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1 obligately intracellular bacterium Ehrlichia chaffeensis.
2 at D. melanogaster is a suitable host for E. chaffeensis.
3  is a tick-borne disease caused by Ehrlichia chaffeensis.
4 -specific expression of the T4S system in E. chaffeensis.
5 iable-length PCR target (VLPT) protein in E. chaffeensis.
6 the protein expression and interaction in E. chaffeensis.
7 -containing vacuoles than in vacuole-free E. chaffeensis.
8 nsis-containing vacuoles and vacuole-free E. chaffeensis.
9 ular basis of the host immune response to E. chaffeensis.
10  by a tick-transmitted rickettsia, Ehrlichia chaffeensis.
11 ost cell-specific antigenic expression by E. chaffeensis.
12 e disease caused by the rickettsia Ehrlichia chaffeensis.
13 was developed for the detection of Ehrlichia chaffeensis.
14 ared with previously described assays for E. chaffeensis.
15 ritical for conferring rapid clearance of E. chaffeensis.
16 d T-cell function on murine resistance to E. chaffeensis.
17 a major antigenic protein (P28) of Ehrlichia chaffeensis.
18  zoonoses caused by infection with Ehrlichia chaffeensis.
19 f both the IL-8 promoter and NF-kappaB by E. chaffeensis.
20 olysaccharide-deficient bacterium, Ehrlichia chaffeensis.
21 _0379 mutant and challenge with wild-type E. chaffeensis 1 month following inoculation with the mutan
22 spectrometry identified the protein as an E. chaffeensis 12.3-kDa hypothetical protein, which was des
23 l tandem repeat DNA-binding domain in the E. chaffeensis 120-kDa tandem repeat protein (TRP120) that
24 olymerase chain reaction (PCR) for Ehrlichia chaffeensis, 2) acute and convalescent serum titers, and
25 genes and that antigenic variation of the E. chaffeensis 28-kDa proteins may result from differential
26  Recently, molecular interactions between E. chaffeensis 47-kDa tandem repeat (TR) protein (TRP47) an
27 . canis and the corresponding ortholog of E. chaffeensis (47 kDa) were identified and the proteins ch
28 01 were positive by PCR assays for Ehrlichia chaffeensis (50 of 217; 23%), Ehrlichia ewingii (44 of 2
29          Major immunoreactive proteins in E. chaffeensis (75-kDa) and E. canis (95-kD) whole-cell lys
30 d a very high prevalence of antibodies to E. chaffeensis (97 of 112; 87%) and a low prevalence of ant
31                                    Ehrlichia chaffeensis, a bacterium that cannot survive outside the
32                                    Ehrlichia chaffeensis, a cholesterol-rich and cholesterol-dependen
33 totropic ehrlichiosis is caused by Ehrlichia chaffeensis, a Gram-negative bacterium lacking lipopolys
34                                    Ehrlichia chaffeensis, a tick-borne rickettsial organism, causes t
35                                    Ehrlichia chaffeensis, a tick-transmitted obligate intracellular r
36                                    Ehrlichia chaffeensis, a tick-transmitted rickettsial agent, cause
37                                    Ehrlichia chaffeensis, a tick-transmitted rickettsial agent, is re
38                                    Ehrlichia chaffeensis, a tick-transmitted rickettsial, is the caus
39    E. ewingii accounted for 20 (91%), and E. chaffeensis accounted for 1 (5%) of the positives.
40                                    Third, E. chaffeensis also inhibited the gene transcription of RAB
41 ning, infectious disease caused by Ehrlichia chaffeensis, an obligate intracellular bacterium that la
42 d mutations by allelic exchange in Ehrlichia chaffeensis, an obligate intracellular tick-borne bacter
43                                    Ehrlichia chaffeensis, an obligate intracellular, tick-transmitted
44                      The genome of Ehrlichia chaffeensis, an obligatory intracellular bacterium that
45                                    Ehrlichia chaffeensis, an obligatory intracellular gram-negative b
46 ns with nonviral pathogens (2 with Ehrlichia chaffeensis and 1 with Mycoplasma pneumoniae).
