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1 tem abnormalities (increased posterior fossa cerebrospinal fluid).
2 or sensing and delivery, in blood plasma and cerebrospinal fluid.
3 ology and tumor DNA extracted from blood and cerebrospinal fluid.
4  A-matrix metalloproteinase-9 pathway(19) in cerebrospinal fluid.
5   Only one patient had SARS-CoV-2 RNA in the cerebrospinal fluid.
6  identify Parkinson's-relevant biomarkers in cerebrospinal fluid.
7  delivery from arterial blood to ventricular cerebrospinal fluid.
8 ndividual is reflected in all tissues and in cerebrospinal fluid.
9 ient detection of TSEs prion protein in goat cerebrospinal fluid.
10 bs, which appear as oligoclonal bands in the cerebrospinal fluid.
11 egative except for increased posterior fossa cerebrospinal fluid.
12 IIB dogs had elevated HS levels in brain and cerebrospinal fluid.
13  aims to characterize the evidence regarding cerebrospinal fluid (1,3)-beta-d-glucan measurement to d
14                                              Cerebrospinal fluid Abeta40 was reduced at 6 months and
15 e chimeric antigen receptor T cells into the cerebrospinal fluid, alone or in combination with azacyt
16                                              Cerebrospinal fluid analyses and neuroimaging can identi
17                                              Cerebrospinal fluid analyses during the acute stage were
18                                              Cerebrospinal fluid analyses were negative for SARS-CoV-
19 ersons who underwent lumbar puncture and had cerebrospinal fluid analysis (January 1, 2008-December 3
20                                              Cerebrospinal fluid analysis reveals lineages not detect
21                                     Same-day cerebrospinal fluid analysis was unremarkable, and unenh
22                                     Same-day cerebrospinal fluid analysis was unremarkable, and unenh
23 y magnetic resonance imaging (MRI) and/or by cerebrospinal fluid analysis.
24 tracranial air (ICA)], water-filled balloon (cerebrospinal fluid and blood) and agarose gel (brain).
25 uencing (mNGS) of RNA and DNA extracted from cerebrospinal fluid and brain tissue now offers another
26 , soluble TREM2 (sTREM2), is abundant in the cerebrospinal fluid and its levels positively correlate
27              We detected inflammation in the cerebrospinal fluid and leptomeninges before the parench
28  able to monitor the changes in DA levels in cerebrospinal fluid and plasma of a mouse model of PD an
29 effector C3 were present at higher levels in cerebrospinal fluid and plasma(8,9) in men than in women
30 cleavage product of TREM2, is elevated in AD cerebrospinal fluid and positively correlates with cogni
31                                   We applied cerebrospinal fluid and serum from PCD patients as well
32                          Additional imaging, cerebrospinal fluid and serum testing, information from
33  pathogens, immune cells, and cytokines into cerebrospinal fluid and the brain.
34  rate (82% vs. 24%) and allowed detection in cerebrospinal fluid and urine.
35 whether nilotinib is safe, and detectable in cerebrospinal fluid, and alters biomarkers and clinical
36 e Mini-Mental Status Exam, protein levels in cerebrospinal fluid, and genotype at the APOE and MAPT l
37 d-glucan and each of several distinct fungi, cerebrospinal fluid, and meningitis.
38                                        While cerebrospinal fluid, and more recently blood, markers of
39  tests were performed on lung, liver, blood, cerebrospinal fluid, and stool samples.
40        Hydrodynamic radii measured in urine, cerebrospinal fluid, and undiluted serum demonstrated th
41  the recognition of oligoclonal bands in the cerebrospinal fluid as a possible marker of disseminatio
42     She was ultimately found to have sterile cerebrospinal fluid ascites which was treated successful
43 r's disease are accessible only via invasive cerebrospinal fluid assays, and reactive oxygen species
44    In vivo tauopathy has been measured using cerebrospinal fluid assays, but these do not provide inf
45 heir phase II metabolites permeate the blood-cerebrospinal fluid barrier (B-CSF-B) of sheep and to pr
46                                    The blood-cerebrospinal fluid barrier (BCSFB) is a highly dynamic
47 luding the blood brain barrier and the blood cerebrospinal fluid barrier, protect the CNS from extern
48      Additional analyses included continuous cerebrospinal fluid biomarker levels to examine the effe
49  to investigate the cognitive impairment and cerebrospinal fluid biomarkers in a follow-up study of p
