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1 sents the first successful purification of a chemoattractant receptor.
2 MP did inhibit the response to formylpeptide chemoattractant receptor.
3 s, we disrupted a gene that encodes a single chemoattractant receptor.
4 CL12 (SDF-1) but does not act as a classical chemoattractant receptor.
5 gand binding pocket of an eicosanoid-binding chemoattractant receptor.
6 otrimeric G protein that couples to the cAMP chemoattractant receptors.
7 to the lung is mediated by G alpha i-coupled chemoattractant receptors.
8 o the lung is an active process dependent on chemoattractant receptors.
9 expression of 'tissue-specific' adhesion and chemoattractant receptors.
10 in transcriptional regulation downstream of chemoattractant receptors.
11 r of seven-membrane span receptors including chemoattractant receptors.
12 of intracellular calcium, and exocytosis by chemoattractant receptors.
13 ory effect was specific for some but not all chemoattractant receptors.
14 yotic cells does not require polarization of chemoattractant receptors.
15 ed from murine eosinophils to identify novel chemoattractant receptors.
16 one of the most thoroughly studied leukocyte chemoattractant receptors.
17 gradient-induced asymmetric distribution of chemoattractant receptors.
18 the family of leukocyte, G protein-coupled, chemoattractant receptors.
19 human L-selectin along with human leukocyte chemoattractant receptors.
20 ty of G alpha(i2) protein that is coupled to chemoattractant receptors.
21 d fibrosarcoma cells downstream of activated chemoattractant receptors.
22 al RBL cells or cells transfected with other chemoattractant receptors.
23 seudopod region induced by G-protein coupled chemoattractant receptors.
30 odes a 130-kD cytosolic protein required for chemoattractant receptor and G protein-mediated activati
31 dification of the cells for their display of chemoattractant receptors and endothelial-cell-binding m
33 involved in PLC beta2 activation by the two chemoattractant receptors and suggest that in COS-7 cell
34 an be phosphorylated following engagement of chemoattractant receptors and suggest that this may be a
36 differences in mRNA for adhesion molecules, chemoattractant receptors, and cytokines compared with o
37 id and selective cross-inactivation of other chemoattractant receptors, and suggest that FPR1 is able
38 tivation and heterologous desensitization of chemoattractant receptors are involved in the inhibition
39 ts at the leading edge of a cell even though chemoattractant receptors are uniformly distributed on t
41 Thus neutrophils do not actively concentrate chemoattractant receptors at the leading edge during che
42 These observations elucidate a novel role of chemoattractant receptor, BLT-1, in promoting monocyte t
43 The G protein-coupled 7 transmembrane (STM) chemoattractant receptors can be inactivated by heterolo
45 lpha and beta chemokine as well as classical chemoattractant receptors can trigger robust adhesion as
46 Unlike most other cellular proteins, the chemoattractant receptor, cAR1, of Dictyostelium is resi
48 hat 6CK/SLC is an agonist for the lymphocyte chemoattractant receptor, CCR7 (EBI-1, BLR-2), previousl
49 urchin species-specific differences in sperm chemoattractant-receptor characteristics we show that re
50 ere enriched in their expression of specific chemoattractant receptors compared with peripheral blood
51 tor shares greatest sequence similarity with chemoattractant receptors compared with prostanoid recep
56 l DC2 (cDC2), we discuss the contribution of chemoattractant receptors (EBI2 or GPR183, S1PR1, and CC
57 mediated by CC chemokine receptor (CCR)9, a chemoattractant receptor expressed at high levels by ess
59 ide receptor 1 (FPR1) is a G protein-coupled chemoattractant receptor expressed mainly on leukocytes.
60 es calcium signaling of a discrete subset of chemoattractant receptors expressed by human leukocytes.
61 n atopic asthmatics to define the pattern of chemoattractant receptor expression on recruited T cells
63 n of CMKLR1 (chemokine-like receptor 1), the chemoattractant receptor for chemerin, and was abrogated
65 ch requires that leukocytes display multiple chemoattractant receptors for successful homing and prov
67 bits chemotaxis of neutrophils by preventing chemoattractant receptors from activating trimeric G pro
68 o IL-8 does not appear to be due to impaired chemoattractant receptor function, as the number of IL-8
74 stinal plasma cells sense oxysterols via the chemoattractant receptor GPR183 and couple their tissue
76 eosinophils (both resting and activated) and chemoattractant receptor homolog expressed on Th2 cells
77 , D-type prostanoid (DP) and, in particular, chemoattractant receptor homologous molecule expressed o
80 through 2 GPCRs, d-type prostanoid 1 and the chemoattractant receptor homologous molecule expressed o
81 ly mediated by the prostaglandin D2 receptor chemoattractant receptor homologous molecule on T(H)2 ce
83 ecules such as thymic stromal lymphopoietin, chemoattractant-receptor homologous molecule expressed o
85 established that interaction of PGD(2) with chemoattractant receptor- homologous molecule expressed
86 4 and IL-5) and chemotactic receptors (CCR4, chemoattractant receptor-homologous molecule expressed o
88 oid receptors (DPs) DP1 and DP2 (also called chemoattractant receptor-homologous molecule expressed o
91 hymic stromal lymphopoietin, as well as oral chemoattractant receptor-homologous molecule expressed o
92 of CD203c(bright), CD63(+), and CD107a(+) on chemoattractant receptor-homologous molecule expressed o
93 T2, the receptor for the cytokine IL-33, and chemoattractant receptor-homologous molecule expressed o
94 D prostanoid receptor 2 (DP2; also known as chemoattractant receptor-homologous molecule expressed o
95 as tested by using TM30089, an antagonist of chemoattractant receptor-homologous molecule expressed o
98 on 3 G protein-coupled receptors, including chemoattractant receptor-homologous molecule expressed o
99 ses through a high-affinity interaction with chemoattractant receptor-homologous molecule expressed o
100 mbrane-spanning/G protein-coupled receptors, chemoattractant receptor-homologous molecule expressed o
102 n eosinophil/mast cell densities, density of chemoattractant receptor-homologous molecule expressed o
103 ndin D2 (PGD2) acting at the CRTH2 receptor (chemoattractant receptor-homologous molecule expressed o
107 SCR1 bound to and physically interacted with chemoattractant receptor-homologous molecule(CRTH2), whi
108 bitors that suppress inflammation (targeting chemoattractant receptor-homologous molecules expressed
109 roperties of a series of clinically relevant chemoattractant receptor-homologous molecules expressed
111 To study the biology of individual leukocyte chemoattractant receptors in a defined lymphoid environm
112 tissue, we performed expression analyses of chemoattractant receptors in a related family that inclu
113 ctions resulting from Galpha(16) coupling of chemoattractant receptors in a transfected cell model sy
114 view recent advances in our understanding of chemoattractant receptors in disease pathogenesis, with
