ライフサイエンスコーパス: cholesterol-rich

1 holesterolemia characterized by unesterified cholesterol-rich abnormal lipoproteins (lamellar/vesicul

2 Although plasma membrane (PM) cholesterol-rich and -poor domains have been isolated by

3 l phase separation in the outer leaf between cholesterol-rich and -poor liquids causes a similar, but

4 omponents (DR5, FADD, and procaspase-8) into cholesterol-rich and ceramide-rich domains known as cave

5 Ehrlichia chaffeensis, a cholesterol-rich and cholesterol-dependent obligate intr

6 The coexistence between cholesterol-rich and cholesterol-poor domains is univers

7 Both cholesterol-rich and triglyceride-rich lipoproteins corr

8 tive at promoting lateral domains, which are cholesterol-rich and unsaturation-rich, respectively.

9 Macrophages were also cholesterol-rich and were able to transfer cholesterol t

10 Lipid rafts are highly ordered, cholesterol-rich, and detergent-resistant microdomains f

11 The PV membrane is cholesterol-rich, and inhibition of host cholesterol met

12 excess lipids derived from the ingestion of cholesterol-rich apoptotic corpses.

13 sclerosis is the subendothelial retention of cholesterol-rich, atherogenic lipoproteins.

14 with the E2 state being likely disfavored in cholesterol-rich bilayers relative to the E1P state beca

15 water transit a leaflet through the DOPC and cholesterol rich boundaries of hexagonally packed DPPC m

16 ) mediate the binding of the CDC monomers to cholesterol-rich cell membranes.

17 mediates the interaction of the CDCs with a cholesterol-rich cell surface.

18 aged with mobile ligand coalesce into large, cholesterol-rich clusters that occupy the central portio

19 ining HDL formed intrahepatically are likely cholesterol-rich compared to the smaller intracellular l

20 -2, and NPC1, but not CI-MPR, similar to the cholesterol-rich compartment in NPC mutant cells.

21 LAT has also been associated with cholesterol-rich condensed lipid domains; However, the p

22 ts, mature nicastrin, APH-1, and PEN-2, with cholesterol-rich detergent insoluble membrane (DIM) doma

23 ins from bovine neutrophils co-isolates with cholesterol-rich, detergent-resistant membrane fragments

24 tion between Talpha and caveolin-1 occurs in cholesterol-rich, detergent-resistant membranes and is l

25 exclusively resides in low-buoyant-density, cholesterol-rich, detergent-resistant membranes that can

26 uencing of different organs of both WHHL and cholesterol-rich diet (Chol)-fed NZW rabbits.

27 ol (HDL-C) compared with those of rats fed a cholesterol-rich diet (HCD).

28 We conclude that a cholesterol-rich diet affects learning speed and perform

29 olling the TFH-germinal center response to a cholesterol-rich diet and uncover a PDL1-dependent mecha

30 Excessive alcohol consumption and cholesterol-rich diet are associated with a high risk of

31 e show that lesions from diabetic pigs fed a cholesterol-rich diet contain abundant insulin-like grow

32 Early animal experiments showed that a cholesterol-rich diet could induce fatty lesion formatio

33 graphy were lower in lesions after feeding a cholesterol-rich diet for 3, 6, and 12 weeks.

34 nces in the plasma lipoprotein response to a cholesterol-rich diet observed in the transgenic rabbits

35 esent study was to determine the effect of a cholesterol-rich diet on learning performance and monito

36 c rabbits (18 +/- 4 mg/dl) were similar, the cholesterol-rich diet raised the non-HDL cholesterol con

37 LDL receptor-deficient mice that were fed a cholesterol-rich diet showed increased cholesterol level

38 After 4 weeks of cholesterol-rich diet, a switch to a control diet for 4

39 en fluorescent protein were fed a control or cholesterol-rich diet, and green fluorescent protein-pos

40 ects on cholesterol metabolism in mice fed a cholesterol-rich diet, including complete resistance to

41 We show that diabetic animals fed a cholesterol-rich diet, like humans, develop severe lesio

42 ts in the Npc1a embryos are ameliorated by a cholesterol-rich diet.

43 d for 8 or 13 weeks and fed chow or high-fat cholesterol-rich diet.

44 9 embryos are substantially ameliorated by a cholesterol-rich diet.

