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1 tment with the 5-HT synthesis inhibitor para-chlorophenylalanine.
2 , where 2Nal is 2-naphthylalanine, 4Cpa is 4-chlorophenylalanine, 3Pal is 3-pyridylalanine, Aph is 4-
4 sequence [PhAc-Tyr1,D-Arg2, Phe(4-Cl)6 (para-chlorophenylalanine), Abu15 (alpha-aminobutyric acid), N
5 inhibitor of phenylalanine hydroxylase, DL-p-chlorophenylalanine, and L-phenylalanine in the diet.
6 observed that systemic 5-HT depletion with p-chlorophenylalanine attenuated mechanical hypersensitivi
8 mice, reduction of 5-HT synthesis with para-chlorophenylalanine increased MES-induced seizure severi
9 ncy in catalyzing genetic incorporation of o-chlorophenylalanine (o-ClF) is better than that for N(ep
10 selective tryptophan hydroxylase inhibitor p-chlorophenylalanine on postnatal days 0 and 1 restored t
12 mined the effect of 5-HT depletion with para-chlorophenylalanine (p-CPA) on the cardiac vagal baroref
14 tion of tryptophan hydroxylase (Tph) using p-chlorophenylalanine (pCPA) or genetic knockout of the ne
15 d either with the serotonin depleting drug p-chlorophenylalanine (PCPA) or with the serotonin reuptak
16 macological depletion of 5-HT stores using p-chlorophenylalanine (PCPA), a 5-HT-synthesis inhibitor,
17 injection of the 5-HT synthesis inhibitor p-chlorophenylalanine (pCPA), which increases feeding, inc
18 s were treated systemically with saline or p-chlorophenylalanine (pCPA, 350 mg/kg) to block 5-HT synt
19 ale rats were treated systemically with para-chlorophenylalanine (pCPA; 350 mg/kg single i.p. injecti
20 p-tyrosine (AMPT, catecholamine depletor), p-chlorophenylalanine (serotonin depletor), prazosin (PRAZ
22 (3) = 3-(3-pyridyl)alanine, and Phe(4CI) = 4-chlorophenylalanine] was followed by the removal of the