47                               Analysis of E. chaffeensis and A. phagocytophilum genome sequences reve
48 btained for HL-60 cells used as hosts for E. chaffeensis and A. phagocytophilum.
49 ested whether cholesterol is required for E. chaffeensis and A. phagocytophilum.
50                                    Ehrlichia chaffeensis and Anaplasma phagocytophilum are agents of
51 olymerase chain reaction assay for Ehrlichia chaffeensis and by the demonstration of morulae within p
52                                    Ehrlichia chaffeensis and E. canis have a small subset of tandem r
53 re observed primarily in the periplasm of E. chaffeensis and E. canis.
54                               Only Ehrlichia chaffeensis and E. ewingii have been thought to cause eh
55 e bond formation (Dsb) proteins of Ehrlichia chaffeensis and Ehrlichia canis were identified which re
56 e proteins have been identified in Ehrlichia chaffeensis and Ehrlichia canis, including three molecul
57 onserved in the omp1 gene locus of Ehrlichia chaffeensis and p30 gene locus of E. canis despite marke
58 serologic cross-reactivity between Ehrlichia chaffeensis and the agent of HGE.
59  N-terminal TR region (18 amino acids) in E. chaffeensis and the complete TR (24 amino acids) in E. c
60     Western immunoblotting using purified E. chaffeensis and the HGE agent as antigens suggested that
61 ompare the genome sequences of strains of E. chaffeensis and to examine the virulence potentials of t
62 ocation of bacterially encoded protein by E. chaffeensis and to identify a specific binding motif and
63 eins in Anaplasma phagocytophilum, Ehrlichia chaffeensis, and Ehrlichia canis.
64 are essential for immunity against Ehrlichia chaffeensis, and protective mechanisms involve blocking
65 tease HtrA was detected on the surface of E. chaffeensis, and TRP120 was degraded by treatment of E.
66            Differential transmigration of E. chaffeensis- and A. phagocytophilum-infected leukocytes
67                                    Ehrlichia chaffeensis AnkA was recently reported to be translocate
68 er membrane proteins (P28 OMPs) of Ehrlichia chaffeensis are encoded by a multigene family.
69 rect immunofluorescence assay with Ehrlichia chaffeensis as the antigenic substrate.
70                                    First, E. chaffeensis avoided stimulation of or repressed the tran
71                                           E. chaffeensis binding to and subsequent infection of monoc
72           A type IV secretion effector of E. chaffeensis blocks mitochondrion-mediated host cell apop
73                                           E. chaffeensis BolA bound to the promoters of genes encodin
74                                           E. chaffeensis bolA complemented a stress-sensitive E. coli
75   During the intracellular development of E. chaffeensis, both P28 and OMP-1F were expressed mostly i
76 y of the mmpA gene in E. canis and Ehrlichia chaffeensis but not in the human granulocytic ehrlichios
77 e reticulate forms of E. canis and Ehrlichia chaffeensis but was notably found on extracellular morul
78  Therefore, we tested the hypothesis that E. chaffeensis can infect adult Drosophila melanogaster.
79                                           E. chaffeensis cannot synthesize phosphatidylcholine or cho
80    The heat-sensitive component of viable E. chaffeensis cells was essential for these signaling even
81                           We assessed the E. chaffeensis clearance from the peritoneum, spleen, and l
82  mice infected with the tick cell-derived E. chaffeensis compared to DH82-grown bacteria.
83                                    Ehrlichia chaffeensis components that induce inflammation and the
84 se five proteins were similar in isolated E. chaffeensis-containing vacuoles and vacuole-free E. chaf
85 d, which was strikingly more prevalent in E. chaffeensis-containing vacuoles than in vacuole-free E.
86                                           E. chaffeensis CtrA bound to the promoters of late-stage tr
87                  Our results suggest that E. chaffeensis CtrA plays a role in co-ordinating developme
88  to be surface exposed), were detected in E. chaffeensis cultured in human monocytic leukemia THP-1 c
89 tein-processing enzymes were expressed by E. chaffeensis cultured in the human promyelocytic leukemia
90 e, suggesting that it is not expressed by E. chaffeensis cultured in THP-1 cells.
