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1 s frequently associated with the presence of cholesterol gallstones.
2  increased biliary cholesterol secretion and cholesterol gallstones.
3 ecifically required for biofilm formation on cholesterol gallstones.
4                 It is also a core protein of cholesterol gallstones.
5 ary lipid metabolism and the pathogenesis of cholesterol gallstones.
6 or uninfected mice fed a chow diet developed cholesterol gallstones.
7  a critical role in 17beta-estradiol-induced cholesterol gallstones.
8 ile revealed cholesterol crystals as well as cholesterol gallstones.
9 trategy for treating hypercholesterolemia or cholesterol gallstones.
10  wild-type mice developed significantly more cholesterol gallstones (27%-80% prevalence) than Rag mic
11  known until recently whether the endemic of cholesterol gallstones among certain southwestern Americ
12 pies that may alleviate biofilm formation on cholesterol gallstones and the chronic carrier state.
13                                              Cholesterol gallstones are associated with slow intestin
14                                              Cholesterol gallstones are more common in women than men
15 odentium and fed a lithogenic diet developed cholesterol gallstones at 80% prevalence by 8 weeks comp
16                Obese persons are at risk for cholesterol gallstones because their bile is saturated w
17  Apob48 or Apob100 reduces susceptibility to cholesterol gallstones by decreasing intestinal absorpti
18 romising strategy for preventing or treating cholesterol gallstones by inhibiting intestinal choleste
19 rome, as hyperglycemia, atherosclerosis, and cholesterol gallstones can all be caused by insulin resi
20                                              Cholesterol gallstone disease (CGD) results from a bioch
21 der cholesterol crystallization and possibly cholesterol gallstone disease (CGD).
22     Estrogen is an important risk factor for cholesterol gallstone disease because women are twice as
23 cus (ABCG5/ABCG8) confers susceptibility for cholesterol gallstone disease in humans.
24                                              Cholesterol gallstone disease is characterized by severa
25 Ralpha are potential therapeutic targets for cholesterol gallstone disease, particularly in women and
26 tible wild-type mice that recapitulate human cholesterol gallstone disease, treatment with a syntheti
27    Because hyperhomocysteinemia can occur in cholesterol gallstone disease, we hypothesized that this
28 dromes in both infants and adults as well as cholesterol gallstone disease.
29 herapeutic target for treating or preventing cholesterol gallstone disease.
30 mbrane composition, such as that observed in cholesterol gallstone disease.Due to the challenges in d
31 l cholesterol absorption and pathogenesis of cholesterol gallstone, dyslipidemia, and diabetes.
32 (LIRKO mice) are markedly predisposed toward cholesterol gallstone formation due to at least two dist
33                                              Cholesterol gallstone formation during weight loss in ob
34  play a major role in the pathophysiology of cholesterol gallstone formation in mice and perhaps huma
35                                              Cholesterol gallstone formation is a complex genetic tra
36                                              Cholesterol gallstone formation is a complex process med
37                Among dieting obese patients, cholesterol gallstone formation is preceded by increases
38 athway has implications for atherosclerosis, cholesterol gallstone formation, and some lipid storage
39 s intestinal cholesterol absorption, hepatic cholesterol gallstone formation, and the accumulation of
40 lesterol hypersecretion as the mechanism for cholesterol gallstone formation, thereby drawing a link
41 gnificant QTLs are detected to control mouse cholesterol gallstone formation.
42 ipid homeostasis and for the pathogenesis of cholesterol gallstone formation.
43 ne which cellular subset was responsible for cholesterol gallstone formation.
44 play an important role in the early stage of cholesterol gallstone formation.
45  high dietary cholesterol and contributes to cholesterol gallstone formation.
46 have small gallbladders and are resistant to cholesterol gallstone formation.
47 nce to diet-induced hypercholesterolemia and cholesterol gallstone formation.
48 gnificant for high-density lipoprotein (HDL) cholesterol, gallstone formation, and obesity.
49 y of the ensembles of their common and rarer cholesterol gallstone genes.
50 lipoprotein (APO)-B gene are associated with cholesterol gallstones in humans.
51 ults in a significantly higher prevalence of cholesterol gallstones in the CCK-1R-null mice.
52 monella can form biofilms on the surfaces of cholesterol gallstones in the gallbladders of mice and h
53 in has a central role in the pathogenesis of cholesterol gallstones, in part because of its ability t
54  have been implicated in the pathogenesis of cholesterol gallstones-including those developing in acr
55                             The formation of cholesterol gallstones is a complex process involving co
56 d fall of medical and contact dissolution of cholesterol gallstones is chronicled.
57 ism, predisposing the mice to development of cholesterol gallstones on a lithogenic diet.
58 the effects of Helicobacter spp infection in cholesterol gallstone pathogenesis in the highly suscept
59 ammation are likely to be crucial factors in cholesterol gallstone pathogenesis, rather then merely t
60 layed statistically significant increases in cholesterol gallstone prevalence compared with uninfecte
61 elicobacter cinaedi , and H rodentium gave a cholesterol gallstone prevalence of 40%, 30%, and 20%, r
62 sorption, biliary cholesterol secretion, and cholesterol gallstone prevalence were compared in knocko
63  modify current views of the pathogenesis of cholesterol gallstones, promote further research on the
64 o and that bile-induced biofilm formation on cholesterol gallstones promotes gallbladder colonization
65                                      In most cholesterol gallstone-prone humans, lithogenic bile carr
66                       To identify additional cholesterol gallstone susceptibility loci, we performed
67 between the metabolic syndrome and increased cholesterol gallstone susceptibility.
68 se ABC genes mapped to regions implicated in cholesterol gallstone susceptibility.
69 1(+/+)) mice fed a lithogenic diet developed cholesterol gallstones that supported biofilm formation
70  of the immune system in the pathogenesis of cholesterol gallstones was not considered a valid topic
71 ecular mechanisms of how estrogen influences cholesterol gallstones, we studied gonadectomized AKR/J