45 ere divided into 3 groups (n=8) and fed with cholesterol-rich diets supplemented with cellulose (CC,

46 When diabetic mice are fed cholesterol-rich diets, on the other hand, they develop

47 cholesterol mixtures suggest that a separate cholesterol rich domain coexists with the DMPC rich doma

48 15% cholesterol content, suggesting that the cholesterol rich domain has a definite stoichiometry and

49 lay a role in sequestering this protein to a cholesterol-rich domain in the membrane.

50 We show that the selective localization of cholesterol-rich domains and associated ganglioside rece

51 d that mouse TRPA1 localizes preferably into cholesterol-rich domains and functional experiments reve

52 tition into submicroscopic sphingolipid- and cholesterol-rich domains called lipid rafts, but the det

53 and integral membrane proteins that organize cholesterol-rich domains called lipid rafts.

54 NAP-22 also protects the cholesterol-rich domains during extraction by methyl-bet

55 The present studies explored the role of cholesterol-rich domains in maintaining this functional

56 sion as well as localization of the virus to cholesterol-rich domains in membranes.

57 the previously described affinity of SCR for cholesterol-rich domains in membranes.

58 When formed, these cholesterol-rich domains in the ER maintain cellular hom

59 in the PM, consistent with the existence of cholesterol-rich domains in the plasma membrane of livin

60 also suggest that Gag assembly must occur in cholesterol-rich domains in the plasma membrane.

61 cluster, we find that the composition of the cholesterol-rich domains is constant, independent of the

62 This study demonstrated that cholesterol-rich domains mediate the actions of early up

63 id components, particularly the formation of cholesterol-rich domains that are thought to be importan

64 conclude that adenylyl cyclase must occur in cholesterol-rich domains to be susceptible to regulation

65 phospholipid monolayers and the tendency of cholesterol-rich domains to form in cholesterol-lipid bi

66 iation of IgE-Fc epsilon RI with specialized cholesterol-rich domains within approximately 4-nm proxi

67 pid rafts enriched in glycosphingolipids and cholesterol-rich domains, but selectively lacking glycos

68 ndent manner, leading to the hypothesis that cholesterol-rich domains, or "lipid rafts," may act as f

69 prefer to partition within the more ordered, cholesterol-rich/DOPC-poor/GM1-rich micrometer-scale pha

70 A cholesterol-rich environment also induces processing of

71 Notably, even in cholesterol-rich environment, iodide transport activity

72 ficance of ordered nanodomains (or rafts) in cholesterol rich eukaryotic cell membranes has only begu

73 hains in gel-fluid bilayers, fluid bilayers, cholesterol-rich fluid bilayers, and gel-fluid bilayers

74 et, and particularly from the consumption of cholesterol-rich foods.

75 that Nox1 and Nox5 but not Nox4, localize in cholesterol-rich fractions in VSMCs.

76 axons is increased by myelin, a specialized, cholesterol-rich glial cell membrane that tightly wraps

77 l-poor ligand that binds to the receptor for cholesterol-rich HDLs, scavenger receptor type B1 (SCARB

78 own AMD risk factors: advanced age, high fat cholesterol-rich (HF-C) diet, and apolipoprotein E (apoE

79 B1), found in lipid rafts, is a receptor for cholesterol-rich high-density lipoproteins (HDL).

80 Caveolae are small, cholesterol-rich, hydrophobic membrane domains, characte

81 Caveolae, cholesterol-rich invaginations of the plasma membrane, h

82 glycolipid rafts and in caveolae, noncoated, cholesterol-rich invaginations on the plasma membrane.

83 directly supported LB monolayers containing cholesterol-rich l(o) phases are inherently unstable whe

84 le concentration, accumulates heavily inside cholesterol-rich late endosomes in Npc1(-/-) cells.

85 the well recognized role and contribution of cholesterol-rich LDL or lipoprotein B particles to the p

86 By contrast, the larger cholesterol-rich LDL particles and all high-density lipo

87 or (LDL-R) mediates the endocytosis of these cholesterol-rich LDL particles into the cell, thereby su

88 version of triglyceride-rich VLDL to smaller cholesterol-rich LDL, arginine-3,500 interacts with the

89 the formation of ordered domains in a SM and cholesterol-rich leaflet can be suppressed by an opposit

90 tein function, contributing, for example, to cholesterol-rich lesions associated with age-related mac

91 LDLR is involved in uptake of cholesterol rich lipid particles from bloodstream.

92 ture of transbilayer couplings in asymmetric cholesterol-rich lipid bilayers, the effects on the lipi

93 F. tularensis live vaccine strain recruits cholesterol-rich lipid domains ("lipid rafts") with cave

94 within lipid rafts; ordered sphingolipid and cholesterol-rich lipid domains believed to exist within

95 that the formation of glycosphingolipid- and cholesterol-rich lipid domains can be driven solely by c

96 f the lipid raft concept is the formation of cholesterol-rich lipid domains.

