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1 in the human challenge model of experimental chancroid.
2  bacterium causing the genital ulcer disease chancroid.
3 imiting the role of EIAs in the diagnosis of chancroid.
4  of the ulcerative lesions characteristic of chancroid.
5 conducted in the experimental swine model of chancroid.
6 in the human challenge model of experimental chancroid.
7 ally transmitted, genital ulcerative disease chancroid.
8 opment of an HgbA subunit vaccine to prevent chancroid.
9 ive bacterium that is the causative agent of chancroid.
10 imals lacked typical microscopic features of chancroid.
11  both swine and human experimental models of chancroid.
12 ive and initiate disease in a human model of chancroid.
13 ve agent of the sexually transmitted disease chancroid.
14 ttenuated in an experimental rabbit model of chancroid.
15 rulent in this animal model for experimental chancroid.
16 n the temperature-dependent rabbit model for chancroid.
17 e sexually transmitted genital ulcer disease chancroid.
18 in the temperature-dependent rabbit model of chancroid.
19 ture-dependent rabbit model for experimental chancroid.
20 thogen that causes the genital ulcer disease chancroid.
21 for use in determining the seroprevalence of chancroid.
22 ture-dependent rabbit model for experimental chancroid.
23 ture-dependent rabbit model for experimental chancroid.
24  Haemophilus ducreyi, the bacterial agent of chancroid.
25 include syndromic treatment for syphilis and chancroid.
26 as in human patients with naturally acquired chancroid.
27 for the tissue destruction characteristic of chancroid.
28 ly stages of the experimental human model of chancroid.
29 crosis and ulcer formation characteristic of chancroid.
30 ucreyi causes the genital ulcerative disease chancroid.
31  have implications for strategies to prevent chancroid.
32            By M-PCR, 33% of 196 patients had chancroid, 29% had syphilitic ulcers, and 10% had genita
33                                              Chancroid (80%), syphilis (8%), and genital herpes (8%)
34                   Haemophilus ducreyi causes chancroid, a genital ulcer disease that facilitates the
35  Haemophilus ducreyi, the etiologic agent of chancroid, a genital ulcer disease, produces a cell-asso
36                   Haemophilus ducreyi causes chancroid, a genital ulcer disease.
37 aemophilus ducreyi is the etiologic agent of chancroid, a genital ulcer disease.
38                   Haemophilus ducreyi causes chancroid, a sexually transmitted cutaneous genital ulce
39                                              Chancroid, a sexually transmitted disease caused by Haem
40 aemophilus ducreyi is the etiologic agent of chancroid, a sexually transmitted disease that increases
41 eyi is a Gram-negative bacterium that causes chancroid, a sexually transmitted disease.
42 aemophilus ducreyi is the etiologic agent of chancroid, a sexually transmitted genital ulcer disease
43                   Haemophilus ducreyi causes chancroid, a sexually transmitted genital ulcer disease
44 aemophilus ducreyi is the etiologic agent of chancroid, a sexually transmitted genital ulcer disease.
45 aemophilus ducreyi is the causative agent of chancroid, a sexually transmitted ulcerative disease.
46 terium that causes the genital ulcer disease chancroid, activates inflammasomes in experimentally inf
47 ic agent of the sexually transmitted disease chancroid, an ulcerative condition implicated in increas
48 reyi causes the sexually transmitted disease chancroid and a chronic limb ulceration syndrome in chil
49                   Haemophilus ducreyi causes chancroid and is a major cause of cutaneous ulcers in ch
50 emolysin is important in the pathogenesis of chancroid and may contribute to ulcer formation, invasio
51             Serum samples from patients with chancroid and other genital ulcer diseases and from norm
52 nsitivity of clinical diagnosis of syphilis, chancroid, and genital herpes was 93%, 53%, and 0% and s
53 n sexually transmitted genital ulcer disease chancroid are characterized by the presence of intraepid
54                                 Syphilis and chancroid are common etiologies of GUD in Malawi.
55 larly for genital ulcer where HSV-2 replaced chancroid as the primary cause.
56 perimental HgbA vaccine efficiently prevents chancroid, as determined by several parameters.