91 rongly with TRP120 in HeLa cells and with E. chaffeensis dense-cored morulae and areas adjacent to mo
92 se data suggest that the host response to E. chaffeensis depends on the source of the bacteria and th
93 late nutrient uptake during intracellular E. chaffeensis development at both temperatures.
94 aplasma phagocytophilum, Ehrlichia canis, E. chaffeensis, E. ewingii, Rickettsia rickettsii, R. conor
95 duce PCR products with DNA extracted from E. chaffeensis-, E. canis-, or E. phagocytophila-infected s
96          When the three human isolates of E. chaffeensis, each belonging to a different genogroup, ar
97     In this study, we determined that the E. chaffeensis effector TRP120 is posttranslationally modif
98                                           E. chaffeensis-EHRL-4-TRIM21 complexes caused significant u
99     With the recent discoveries of Ehrlichia chaffeensis, Ehrlichia ewingii, and "Borrelia lonestari,
100 horetic mobility shift assays revealed an E. chaffeensis-encoded protein that specifically bound to t
101 e obligate intracellular bacterium Ehrlichia chaffeensis, even when administered well after infection
102                                           E. chaffeensis experiences temperature changes during trans
103          These novel findings reveal that E. chaffeensis exploits canonical and noncanonical Wnt path
104  survive and replicate within phagocytes, E. chaffeensis exploits the host cell by modulating a numbe
105 st and the most comprehensive analysis of E. chaffeensis-expressed proteins.
106                                           E. chaffeensis expresses a sensor kinase, PleC, and a cogna
107 the first to examine genetic variation in E. chaffeensis from a nonhuman vertebrate host.
108 was downregulated prior to the release of E. chaffeensis from host THP-1 cells and was upregulated at
109 de variation than previously reported for E. chaffeensis from infected humans or ticks.
110 rum titers, and 3) in vitro cultivation of E chaffeensis from peripheral blood.
111 of tandem repeats, were characterized for E. chaffeensis from white-tailed deer (Odocoileus virginian
112 ngly different among the three strains of E. chaffeensis: gamma interferon, CCL5, CXCL1, CXCL2, CXCL7
113 utagenesis is a valuable tool in defining E. chaffeensis genes critical for its persistent growth in
114 n methods to investigate the functions of E. chaffeensis genes.
115                  In all, 278 genes of the E. chaffeensis genome were verified as functional genes.
116              Bioinfomatic analysis of the E. chaffeensis genome, however, predicted genes encoding 15
117  one-fourth of all predicted genes of the E. chaffeensis genome, validating that they are functionall
118 us transcription across the entire Ehrlichia chaffeensis genome.
119 e deletion and insertion mutations in the E. chaffeensis genome.
120 nses to another outer membrane protein of E. chaffeensis (GP120) showed similar temporal and quantita
121                         E. canis gp36 and E. chaffeensis gp47 were differentially expressed only on t
122 ptide repeat units from E. canis gp36 and E. chaffeensis gp47 were substantially less immunoreactive
123                 We recently reported that E. chaffeensis grown in tick cells expresses different prot
124 rmissive and nonpermissive conditions for E. chaffeensis growth.
125                                           E. chaffeensis has a multigene family of major outer membra
126 articular, despite its small genome size, E. chaffeensis has four tandem virB6 paralogs (virB6-1, -2,
127 ely related A. phagocytophilum and Ehrlichia chaffeensis have been shown to localize to the host cell
128                            Whereas Ehrlichia chaffeensis (HME) often causes meningoencephalitis, this
129 atment of E. chaffeensis with recombinant E. chaffeensis HtrA.
130                                    All 16 E. chaffeensis IFA-positive sera reacted with rP30.
131 rditis and multiorgan failure from Ehrlichia chaffeensis in a previously healthy adolescent is descri
132  muris strain that are closely related to E. chaffeensis in C57BL/6 mice.
