コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 tem abnormalities (increased posterior fossa cerebrospinal fluid).
2 or sensing and delivery, in blood plasma and cerebrospinal fluid.
3 ology and tumor DNA extracted from blood and cerebrospinal fluid.
4 A-matrix metalloproteinase-9 pathway(19) in cerebrospinal fluid.
5 Only one patient had SARS-CoV-2 RNA in the cerebrospinal fluid.
6 identify Parkinson's-relevant biomarkers in cerebrospinal fluid.
7 delivery from arterial blood to ventricular cerebrospinal fluid.
8 ndividual is reflected in all tissues and in cerebrospinal fluid.
9 ient detection of TSEs prion protein in goat cerebrospinal fluid.
10 bs, which appear as oligoclonal bands in the cerebrospinal fluid.
11 egative except for increased posterior fossa cerebrospinal fluid.
12 IIB dogs had elevated HS levels in brain and cerebrospinal fluid.
13 aims to characterize the evidence regarding cerebrospinal fluid (1,3)-beta-d-glucan measurement to d
15 e chimeric antigen receptor T cells into the cerebrospinal fluid, alone or in combination with azacyt
19 ersons who underwent lumbar puncture and had cerebrospinal fluid analysis (January 1, 2008-December 3
24 tracranial air (ICA)], water-filled balloon (cerebrospinal fluid and blood) and agarose gel (brain).
25 uencing (mNGS) of RNA and DNA extracted from cerebrospinal fluid and brain tissue now offers another
26 , soluble TREM2 (sTREM2), is abundant in the cerebrospinal fluid and its levels positively correlate
28 able to monitor the changes in DA levels in cerebrospinal fluid and plasma of a mouse model of PD an
29 effector C3 were present at higher levels in cerebrospinal fluid and plasma(8,9) in men than in women
30 cleavage product of TREM2, is elevated in AD cerebrospinal fluid and positively correlates with cogni
35 whether nilotinib is safe, and detectable in cerebrospinal fluid, and alters biomarkers and clinical
36 e Mini-Mental Status Exam, protein levels in cerebrospinal fluid, and genotype at the APOE and MAPT l
41 the recognition of oligoclonal bands in the cerebrospinal fluid as a possible marker of disseminatio
42 She was ultimately found to have sterile cerebrospinal fluid ascites which was treated successful
43 r's disease are accessible only via invasive cerebrospinal fluid assays, and reactive oxygen species
44 In vivo tauopathy has been measured using cerebrospinal fluid assays, but these do not provide inf
45 heir phase II metabolites permeate the blood-cerebrospinal fluid barrier (B-CSF-B) of sheep and to pr
47 luding the blood brain barrier and the blood cerebrospinal fluid barrier, protect the CNS from extern
49 to investigate the cognitive impairment and cerebrospinal fluid biomarkers in a follow-up study of p
52 can result in very low folate levels in the cerebrospinal fluid causing childhood neurodegenerative
53 study, we assessed the levels of ventricular cerebrospinal fluid-cfmtDNA (vCSF-cfmtDNA) in a diverse
55 ated tau concentrations in serial samples of cerebrospinal fluid collected from participants who were
58 sed of amyloid beta (Abeta) peptides and the cerebrospinal fluid concentrations of those peptides are
63 The first comprises an interconnected set of cerebrospinal-fluid-contacting DA nuclei surrounding the
64 nifesting in abnormal cerebral perfusion and cerebrospinal fluid convection, present observation pres
65 ics of the alpha-synuclein aggregates in the cerebrospinal fluid could be used to readily distinguish
66 ositive participants, 3 of whom had negative cerebrospinal fluid CrAg tests from lumbar punctures (LP
69 lyzed associations between PiB retention and cerebrospinal fluid (CSF) Abeta concentrations in 17 M(+
70 Participants were enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis cond
71 Participants were enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis that
73 ricles, is considered a disease of excessive cerebrospinal fluid (CSF) accumulation and thereby treat