97 proteins (Envs) of HIV-1 are embedded in the cholesterol-rich lipid membrane of the virus.

98 e that co-localization of TRPV1 with TLR4 to cholesterol-rich lipid membrane rafts in nociceptors is

99 ctor of Streptococcus pneumoniae, perforates cholesterol-rich lipid membranes.

100 alters plasma membrane fluidity by reducing cholesterol-rich lipid microdomains, while alkaline pH e

101 l nitric oxide synthase (eNOS), localizes to cholesterol-rich lipid raft domains of the plasma membra

102 alization is mediated by caveolae, which are cholesterol-rich lipid raft domains.

103 lity, we identified an Akt1 subpopulation in cholesterol-rich lipid raft fractions prepared from LNCa

104 the non-raft region, the membrane region of cholesterol-rich lipid raft markedly weakens the membran

105 rters, especially EAAT2, are associated with cholesterol-rich lipid raft microdomains of the plasma m

106 associates in a Ca(2+) dependent manner with cholesterol-rich lipid raft microdomains.

107 e factor at the cell surface is localized in cholesterol-rich lipid rafts and extensively colocalized

108 he possibility that microvesicles arise from cholesterol-rich lipid rafts and found that both TF and

109 udy, we show that E-selectin is localized in cholesterol-rich lipid rafts at the cell surface, and th

110 tracellular cysteines, partitioning CD2 into cholesterol-rich lipid rafts constitutively, human CD2 h

111 in the lipid raft fractions suggesting that cholesterol-rich lipid rafts mediate PKC-triggered NET i

112 Many enveloped viruses bud from cholesterol-rich lipid rafts on the cell membrane.

113 human prostate cancer (LNCaP) cells contain cholesterol-rich lipid rafts that mediate epidermal grow

114 Numerous enveloped viruses utilize cholesterol-rich lipid rafts to bud from the host cell m

115 P2X7 receptors associate with cholesterol-rich lipid rafts, but it is unclear how this

116 irus, which is associated with controversial cholesterol-rich lipid rafts.

117 ETEC vesicle endocytosis depended on cholesterol-rich lipid rafts.

118 dicating that the channels are targeted into cholesterol-rich lipid rafts.

119 of SNAP-23 is associated with non-caveolar, cholesterol-rich lipid rafts.

120 nces or the organization of sphingolipid and cholesterol-rich lipid rafts.

121 ferrin receptor and often were juxtaposed to cholesterol-rich lipid rafts.

122 n-mediated bacterial internalization through cholesterol-rich lipid rafts.

123 yn kinase, was dependent on the integrity of cholesterol-rich lipid rafts.

124 esis and is known to be tightly regulated by cholesterol-rich "lipid rafts." Collectively, these data

125 Recent studies have implicated cholesterol-rich, lipid raft microdomains in survival si

126 LDL, the most abundant cholesterol-rich lipoprotein in plasma, is causally link

127 late in atherosclerotic plaques, internalize cholesterol-rich lipoprotein particles, and evolve into

128 o prepare an apoE peptide that bound to both cholesterol-rich lipoproteins and lipoprotein receptors,

129 re, this peptide selectively associated with cholesterol-rich lipoproteins and mediated their acute c

130 ran sulfate participates in the clearance of cholesterol-rich lipoproteins as well.

131 nding and clearance of both triglyceride and cholesterol-rich lipoproteins from the circulation.

132 atherogenic because of their ability to trap cholesterol-rich lipoproteins in vitro.

133 cellular cholesterol homeostasis by binding cholesterol-rich lipoproteins through their apoB and apo

134 e into the subendothelial space, internalize cholesterol-rich lipoproteins, and become foam cells by

135 erforming "receptor-mediated endocytosis" of cholesterol-rich lipoproteins.