133 This is the first demonstration of RNA of E. chaffeensis in infected blood and acquisition-fed male,
134                Entry and proliferation of E. chaffeensis in THP-1 cells were significantly blocked by
135 irB6 proteins and VirB9 were expressed by E. chaffeensis in THP-1 cells, and amounts of these five pr
136       Only one paralog was transcribed by E. chaffeensis in three developmental stages of Amblyomma a
137        Multiple lipoproteins expressed by E. chaffeensis in vitro and in vivo may play key roles in p
138 ial in preventing lysosomal fusion of the E. chaffeensis inclusion compartment.
139 treatment induced lysosomal fusion of the E. chaffeensis inclusion in a human monocytic leukemia cell
140                                           E. chaffeensis induced rapid tyrosine phosphorylation of PL
141                                  Although E. chaffeensis induces the generation of several cytokines
142  strongly with antibodies in sera from an E. chaffeensis-infected dog and human monocytotropic ehrlic
143 ofluorescent microscopy in the nucleus of E. chaffeensis-infected host cells and was detected in nucl
144                                      More E. chaffeensis-infected monocytes transmigrated than uninfe
145        Analyses of human sera showed that E. chaffeensis-infected patients also generated serological
146  from E. canis-infected dogs but not from E. chaffeensis-infected patients.
147                                           E. chaffeensis infection activated the phosphatidylinositol
148 hibitor, globomycin, was found to inhibit E. chaffeensis infection and lipoprotein processing in HL-6
149 1, which were strongly upregulated during E. chaffeensis infection and were also upregulated by direc
150 ate that huMAbs are capable of inhibiting E. chaffeensis infection by distinct effector mechanisms: e
151                              Diagnosis of E. chaffeensis infection by indirect immunofluorescence ass
152                                           E. chaffeensis infection did not result in dramatic changes
153                                           E. chaffeensis infection in a human monocyte cell line (THP
154                Previous studies of Ehrlichia chaffeensis infection in the mouse have demonstrated tha
155 ated the effects of c-di-GMP signaling on E. chaffeensis infection of the human monocytic cell line T
156 t susceptible SCID mice from fatal Ehrlichia chaffeensis infection, an observation that has been hypo
157                                     After E. chaffeensis infection, canonical and noncanonical Wnt si
158  knockouts, when challenged with a second E. chaffeensis infection.
159 ed molecular patterns and pathogenesis of E. chaffeensis infection.
160  macrophage-tropic bacterium, we assessed E. chaffeensis infections in three mouse strains with diffe
161 tyldimethysilyl)-c-di-GMP (CDGA) inhibits E. chaffeensis internalization into host cells by facilitat
162                                    Ehrlichia chaffeensis invades and survives in phagocytes by modula
163                  Our results suggest that E. chaffeensis invasion is regulated by c-di-GMP signaling,
164                                    Ehrlichia chaffeensis is an obligate intracellular bacterium that
165                                    Ehrlichia chaffeensis is an obligate intracellular bacterium that
166                                    Ehrlichia chaffeensis is an obligate intracellular bacterium that
167                                    Ehrlichia chaffeensis is an obligate, intracellular bacterium, tra
168                                    Ehrlichia chaffeensis is an obligately intracellular bacterium tha
169                                    Ehrlichia chaffeensis is an obligately intracellular bacterium tha
170                                    Ehrlichia chaffeensis is an obligately intracellular bacterium tha
171                                    Ehrlichia chaffeensis is an obligately intracellular gram-negative
172                                    Ehrlichia chaffeensis is an obligately intracellular Gram-negative
173                                    Ehrlichia chaffeensis is an obligatory intracellular and cholester
174                                    Ehrlichia chaffeensis is an obligatory intracellular bacterium of
175                                    Ehrlichia chaffeensis is an obligatory intracellular bacterium whi
176           We previously demonstrated that E. chaffeensis is capable of growing in Drosophila S2 cells
177                        We found here that E. chaffeensis is dependent on host glycerolipid biosynthes
178                    The data indicate that E. chaffeensis is exposed to the extracellular milieu durin
179 n this study suggest that the membrane of E. chaffeensis is very complex, having many expressed prote
180 obligatory intracellular pathogen, Ehrlichia chaffeensis, is characterized by formation of bacterial
181                                  Although E. chaffeensis lacks entire lipopolysaccharide and most pep
182 l cytokines and chemokines by leukocytes, E. chaffeensis lacks lipopolysaccharide and peptidoglycan.