50  I clinical trial and engages TREM2 based on cerebrospinal fluid biomarkers.
51            No virus was detected in serum or cerebrospinal fluid, but postmortem analysis confirmed d
52  can result in very low folate levels in the cerebrospinal fluid causing childhood neurodegenerative
53 study, we assessed the levels of ventricular cerebrospinal fluid-cfmtDNA (vCSF-cfmtDNA) in a diverse
54 everal important clinical, neuroimaging, and cerebrospinal fluid characteristics.
55 ated tau concentrations in serial samples of cerebrospinal fluid collected from participants who were
56                     Sleep loss increases the cerebrospinal fluid concentration of amyloid-beta and ta
57                                              Cerebrospinal fluid concentrations of GS and GFAP were m
58 sed of amyloid beta (Abeta) peptides and the cerebrospinal fluid concentrations of those peptides are
59 safe and achieves pharmacologically relevant cerebrospinal fluid concentrations.
60  areas, without interfering with drug plasma/cerebrospinal fluid concentrations.
61                                              Cerebrospinal fluid contacting neurons (CSF-cNs) represe
62           We observed expression of PSST1 in cerebrospinal fluid-contacting (CSF-c) neurons of the hy
63 The first comprises an interconnected set of cerebrospinal-fluid-contacting DA nuclei surrounding the
64 nifesting in abnormal cerebral perfusion and cerebrospinal fluid convection, present observation pres
65 ics of the alpha-synuclein aggregates in the cerebrospinal fluid could be used to readily distinguish
66 ositive participants, 3 of whom had negative cerebrospinal fluid CrAg tests from lumbar punctures (LP
67 imary endpoint was mean rate of clearance of cerebrospinal fluid cryptococcal infection (EFA).
68                                              Cerebrospinal fluid (CSF) (1,3)-beta-d-glucan measuremen
69 lyzed associations between PiB retention and cerebrospinal fluid (CSF) Abeta concentrations in 17 M(+
70     Participants were enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis cond
71     Participants were enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis that
72           Individuals enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis were
73 ricles, is considered a disease of excessive cerebrospinal fluid (CSF) accumulation and thereby treat
74 xchange between interstitial fluid (ISF) and cerebrospinal fluid (CSF) along the paravascular spaces
75 is a source of secreted signaling factors in cerebrospinal fluid (CSF) and a key barrier between bloo
76 use brain that manifests as pro-inflammatory cerebrospinal fluid (CSF) and accumulation of ChP macrop
77 quencing (scRNA-Seq) was performed on paired cerebrospinal fluid (CSF) and blood from subjects with r
78 as to evaluate the diagnostic performance of cerebrospinal fluid (CSF) and blood KLK8 for AD and mild
79     We examined immune activation markers in cerebrospinal fluid (CSF) and blood specimens collected
80 s for visualizing CAA and assessing Abeta in cerebrospinal fluid (CSF) and brain.
81 use brain interstitial fluid (ISF) and human cerebrospinal fluid (CSF) and chronic sleep deprivation
82  1 (CHI3L1), markers of glial activation, in cerebrospinal fluid (CSF) and plasma and determined the
83                                        Novel cerebrospinal fluid (CSF) and plasma biomarkers are amon
84                                              Cerebrospinal fluid (CSF) and plasma from 35 patients wi
85 n individual HIV-1 polymerase genomes in the cerebrospinal fluid (CSF) and plasma in individuals with
86                       HIV-1 evolution in the cerebrospinal fluid (CSF) and plasma may result in disco
87 tigated a broad set of protein biomarkers in cerebrospinal fluid (CSF) and plasma using a highly sens
88                                       Whilst cerebrospinal fluid (CSF) and positron emission tomograp
89                               We studied the cerebrospinal fluid (CSF) and serum biochemical inflamma
90                                        Sera, cerebrospinal fluid (CSF) and tissue samples from 100 fa
91 or identifying Mycobacterium tuberculosis in cerebrospinal fluid (CSF) are inadequate.