115 G-protein-coupled receptors that function as chemoattractant receptors in innate immune responses.
116 s, LPS selectively modulates the function of chemoattractant receptors in microglia and may promote h
118 mphocyte trafficking, but the involvement of chemoattractant receptors in the physiologic recruitment
119 or CCR6 and CXCR3, in conjunction with other chemoattractant receptors, in the recruitment of inflamm
120 eutrophils infiltrate the joint via multiple chemoattractant receptors, including the leukotriene B(4
122 inhibitor, staurosporine, completely blocked chemoattractant receptor-induced phosphorylation of L-se
123 is study, we have investigated whether human chemoattractant receptors internalize via clathrin-coate
125 tion of siRNA to silence a G protein-coupled chemoattractant receptor involved in inflammation and su
129 eraction with D prostanoid receptor (DP) and chemoattractant receptor-like molecule expressed on Th2
130 or receptors, D prostanoid receptor (DP) and chemoattractant receptor-like molecule on the Th2 cell.
131 phosphorylation in cross-desensitization of chemoattractant receptors, M2CXCR1 was coexpressed with
132 is that interaction between autoantigens and chemoattractant receptors may be an important step in th
134 zed that signaling through G protein-coupled chemoattractant receptors may stimulate cytokine product
135 ver, the regulatory role of TRPC channels in chemoattractant receptor-mediated signaling has not yet
136 ymmetric macromolecular complex formation of chemoattractant receptors mediates gradient sensing and
137 ress this issue, we purified cAR1, the major chemoattractant receptor of Dictyostelium discoideum by
138 ere that elimination of phosphorylation of a chemoattractant receptor of Dictyostelium, either by sit
139 ormyl peptide receptor 2 (FPR2), a classical chemoattractant receptor of G-protein-coupled receptors,
141 g with HIV-1 fusion cofactors, downregulates chemoattractant receptors on monocytes by a CD4-dependen
143 20, implying the contribution of alternative chemoattractant receptors other than spingosine 1-phosph
144 he apparent functional redundancy with other chemoattractant-receptor pairs in vitro, LTB(4) and BLTR
156 rophil migration requires B(2) integrins and chemoattractant receptor signaling for motility and dire
157 pear to be critical downstream components of chemoattractant receptor signaling in human neutrophils,
159 sensitivity to background signals, prolonged chemoattractant receptor signaling, and inappropriate CX
162 or chemotaxis and phagocytosis indicate that chemoattractant receptor-signaling is not essential for
164 esponding to fMLF, a peptide agonist for two chemoattractant receptor subtypes, formyl peptide recept
165 egulation of Th1/2 effector tissue-targeting chemoattractant receptors such as CCR4, CCR5, CXCR6, and
166 ollowing the activation of G-protein coupled chemoattractant receptors such as CXCR2 whose signaling
168 B4, did not inhibit the responses of peptide chemoattractant receptors, suggesting distinct signaling
170 Thus, the 5-HIAA-GPR35 axis is an eosinophil chemoattractant receptor system that modulates the clear
171 lls that differentially express adhesion and chemoattractant receptors targeting them to the correspo
172 rect T cell expression of these adhesion and chemoattractant receptors, targeting the resulting effec
175 he innate immune system express adhesion and chemoattractant receptors that allow them to migrate dir
177 uishable by their expression of adhesion and chemoattractant receptors that directed their homing to
178 cking programs (combinations of adhesion and chemoattractant receptors) that target their migration t
179 geted disruption of two abundantly expressed chemoattractant receptors, the receptors for C3a and C5a
180 the expression and function of a variety of chemoattractant receptors through a process of heterolog
181 CAM-1, suggesting common mechanisms coupling chemoattractant receptors to activation of distinct inte
184 aling pathway leading from G protein-coupled chemoattractant receptors to the generation of oxidants
185 gest that Rho participates in signaling from chemoattractant receptors to trigger rapid adhesion in l
188 of the overall structure of other chemokine/chemoattractant receptors, underscoring an important evo
189 ally expressed in neutrophils, and acts as a chemoattractant receptor via an Akt-dependent pathway.
190 vation takes place through G protein-coupled chemoattractant receptors via a pathway that requires ho
191 , following segmental allergen challenge, no chemoattractant receptor was specifically increased.