183                          To study in vivo E. chaffeensis lipoprotein expression and host immune respo
184 ponses to E. chaffeensis lipoproteins, 13 E. chaffeensis lipoprotein genes were cloned into a mammali
185 n expression and host immune responses to E. chaffeensis lipoproteins, 13 E. chaffeensis lipoprotein
186                       Removal of PBP from E. chaffeensis lysate using penicillin affinity column and
187     Western blot analysis of E. canis and E. chaffeensis lysates with the anti-rMmpA serum resulted i
188                                      When E. chaffeensis matures into an infectious form, morulae bec
189                  Western blot analysis of E. chaffeensis membrane and soluble fractions using antibod
190    To investigate the surface proteins of E. chaffeensis, membrane-impermeable, cleavable Sulfo-NHS-S
191 n TRP32 and host targets localized to the E. chaffeensis morulae or in the host cell cytoplasm adjace
192  study, we assessed two clonally purified E. chaffeensis mutants with insertions within the genes Ech
193             [(32)P]c-di-GMP bound several E. chaffeensis native proteins and two E. chaffeensis recom
194  and was located downstream of two Ehrlichia chaffeensis omp-1 homologs and a decarboxylase gene (ubi
195 ptides are conserved between E. muris and E. chaffeensis OMP-19, and they elicited IFN-gamma producti
196  if the ELISA is positive), we identified E. chaffeensis or a serologically and antigenically similar
197 ta or Wisconsin were found not to be from E. chaffeensis or E. ewingii and instead to be caused by a
198 lar to those of human infection by Ehrlichia chaffeensis or EMLA.
199 acteria, such as Anaplasma platys, Ehrlichia chaffeensis, Orientia tsutsugamushi, and Rickettsia rick
200 l, membrane, and immunogenic proteomes of E. chaffeensis originating from macrophage and tick cell cu
201  different between mice infected with the E. chaffeensis originating from tick cells or macrophages.
202         Here we demonstrate that isolated E. chaffeensis outer membranes have porin activities, as de
203 P) with DNA sequencing revealed an Ehrlichia chaffeensis p200 interaction located within host promote
204           Homologous sequences for Ehrlichia chaffeensis p28 were compared to sequences of primers de
205 plasma phagocytophilum MSP2 (p44), Ehrlichia chaffeensis p28-OMP, Ehrlichia canis p30, and Ehrlichia
206         The cytokine-inducing activity by E. chaffeensis PBP provides novel insights into pathogen-as
207                               Recombinant E. chaffeensis PleD showed diguanylate cyclase activity as
208 e white-tailed deer agent, and additional E. chaffeensis-positive samples).
209                     The discovery of many E. chaffeensis proteins crucial for its continuous in vivo
210            The surface proteins of Ehrlichia chaffeensis provide an important interface for pathogen-
211 al E. chaffeensis native proteins and two E. chaffeensis recombinant I-site proteins, and this bindin
212 e sensitive than the PCR for detection of E. chaffeensis regardless of the nature of the specimens.
213 ule inhibitor had a significant impact on E. chaffeensis replication and recruitment of the TRP120-in
214 cleotide salvage pathway is essential for E. chaffeensis replication and that it may be important for
215 on of the uridine-cytidine kinase affects E. chaffeensis replication in human macrophages.
216 e obligate intracellular bacterium Ehrlichia chaffeensis resides in early endosome-like vacuoles and
217 ns from macrophage- and tick cell-derived E. chaffeensis, respectively.