92 d for distribution and antiviral activity in cerebrospinal fluid (CSF) as well as neurocognitive (NC)
93 d for distribution and antiviral activity in cerebrospinal fluid (CSF) as well as neurocognitive (NC)
94 alised brain structure which forms the blood-cerebrospinal fluid (CSF) barrier.
95                            Associations with cerebrospinal fluid (CSF) biomarkers (amyloid beta1-42,
96 ortality, and B cell phenotypes in blood and cerebrospinal fluid (CSF) by flow cytometry in HIV-infec
97 alitis, diagnosed by detection of VZV DNA in cerebrospinal fluid (CSF) by PCR and concomitant symptom
98  contribute to memory consolidation, whereas cerebrospinal fluid (CSF) clears metabolic waste product
99              However, we recently found that cerebrospinal fluid (CSF) concentration of the "social"
100 dy were to determine longitudinal changes in cerebrospinal fluid (CSF) concentrations of MMPs after a
101                                          The cerebrospinal fluid (CSF) contains an extracellular thre
102                           Circulation of the cerebrospinal fluid (CSF) contributes to body axis forma
103                 Extravasated erythrocytes in cerebrospinal fluid (CSF) critically contribute to the p
104 gate whether neurodegenerative biomarkers in cerebrospinal fluid (CSF) differentiate patients with su
105 les approach in order to investigate how the cerebrospinal fluid (CSF) enters the brain through a per
106                    The incidence of abnormal cerebrospinal fluid (CSF) flow dynamics in children with
107                       Lymphocyte activity in cerebrospinal fluid (CSF) from 4 cohorts of HTT gene exp
108 e intent of this study was to investigate if cerebrospinal fluid (CSF) from autoimmune encephalitis (
109 ed rat hippocampal neurons were treated with cerebrospinal fluid (CSF) from patients with anti-NMDAR
110 with higher cryptococcal antigen levels, the cerebrospinal fluid (CSF) fungal burden by quantitative
111 agenomic next-generation sequencing (NGS) of cerebrospinal fluid (CSF) has the potential to identify
112                                              Cerebrospinal fluid (CSF) hcrt-1 measurement has been we
113 uitary morphologic structure, and aqueductal cerebrospinal fluid (CSF) hydrodynamics relative to spac
114  pituitary deformation, augmented aqueductal cerebrospinal fluid (CSF) hydrodynamics, and expansion o
115 horylation in the choroid plexus and reduces cerebrospinal fluid (CSF) hypersecretion in a model of p
116 bound concentrations of bictegravir (BIC) in cerebrospinal fluid (CSF) in 15 asymptomatic, virologica
117 p-tau) and amyloid-beta (Abeta) 42 levels in cerebrospinal fluid (CSF) in the Amsterdam Dementia Coho
118 t microbiologically confirmed bloodstream or cerebrospinal fluid (CSF) infection between 24 h after r
119 -infection influenced clinical presentation, cerebrospinal fluid (CSF) inflammation, and outcome from
120 (GS) hypothesis states that advective driven cerebrospinal fluid (CSF) influx from the perivascular s
121 erivascular spaces that promotes movement of cerebrospinal fluid (CSF) into the brain and clearance o
122                               Tau protein in cerebrospinal fluid (CSF) is a central and relevant biom
123               Magnesium isotopic analysis of cerebrospinal fluid (CSF) is a potentially interesting a
124 r the absence of SARS-CoV-2 particles in the cerebrospinal fluid (CSF) is a topic of debate.
125                                              Cerebrospinal fluid (CSF) is a vital liquid, providing n
126                      As testing for HHV-6 in cerebrospinal fluid (CSF) is more readily available usin
127                                              Cerebrospinal fluid (CSF) is produced in the cerebral ve
128  that is released from erythrocytes into the cerebrospinal fluid (CSF) is suggested to cause vasocons
129 led perivascular spaces (PVS), through which cerebrospinal fluid (CSF) is transported from the subara
130 ctivity (EFA) of Cryptococcus clearance from cerebrospinal fluid (CSF) is used as a surrogate endpoin
131  neither the relationship between plasma and cerebrospinal fluid (CSF) KP metabolites nor their assoc
132 vel demographic and clinical information and cerebrospinal fluid (CSF) laboratory results.
