ライフサイエンスコーパス: cholesterol metabolism

1 ssociated protein required for regulation of cholesterol metabolism.

2 Bile acids are important end products of cholesterol metabolism.

3 lular matrix, endocrine system, immunity and cholesterol metabolism.

4 ) to plasma HDLs, suggesting a major role in cholesterol metabolism.

5 DNL, inhibition of fatty acid oxidation and cholesterol metabolism.

6 d in HCC, influencing its ability to harness cholesterol metabolism.

7 esterol absorbers with intact homeostasis of cholesterol metabolism.

8 effective insecticides targeting the insect cholesterol metabolism.

9 t inactivation of ABCA1 and dysregulation of cholesterol metabolism.

10 risk factors for tooth loss and measures of cholesterol metabolism.

11 control of multiple genes in lipoprotein and cholesterol metabolism.

12 cholesterol 7alpha-hydroxylase (CYP7A1) and cholesterol metabolism.

13 controls multiple mechanisms associated with cholesterol metabolism.

14 lation of genes implicated in fatty acid and cholesterol metabolism.

15 eceptor (LXR), both of which control hepatic cholesterol metabolism.

16 nd that they function in four subpathways of cholesterol metabolism.

17 c activity of TTF-1 and the role of TTF-1 in cholesterol metabolism.

18 ) to plasma HDLs, suggesting a major role in cholesterol metabolism.

19 research on apoE has focused on its role in cholesterol metabolism.

20 lipidemias and in the regulation of cellular cholesterol metabolism.

21 95 loci that associate with control of lipid/cholesterol metabolism.

22 e, revealing a relationship between CB1R and cholesterol metabolism.

23 e effective than ezetimibe alone in altering cholesterol metabolism.

24 une and inflammatory responses as well as on cholesterol metabolism.

25 ome, an X-linked dominant disorder of distal cholesterol metabolism.

26 e properties independent of their effects on cholesterol metabolism.

27 ndent on a metabolic priming event driven by cholesterol metabolism.

28 irulence of M. tuberculosis, is required for cholesterol metabolism.

29 and, thus, methylmalonyl CoA increasing upon cholesterol metabolism.

30 fects of 3 phytosterol intakes on whole-body cholesterol metabolism.

31 tionally suggest a role of sEH in regulating cholesterol metabolism.

32 ant feeding may program long-term changes in cholesterol metabolism.

33 a number of genes regulating fatty acid and cholesterol metabolism.

34 and plays a central role in phospholipid and cholesterol metabolism.

35 s dissociation of HDL apolipoprotein and HDL cholesterol metabolism.

36 d LXR reporter activity, suggesting roles in cholesterol metabolism.

37 ne protease family with an important role in cholesterol metabolism.

38 n and evaluated plasma, hepatic, and biliary cholesterol metabolism.

39 ess, they likely serve distinct functions in cholesterol metabolism.

40 ppears to play an important role in cellular cholesterol metabolism.

41 lation of bile salts and plays a key role in cholesterol metabolism.

42 between a Swi/Snf complex and regulation of cholesterol metabolism.

43 isin serine protease with a putative role in cholesterol metabolism.

44 gulation of genes involved in fatty acid and cholesterol metabolism.

45 rfamily and function as master regulators of cholesterol metabolism.

46 n of genes involved in phototransduction and cholesterol metabolism.

47 previously identified as being connected to cholesterol metabolism.

48 affects the expression of genes involved in cholesterol metabolism.

49 glyoxalase I (GLO1) as a gene implicated in cholesterol metabolism.

50 esterol ester, validating a role for GLO1 in cholesterol metabolism.

51 regulatory axis in control of bile acid and cholesterol metabolism.

52 apolipoprotein E4 (apoE4), which influences cholesterol metabolism.

53 inhibition of enhanced renal fatty acid and cholesterol metabolism.

54 ing of the molecular links between SORT1 and cholesterol metabolism.

55 sociated with multiple indicators of altered cholesterol metabolism.

56 The liver plays a key role in cholesterol metabolism.

57 egulates bile acid synthesis, transport, and cholesterol metabolism.

58 (+) T cells can be potentiated by modulating cholesterol metabolism.

59 s a link between inflammation and macrophage cholesterol metabolism.

60 on CXCR4 chemokine receptor, autophagy, and cholesterol metabolism.

61 analysis predicted that HNF-1beta regulates cholesterol metabolism.

