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1 lication of a three-state "hopping" model of chain migration.
2 , and blockade of Reelin signaling increased chain migration.
3 r of D-1 that forms nonenzymatically by acyl chain migration.
4 e equivalent to that previously described as chain migration.
5  cell-cell interactions underlying homotypic chain migration.
6 ar protrusions and disrupted directional and chain migration.
7  blockade abrogates their pathology-directed chain migration, a developmentally relevant mode of tang
8 pus, moreover, likely contributes to ectopic chain migration and aberrant integration of newborn DGCs
9                   Oxidative stress increased chain migration and neurosphere size while decreasing ne
10 escent NSCs enhances proliferation, promotes chain migration and transmigration, and activates intrac
11 gly, lamellipodia formation and in vivo ENCC chain migration defects are rescued by inhibition of ROC
12                             We find that the chain migration flux and metal deposition rate are key f
13 NSCs expressing reelin did show a pattern of chain migration in the reeler mouse cortex.
14 o associated with neurons undergoing ectopic chain migration into the ischemic striatum and cerebral
15 ains under these conditions, suggesting that chain migration is characteristic of SVZ precursors.
16                                              Chain migration is inhibited by blocking the alpha 6beta
17                                 Thus, during chain migration, neural precursors moved associated with
18 he epileptic dentate gyrus leads to aberrant chain migration of DGC precursors.
19 ctopic DGCs arise as a result of an aberrant chain migration of neural progenitors.
20 nsive network of pathways for the tangential chain migration of neuronal precursors throughout the la
21 other cellular functions of NPCs, namely the chain migration of NPCs from neurospheres, perhaps as a
22  the adult human SVZ appears to be devoid of chain migration or large numbers of newly formed young n
23 generate neuroblasts that undergo restricted chain migration over an extended distance of more than 2
24  and initiate the transition from tangential chain migration to individual radial migration outside o
25  upon cleavage of the beta-agostic bonds and chain-migration via beta-H elimination.
26  confirmed using different markers, neuronal chain migration was not demonstrated, and no serial reco
27 ere morphology (neurosphere number, size and chain migration) were assessed in response to cytotoxic