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1 ing of changes in a single cell induced by a chemical agent.
2 teins are extremely stable to heat, acid and chemical agents.
3 attle and chickens, and also to carcinogenic chemical agents.
4 y than wild-type controls in response to the chemical agents.
5 portunities for concentrating and separating chemical agents.
6 microbial pathogens requires alternatives to chemical agents.
7 ples whose stiffnesses were manipulated with chemical agents.
8  this is accomplished with surface-modifying chemical agents.
9 ting the physiologically relevant targets of chemical agents.
10  a wide spectrum of biological, physical and chemical agents.
11 s anthropogenic noise, artificial lights and chemical agents.
12 25(OH)D in the therapeutic use against these chemical agents.
13  or under the action of various physical and chemical agents.
14 ks, it is reversible and can be modulated by chemical agents.
15 o identify the macromolecular targets of new chemical agents.
16 ave so far been identified are determined by chemical agents.
17            It is stable against a variety of chemical agents.
18 nmentally benign production of key synthetic chemical agents.
19  resistant to drought and other physical and chemical agents.
20 robust cell wall that is impermeable to many chemical agents.
21 e (+1.25 V) to release precise quantities of chemical agents.
22  for the metabolism of nutrients, drugs, and chemical agents.
23 y disrupting the partnering with peptide and chemical agents.
24 in their biochemical optima and responses to chemical agents.
25 s frequently damaged by various physical and chemical agents.
26 e the potential health impacts of individual chemical agents?
27 of IFI16 reduced lytic gene induction by the chemical agent 12-O-tetradecoylphorbol-13-acetate (TPA).
28                   Here we identify the first chemical agent able to act upstream of the proteasome to
29 mechanisms have been proposed: movement of a chemical agent and a pressure wave through the vasculatu
30 es practical guidance for selecting suitable chemical agents and conditions.
31 ain sequences important for gene response to chemical agents and fungal attack.
32 it a target for the rational design of novel chemical agents and genetic modifications that improve p
33  macronucleus in response to DSBs induced by chemical agents and in the micronucleus during prophase
34 rium appears to be relatively insensitive to chemical agents and inflammatory mediators, in contrast
35  major DNA repair pathway for SSB induced by chemical agents and ionizing radiation, we initially ass
36 ion was measured after cells were exposed to chemical agents and radiation and after HO endonuclease
37 in sampling hundreds of chemicals, including chemical agents and their signatures, pharmaceutics, met
38 rrent methods require proteolytic enzymes or chemical agents and typically a second reagent to discon
39 nduced by endonucleases, ionizing radiation, chemical agents, and mechanical forces or by replication
40 P expression sensitizes cells to a number of chemical agents, and mutations at predicted channel-faci
41 ces obviate bacterial resistance common with chemical agents, and therefore a robust and stable means
42 lucidate the roles of shear stress, specific chemical agents, and thermal fluctuations in cytoskeleto
43 aptation under the effect of physical and/or chemical agents, and thus function as adaptive polymers
44                 A wide variety of antifungal chemical agents are available; however, the side effects
45 umors by inducing the viral lytic cycle with chemical agents are hindered by inefficient responses to
46 city of 97%) in assessing whether particular chemical agents are irritating or not for human skin.
47                     Endogenous and exogenous chemical agents are known to compromise the integrity of
48                                        These chemical agents are known to confer protection on heart
49 the application of antidepressants and other chemical agents as well as physical exercise to engrafti
50 ne that does not require the presence of the chemical agent before or during UVR exposure.
51 dged to destroy approximately 25,000 tons of chemical agents by the end of the decade.
52 esult if, using this accumulating knowledge, chemical agents can be developed that can enhance repair
53                                      Certain chemical agents can force some tumor cells to resume the
54             However, DNA structures bound to chemical agents cannot be PCR-amplified, and therefore a
55 ied to CNVs monitoring in cells exposed to a chemical agent capable of deletion induction, such as ci
56 tudy, we have demonstrated that a variety of chemical agents capable of denaturing or dissociating pr
57 y neurons grown in culture to the excitatory chemical agent capsaicin was examined.
58 control elements, and a unique modulation by chemical agents, cytokines, and serum-factors.
59 ilayer, deformable microfluidic channels for chemical agent delivery, electrical interconnects throug
60 he wasabi receptor) is a detector of noxious chemical agents encountered in our environment or produc
61 li (errors in DNA replication, UV radiation, chemical agents, etc.) is normally detected by special c
62                 Physical stressors (heat) or chemical agents (ethanol) can render virions noninfectio
63 s were examined, incorporating variations in chemical agents, exposure durations, and conditions.
64 ould facilitate development of site-directed chemical agents for bioimaging or therapeutic applicatio
65 our newly discovered p18SMIs represent novel chemical agents for murine and human HSCs ex vivo expans
66 col that does not rely on the use of glue or chemical agents for muscle and tendon fiber immobilizati
67                                       Use of chemical agents for scar homogenization represents an al
68 -renewal and may help develop more effective chemical agents for therapeutic expansion of HSC.
69                      Metabolites and certain chemical agents (for example methyl methanesulfonate) ca
70 uses extreme sensitivity to the [PSI]-curing chemical agent guanidine hydrochloride.
71 l residues of proteins with a broad range of chemical agents has been proposed to be dependent on the
72                                     Although chemical agents have long been the primary method for in
73 ed by the action of a variety of physical or chemical agents, have led to the definition of a folded
74 on of these metal-peptide interactions using chemical agents holds considerable promise as a therapeu
75 sis of mass flow in the xylem transporting a chemical agent, however, is able to reproduce experiment
76                  Cases of ocular exposure to chemical agents in children younger than 18 years during
77  threshold VC exposure and the role of other chemical agents in TASH are as yet unknown.
