamate, and cerebrospinal fluid glutamine levels were increased.

191 brain ventricular system reported to govern cerebrospinal fluid homeostasis [13, 14], neurogenesis [

192 an adaptive immune response in the blood and cerebrospinal fluid in Alzheimer's disease and provide e

193 Proteins from this module were elevated in cerebrospinal fluid in early stages of the disease.

194 es in ratios comparable to those secreted to cerebrospinal fluid in human, however the protocol to ac

195 chemosensitive glial cells that contact the cerebrospinal fluid in the third ventricle and send proc

196 NMDAR antibody seropositive have coexisting cerebrospinal fluid inflammatory changes or other paracl

197 mmune, the presence of GAD antibodies in the cerebrospinal fluid is sufficient to confirm a pathogeni

198 ear or sinus infections (43 of 120, 36%) and cerebrospinal fluid leakage (37 of 116, 32%).

199 ious side effects, including hemorrhages and cerebrospinal fluid leakage.

200 in patients with ear or sinus infections and cerebrospinal fluid leakage.

201 Possible side effects include worsening cerebrospinal fluid leaks.

202 tate at multiple sites of the tau protein in cerebrospinal fluid markers across four decades of disea

203 l cut point, 1.55 SUVR), and comparison with cerebrospinal fluid measures of amyloid-beta (optimal cu

204 ussian mixture modeling, and comparison with cerebrospinal fluid measures of amyloid-beta, specifical

205 Significant changes of cerebrospinal fluid microRNAs that control inflammation,

206 ses the technical advantages and pitfalls of cerebrospinal fluid mNGS in the context of patients with

207 c during head and neck movements and promote cerebrospinal fluid motion; however, their role in neuro

208 ay a critical role in mucociliary clearance, cerebrospinal fluid movement, and fertility.

209 impairment, and increased protein content in cerebrospinal fluid occurred in anti-Drebrin-seropositiv

210 ens from the brains of people with AD and in cerebrospinal fluid of individuals diagnosed with AD.

211 from both postmortem brains and detection in cerebrospinal fluid of infected individuals.

212 specificity of clonally expanded TCRs in the cerebrospinal fluid of patients with Alzheimer's disease

213 lonally expanded CD8(+) T(EMRA) cells in the cerebrospinal fluid of patients with Alzheimer's disease

214 IL-11(+)CD4(+) cells accumulate in the cerebrospinal fluid of patients with early relapsing-rem

215 Moreover, AICAr is present in the cerebrospinal fluid of patients with LND, but not in con

216 n the concentration range that occurs in the cerebrospinal fluid of patients with SAH.

217 he endocannabinoid anandamide is elevated in cerebrospinal fluid of patients with schizophrenia.

218 esioned Macaca mulatta, and in the serum and cerebrospinal fluid of PD patients.

219 We show these profiles mirror in cerebrospinal fluid of people with DS.

220 e screening tool than the currently approved cerebrospinal fluid or amyloid beta positron emission to

221 The diagnosis was supported by cerebrospinal fluid or amyloid positron emission tomogra

222 to amyloid-beta or tau pathology measured in cerebrospinal fluid or by positron emission tomography(2

223 radation, with a half-life of ~5 days in rat cerebrospinal fluid or serum-containing medium.

224 ance imaging or computed tomography imaging, cerebrospinal fluid parameters and clinical endpoints.

225 inical improvement along with a reduction of cerebrospinal fluid parameters was observed after high-d

226 More abnormal levels of baseline cerebrospinal fluid phosphorylated tau and subthreshold

227 n the processes of notochord development and cerebrospinal fluid physiology, and how defects in those

228 Cerebrospinal fluid pleocytosis and protein elevation we

229 A cerebrospinal fluid polymerase chain reaction (PCR) for

230 jury biomarker soluble PDGFRbeta(7,8) in the cerebrospinal fluid predicted future cognitive decline i

231 By altering embryonic cerebrospinal fluid pressure during development, we foun

232 ura mater-surround the CNS, encompassing the cerebrospinal fluid produced by the choroid plexus epith

233 Interstitial and cerebrospinal fluid produced in the CNS is continuously

234 We identified and replicated 10 cerebrospinal fluid proteins including IL-12B, CD5, MIP-

235 Comparisons of brain tissue and cerebrospinal fluid proteomes reveal biomarker candidate

236 tched by changes in surrounding amniotic and cerebrospinal fluid proteomes.

237 Multiplexed DIA analysis of cerebrospinal fluid revealed the dynamic proteome change

238 n this study of >2,000 brains and nearly 400 cerebrospinal fluid samples by quantitative proteomics,

239 kynurenine (Kyn) and tryptophan (Trp) in 221 cerebrospinal fluid samples from patients with bacterial

240 detect aggregated Abeta in brain tissue and cerebrospinal fluid samples of AD mice.

241 metagenomic sequencing testing requested on cerebrospinal fluid samples submitted to an outside refe

242 nsion in infants with CHD were identified in cerebrospinal fluid spaces (p < 0.025).

