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1 in meiotic chromosome cohesion, as a spindle checkpoint component.
2 with its regulatory protein Bud14 as a novel checkpoint component.
3 ts cells in metaphase independently of other checkpoint components.
4 her B-type cyclins in the absence of spindle checkpoint components.
5 fferential genetic interactions with spindle checkpoint components.
6 or by inactivating a subset of recombination checkpoint components.
7 netochores and the subsequent recruitment of checkpoint components.
8  cell division and death by associating with checkpoint components, although their mode of regulation
9                             Spindle assembly checkpoint components and a checkpoint-independent funct
10 sion is reduced in the absence of DNA damage checkpoint components and nonhomologous end-joining (NHE
11                  Unusually, spindle assembly checkpoint components are required for the Topo II check
12 and a portion of Mad2, all essential mitotic-checkpoint components, are stably bound elements of unat
13 d kinetochore; consequently, accumulation of checkpoint components at spindle poles increases markedl
14 localizes and genetically interacts with the checkpoint components Aurora B and ANCHR, and the abscis
15 -CIN) we down-regulated the spindle assembly checkpoint component BUB1 and the mitotic cohesin SMC1A,
16  isolated a Xenopus homologue of the spindle checkpoint component Bub1, and investigated its role in
17 ion, kinetochore localization of the spindle checkpoint components BubR1, Mad2, and Cenp-E is diminis
18 that PP1(Dis2)-mediated dephosphorylation of checkpoint components forms a novel spindle checkpoint s
19    We show that san-1 functions as a spindle checkpoint component in C. elegans.
20             We have isolated mad2, a spindle checkpoint component in fission yeast, and shown that ma
21  limit APC/C activity when kinetochores lack checkpoint components in early mitosis.
22                               To ask how the checkpoint components induce metaphase arrest, we constr
23                                        A key checkpoint component is the mitotic arrest-deficient pro
24 asm, yet kinetochore localization of spindle checkpoint components is required for proper recovery fr
25  mitotic arrest by dominant-negative spindle checkpoint components led to a failure in completion of
26            Reduced expression of the mitotic checkpoint component Mad1 causes aneuploidy and promotes
27  mitotic regulator cyclin B1 and the spindle checkpoint component Mad1 was independently described by
28                                          The checkpoint components Mad1 and Mad2 associate with unatt
29 ad2p tightly associates with another spindle checkpoint component, Mad1p.
30                                              Checkpoint component Mad2 localizes selectively to unatt
31 ) mice by reducing levels of another mitotic checkpoint component, Mad2, is now shown to result in el
32        We show that the roles of two spindle checkpoint components, Mad2 and Mad3, differ in meiosis
33  synthesis of proteins including the mitotic checkpoint component MAD2L1 was negatively regulated.
34 r data indicate that some well-characterized checkpoint components maintain minisatellite stability i
35 lacking functional SAN-1 or a second spindle checkpoint component, MDF-2, failed to arrest the cell c
36 he function of ribonucleotide reductase as a checkpoint component of S progression was studied in yea
37 he checkpoint, apparently by moving critical checkpoint components off kinetochores.
38               The history of Bub1, a spindle checkpoint component, reveals a spectacular case of para
39 estore the checkpoint, suggesting that other checkpoint component(s) were depleted from the extract t
40        Our results revealed that a subset of checkpoint components, specifically MRC1, CSM3, TOF1, DD
41    Experimental observations have shown that checkpoint components stream from attached kinetochores
42 5CDC/hCdc20, a protein known to link spindle checkpoint components such as MAD2 to anaphase promoting
43      The protein kinase Bub1 is an essential checkpoint component that resides at kinetochores during
44                                Mad2 is a key checkpoint component that undergoes conformational activ
45 controlled specifically by Bub2p, one of the checkpoint components that is conserved in fission yeast
46 K-p90(Rsk) cascade utilizes spindle-assembly-checkpoint components to effect metaphase arrest.
47 hat hPer2 directly acts on the hp53 node, as checkpoint components upstream of hp53 remained active i
48 /Rad17/Mec3 complex and Rad24 are DNA damage checkpoint components with limited homology to replicati