218                               Addition of E. chaffeensis resulted in rapid increases in the level of
219 chia muris, a pathogen closely related to E. chaffeensis, resulted in anemia, thrombocytopenia, and a
220 use of template DNA extracted from Ehrlichia chaffeensis, Rickettsia rickettsii, and Bartonella hense
221 ae) two-hybrid analysis demonstrated that E. chaffeensis-secreted tandem repeat protein 120 (TRP120)
222                                    Ehrlichia chaffeensis secretes tandem repeat protein (TRP) effecto
223 es, identity of the primers to homologous E. chaffeensis sequences, and the availability of similarly
224 he members of the p28 multigene family of E. chaffeensis, sera from two beagle dogs experimentally in
225 n leukemia cell line THP-1 was exposed to E. chaffeensis, significant upregulation of IL-8, IL-1beta,
226                                           E. chaffeensis-specific cytotoxic T cells were not detected
227                                       The E. chaffeensis-specific immunoglobulin G response was consi
228 S6-Cd4(tm1Knw) mice also developed active E. chaffeensis-specific immunoglobulin G responses that wer
229 ay of 147,027 chromosome positions of the E. chaffeensis strain Arkansas genome.
230                                       The E. chaffeensis strain Wakulla induces diffuse hepatitis wit
231 equence polymorphisms in several genes in E. chaffeensis strains have been reported, global genomic d
232                                           E. chaffeensis strains induce strikingly variable inflammat
233 f host cytokine and chemokine profiles by E. chaffeensis strains underlies the distinct host liver di
234   Genomic DNA was extracted from purified E. chaffeensis strains Wakulla and Liberty, and comparative
235                                  When the E. chaffeensis strains were inoculated into severe combined
236 revealed distinct virulence phenotypes of E. chaffeensis strains with defined genome sequences.
237  responses and the in vivo replication of E. chaffeensis suggests that D. melanogaster is a suitable
238                        E. coli expressing E. chaffeensis SurE exhibited increased resistance to osmot
239 proteins provides novel insights into the E. chaffeensis surface and lays the foundation for rational
240                                Additional E. chaffeensis surface proteins detected were OMP85, hypoth
241                     The identification of E. chaffeensis surface-exposed proteins provides novel insi
242                                       The E. chaffeensis T1S effector TRP120 is conjugated to SUMO at
243          Recently we have determined that E. chaffeensis tandem repeat proteins (TRPs) are type 1 sec
244       Recently, molecular interactions of E. chaffeensis tandem repeat proteins 47 and 120 (TRP47 and
245 hat documents that insertion mutations in E. chaffeensis that cause attenuated growth confer protecti
246 ve proteins of Ehrlichia canis and Ehrlichia chaffeensis that have been characterized include a famil
247  tick-borne zoonoses, is caused by Ehrlichia chaffeensis that lacks endotoxin and peptidoglycan.
248 ive dogs, or when dogs were infected with E. chaffeensis, the animals developed delayed-type hypersen
249           Infection of humans with Ehrlichia chaffeensis, the etiologic agent of human monocytic ehrl
250                                    Ehrlichia chaffeensis, the etiologic agent of human monocytic ehrl
251                                    Ehrlichia chaffeensis, the etiologic agent of human monocytic ehrl
252 sma (Ehrlichia) phagocytophila and Ehrlichia chaffeensis, the etiologic agents of granulocytic and mo
253 t amplify the 200-bp target amplicon from E. chaffeensis, the human granulocytic ehrlichiosis agent,
254 found in an intravacuolar pathogen Ehrlichia chaffeensis, the tick-borne causative agent of human mon
255  two surface-expressed antigens of Ehrlichia chaffeensis, the variable-length PCR target (VLPT) and t
256        In the rickettsial pathogen Ehrlichia chaffeensis, the virBD genes are split into two operons,
257                                           E. chaffeensis, therefore, can recruit interacting signal-t
258 ndocytosis directs A. phagocytophilum and E. chaffeensis to an intracellular compartment secluded fro
259 d and compared in 12 clinical isolates of E. chaffeensis to determine allele variation.
260 mblyomma americanum ticks before or after E. chaffeensis transmission to naive dogs.