62 criptional network that regulates intrarenal cholesterol metabolism.

63 ation, protein translation, cell growth, and cholesterol metabolism.

64 rfering RNA knockdown, of enzymes regulating cholesterol metabolism.

65 code enzymes or carrier proteins involved in cholesterol metabolism, 3 participate in fatty acid meta

66 ing PCSK9-a key protein in the regulation of cholesterol metabolism(6-8)-can boost the response of tu

67 f LC-PUFA biosynthesis (elovl and fads2) and cholesterol metabolism (abca1) are regulated by Lxr and

68 ed in lipid metabolism (Ppargamma, Angptl4), cholesterol metabolism (Abcg5/8), gastrointestinal homeo

69 ly, blocking the ability of IFN to reprogram cholesterol metabolism abrogates cellular protection and

70 se results indicate that bilirubin regulates cholesterol metabolism, adipokines and PPARgamma levels,

71 of BAs in improving insulin sensitivity and cholesterol metabolism after BPD.

72 heimer's disease (AD) may arise from altered cholesterol metabolism, although the molecular pathways

73 Brazil has a metabolic effect on endogenous cholesterol metabolism and a protector effect on develop

74 uctions in Npc1 protein, as well as abnormal cholesterol metabolism and altered glycolipid expression

75 pidemiological studies suggest links between cholesterol metabolism and Alzheimer's disease (AD), wit

76 s across pan-cancer, while the alteration in cholesterol metabolism and arachidonic acid metabolism d

77 However, the precise role of BAT in plasma cholesterol metabolism and atherosclerosis development r

78 onsiderable interest in defining its role in cholesterol metabolism and atherosclerosis.

79 og 1 (LRH-1; NR5A2) is a potent regulator of cholesterol metabolism and bile acid homeostasis.

80 We found that hepatic JNK deficiency alters cholesterol metabolism and bile acid synthesis, conjugat

81 argets in cardiovascular disease, regulating cholesterol metabolism and bile acid transport and metab

82 and viral pathogens may modulate macrophage cholesterol metabolism and cardiovascular disease.

83 c role of the CNS in the control of systemic cholesterol metabolism and circulating plasma lipids lev

84 a specific pathway involved in regulation of cholesterol metabolism and clearance.

85 of allelic variation in regulating lipid and cholesterol metabolism and could potentially provide a p

86 ptors (LXR) alpha and beta are regulators of cholesterol metabolism and determinants of atheroscleros

87 may offer a novel and safe means of managing cholesterol metabolism and diet induced dyslipidaemia, a

88 o pathogenic bacteria, and are essential for cholesterol metabolism and embryonic development in huma

89 cesses whereas ATF4 uniquely associates with cholesterol metabolism and endoplasmic reticulum (ER) st

90 at are important hormones that regulate host cholesterol metabolism and energy balance via several nu

91 roles of NRs in cholangiocyte physiology and cholesterol metabolism and flux.

92 gulate numerous biological processes such as cholesterol metabolism and hepatocyte signaling pathways

93 This review focuses on how the link between cholesterol metabolism and higher-order brain function w

94 holesterol-24-hydroxylase is responsible for cholesterol metabolism and homeostasis in the human brai

95 ion and progression of AD has been linked to cholesterol metabolism and inflammation, processes that

96 pulmonary homeostasis, balancing both lipid/cholesterol metabolism and inflammatory responses.

97 metabolism is understood, its role in brain cholesterol metabolism and its impact on AD development

98 ion suggests roles for PLTP in both cellular cholesterol metabolism and lipoprotein retention on extr

99 al study, in which we examined parameters of cholesterol metabolism and liver function values in seru

100 HSL is involved in regulating intracellular cholesterol metabolism and making unesterified cholester

101 cate apolipoprotein E, the endocytic system, cholesterol metabolism and microglial activation as Abet

102 The dual problem of perturbed cholesterol metabolism and mitochondrial dysfunction cou

103 The altered transcripts are involved with cholesterol metabolism and mitochondrial function.

104 mechanistic connection between AIBP-mediated cholesterol metabolism and Notch signaling, implicating

105 s critical for the neuroendocrine control of cholesterol metabolism and plasma lipid levels.

106 anced the effects of ezetimibe on whole-body cholesterol metabolism and plasma low-density lipoprotei

107 CYP125A1 is a key enzyme in cholesterol metabolism and plays a crucial role in circu

108 cid metabolism, arachidonic acid metabolism, cholesterol metabolism and PPAR signaling.

