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1 s if no tumor was seen in mammary fat pad or chest wall).
2 hat was uninvolved in the surgical incision (chest wall).
3 mpectomy breast or (optional) postmastectomy chest wall.
4 ral space to involve the soft tissues of the chest wall.
5 nd complications like bronchospasm and stiff chest wall.
6 Saline injections protected the skin and/or chest wall.
7 e breathlessness and unremitting pain in the chest wall.
8 c forces in the pulmonary parenchyma and the chest wall.
9 %) of 869 patients had primary tumors of the chest wall.
10 ithin the first hour after disruption of the chest wall.
11 ess, and contrast in lungs, mediastinum, and chest wall.
12 four noncardiac sites on the left and right chest wall.
13 erythema developing on the skin of his right chest wall.
14 lly determined to be fixed to the underlying chest wall.
15 or guidance at biopsy of masses abutting the chest wall.
16 t on the static mechanical properties of the chest wall.
17 l variations in the bone or cartilage of the chest wall.
18 mic elastances of the respiratory system and chest wall.
19 thigh and soft-tissue swelling in the right chest wall.
20 thigh and soft-tissue swelling in the right chest wall.
21 in treatment-refractory breast cancer of the chest wall.
22 ation revealed well-healed VATS scars in the chest wall.
23 actions (over 1 week) to the whole breast or chest wall.
24 3) transpulmonary open lung approach, stiff chest wall.
25 oid shape with the long axis parallel to the chest wall (10 of 11), well-defined margins (eight of 11
26 : 1) conventional open lung approach, normal chest wall; 2) conventional open lung approach, stiff ch
30 (38% v 24%, P < .001), and receive adjuvant chest wall (89% v 78%, P = .024) and nodal radiation (82
33 a total dose of 50.0 Gy was delivered to the chest wall (after mastectomy) or to the whole breast (fo
34 e total respiratory system P-V curve for the chest wall allows for calculation of an airway pressure
37 ide effect of radiotherapy of intrathoracic, chest wall and breast tumors when radiation fields encom
38 l components of tidal volume (left and right chest wall and diaphragm, and left and right lung tidal
40 ssures, heavily affected by elastance of the chest wall and lung, respectively, plays a central role.
41 gentle mechanical excitation of the external chest wall and measured the lung surface wave speed with
42 schedule, without stratification, to receive chest wall and nodal irradiation at a dose of 50 Gy in 2
43 T reported, delivered in 11 fractions to the chest wall and nodes and 15 fractions inclusive of a boo
44 in trials of radiotherapy (generally to the chest wall and regional lymph nodes), with similar absol
45 who required postoperative RT to the breast/chest wall and regional lymphatics and who were consider
49 two positive end-expiratory pressure levels, chest wall and respiratory system elastances were calcul
50 functional residual capacity, blood volume, chest wall and spinal soft-tissue mobility, and cardiac
52 ur secondary to afferent feedback from lungs/chest wall and that compensation for more negative inspi
53 11 fractions of 3.33 Gy over 11 days to the chest wall and the draining regional lymph nodes, follow
54 us cysticercosis involving the left anterior chest wall and the first case with high resolution ultra
55 t that presented with a firm swelling in the chest wall and was histopathologically confirmed to have
57 de involvement, skin and/or nipple invasion, chest wall and/or pectoralis muscle invasion, or contral
58 as defined as tumor recurrence involving the chest wall and/or the ipsilateral supraclavicular/axilla
60 and adjacent organ spread (eg, lymph nodes, chest wall), and distant disease to remote metastases (b
61 oid colon and one epithelioid sarcoma of the chest wall), and three were hematopoietic malignancies.
62 olumetric parameters were derived from lung, chest wall, and diaphragm segmentations, and parameter c
64 s Ewing's sarcoma (ES), Askin's tumor of the chest wall, and peripheral primitive neuroectodermal tum
65 y to emphysema, marked hyperinflation of the chest wall, and regional heterogeneity in the distributi
66 leads to structural underdevelopment of the chest wall, and results in increased, rather than decrea
68 smatch between the sizes of the lung and the chest wall, and the effects of LVRS are almost exclusive
71 gm, increase its area of apposition with the chest wall, and thereby improve its mechanical function.
74 patient-reported moderate or severe breast, chest wall, and/or shoulder pain in combination with mil
75 l; 2) conventional open lung approach, stiff chest wall; and 3) transpulmonary open lung approach, st
78 Mechanical interactions between lung and chest wall are important determinants of respiratory fun
79 t (commotio cordis), we sought to define the chest wall areas important in the initiation of ventricu
80 owing of the time-intensity curves caused by chest wall attenuation of the echocardiographic signal,
81 ore effective preventive strategies (such as chest wall barriers) to achieve protection from ventricu
85 ures analyzed were shape, orientation to the chest wall, border characteristics, echogenicity, homoge
87 ntrol in patients with bone, lymph node, and chest wall/breast/skin metastases at baseline was observ
91 time, provides a stable early postoperative chest wall, causes only mild postoperative pain, and pro
92 sudden death due to low-energy trauma to the chest wall (commotio cordis) has been described in young
93 ve lung disease is in part caused by reduced chest wall compliance (C(W)), believed to reflect stiffe
94 e that DP(AW) is influenced by reductions in chest wall compliance and by underlying lung properties.