133                                              Cerebrospinal fluid (CSF) lactate levels can differentia
134                                              Cerebrospinal fluid (CSF) leakage is a risk factor for d
135 s within the BACE2 locus are associated with cerebrospinal fluid (CSF) levels of Abeta1-42.
136                                    Serum and cerebrospinal fluid (CSF) levels of alpha-fetoprotein an
137 volume, cortical surface area and thickness, cerebrospinal fluid (CSF) levels of amyloid-beta (Abeta(
138 ing the A/T/N classification, relying on the cerebrospinal fluid (CSF) levels of beta-amyloid peptide
139              Here we simultaneously measured cerebrospinal fluid (CSF) levels of established amyloid
140       Here, we investigated the potential of cerebrospinal fluid (CSF) lipocalin 2 (LCN2), a secreted
141 ogression from normal aging to early AD, and cerebrospinal fluid (CSF) miR-195 levels of MCI subjects
142         We aimed to investigate the value of cerebrospinal fluid (CSF) neuronal pentraxins (NPTXs), a
143 lobin-haptoglobin complexes were measured in cerebrospinal fluid (CSF) of 44 patients with aSAH and r
144 d examined the immune cell profiles from the cerebrospinal fluid (CSF) of Neuro-COVID patients compar
145                                              Cerebrospinal fluid (CSF) of patients with anti-NMDAR en
146 e blood as well as on ventricular and lumbar cerebrospinal fluid (CSF) of pediatric patients treated
147 lf-lives and permeability into the brain and cerebrospinal fluid (CSF) of recombinant human (rh) proS
148                                              Cerebrospinal fluid (CSF) oligoclonal bands were rare in
149  medications, coexisting medical conditions, cerebrospinal fluid (CSF) opening pressure, treatments,
150 ociated with increased NFL concentrations in cerebrospinal fluid (CSF) or plasma in MCI Abeta+ and AD
151 fied two miR-298 SNPs associated with higher cerebrospinal fluid (CSF) p-tau and lower CSF Abeta42 le
152                                              Cerebrospinal fluid (CSF) p-tau181 (tau phosphorylated a
153 ut not significantly different compared with cerebrospinal fluid (CSF) P-tau217, CSF P-tau181, and ta
154 >=100 cells/muL by clinical characteristics, cerebrospinal fluid (CSF) parameters, and 18-week surviv
155                                              Cerebrospinal fluid (CSF) physiology is important for th
156      Testing was restricted to patients with cerebrospinal fluid (CSF) pleocytosis.
157  of multiple sclerosis (MS), but whether the cerebrospinal fluid (CSF) profile can help to identify p
158                                              Cerebrospinal fluid (CSF) protects the central nervous s
159 t genome-wide association study (GWAS) of 59 cerebrospinal fluid (CSF) proteins with a connection to
160 oimaging Initiative (ADNI) participants with cerebrospinal fluid (CSF) Ptau collected at baseline, di
161 h diagnostic value of a second generation of cerebrospinal fluid (CSF) Real-Time Quaking-Induced Conv
162    We summarize recent work illuminating how cerebrospinal fluid (CSF) regulates brain function.
163 gadoteric acid (Gd-DOTA) administration into cerebrospinal fluid (CSF) requires pre-contrast data for
164 r change in profile and concentration in the cerebrospinal fluid (CSF) resulted from fluctuations in
165 te the diagnostic accuracy of NAA tests with cerebrospinal fluid (CSF) samples against that of cultur
166 ament light (NFL) chain in serum, plasma and cerebrospinal fluid (CSF) samples collected over 12 mont
167             Routine EV/PeV RT-PCR testing of cerebrospinal fluid (CSF) samples in children impacted c
168 erial diseases in The Gambia where blood and cerebrospinal fluid (CSF) samples of hospitalized partic
169 tive viral load assays of plasma, semen, and cerebrospinal fluid (CSF) samples to detect HIV-1 RNA.
170 e, and neurologic examination, with elective cerebrospinal fluid (CSF) sampling, brain diffusion tens
171 re used: (1) regression of white matter (WM)/cerebrospinal fluid (CSF) signals only (BASE); (2) WM/CS
172  outcome was the change from baseline in the cerebrospinal fluid (CSF) SOD1 concentration at day 85.