109 t liver-specific microRNA (miRNA), regulates cholesterol metabolism and promotes hepatitis C virus (H

110 e clarify the relationship between disrupted cholesterol metabolism and reduced SHH signalling in SLO

111 in conserved in several proteins involved in cholesterol metabolism and signaling.

112 ory role for the GPR146/ERK axis in systemic cholesterol metabolism and suggest that GPR146 inhibitio

113 ort a role for SUGP1 as a novel regulator of cholesterol metabolism and suggest that it contributes t

114 tes migration, the inflammatory response and cholesterol metabolism and suggest that targeting Akt2 i

115 Our study reveals that miR-146a regulates cholesterol metabolism and tempers chronic inflammatory

116 c Nnmt expression in vivo alters glucose and cholesterol metabolism and that the metabolic effects of

117 enes and activation of genes associated with cholesterol metabolism and the p53 pathway in CDK19 knoc

118 suggests the occurrence of changes in brain cholesterol metabolism and the potential utility of usin

119 ies have focused on three general areas: HDL-cholesterol metabolism and the reverse cholesterol trans

120 d offers a potential link between disordered cholesterol metabolism and the synapse loss seen in neur

121 However, the changes in cholesterol metabolism and their role during PDAC progre

122 mmon environmental toxicants may also impair cholesterol metabolism and thereby possibly contribute t

123 pression of genes linked to neuroprotection, cholesterol metabolism and tissue remodeling.

124 oci in genes that have putative functions in cholesterol metabolism and transport, and sulfonylation

125 Variants associated with cholesterol metabolism and type 1 diabetes showed simila

126 ipid metabolism and plays a profound role in cholesterol metabolism and weight gain in the host.

127 atocytes and adipocytes via enhanced hepatic cholesterol metabolism and white fat browning.

128 Inflammation, oxidative damage, cholesterol metabolism and/or impaired function of retin

129 tone group was characterized by deteriorated cholesterol metabolism, and accumulation of cholestanol,

130 As EPHX2 is known to influence cholesterol metabolism, and AN is often associated with

131 gulation, signal transduction, bile acid and cholesterol metabolism, and control of apoptosis.

132 Changes in cholesterol metabolism are common hallmarks of neurodeve

133 A number of key genes involved in cholesterol metabolism are known to undergo functionally

134 Retinoid and cholesterol metabolism are linked in stellate cells by t

135 al therapies targeting different pathways in cholesterol metabolism are now available, ranging from s

136 fects of different intakes of phytosterol on cholesterol metabolism are uncertain.

137 od, we applied it using 21 genes involved in cholesterol metabolism as "bait" to "fish out" (or ident

138 Collectively, our data define cholesterol metabolism as an integral metabolic pathway

139 l- and LDL-cholesterol, and with AT-specific cholesterol metabolism-associated lipids [arachidonoyl c

140 he role of RA in important processes such as cholesterol metabolism, bile acid secretion, and oncogen

141 he role of T3 in important processes such as cholesterol metabolism, bile acid secretion, oncogenesis

142 ved in oxidative stress response basally and cholesterol metabolism both basally and under stress.

143 LXR signaling not only regulates macrophage cholesterol metabolism but also impacts antimicrobial re

144 eveal a critical role for miR-122 in fat and cholesterol metabolism but suggest that other metabolic

145 e of LCLs for the study of statin effects on cholesterol metabolism, but suggest that drug effects on

146 rter A1 (ABCA1) plays a critical role in HDL cholesterol metabolism, but the mechanism by which it tr

147 Plg in the regulation of gene expression and cholesterol metabolism by macrophages and identify Plg-m

148 We show that NBEAL1 regulates cholesterol metabolism by modulating LDLR expression in

149 e protein SAA plays an important role in HDL cholesterol metabolism by promoting cellular cholesterol

150 AT activation and highlight the relevance of cholesterol metabolism by the host for diet-induced chan

151 brain is directly involved in the control of cholesterol metabolism by the liver.