96 luding a superiorly placed larynx, increased chest wall compliance, ventilation-perfusion mismatching
98 s of the respiratory system and its lung and chest wall components during passive ventilation did not
99 spiratory system and the respective lung and chest wall components or in terms of dynamic elastances
100 nics of the respiratory system into lung and chest-wall components, using the rapid occlusion techniq
101 S guidance was used for lesions abutting the chest wall; computed tomographic (CT) guidance was used
105 velopmental delay, chronic lung disease, and chest wall deformity are all seen with increased frequen
106 elain aorta, previous mediastinal radiation, chest wall deformity, and potential for injury to previo
107 ations of Marfan syndrome include scoliosis, chest wall deformity, dural ectasia, joint hypermobility
110 e were poor predictors of MRI tidal volumes (chest wall, diaphragm, and left and right separately), b
111 ed moderate correlations with tidal volumes (chest wall, diaphragm, and left and right separately).
116 ss invasive surgical procedures for lung and chest wall diseases has warranted earlier intervention,
117 nary fibrosis, sarcoidosis, neuromuscular or chest wall disorders, and disorders of ventilatory contr
119 cycle ergometer, and relative abdominal and chest wall displacements were measured by respiratory in
121 tized pigs were assigned randomly to undergo chest wall dissection alone or chest wall dissection and
122 ly to undergo chest wall dissection alone or chest wall dissection and bilateral fractures of ribs wi
127 e first case of necrotizing fasciitis of the chest wall due to infection with S. marcescens that init
128 chanics of the respiratory system, lung, and chest wall during passive ventilation at usual ventilato
129 onstrate that lesions can be detected at the chest-wall edge despite variance artifacts, and fine str
130 n geometry limits sampling statistics at the chest-wall edge of the camera, resulting in high varianc
131 Previously published methods to assess the chest wall effect on total respiratory system pressure-v
132 re is the lung-distending pressure, and that chest wall elastance may vary among individuals, a physi
141 rated tissue, collapsed tissue, and lung and chest wall elastances were similar between the two group
145 s is of modest entity and leads to a greater chest wall expansion than lung reduction, without affect
147 surgery and brought out through the anterior chest wall for potential diagnostic and therapeutic use
154 n an experimental model of sudden death from chest wall impact (commotio cordis), we sought to define
155 yndrome of sudden death caused by low-energy chest wall impact, may account for a significant percent
156 individual vulnerability to VF triggered by chest wall impact, with a distinct minority being unique
157 Sudden death due to relatively innocent chest-wall impact has been described in young individual
158 model of commotio cordis, sudden death with chest-wall impact, we sought to systematically evaluate
161 animals (14%) had >50% occurrence of VF with chest wall impacts, and only 7 (5%) had >80% occurrence
165 naked plasmid DNA, via a minimally invasive chest wall incision, is safe and may lead to reduced sym
167 hing and have either severe pneumonia (lower chest wall indrawing) or very severe pneumonia (central
171 ma Scale of less than or equal to 13, having chest wall instability or deformity, pelvic fracture, an
172 pleurodesis, asthma exacerbations, detecting chest wall invasion by tumours, lung biopsy, estimating
175 ith chest-wall irradiation and 81.9% with no chest-wall irradiation according to 10-year Kaplan-Meier
176 ot result in higher overall survival than no chest-wall irradiation among patients with intermediate-
179 ndomized trial, we evaluated the omission of chest-wall irradiation in women with "intermediate-risk"
183 er and Permutt that "resizing of the lung to chest wall" is the primary mechanism by which LVRS impro
186 n of asymptomatic, palpable, focal, anterior chest wall lesions in otherwise healthy children were re
191 isease regression could be induced in murine chest wall mammary cancers with a topical toll-like rece
192 eedle aspiration biopsy of the left anterior chest wall mass was nondiagnostic, and lumbar puncture a
195 , elevated esophageal pressures suggest that chest wall mechanical properties often contribute substa
199 ic radiotherapy in reducing the incidence of chest wall metastases (CWM) after a procedure in MPM.
200 ession in treatment-refractory breast cancer chest wall metastases but responses are short-lived.