243 Cerebrospinal fluid specimens from 29 cases were tested

244 Cerebrospinal fluid specimens were analyzed by culture a

245 oton imaging in rodents to show instead that cerebrospinal fluid surrounding the brain enters the tis

246 Cerebrospinal fluid testing showed normal cell counts, a

247 novel protein changes in Parkinson's disease cerebrospinal fluid that may be exploited for understand

248 In contact with the cerebrospinal fluid through their cell bodies, they send

249 source of miR-204 that is released into the cerebrospinal fluid to control number of NSCs within the

250 use of (1,3)-beta-d-glucan measurement with cerebrospinal fluid to diagnose fungal meningitis.

251 lammation, and increase AD biomarkers in the cerebrospinal fluid to similar levels observed in patien

252 faster in female youths than in male youths; cerebrospinal fluid volume expanded faster in most cereb

253 mian white matter expansion, and accelerated cerebrospinal fluid volumes expansion of anterior lobule

254 Cerebrospinal fluid was collected from children <5 years

255 The fungal clearance rate from cerebrospinal fluid was similar between groups (0.43 -lo

256 s of aggregates that were amplified from the cerebrospinal fluid were similar to those of aggregates

257 Levels of SOD1 in cerebrospinal fluid were transiently and only slightly l

258 ect alpha-synuclein aggregates in samples of cerebrospinal fluid with high sensitivity and specificit

259 tem abnormalities (increased posterior fossa cerebrospinal fluid).

260 ion of T cells, B cells, and blinatumomab in cerebrospinal fluid, (iv) blinatumomab-induced T-cell ro

261 e chimeric antigen receptor T cells into the cerebrospinal fluid, alone or in combination with azacyt

262 whether nilotinib is safe, and detectable in cerebrospinal fluid, and alters biomarkers and clinical

263 e Mini-Mental Status Exam, protein levels in cerebrospinal fluid, and genotype at the APOE and MAPT l

264 d-glucan and each of several distinct fungi, cerebrospinal fluid, and meningitis.

265 While cerebrospinal fluid, and more recently blood, markers of

266 tests were performed on lung, liver, blood, cerebrospinal fluid, and stool samples.

267 Hydrodynamic radii measured in urine, cerebrospinal fluid, and undiluted serum demonstrated th

268 No virus was detected in serum or cerebrospinal fluid, but postmortem analysis confirmed d

269 ry of alpha-glucan material in healthy human cerebrospinal fluid, establishing a novel methodological

270 h nonscintigraphic biomarkers from blood and cerebrospinal fluid, have provided an opportunity to inv

271 ll polymeric micelles exhibited stability in cerebrospinal fluid, highlighting the potential for loca

272 cords were excluded, as they did not examine cerebrospinal fluid, included animals, or focused on non

273 Cs) for testing postmortem specimens (blood, cerebrospinal fluid, lung tissue, respiratory tract swab

274 We observed neither SARS-CoV-2 RNA in the cerebrospinal fluid, nor intrathecal IgG synthesis, but

275 as are located adjacent to and bathed by the cerebrospinal fluid, presenting an opportunity for locor

276 ase of levels of neurofilaments in patients' cerebrospinal fluid, suggest that IgLON5 IgG, unlike oth

277 ical microbiology laboratory testing data of cerebrospinal fluid, the expected August/September/Octob

278 tetrameric carrier of thyroxine in blood and cerebrospinal fluid, the pathogenic aggregation of which

279 ut mice have low levels of circulating OT in cerebrospinal fluid, which can be corrected by the oral

280 n of phosphorylated tau and amyloid in their cerebrospinal fluid, yielding neurotypical and preclinic

281 study, we assessed the levels of ventricular cerebrospinal fluid-cfmtDNA (vCSF-cfmtDNA) in a diverse

282 We observed expression of PSST1 in cerebrospinal fluid-contacting (CSF-c) neurons of the hy

283 tion of human ChP organoids, which produce a cerebrospinal fluid-like secretion and recapitulate barr

284 and within sulci, suggesting an inflammatory cerebrospinal fluid-mediated lesion pathogenesis.

285 or sensing and delivery, in blood plasma and cerebrospinal fluid.

286 ology and tumor DNA extracted from blood and cerebrospinal fluid.

287 A-matrix metalloproteinase-9 pathway(19) in cerebrospinal fluid.

288 Only one patient had SARS-CoV-2 RNA in the cerebrospinal fluid.

289 identify Parkinson's-relevant biomarkers in cerebrospinal fluid.

290 delivery from arterial blood to ventricular cerebrospinal fluid.

291 ndividual is reflected in all tissues and in cerebrospinal fluid.

292 ient detection of TSEs prion protein in goat cerebrospinal fluid.

293 bs, which appear as oligoclonal bands in the cerebrospinal fluid.

294 egative except for increased posterior fossa cerebrospinal fluid.

295 IIB dogs had elevated HS levels in brain and cerebrospinal fluid.

296 The first comprises an interconnected set of cerebrospinal-fluid-contacting DA nuclei surrounding the

297 eding activities in patient biosamples, e.g. cerebrospinal fluids (CSF), and study their correlation

298 ecovered from the bronchoalveolar lavage and cerebrospinal fluids of critically ill patients.

299 amount of target molecules in the blood and cerebrospinal fluids samples.

300 ithin the cingulum bundle of hippocampus and cerebrospinal tracts were the most robust deficits in in