152 Thus, cholesterol metabolism by these microbes may play import

153 this study augments our understanding of how cholesterol metabolism can modulate a neuroprotective me

154 ons in mammals, including neuronal survival, cholesterol metabolism, cell differentiation and tumor d

155 nt insulin secretion, glucose metabolism and cholesterol metabolism, compared to the high-fat control

156 autoimmunity; however, how the regulation of cholesterol metabolism contributes to autoimmunity is un

157 ors (LXRs) are transcriptional regulators of cholesterol metabolism, controlling cholesterol flow int

158 We define a mechanism by which cholesterol metabolism controls the development and diff

159 dy, we tested the hypothesis that hepatic ER cholesterol metabolism differentially regulates ER stres

160 We report alterations in sphingolipid and cholesterol metabolism during normal brain aging and in

161 ion to classic transcriptional regulation of cholesterol metabolism (e.g. by SREBP and LXR), members

162 ned dietary and pharmacological treatment on cholesterol metabolism emphasizes the potential importan

163 s of 2 nuclear hormone receptors involved in cholesterol metabolism, establishing a plausible mechani

164 ed immune response, high-density lipoprotein cholesterol metabolism, extracellular matrix, and angiog

165 athways, such as fatty acid, eicosanoid, and cholesterol metabolism; fibrinolytic regulation; cell gr

166 Hepatitis C virus (HCV) subverts host cholesterol metabolism for key processes in its lifecycl

167 ndings demonstrate the importance of retinal cholesterol metabolism for maintenance of the normal ret

168 The mechanism whereby cholesterol metabolism functionally impacts neurodegener

169 a significant association between IL-10 and cholesterol metabolism gene expression.

170 , these data suggest that alterations of the cholesterol metabolism gene network represent a molecula

171 that alterations in a network of coexpressed cholesterol metabolism genes are a signature feature of

172 to maintain the expression of fatty acid and cholesterol metabolism genes under hypoxic conditions.

173 As expected, TO-901317 upregulated several cholesterol metabolism genes, but it also decreased expr

174 in cholesterol content and the expression of cholesterol metabolism genes.

175 racellular matrix formation, oocyte meiosis, cholesterol metabolism, glycolysis/gluconeogenesis, and

176 Cholesterol metabolism has been implicated in prostate c

177 Cholesterol metabolism has been implicated in the pathog

178 controls cell proliferation; disruptions in cholesterol metabolism have been associated with the dev

179 metabolic consequences of a leaking BBB for cholesterol metabolism have not been studied previously.

180 A implicated in regulation of fatty acid and cholesterol metabolism, hepatitis C infection, and hepat

181 , glucagon participates in the regulation of cholesterol metabolism; however, the molecular pathways

182 Recent work suggests that cholesterol metabolism impacts innate immune responses a

183 te and high doses favorably alter whole-body cholesterol metabolism in a dose-dependent manner.

184 ggests that both HIV and ART affect monocyte cholesterol metabolism in a pattern consistent with accu

185 TSHB mRNA is linked to cholesterol metabolism in adipose tissue.

186 Higher levels of apoE and associated cholesterol metabolism in APOE2 carriers might contribut

187 However, spatial cholesterol metabolism in brain and the resulting sterol

188 under stress, we propose that suppression of cholesterol metabolism in cancer cells should elicit syn

189 Here, we report a significant role of cholesterol metabolism in cancer metastasis.

190 nity, Ito et al. (2016) show that defects in cholesterol metabolism in CD11c(+) immune cells result i

191 ere, we show that DENV infection manipulated cholesterol metabolism in cells residing in low-oxygen m

192 d the effects of individual plant sterols on cholesterol metabolism in cultured adrenal cells.

193 apeutic in improving host glucose, lipid and cholesterol metabolism in diet induced obese rodents.

194 s known to be a major pathway of lipoprotein cholesterol metabolism in experimental animals and human

195 little is known about mechanisms regulating cholesterol metabolism in fat cells.

196 powerful tool for future studies of spatial cholesterol metabolism in healthy and diseased tissues.

197 tigate the relationship between TSH mRNA and cholesterol metabolism in human adipose tissue (AT).

198 ne factor that is modulated in parallel with cholesterol metabolism in human AT.

199 evels in plasma from atopic patients (AP) on cholesterol metabolism in human macrophages as compared

200 ations highlights a role for the gene in LDL cholesterol metabolism in humans and shows the usefulnes

201 ation studies (GWAS) as a novel regulator of cholesterol metabolism in humans.

202 ha is directly involved in the regulation of cholesterol metabolism in macrophages and plays an impor

203 For that reason, cholesterol metabolism in macrophages has attracted cons

204 aimed to identify novel miRNAs that regulate cholesterol metabolism in macrophages stimulated with LX

205 macrophage inflammation/chemotaxis and lipid/cholesterol metabolism in MAKO/LDLRKO mice.