173  Among suspected cases, 16 885 (92.5%) had a cerebrospinal fluid (CSF) specimen collected, 13 625 (80
174  developing Alzheimer disease (AD) and lower cerebrospinal fluid (CSF) tau biomarker levels.
175  disease (AD) has been suggested in GWASs of cerebrospinal fluid (CSF) tau levels and longitudinal ch
176                   Elevated concentrations of cerebrospinal fluid (CSF) tau, a marker of axonal injury
177         We measured the relationship between cerebrospinal fluid (CSF) TB bacillary load with mortali
178 A-based quantification of human PrP in human cerebrospinal fluid (CSF) to serve as a biomarker for Pr
179              For diagnosis of neurosyphilis, cerebrospinal fluid (CSF) TP-PA has similar performance
180                         Molecular testing of cerebrospinal fluid (CSF) using the BioFire FilmArray me
181         Pharmacokinetic data from plasma and cerebrospinal fluid (CSF) were analyzed with nonlinear m
182 l etiology, yet EV RNA is rarely detected in cerebrospinal fluid (CSF)(2).
183 t cohorts to identify protein alterations in cerebrospinal fluid (CSF), a proximal site to pathology.
184  beta-amyloid(42) and tau were quantified in cerebrospinal fluid (CSF), and white matter hyperintensi
185 ed directly from clinical specimens, such as cerebrospinal fluid (CSF), blood, or urine.
186                          In blood plasma and cerebrospinal fluid (CSF), most proteins, including cand
187        No studies have examined C4 levels in cerebrospinal fluid (CSF), presumably a better biofluid
188                                           In cerebrospinal fluid (CSF), we measured 3 macrophage and
189 e and is characterized by enlargement of the cerebrospinal fluid (CSF)-filled brain ventricles result
190 re hydrocephalus (iNPH), an expansion of the cerebrospinal fluid (CSF)-filled brain ventricles.
191                      Cancer cells within the cerebrospinal fluid (CSF)-filled leptomeninges face subs
192 n body fluids such as human blood plasma and cerebrospinal fluid (CSF).
193  has not been prospectively studied in human cerebrospinal fluid (CSF).
194 ter, and cortical regions and nonventricular cerebrospinal fluid (CSF).
195 ry end point was HTT(Rx) pharmacokinetics in cerebrospinal fluid (CSF).
196 s spp. were quantified directly from patient cerebrospinal fluid (CSF).
197 istering drugs intrathecally directly to the cerebrospinal fluid (CSF).
198 ce, as revealed by its increased presence in cerebrospinal fluid (CSF).
199  analysis by mass spectrometry in plasma and cerebrospinal fluid (CSF).
200 rylated at threonine 181 (p-tau), and NFL in cerebrospinal fluid (CSF); and one or more of PET with (
201 eding activities in patient biosamples, e.g. cerebrospinal fluids (CSF), and study their correlation
202                                    Serum and cerebrospinal fluid cytokine levels were generally simil
203 gh titers of IgG autoantibodies in serum and cerebrospinal fluid directed against the nuclei of Purki
204                      Here, we tested whether cerebrospinal fluid entry into the glymphatic system, a
205 ry of alpha-glucan material in healthy human cerebrospinal fluid, establishing a novel methodological
206  in vivo biomarkers of AD, whether by serum, cerebrospinal fluid examination or PET, have transformed
207 atic system, a brain-wide interstitial fluid-cerebrospinal fluid exchange described in rodents, exist
208                      This study analyzed the cerebrospinal fluid features of 31 COVID-19 patients wit
209 athological, clinical, molecular imaging and cerebrospinal fluid features of the most common neurodeg
210 s to irregularities in motile cilia-mediated cerebrospinal fluid flow [7-9].
211                                Age, positive cerebrospinal fluid for oligoclonal bands, infratentoria
212 suffered from errors due to heterogeneity of cerebrospinal fluid fraction and partial volume.
