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1 might be mediated by a diffusible but labile chemical mediator.
2 lose both itch and pain reactions to potent chemical mediators.
3 d by distinct cellular events and endogenous chemical mediators.
4 nputs from the site of injury and release of chemical mediators.
5 thelial cell response to changes of external chemical mediators.
6 via alteration of the biosynthesis of local chemical mediators.
7 accompanied by release of a wide variety of chemical mediators.
8 een studied mainly at the level of cells and chemical mediators.
9 agents that block or inhibit proinflammatory chemical mediators.
14 r, our data provide strong evidence that the chemical mediator CCL2 is released from tumor cells and
15 sues is regulated by the local production of chemical mediators collectively referred to as chemoattr
17 Given their potent bioactions, these new chemical mediator families were termed resolvins and pro
19 to homeostasis is in part due to endogenous chemical mediators, for example, lipoxins, resolvins, an
25 portant role in the metabolism of endogenous chemical mediators involved in the regulation of blood p
26 , we show that consumption or degradation of chemical mediators moderates interaction strengths and p
27 al activity often relies critically on these chemical mediators, obscuring the contribution of voltag
29 rbonate (ONOOCO(2)(-)), a macrophage-derived chemical mediator of inflammation, and the classical one
33 Consequently, tannins may be considered as chemical mediators of tolerance rather than resistance.
34 s to degranulation and consequent release of chemical mediators on rat basophilic leukemia cells.
36 city and in-situ bacterial detection with no chemical mediators, represents a versatile approach for
38 To identify and measure recently described chemical mediators, termed specialized pro-resolving med
40 and finds the reactive oxygen species a key chemical mediator that participates in or facilitates ne
42 is involved in the metabolism of endogenous chemical mediators that play an important role in blood
43 is involved in the metabolism of endogenous chemical mediators that play an important role in inflam
44 ism of arachidonic acid epoxides, endogenous chemical mediators that play important roles in blood pr
45 he site of injury, where these cells release chemical mediators that promote inflammatory processes i
46 when self-limited, produces a superfamily of chemical mediators that stimulate resolution of the resp