261 anonical and noncanonical Wnt pathways by E. chaffeensis TRP effectors stimulates phagocytosis and pr
262 r closely related interacting partners of E. chaffeensis TRP32, TRP47, and TRP120 demonstrate a molec
263                                           E. chaffeensis TRP75 and E. canis TRP95 were immunoprecipit
264 c C terminus of E. canis TRP95 but not in E. chaffeensis TRP75.
265  two-hybrid analysis demonstrated that an E. chaffeensis type 1 secretion system substrate, TRP32, in
266 afficked to autophagosomes induced by the E. chaffeensis type IV secretion system effector Etf-1, whi
267      The current study identified a novel E. chaffeensis ubiquitin ligase and revealed an important r
268                                   Second, E. chaffeensis up-regulated NF-kappaB and apoptosis inhibit
269 rly Ehrlichia) phagocytophilum and Ehrlichia chaffeensis, upon infection of humans, replicate in host
270                 We recently reported that E. chaffeensis utilizes a type 1 secretion (T1S) system to
271 lycoprotein (gp19) ortholog of the Ehrlichia chaffeensis variable-length PCR target (VLPT) protein.
272                  The results suggest that E. chaffeensis VirB9, the quadruple VirB6 proteins, and the
273       This is the most extensive study of E. chaffeensis VLPT and 120-kDa antigen gene genetic variat
274 l-terminal amino acid homology (59%) with E. chaffeensis VLPT and the same chromosomal location; howe
275 e same chromosomal location; however, the E. chaffeensis VLPT gene (594 bp) has tandem repeats that a
276           These results indicate that the E. chaffeensis Wakulla strain can induce inflammatory respo
277 ected, and EHRL-4-mediated degradation of E. chaffeensis was abrogated by the autophagy inhibitor 3-m
278 on of penicillin-binding protein (PBP) in E. chaffeensis was analyzed by reverse-transcription polyme
279                          Macrophage-grown E. chaffeensis was cleared in 2 weeks from the peritoneum,
280 ocytotropic ehrlichiosis caused by Ehrlichia chaffeensis was reported in 1987.
281                                           E. chaffeensis was sensitive to closantel, an HK inhibitor.
282                                    Ehrlichia chaffeensis was suspected as the causal agent but was no
283                      Expression of PBP by E. chaffeensis was upregulated during its intracellular lif
284                                        In E. chaffeensis we predicted three pairs of putative two-com
285 ria and that of macrophages infected with E. chaffeensis, we have identified few genes that are commo
286  sensitivities for A. phagocytophilum and E. chaffeensis were 93% and 84%, respectively, and specific
287 ter membrane protein gene (p28) of Ehrlichia chaffeensis were analyzed to determine the mechanism of
288  and virB6) of both A. phagocytophila and E. chaffeensis were arranged downstream from a sodB gene an
289                     Targeted mutations in E. chaffeensis were created to disrupt two genes, and also
290                  Antibodies reactive with E. chaffeensis were detected in 14 (67%) of the 21 PCR-posi
291  beagle dogs experimentally infected with E. chaffeensis were evaluated for the presence of specific
292 hromatography purified, and native PBP of E. chaffeensis were investigated for their ability to induc
293  five genes in both A. phagocytophila and E. chaffeensis were polycistronically transcribed and contr
294 ominant outer membrane proteins of Ehrlichia chaffeensis were transcribed in blood monocytes of dogs
295 phagocytophilum and one of B. microti and E. chaffeensis, which were confirmed by the LDTs.
296                  Although pretreatment of E. chaffeensis with CDGA did not reduce bacterial binding t
297 , and TRP120 was degraded by treatment of E. chaffeensis with recombinant E. chaffeensis HtrA.
298 d by Anaplasma phagocytophilum and Ehrlichia chaffeensis with the ompA, 17-kDa surface antigen gene,
299 ng regulates aggregation and sessility of E. chaffeensis within the inclusion through stabilization o
300  hypothesis that immune responses against E. chaffeensis would be different if the mice are challenge

 
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