74 xchange between interstitial fluid (ISF) and cerebrospinal fluid (CSF) along the paravascular spaces
75 is a source of secreted signaling factors in cerebrospinal fluid (CSF) and a key barrier between bloo
76 use brain that manifests as pro-inflammatory cerebrospinal fluid (CSF) and accumulation of ChP macrop
77 quencing (scRNA-Seq) was performed on paired cerebrospinal fluid (CSF) and blood from subjects with r
78 as to evaluate the diagnostic performance of cerebrospinal fluid (CSF) and blood KLK8 for AD and mild
81 use brain interstitial fluid (ISF) and human cerebrospinal fluid (CSF) and chronic sleep deprivation
82 1 (CHI3L1), markers of glial activation, in cerebrospinal fluid (CSF) and plasma and determined the
85 n individual HIV-1 polymerase genomes in the cerebrospinal fluid (CSF) and plasma in individuals with
87 tigated a broad set of protein biomarkers in cerebrospinal fluid (CSF) and plasma using a highly sens
92 d for distribution and antiviral activity in cerebrospinal fluid (CSF) as well as neurocognitive (NC)
93 d for distribution and antiviral activity in cerebrospinal fluid (CSF) as well as neurocognitive (NC)
96 ortality, and B cell phenotypes in blood and cerebrospinal fluid (CSF) by flow cytometry in HIV-infec
97 alitis, diagnosed by detection of VZV DNA in cerebrospinal fluid (CSF) by PCR and concomitant symptom
98 contribute to memory consolidation, whereas cerebrospinal fluid (CSF) clears metabolic waste product
100 dy were to determine longitudinal changes in cerebrospinal fluid (CSF) concentrations of MMPs after a
104 gate whether neurodegenerative biomarkers in cerebrospinal fluid (CSF) differentiate patients with su
105 les approach in order to investigate how the cerebrospinal fluid (CSF) enters the brain through a per
108 e intent of this study was to investigate if cerebrospinal fluid (CSF) from autoimmune encephalitis (
109 ed rat hippocampal neurons were treated with cerebrospinal fluid (CSF) from patients with anti-NMDAR
110 with higher cryptococcal antigen levels, the cerebrospinal fluid (CSF) fungal burden by quantitative
111 agenomic next-generation sequencing (NGS) of cerebrospinal fluid (CSF) has the potential to identify
113 uitary morphologic structure, and aqueductal cerebrospinal fluid (CSF) hydrodynamics relative to spac
114 pituitary deformation, augmented aqueductal cerebrospinal fluid (CSF) hydrodynamics, and expansion o
115 horylation in the choroid plexus and reduces cerebrospinal fluid (CSF) hypersecretion in a model of p
116 bound concentrations of bictegravir (BIC) in cerebrospinal fluid (CSF) in 15 asymptomatic, virologica
117 p-tau) and amyloid-beta (Abeta) 42 levels in cerebrospinal fluid (CSF) in the Amsterdam Dementia Coho
118 t microbiologically confirmed bloodstream or cerebrospinal fluid (CSF) infection between 24 h after r
119 -infection influenced clinical presentation, cerebrospinal fluid (CSF) inflammation, and outcome from
120 (GS) hypothesis states that advective driven cerebrospinal fluid (CSF) influx from the perivascular s
121 erivascular spaces that promotes movement of cerebrospinal fluid (CSF) into the brain and clearance o
128 that is released from erythrocytes into the cerebrospinal fluid (CSF) is suggested to cause vasocons
129 led perivascular spaces (PVS), through which cerebrospinal fluid (CSF) is transported from the subara
130 ctivity (EFA) of Cryptococcus clearance from cerebrospinal fluid (CSF) is used as a surrogate endpoin
131 neither the relationship between plasma and cerebrospinal fluid (CSF) KP metabolites nor their assoc
137 volume, cortical surface area and thickness, cerebrospinal fluid (CSF) levels of amyloid-beta (Abeta(
138 ing the A/T/N classification, relying on the cerebrospinal fluid (CSF) levels of beta-amyloid peptide
141 ogression from normal aging to early AD, and cerebrospinal fluid (CSF) miR-195 levels of MCI subjects