213 CA assay can discriminate between samples of cerebrospinal fluid from patients diagnosed with Parkins
214             Nearer to the site of pathology, cerebrospinal fluid from patients with NSAb-mediated dis
215 sis secreted higher levels of glutamate, and cerebrospinal fluid glutamine levels were increased.
216 h nonscintigraphic biomarkers from blood and cerebrospinal fluid, have provided an opportunity to inv
217 ll polymeric micelles exhibited stability in cerebrospinal fluid, highlighting the potential for loca
218  brain ventricular system reported to govern cerebrospinal fluid homeostasis [13, 14], neurogenesis [
219 an adaptive immune response in the blood and cerebrospinal fluid in Alzheimer's disease and provide e
220   Proteins from this module were elevated in cerebrospinal fluid in early stages of the disease.
221 es in ratios comparable to those secreted to cerebrospinal fluid in human, however the protocol to ac
222  chemosensitive glial cells that contact the cerebrospinal fluid in the third ventricle and send proc
223 cords were excluded, as they did not examine cerebrospinal fluid, included animals, or focused on non
224  NMDAR antibody seropositive have coexisting cerebrospinal fluid inflammatory changes or other paracl
225 mmune, the presence of GAD antibodies in the cerebrospinal fluid is sufficient to confirm a pathogeni
226 ion of T cells, B cells, and blinatumomab in cerebrospinal fluid, (iv) blinatumomab-induced T-cell ro
227 ear or sinus infections (43 of 120, 36%) and cerebrospinal fluid leakage (37 of 116, 32%).
228 ious side effects, including hemorrhages and cerebrospinal fluid leakage.
229 in patients with ear or sinus infections and cerebrospinal fluid leakage.
230      Possible side effects include worsening cerebrospinal fluid leaks.
231 tion of human ChP organoids, which produce a cerebrospinal fluid-like secretion and recapitulate barr
232 Cs) for testing postmortem specimens (blood, cerebrospinal fluid, lung tissue, respiratory tract swab
233 tate at multiple sites of the tau protein in cerebrospinal fluid markers across four decades of disea
234 l cut point, 1.55 SUVR), and comparison with cerebrospinal fluid measures of amyloid-beta (optimal cu
235 ussian mixture modeling, and comparison with cerebrospinal fluid measures of amyloid-beta, specifical
236 and within sulci, suggesting an inflammatory cerebrospinal fluid-mediated lesion pathogenesis.
237                       Significant changes of cerebrospinal fluid microRNAs that control inflammation,
238 ses the technical advantages and pitfalls of cerebrospinal fluid mNGS in the context of patients with
239 c during head and neck movements and promote cerebrospinal fluid motion; however, their role in neuro
240 ay a critical role in mucociliary clearance, cerebrospinal fluid movement, and fertility.
241    We observed neither SARS-CoV-2 RNA in the cerebrospinal fluid, nor intrathecal IgG synthesis, but
242 impairment, and increased protein content in cerebrospinal fluid occurred in anti-Drebrin-seropositiv
243 ens from the brains of people with AD and in cerebrospinal fluid of individuals diagnosed with AD.
244 from both postmortem brains and detection in cerebrospinal fluid of infected individuals.
245 specificity of clonally expanded TCRs in the cerebrospinal fluid of patients with Alzheimer's disease
246 lonally expanded CD8(+) T(EMRA) cells in the cerebrospinal fluid of patients with Alzheimer's disease
247       IL-11(+)CD4(+) cells accumulate in the cerebrospinal fluid of patients with early relapsing-rem
248            Moreover, AICAr is present in the cerebrospinal fluid of patients with LND, but not in con
249 n the concentration range that occurs in the cerebrospinal fluid of patients with SAH.
250 he endocannabinoid anandamide is elevated in cerebrospinal fluid of patients with schizophrenia.
251 esioned Macaca mulatta, and in the serum and cerebrospinal fluid of PD patients.
252             We show these profiles mirror in cerebrospinal fluid of people with DS.
253 ecovered from the bronchoalveolar lavage and cerebrospinal fluids of critically ill patients.
254 e screening tool than the currently approved cerebrospinal fluid or amyloid beta positron emission to
255               The diagnosis was supported by cerebrospinal fluid or amyloid positron emission tomogra
256 to amyloid-beta or tau pathology measured in cerebrospinal fluid or by positron emission tomography(2
257 radation, with a half-life of ~5 days in rat cerebrospinal fluid or serum-containing medium.