78              These questions, as well as how chemical agents, including therapeutic substances, might
79   As the public pressure to limit the use of chemical agents increases, the control of thinning becom
80 al route for a "quick fix." Thus, the use of chemical agents is on the rise.
81                               Conditions and chemical agents known to activate ER stress response (ER
82 ims from severe sunburn or exposure to other chemical agents known to trigger the p38 pathway.
83 wn to impart cellular resistance to multiple chemical agents, many of which are commonly used in canc
84 d previous research has suggested that these chemical agents may disrupt circulating levels of total
85 estriction enzyme Pvu II or the DNA-damaging chemical agents methyl methanesulfonate (MMS) or 4-nitro
86 he combined actions of ricin holotoxin and a chemical agent, N,N'-dimethylethylenediamine.
87                          Sulfur mustard is a chemical agent of high military and terroristic signific
88 data, could be used to predict the effect of chemical agents on mobility, adhesion, and proliferation
89 sment of the effects of pH, temperature, and chemical agents on the PPO activity as well as character
90  the presence and concentration of different chemical agents or drugs in preimplantation tissues.
91 ly, the reduction of GO has relied on either chemical agents or high temperature treatment.
92                          Introduction of new chemical agents or reactive processes can initiate compl
93     The blockade of NF-kappaB activation via chemical agents or the overexpression of the mutant form
94 noflagellates is evoked by a factor (i.e., a chemical agent) present in a homogenate of host tissue.
95  imaging routine for distinguishing multiple chemical agents, regardless of contrast similarity.
96 ently alert the cell to an array of reactive chemical agents, regardless of their structure.
97    The increase in excitability is caused by chemical agents released at the site of injury.
98  hypochlorous acid is hypothesized to be the chemical agent responsible for the observed (destruction
99                 High resolution mapping with chemical agents selective for non-B DNA provides evidenc
100 vironment and that only a minute fraction of chemical agents share the ability to act on this vulnera
101 heroids and evaluation of skin toxicity with chemical agents showed a correlation with clinical resul
102 elp repair damage to DNA caused by exogenous chemical agents such as chloroacetaldehyde.
103 tiate in vivo by treatment with a variety of chemical agents such as N, N-hexamethylene bisacetamide
104 concept of enhancing antibody sensitivity by chemical agents that affect the structural stability of
105 onoxide-releasing molecules (photoCORMs) are chemical agents that allow for precise spatial and tempo
106                   A number of detergents and chemical agents that are capable of breaking ionic and h
107 s susceptible to damage by a wide variety of chemical agents that are generated either as byproducts
108 be readily manipulated, including the use of chemical agents that cannot be safely administered to li
109 duced by ultraviolet irradiation and various chemical agents that cause bulky adducts.
110 he development of aneuploidy when exposed to chemical agents that disrupt the mitotic spindle and pre
111                High-throughput screening for chemical agents that exert greater inhibitory effects ag
112            Consequently, there is a need for chemical agents that increase DNA damage by ionizing rad
113         Several environmental conditions and chemical agents that influence the expression of VEGF ca
114                                              Chemical agents that inhibit infected cells from enterin
115 t can therefore be used for the screening of chemical agents that may have gamma-globin gene inducibi
116 nting challenge because of the wide array of chemical agents that must be screened.
117  modulus, and resistance to heat, flame, and chemical agents that normally degrade conventional macro
118                      Typically, control uses chemical agents that often are ineffective, harmful to n
119 ratumor expression of KDM5B and screened for chemical agents that phenocopy this effect.
120 ey advances in the design and development of chemical agents that show promise in blocking the action
121 Tosis after exposure to diverse microbes and chemical agents that stimulate ion flux.
122 e in vitro pharmacological evaluation of new chemical agents that target bacterial transcription.
123          The focus of this Perspective is on chemical agents that target the most common mechanisms o
124 ed for targeted delivery of radioisotopes or chemical agents to diseased tissues.
125 Moreover, our results suggest that designing chemical agents to disrupt Rhl-Pqs crosstalk could be an
126 lar ion concentrations, and the screening of chemical agents to identify molecules targeting ion tran
127  is the use of specific natural or synthetic chemical agents to reverse, suppress, or prevent progres
128  pyelonephritis by delivering bacteriostatic chemical agents to the lower urinary system.
129 with the modes of application of anti-plaque chemical agents to their interdental and subgingival sit
130 throughput approaches evaluating toxicity of chemical agents toward bacteria typically rely on optica
131                    We therefore suggest that chemical agents transported by mass flow within the xyle
132 ated release were dependent upon the type of chemical agent used to evoke the release.
133 ther of toxicology Paracelsus (1493-1541) on chemical agents used as therapeutics, "the dose makes th
134                                  The primary chemical agents used for controlling mosquitoes are inse
135 bon monoxide-releasing molecules (CORMs) are chemical agents used to administer CO as an endogenous,
136                            As representative chemical agents, we selected a chemotherapeutic drug (ci
137 roblem associated with CF and to identifying chemical agents, which correct this problem.
138                                       Unlike chemical agents, which typically lead to violent disease
139             They merge the toxicity of known chemical agents with the specificity of monoclonal antib
140 biological targets or signaling pathways and chemical agents, with a focus on small molecules, to ach

 
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