143 lobin-haptoglobin complexes were measured in cerebrospinal fluid (CSF) of 44 patients with aSAH and r
144 d examined the immune cell profiles from the cerebrospinal fluid (CSF) of Neuro-COVID patients compar
146 e blood as well as on ventricular and lumbar cerebrospinal fluid (CSF) of pediatric patients treated
147 lf-lives and permeability into the brain and cerebrospinal fluid (CSF) of recombinant human (rh) proS
149 medications, coexisting medical conditions, cerebrospinal fluid (CSF) opening pressure, treatments,
150 ociated with increased NFL concentrations in cerebrospinal fluid (CSF) or plasma in MCI Abeta+ and AD
151 fied two miR-298 SNPs associated with higher cerebrospinal fluid (CSF) p-tau and lower CSF Abeta42 le
153 ut not significantly different compared with cerebrospinal fluid (CSF) P-tau217, CSF P-tau181, and ta
154 >=100 cells/muL by clinical characteristics, cerebrospinal fluid (CSF) parameters, and 18-week surviv
157 of multiple sclerosis (MS), but whether the cerebrospinal fluid (CSF) profile can help to identify p
159 t genome-wide association study (GWAS) of 59 cerebrospinal fluid (CSF) proteins with a connection to
160 oimaging Initiative (ADNI) participants with cerebrospinal fluid (CSF) Ptau collected at baseline, di
161 h diagnostic value of a second generation of cerebrospinal fluid (CSF) Real-Time Quaking-Induced Conv
163 gadoteric acid (Gd-DOTA) administration into cerebrospinal fluid (CSF) requires pre-contrast data for
164 r change in profile and concentration in the cerebrospinal fluid (CSF) resulted from fluctuations in
165 te the diagnostic accuracy of NAA tests with cerebrospinal fluid (CSF) samples against that of cultur
166 ament light (NFL) chain in serum, plasma and cerebrospinal fluid (CSF) samples collected over 12 mont
168 erial diseases in The Gambia where blood and cerebrospinal fluid (CSF) samples of hospitalized partic
169 tive viral load assays of plasma, semen, and cerebrospinal fluid (CSF) samples to detect HIV-1 RNA.
170 e, and neurologic examination, with elective cerebrospinal fluid (CSF) sampling, brain diffusion tens
171 re used: (1) regression of white matter (WM)/cerebrospinal fluid (CSF) signals only (BASE); (2) WM/CS
172 outcome was the change from baseline in the cerebrospinal fluid (CSF) SOD1 concentration at day 85.
173 Among suspected cases, 16 885 (92.5%) had a cerebrospinal fluid (CSF) specimen collected, 13 625 (80
175 disease (AD) has been suggested in GWASs of cerebrospinal fluid (CSF) tau levels and longitudinal ch
178 A-based quantification of human PrP in human cerebrospinal fluid (CSF) to serve as a biomarker for Pr
183 t cohorts to identify protein alterations in cerebrospinal fluid (CSF), a proximal site to pathology.
184 beta-amyloid(42) and tau were quantified in cerebrospinal fluid (CSF), and white matter hyperintensi
189 e and is characterized by enlargement of the cerebrospinal fluid (CSF)-filled brain ventricles result
200 rylated at threonine 181 (p-tau), and NFL in cerebrospinal fluid (CSF); and one or more of PET with (
201 eding activities in patient biosamples, e.g. cerebrospinal fluids (CSF), and study their correlation
203 gh titers of IgG autoantibodies in serum and cerebrospinal fluid directed against the nuclei of Purki
205 ry of alpha-glucan material in healthy human cerebrospinal fluid, establishing a novel methodological
206 in vivo biomarkers of AD, whether by serum, cerebrospinal fluid examination or PET, have transformed
207 atic system, a brain-wide interstitial fluid-cerebrospinal fluid exchange described in rodents, exist
209 athological, clinical, molecular imaging and cerebrospinal fluid features of the most common neurodeg
213 CA assay can discriminate between samples of cerebrospinal fluid from patients diagnosed with Parkins
215 sis secreted higher levels of glutamate, and cerebrospinal fluid glutamine levels were increased.
