ライフサイエンスコーパス: cervical cord

1 ation, and astrocytic gliosis in the injured cervical cord.

2 nia or optogenetic photoactivation of the C4 cervical cord.

3 l and lateral columns and grey matter of the cervical cord.

4 rsal column extending from the lumbar to the cervical cord.

5 injury to the craniocervical junction or the cervical cord.

6 sion load and the presence of atrophy of the cervical cord.

7 ng to target sensorimotor convergence in the cervical cord.

8 ding motor and spinal sensory stimuli in the cervical cord.

9 ns in the upper cervical cord, who underwent cervical cord (1) H-magnetic resonance spectroscopy.

10 of all labeled cells were located within the cervical cord, 18% in thoracic cord, and 19% in the lumb

11 althy volunteers) underwent spinal cord MRI (cervical cord: 3D T1, 3D T2, diffusion tensor imaging an

12 tion of the corticospinal tract (CST) in the cervical cord above a traumatic lesion and explored its

13 e-specific blood perfusion impairment of the cervical cord above the compression site in patients wit

14 to the biochemical changes in the atrophied cervical cord after spinal cord injury.

15 al area (LPb), and that most of those in the cervical cord also belong to the spinothalamic tract.

16 s showed progressive degenerative changes in cervical cord and brain morphometry across the sensory s

17 DTI of the cervical cord and brain provided measurements of fractio

18 the spinal cord (SC) and the brain, with the cervical cord and brain stem as the reference region, re

19 nificantly lower in both CSTs throughout the cervical cord and brain when compared with controls (p</

20 rest (ROI) included the bilateral CST in the cervical cord and brain.

21 om sporadic PAPT in having marked atrophy of cervical cord and brainstem with corticospinal signs but

22 ance spectroscopy and q-space imaging of the cervical cord and conventional brain and spinal magnetic

23 ing single-voxel (1)H-MR spectroscopy of the cervical cord and diffusion-based tractography of the ma

24 -like immunoreactivity in all laminae of the cervical cord and in laminae I and II and the ventral ho

25 expression in laminae I-II of the lumbar and cervical cord and in the rostral ventromedial medulla in

26 ues in the PAG and greater R2* values in the cervical cord are associated with NP intensity.

27 hippocampal (0.000031, p = 0.044) and upper cervical cord area (UCCA) loss (1.482887, p = 0.005), co

28 data provided information on cross-sectional cervical cord area and volumetric brain changes in 30 in

29 A smaller cervical cord area was associated with impaired upper li

30 inal cord gray and white matter areas, upper cervical cord area, and the ratio of gray matter to the

31 a, and the ratio of gray matter to the upper cervical cord area.

32 any training interventions and the amount of cervical cord atrophy above the injury, length of post-t

33 Neutral MRI revealed LOA in 64.7%, lower cervical cord atrophy in all patients, T2 hyperintensity

34 association between perfusion impairment and cervical cord atrophy indicates that changes in hemodyna

35 Cervical cord atrophy was also assessed by using an acti

36 In patients with MS, cervical cord atrophy was correlated with clinical disab

37 PAPT is associated with marked brainstem and cervical cord atrophy with corticospinal tract findings,

38 chment of posterior dura, asymmetrical lower cervical cord atrophy, T2 hyperintensity, and loss of ce

39 of attachment (LOA) of posterior dura, lower cervical cord atrophy, T2 hyperintensity, loss of cervic

40 increased corticospinal axon density in the cervical cord below the level of the injury relative to

41 g syndrome that involves the optic nerve and cervical cord but differs pathologically from multiple s

42 n patients with non-myelopathic degenerative cervical cord compression (NMDCCC), i.e., without clinic

43 Seven of the 366 patients had cervical cord contusions, four patients had ligamentous

44 ated with Ambulation Index, whereas only the cervical cord correlated with disease duration (p < 0.05

45 IVIM were applied in all participants in the cervical cord (covering C1-C3 levels) to determine white

46 dy disease showed a significant reduction in cervical cord cross-sectional area (P = 0.038), thoracic

47 SC total cervical cord cross-sectional area (TCA), gray matter ar

48 Upper cervical cord cross-sectional area (UCCA), brain and spi

49 Cervical cord cross-sectional area associated with disab

50 global and regional brain volumes and upper cervical cord cross-sectional area that are highly repro

51 tures of patients with HSP who had brain and cervical cord damage were also investigated.