206 f intestinal SIRT1 in systemic bile acid and cholesterol metabolism in mice.

207 pothesis that inhibition of ACAT may improve cholesterol metabolism in NS.

208 e models to determine the drivers of altered cholesterol metabolism in PDAC and the consequences of i

209 ide direct evidence that A2E causes aberrant cholesterol metabolism in RPE cells which could likely c

210 ral organoids as a tool to study the role of cholesterol metabolism in SHH signaling.

211 To better understand cholesterol metabolism in situ across the complex functi

212 The key roles of cholesterol metabolism in the activation of inflammasome

213 at one of the initial reactions of anaerobic cholesterol metabolism in the beta-proteobacterium Stero

214 he blood-brain barrier (BBB) is critical for cholesterol metabolism in the brain, preventing uptake o

215 plays a central role in regulating apoE and cholesterol metabolism in the CNS via LRP1 and establish

216 t serum desmosterol is a marker of disturbed cholesterol metabolism in the liver.

217 direct evidence of a pivotal role of altered cholesterol metabolism in the pathogenesis of motor-neur

218 Recent results implicating cholesterol metabolism in the pathophysiology of Alzheim

219 ata indicate an independent role for APOE in cholesterol metabolism in the periphery relative to the

220 MDV-infected chickens; however, the role of cholesterol metabolism in the replication and spread of

221 um mineral, we examined the role of cellular cholesterol metabolism in vascular cell mineralization.

222 modulate HMGCR and contribute to control of cholesterol metabolism in whole animals is unknown.

223 iously implicated in Alzheimer's disease and cholesterol metabolism, in integrating cellular response

224 iency has profound effects on murine hepatic cholesterol metabolism, including hypersensitivity to di

225 R-BI) shows a variety of effects on cellular cholesterol metabolism, including increased selective up

226 for a number of well-known relationships in cholesterol metabolism, including the epidemiological re

227 ing cassette transporter A1 (ABCA1)-mediated cholesterol metabolism, increase reverse cholesterol tra

228 olved in metabolism (e.g., sugar, lipid, and cholesterol metabolism), inflammation, and fibrosis.

229 transfected cells to elucidate how altering cholesterol metabolism influences APP processing.

230 ation of all PA strains was blocked by three cholesterol metabolism inhibitors (P < 0.01).

231 aluated by pretreatment of hTCEpi cells with cholesterol metabolism inhibitors.

232 singly, GBA2 deficiency leaves bile acid and cholesterol metabolism intact, instead causing lipid acc

233 physiological function, yet dysregulation of cholesterol metabolism is associated with diseases such

234 r, the results suggest that up-regulation of cholesterol metabolism is essential for matrix mineraliz

235 Dysregulated cholesterol metabolism is implicated in a number of neur

236 Aberrant lipid and cholesterol metabolism is involved in prostate cancer de

237 Yet whether these play a role in cholesterol metabolism is largely unknown.

238 Impact on cholesterol metabolism is not limited to the infected ce

239 me in interaction between pathogens and host cholesterol metabolism is pathogens targeting lipid raft

240 Cholesterol metabolism is subject to complex transcripti

241 An important event in cholesterol metabolism is the efflux of cellular cholest

242 Cholesterol metabolism is tightly regulated at the cellu

243 Cholesterol metabolism is tissue specific, and its signi

244 gh much of the biology of APOE in peripheral cholesterol metabolism is understood, its role in brain

245 ntribution of individual cell types to brain cholesterol metabolism is unknown.

246 ions as a bile acid (BA) sensor coordinating cholesterol metabolism, lipid homeostasis, and absorptio

247 Cholesterol metabolism may be involved in pediatric gall

248 The cellular control of cholesterol metabolism mediated by lipoproteins was firs

249 ction, and that pharmaceuticals that inhibit cholesterol metabolism might be valuable in therapy of A

250 raises the possibility that abnormalities in cholesterol metabolism might underlie some cases of huma

251 ys involved in lesion development, including cholesterol metabolism, mitochondrial oxidative phosphor

252 Advances in cholesterol biology suggest that cholesterol metabolism modulates beta-amyloid production

253 is cholesterol-rich, and inhibition of host cholesterol metabolism negatively impacts PV biogenesis

254 Here, we examined the roles in cholesterol metabolism of two other cytochrome P450 enzy

255 ffects of apolipoprotein E (APOE) levels and cholesterol metabolism on disease development.