258 ance imaging or computed tomography imaging, cerebrospinal fluid parameters and clinical endpoints.
259 inical improvement along with a reduction of cerebrospinal fluid parameters was observed after high-d
260             More abnormal levels of baseline cerebrospinal fluid phosphorylated tau and subthreshold
261 n the processes of notochord development and cerebrospinal fluid physiology, and how defects in those
262                                              Cerebrospinal fluid pleocytosis and protein elevation we
263                                            A cerebrospinal fluid polymerase chain reaction (PCR) for
264 jury biomarker soluble PDGFRbeta(7,8) in the cerebrospinal fluid predicted future cognitive decline i
265 as are located adjacent to and bathed by the cerebrospinal fluid, presenting an opportunity for locor
266                        By altering embryonic cerebrospinal fluid pressure during development, we foun
267 ura mater-surround the CNS, encompassing the cerebrospinal fluid produced by the choroid plexus epith
268                             Interstitial and cerebrospinal fluid produced in the CNS is continuously
269              We identified and replicated 10 cerebrospinal fluid proteins including IL-12B, CD5, MIP-
270              Comparisons of brain tissue and cerebrospinal fluid proteomes reveal biomarker candidate
271 tched by changes in surrounding amniotic and cerebrospinal fluid proteomes.
272                  Multiplexed DIA analysis of cerebrospinal fluid revealed the dynamic proteome change
273 n this study of >2,000 brains and nearly 400 cerebrospinal fluid samples by quantitative proteomics,
274 kynurenine (Kyn) and tryptophan (Trp) in 221 cerebrospinal fluid samples from patients with bacterial
275  detect aggregated Abeta in brain tissue and cerebrospinal fluid samples of AD mice.
276  metagenomic sequencing testing requested on cerebrospinal fluid samples submitted to an outside refe
277  amount of target molecules in the blood and cerebrospinal fluids samples.
278 nsion in infants with CHD were identified in cerebrospinal fluid spaces (p < 0.025).
279                                              Cerebrospinal fluid specimens from 29 cases were tested
280                                              Cerebrospinal fluid specimens were analyzed by culture a
281 ase of levels of neurofilaments in patients' cerebrospinal fluid, suggest that IgLON5 IgG, unlike oth
282 oton imaging in rodents to show instead that cerebrospinal fluid surrounding the brain enters the tis
283                                              Cerebrospinal fluid testing showed normal cell counts, a
284 novel protein changes in Parkinson's disease cerebrospinal fluid that may be exploited for understand
285 ical microbiology laboratory testing data of cerebrospinal fluid, the expected August/September/Octob
286 tetrameric carrier of thyroxine in blood and cerebrospinal fluid, the pathogenic aggregation of which
287                          In contact with the cerebrospinal fluid through their cell bodies, they send
288  source of miR-204 that is released into the cerebrospinal fluid to control number of NSCs within the
289  use of (1,3)-beta-d-glucan measurement with cerebrospinal fluid to diagnose fungal meningitis.
290 lammation, and increase AD biomarkers in the cerebrospinal fluid to similar levels observed in patien
291 rkers of neuroinflammation and injury in the cerebrospinal fluid (tumor necrosis factor-alpha, kynure
292 faster in female youths than in male youths; cerebrospinal fluid volume expanded faster in most cereb
293 mian white matter expansion, and accelerated cerebrospinal fluid volumes expansion of anterior lobule
294                                              Cerebrospinal fluid was collected from children <5 years
295               The fungal clearance rate from cerebrospinal fluid was similar between groups (0.43 -lo
296 s of aggregates that were amplified from the cerebrospinal fluid were similar to those of aggregates
297                            Levels of SOD1 in cerebrospinal fluid were transiently and only slightly l
298 ut mice have low levels of circulating OT in cerebrospinal fluid, which can be corrected by the oral
299 ect alpha-synuclein aggregates in samples of cerebrospinal fluid with high sensitivity and specificit
300 n of phosphorylated tau and amyloid in their cerebrospinal fluid, yielding neurotypical and preclinic

 
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