216 h nonscintigraphic biomarkers from blood and cerebrospinal fluid, have provided an opportunity to inv
217 ll polymeric micelles exhibited stability in cerebrospinal fluid, highlighting the potential for loca
218 brain ventricular system reported to govern cerebrospinal fluid homeostasis [13, 14], neurogenesis [
219 an adaptive immune response in the blood and cerebrospinal fluid in Alzheimer's disease and provide e
221 es in ratios comparable to those secreted to cerebrospinal fluid in human, however the protocol to ac
222 chemosensitive glial cells that contact the cerebrospinal fluid in the third ventricle and send proc
223 cords were excluded, as they did not examine cerebrospinal fluid, included animals, or focused on non
224 NMDAR antibody seropositive have coexisting cerebrospinal fluid inflammatory changes or other paracl
225 mmune, the presence of GAD antibodies in the cerebrospinal fluid is sufficient to confirm a pathogeni
226 ion of T cells, B cells, and blinatumomab in cerebrospinal fluid, (iv) blinatumomab-induced T-cell ro
231 tion of human ChP organoids, which produce a cerebrospinal fluid-like secretion and recapitulate barr
232 Cs) for testing postmortem specimens (blood, cerebrospinal fluid, lung tissue, respiratory tract swab
233 tate at multiple sites of the tau protein in cerebrospinal fluid markers across four decades of disea
234 l cut point, 1.55 SUVR), and comparison with cerebrospinal fluid measures of amyloid-beta (optimal cu
235 ussian mixture modeling, and comparison with cerebrospinal fluid measures of amyloid-beta, specifical
238 ses the technical advantages and pitfalls of cerebrospinal fluid mNGS in the context of patients with
239 c during head and neck movements and promote cerebrospinal fluid motion; however, their role in neuro
241 We observed neither SARS-CoV-2 RNA in the cerebrospinal fluid, nor intrathecal IgG synthesis, but
242 impairment, and increased protein content in cerebrospinal fluid occurred in anti-Drebrin-seropositiv
243 ens from the brains of people with AD and in cerebrospinal fluid of individuals diagnosed with AD.
245 specificity of clonally expanded TCRs in the cerebrospinal fluid of patients with Alzheimer's disease
246 lonally expanded CD8(+) T(EMRA) cells in the cerebrospinal fluid of patients with Alzheimer's disease
250 he endocannabinoid anandamide is elevated in cerebrospinal fluid of patients with schizophrenia.
254 e screening tool than the currently approved cerebrospinal fluid or amyloid beta positron emission to
256 to amyloid-beta or tau pathology measured in cerebrospinal fluid or by positron emission tomography(2
258 ance imaging or computed tomography imaging, cerebrospinal fluid parameters and clinical endpoints.
259 inical improvement along with a reduction of cerebrospinal fluid parameters was observed after high-d
261 n the processes of notochord development and cerebrospinal fluid physiology, and how defects in those
264 jury biomarker soluble PDGFRbeta(7,8) in the cerebrospinal fluid predicted future cognitive decline i
265 as are located adjacent to and bathed by the cerebrospinal fluid, presenting an opportunity for locor
267 ura mater-surround the CNS, encompassing the cerebrospinal fluid produced by the choroid plexus epith
273 n this study of >2,000 brains and nearly 400 cerebrospinal fluid samples by quantitative proteomics,
274 kynurenine (Kyn) and tryptophan (Trp) in 221 cerebrospinal fluid samples from patients with bacterial
276 metagenomic sequencing testing requested on cerebrospinal fluid samples submitted to an outside refe
281 ase of levels of neurofilaments in patients' cerebrospinal fluid, suggest that IgLON5 IgG, unlike oth
282 oton imaging in rodents to show instead that cerebrospinal fluid surrounding the brain enters the tis
284 novel protein changes in Parkinson's disease cerebrospinal fluid that may be exploited for understand
285 ical microbiology laboratory testing data of cerebrospinal fluid, the expected August/September/Octob
286 tetrameric carrier of thyroxine in blood and cerebrospinal fluid, the pathogenic aggregation of which
288 source of miR-204 that is released into the cerebrospinal fluid to control number of NSCs within the
290 lammation, and increase AD biomarkers in the cerebrospinal fluid to similar levels observed in patien
291 rkers of neuroinflammation and injury in the cerebrospinal fluid (tumor necrosis factor-alpha, kynure
292 faster in female youths than in male youths; cerebrospinal fluid volume expanded faster in most cereb
293 mian white matter expansion, and accelerated cerebrospinal fluid volumes expansion of anterior lobule
296 s of aggregates that were amplified from the cerebrospinal fluid were similar to those of aggregates
298 ut mice have low levels of circulating OT in cerebrospinal fluid, which can be corrected by the oral
299 ect alpha-synuclein aggregates in samples of cerebrospinal fluid with high sensitivity and specificit
300 n of phosphorylated tau and amyloid in their cerebrospinal fluid, yielding neurotypical and preclinic