52 In the cervical cord, divergent long descending propriospinal n

53 sion loads, age, sex, and disease duration), cervical cord GM areas had the strongest correlation wit

54 In multiple sclerosis (MS), upper cervical cord gray matter (GM) atrophy correlates more s

55 racic cord cross-sectional area (P = 0.043), cervical cord grey matter (P = 0.011), magnetization tra

56 These unfolded cervical cord images were coregistered into a common sta

57 Each subject underwent quantitative cervical cord imaging.

58 all patients, T2 hyperintensity in the lower cervical cord in 35.2% of patients, and loss of cervical

59 sensitive to remote perfusion changes in the cervical cord in DCM and may serve as neuroimaging bioma

60 s was thin compared to HVs, whereas only the cervical cord in MS patients was thinner than in HVs (p

61 Reduced FA of the cervical cord in patients with SCI was associated with s

62 lanning, but its routine use for imaging the cervical cord in shaken, abused infants without clinical

63 of low signal intensity that compressed the cervical cord in six patients.

64 olumes and magnetization transfer ratio) and cervical cord involvement.

65 f the regional distribution of damage in the cervical cord is feasible and might improve our understa

66 l nucleus synapse in the brainstem and upper cervical cord is the most likely site of action for brai

67 Patients showed lower tNAA of the cervical cord, lower connectivity and lower fractional a

68 A significant difference in cervical cord mean fractional anisotropy (FA) was found

69 lesions in an MS cord are seen in the upper cervical cord, most of the pathology in HAM/TSP is seen

70 A greater number of cervical cord neurons responded to hypocretin than anoth

71 alities in the glutamatergic pathways in the cervical cord of early primary progressive multiple scle

72 rror corrected for multiple comparisons) and cervical cord (P < .001) in patients with HSP relative t

73 x, while increases in R2* are evident in the cervical cord, periaqueductal grey (PAG), thalamus and a

74 and longitudinally extensive lesions in the cervical cord pointed towards a diagnosis of MOG-antibod

75 e of longitudinally extensive lesions in the cervical cord predicted MOG-antibody disease versus AQP4

76 ng left-right interactions within lumbar and cervical cords, promote left-right synchronization neces

77 copy and diffusion-based tractography of the cervical cord provide measures that are sensitive to the

78 onal degeneration of the CST in the atrophic cervical cord, proximal to the site of injury, parallels

79 Fourteen patients at the onset of a cervical cord relapse with at least one lesion between C

80 jor spinal cord pathways, in patients with a cervical cord relapse, differed from controls and correl

81 ge occurring in this area in patients with a cervical cord relapse.

82 cores (B = -0.07, P = 0.0440, R2 = 0.20) and cervical cord spinothalamic tract fractional anisotropy

83 Baseline 3.0T brain and cervical cord T2-weighted and three-dimensional T1-weigh

84 ord, but were 10 times more prevalent in the cervical cord than the lumbar cord.

85 sition or the trigeminal subnucleus caudalis-cervical cord (Vc/C1) junction region in the lower brain

86 ts, cortical lesions, grey matter volume and cervical cord volume explained 60% of the variance of th

87 The upper cervical cord volumes (r = -0.39, P < 0.01), but not the

88 red cortex that innervated the contralateral cervical cord was five times that of controls, and in th

89 lution magnetic resonance (MR) images of the cervical cord were acquired from 45 patients with RR MS,

90 istribution patterns on the two sides of the cervical cord were compared.

91 rexin 1 receptor protein in the ventral C3-5 cervical cord were statistically diminished in WNV-infec

92 ongitudinally extensive lesions in the upper cervical cord, who underwent cervical cord (1) H-magneti