256 ts recent findings on the impact of aberrant cholesterol metabolism on platelet biogenesis and activi

257 es or other small molecules to components of cholesterol metabolism or regulation.

258 rofiling unravelled novel roles for GSTO1 in cholesterol metabolism, oxidative and endoplasmic stress

259 tabolism, the thyroid hormone pathway, lipid/cholesterol metabolism, oxidative stress, immune respons

260 s been implicated in a variety of endogenous cholesterol metabolism pathways including the following

261 for albumin and a serum protein involved in cholesterol metabolism, PCSK9, demonstrating the potenti

262 Taken together, our results indicate that cholesterol metabolism plays a crucial role in GCT diffe

263 Given an essential role that host cholesterol metabolism plays in pathogen development, ta

264 binding protein (SREBP), a key regulator of cholesterol metabolism proteins such as PCSK9, HMG-CoA r

265 diverse cancers, including those involved in cholesterol metabolism, providing correlative support fo

266 While previous studies have shown that cholesterol metabolism regulates APP processing to Abeta

267 ol concentration and decreased expression of cholesterol metabolism related genes Abcg5, Abcg8, Abcg1

268 e targets for pharmaceutical intervention in cholesterol metabolism-related disease processes.

269 nd identified many potential tissue-specific cholesterol metabolism-related genes.

270 Inherited gene mutations in cholesterol metabolism result in a severe autoinflammato

271 ges, with up-regulation of genes involved in cholesterol metabolism, scavenger receptors, MERTK, and

272 n of the master transcriptional regulator of cholesterol metabolism, SREBP-2, almost as effectively a

273 lular functions, including the regulation of cholesterol metabolism, steroidogenesis, and apoptosis.

274 egulated expression of genes associated with cholesterol metabolism, such as cholesterol-25-hydroxyla

275 portance of SREBF2 and miR-33a in regulating cholesterol metabolism suggests that TTF-1 may be a modu

276 A diverse array of genetic disorders of cholesterol metabolism support this claim as do multiple

277 Liver X receptors (LXRs) are regulators of cholesterol metabolism that also modulate immune respons

278 monly used pharmaceuticals induce changes in cholesterol metabolism that are similar to changes induc

279 e fadA5 mutant is a consequence of disrupted cholesterol metabolism that is essential only in the per

280 By enhancing the SREBP-mediated cholesterol metabolism, this unique mechanism may contri

281 the role of microRNA (miRNAs) in regulating cholesterol metabolism through ABC transporters.

282 ere that pathogens interfere with macrophage cholesterol metabolism through inhibition of the LXR sig

283 ze SAA and, further, that SAA influences HDL cholesterol metabolism through its inhibitory effects on

284 xa, from prions to protozoa, target cellular cholesterol metabolism to advance their own development

285 Many of these viruses manipulate host cholesterol metabolism to facilitate their replication.

286 ential for biological processes ranging from cholesterol metabolism to host defense, the in vivo impo

287 has led to promising new therapies targeting cholesterol metabolism, triglyceride production, hepatic

288 This study sheds light into microbial cholesterol metabolism under anoxic conditions.

289 Altering cholesterol metabolism using statins decreased the gener

290 miR-144 regulates cholesterol metabolism via suppressing ABCA1 expression

291 A role for PCSK9 in cholesterol metabolism was proposed from the expression

292 Rosi induction of ABCA1, which regulates cholesterol metabolism, was dependent upon SNP rs4743771

293 arkers of cholesterol absorption and hepatic cholesterol metabolism were assessed together with globa

294 ortantly, 4 of 6 genes associated with lipid/cholesterol metabolism were significantly dysregulated b

295 Genes involved in cholesterol metabolism were similarly regulated between

296 s function is very elegantly demonstrated in cholesterol metabolism where miRNAs reducing cellular ch

297 stress, resulting in perturbed ceramide and cholesterol metabolism which, in turn, triggers a neurod

298 physiological link between ASBT and hepatic cholesterol metabolism, which led to the clinical invest

299 nection between prion infection and cellular cholesterol metabolism, which plays an important role in

300 ockdown on the PPARalpha-LXRalpha pathway of cholesterol metabolism with MK886 (a selective inhibitor