57 iological agent of the genital ulcer disease chancroid, binds extracellular matrix components, includ
58  content, and antibiotic susceptibility, the chancroid cases in New Orleans were found to be due to a
59  Haemophilus ducreyi, the etiologic agent of chancroid, chemically and immunologically resembles huma
60               Pooled sera from patients with chancroid contain antibodies to a Haemophilus ducreyi an
61 n the temperature-dependent rabbit model for chancroid, did not produce either LspA1 or LspA2 in vitr
62  sp. antigens, suggesting that patients with chancroid do not develop an H. ducreyi-specific antibody
63  Haemophilus ducreyi, the etiologic agent of chancroid, expresses lipooligosaccharides (LOS) that are
64  Haemophilus ducreyi, the etiologic agent of chancroid, expresses variants of several key virulence f
65           Azithromycin prevents experimental chancroid for nearly 2 months.
66  Haemophilus ducreyi, the causative agent of chancroid, harbors a homolog of csrA.
67   As syphilis rates have increased globally, chancroid has dramatically declined as a cause of genita
68 aemophilus ducreyi, the etiological agent of chancroid, has a strict requirement for heme, which it a
69  Haemophilus ducreyi, the etiologic agent of chancroid, has an obligate requirement for heme.
70 e sexually transmitted genital ulcer disease chancroid, has been shown to associate with dermal colla
71  Haemophilus ducreyi, the etiologic agent of chancroid, has been shown to form microcolonies when cul
72 ic agent of the sexually transmitted disease chancroid, has been shown to inhibit phagocytosis of bot
73  Haemophilus ducreyi, the causative agent of chancroid, has homologs of both relA and spoT, which pri
74 reyi causes the sexually transmitted disease chancroid in adults and cutaneous ulcers in children.
75 eviously we demonstrated in a swine model of chancroid infection that an HgbA vaccine conferred compl
76      Accordingly, we used the swine model of chancroid infection to examine the impact of multiple in
77 mal and immune cell-depleted swine models of chancroid infection.
78 of the lipooligosaccharide (LOS) molecule in chancroid infections, we constructed mutants defective i
79 r, neither culture nor PCR is feasible where chancroid is endemic.
80  this a valuable bedside tool in areas where chancroid is endemic.
81                       The etiologic agent of chancroid is Haemophilus ducreyi.
82                      These data suggest that chancroid is prevalent in some US cities and that person
83  Haemophilus ducreyi, the causative agent of chancroid, is highly resistant to the complement-mediate
84 oids killing and clearance by macrophages in chancroid lesions and inguinal lymph nodes.
85                                              Chancroid lesions are deep necrotic ulcers with an immun
86 e keratinocyte cytopathology associated with chancroid lesions in the pig.
87                    Thus, prompt treatment of chancroid may abrogate increases in viral replication as
88                  Neither naturally occurring chancroid nor experimental infection with H. ducreyi res
89 on EIA had a sensitivity and specificity for chancroid of 53% and 71%, while the LOS EIA had a sensit
90 ined from healthy subjects and patients with chancroid or other genital ulcer diseases contained anti
91 esions had histology typical of experimental chancroid or were culture positive.
92 e the importance of keratinocyte adhesion in chancroid pathogenesis so we tested the wild-type and ds
93 strains of H. ducreyi in our swine models of chancroid pathogenesis.
94 he cause of the sexually transmitted disease chancroid produces a lipooligosaccharide (LOS) containin
95  Haemophilus ducreyi, the causative agent of chancroid, produces a hemolysin, whose role in virulence
96  Haemophilus ducreyi, the causative agent of chancroid, produces a lipooligosaccharide (LOS) which te
97 reyi, which causes the genital ulcer disease chancroid, requires high basal levels of the 60-kDa heat
98 Haemophilus ducreyi (the infectious agent of chancroid) responsible for the activation and attachment
99 ed-type hypersensitivity (DTH) and bacterial chancroid skin lesions express both CCR4 and CLA, but on
100 n the temperature-dependent rabbit model for chancroid, the lspA1 lspA2 double mutant was substantial
101      Although the subjects had no history of chancroid, their immune responses resembled delayed-type
102 f Haemophilus ducreyi in the pathogenesis of chancroid, Tn916 mutants of H. ducreyi 35000 defective i
103  between host and bacterial contributions to chancroid ulcer formation.
104 at the host immune response was required for chancroid ulceration, while bacterial products were at l
105                                              Chancroid ulcerations are difficult to distinguish from
106 pite the presence of these phagocytic cells, chancroid ulcers can persist for months and live H. ducr
107 cutaneous pathogen, H. ducreyi is present in chancroid ulcers that are characterized by extensive neu
108 rotection in the experimental swine model of chancroid, using either Freund's or monophosphoryl lipid
109 rum specimens from patients with and without chancroid were assayed to determine optimum sensitivity
110 M-PCR findings; none of 5 suspected cases of chancroid were positive by M-PCR and none of 1 for syphi
111                             Two outbreaks of chancroid were recently studied in the United States, on
112 ultivariate analysis indicated that men with chancroid were significantly more likely than male patie
113 ture-dependent rabbit model for experimental chancroid, whereas another H. ducreyi mutant with inacti
114 reyi causes the sexually transmitted disease chancroid, which facilitates HIV-1 transmission.
115                   Haemophilus ducreyi causes chancroid, which facilitates transmission of human immun

 
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