202 ve tissue resulting from chronically reduced chest wall motion in the presence of respiratory muscle
203 We hypothesized that chronic limitation of chest wall motion in young children with NMD leads to st
204 improvement in diaphragm speed and range of chest wall movement during respiration aftere treatment
206 ial blood pressure, central venous pressure, chest wall movement, electrocardiography, electromyograp
207 breasts revealed structures corresponding to chest-wall muscle, fibroglandular, and adipose tissues i
208 zed botulinum toxin (BT) infiltration of the chest wall musculature after mastectomy would create a p
209 c (n = 3), sternal (n = 34), breast (n = 3), chest wall (n = 18), abdominal wall (n = 1), and perinea
211 , we sought to characterize influence of the chest wall on Ppl and transpulmonary pressure (PL) in pa
213 nal soft-tissue PNTs from the upper back and chest wall, one retiform soft tissue variant from the sc
216 nce of a local recurrence elsewhere (eg, the chest wall or regional nodes) after mastectomy were of c
220 was defined as ipsilateral in breast, skin, chest wall, or regional nodal recurrence without concurr
221 rative fluid collections in the mediastinum, chest wall, or retroperitoneum; (b) malignancies that we
222 p mobilize secretions include high frequency chest wall oscillation and intrapulmonary percussive ven
223 /cm2 at 1 MHz for 15 min) was applied to the chest wall overlying the myocardium during intravenous i
224 reatment-related adverse events (three [10%] chest wall pain, two [6%] dyspnoea or cough, and one [3%
226 ded bone, liver, contralateral axilla, lung, chest wall, pelvis, and the subpectoral, supraclavicular
227 essure-volume relationships for the lung and chest wall, pleural pressures generated during active re
236 scitation is recommended, because incomplete chest wall recoil from leaning may decrease venous retur
237 ances negative intrathoracic pressure during chest wall recoil or the decompression phase, leading to
240 horax (total separation of the lung from the chest wall), recruited at 31 French hospitals from 2009
241 this is clinically studied for treatment of chest wall recurrence of breast cancer, however with var
246 Following inclination, manual loading of the chest wall restored C rs and driving pressure to baselin
248 rence occurs in one-quarter of children with chest wall sarcoma and is independent of tumor type.
250 w of 175 children (median age 13 years) with chest wall sarcoma treated at seventeen Pediatric Surgic
252 y open lung approach minimized the impact of chest wall stiffening on alveolar recruitment without ca
253 tized and placed prone in a sling to receive chest wall strikes with a ball propelled at 30 to 40 mph
254 thetized, placed prone in a sling to receive chest-wall strikes during the vulnerable time window dur
256 ation mechanism, (3) distracting injury, (4) chest wall tenderness, (5) sternal tenderness, (6) thora
257 op edema and lymphocytic infiltration in the chest wall that appears to originate around lymphatics.
258 ent be designed to cover all portions of the chest wall that overlie the heart, even during body move
259 ur lesions (19 parenchymal, six pleural, six chest wall, three mediastinal) were amenable to US-guide
260 icted one or more variations in the anterior chest wall: titled sternum (n = 29), prominent convexity
261 ratory muscle function, enables the lung and chest wall to act more effectively as a pump, thereby in
263 ollowing inclination, we manually loaded the chest wall to determine if C rs increased or paradoxical
268 , and lung collapse can be acutely evoked by chest wall trauma, pneumothorax or airway compression.
269 sm in sudden death resulting from low-energy chest-wall trauma in young people during sporting activi
270 were two patients with isolated ipsilateral chest wall tumor recurrences (2 of 67; crude rate, 3%).
272 We reviewed our experience with pediatric chest wall tumors (CWTs) to identify variables associate
277 L x kg(-1) x min(-1) delivered with external chest wall vibration (29 Hz, 2 mm amplitude) of the depe
278 During each protocol, we applied in-phase chest wall vibration (CW) randomly alternating with one
281 V, the smallest amplitude to achieve visible chest wall vibration was used, and the frequency was set
284 o cordis in which a low-energy impact to the chest wall was produced by a wooden object the size and
285 essed normal tissue effects in the breast or chest wall was reported for 98 of 986 (9.9%) 40 Gy patie
287 n unremitting progression of limb, neck, and chest wall weakness and wasting that commenced and remai
290 ts < or =30 years of age with ES/PNET of the chest wall were entered in 2 consecutive protocols.
293 stances of the respiratory system, lung, and chest wall were observed between the two groups or when
295 unusual case of lymphocele of the left upper chest wall which was discovered incidentally during lymp
296 terior part of the breast and those near the chest wall, which can be inaccessible with standard grid
297 ells are viewed through a skin-flap over the chest wall, while contralateral micrometastases were ima
298 ntinuous negative pressure as applied to the chest wall with a poncho cuirass in different postures a
299 eathing, along with visible indrawing of the chest wall with or without fast breathing for age).
300 us cysticercosis involving the left anterior chest wall' with high resolution ultrasound findings.