136 solated from both cells and sphingolipid and cholesterol-rich liposomes (SCRLs) in association with d

137 Similar cholesterol-rich liposomes are found in early developing

138 Inoculation of mice with cholesterol-rich liposomes containing the adjuvant monop

139 with cholesterol esterase converts LDL into cholesterol-rich liposomes having >90% cholesterol in un

140 esterase-mediated transformation of LDL into cholesterol-rich liposomes is an LDL modification that:

141 We now show that cholesterol-rich liposomes produced from cholesterol est

142 arly, binding of anti-cholesterol A to small cholesterol-rich liposomes resulted in the appearance of

143 effect on SLO-mediated poration of synthetic cholesterol-rich liposomes.

144 oglycan complex formation, and conversion to cholesterol-rich liposomes.

145 Sphingolipid- and cholesterol-rich liquid-ordered (Lo) lipid domains (raft

146 ne of the main properties distinguishing the cholesterol-rich liquid-ordered (Lo) phase from the liqu

147 We showed previously that cholesterol-rich liquid-ordered domains with lipid compo

148 ximately 2, consistent with the zone being a cholesterol-rich liquid-ordered phase.

149 Cholesterol-rich, liquid-ordered (L(o)) domains are beli

150 ecognition is barely detectable in analogous cholesterol-rich, liquid-ordered (l0) bilayers.

151 e consistent with condensed complex-rich and cholesterol-rich liquids.

152 ein receptor (LDLR) is involved in uptake of cholesterol rich low-density lipoprotein (LDL) particles

153 esis and through receptor-mediated uptake of cholesterol-rich low density lipoprotein (LDL).

154 ce receptor that mediates the endocytosis of cholesterol-rich low-density lipoproteins, allowing cell

155 s DA efflux and enhances DAT localization in cholesterol rich membrane microdomains.

156 ation of the small GTPase p21Ras to GM1- and cholesterol-rich membrane areas.

157 Recognition of the cholesterol-rich membrane by CDCs is a surprisingly subt

158 tumoral protein levels - a major protein of cholesterol-rich membrane domains - and Trastuzumab-drug

159 emonstrate modifications of the structure of cholesterol-rich membrane domains and the association of

160 Disruption of cholesterol-rich membrane domains by filipin inhibits Pl

161 ndings demonstrate that Francisella requires cholesterol-rich membrane domains for entry into and pro

162 sterol depletion studies indicate a role for cholesterol-rich membrane domains in the formation/maint

163 at specific localization of the F protein in cholesterol-rich membrane domains is not required for ce

164 By directly monitoring cholesterol-rich membrane domains with a fluorescently t

165 disease virus (NDV) fusion (F) protein with cholesterol-rich membrane domains, its localization in d

166 d by CD81 oligomerization, partitioning into cholesterol-rich membrane domains, or other, lateral pro

167 cal for the trafficking of these proteins to cholesterol-rich membrane domains, which leads to cleava

168 st in part because it directs the protein to cholesterol-rich membrane domains.

169 actin and myosin II filament organization at cholesterol-rich membrane domains.

170 tosis that has been shown to colocalize with cholesterol-rich membrane domains.

171 active as long as GPI-ACE was sequestered in cholesterol-rich membrane domains.

172 anifestations of receptor co-localization in cholesterol-rich membrane domains.

173 for the formation of signaling platforms in cholesterol-rich membrane domains.

174 T adopts an outward facing conformation in a cholesterol-rich membrane environment, suggesting a nove

175 naptosomes and transfected cells, DAT was in cholesterol-rich membrane fractions after mild detergent

176 mitoyl-sn-glycero-3-phosphocholine (DPPC) in cholesterol-rich membrane leaflets.

177 lex of Toxoplasma is immobilized within this cholesterol-rich membrane likely extends to closely rela

178 cells, suggesting that it may partition into cholesterol-rich membrane microdomains (lipid rafts), it

179 Disruption of cholesterol-rich membrane microdomains by acute exposure

180 graphy and remain intact after disruption of cholesterol-rich membrane microdomains by methyl-beta-cy

181 Exosome uptake depends on cholesterol-rich membrane microdomains called lipid raft

182 The targeting of ion channels to cholesterol-rich membrane microdomains has emerged as a

183 Detection of cholesterol-rich membrane microdomains is confirmed by o

184 nding, TrkA translocates and concentrates in cholesterol-rich membrane microdomains or lipid rafts, f

185 Our results indicate that cholesterol-rich membrane microdomains play a role in tr

186 Neither luminal acidification nor cholesterol-rich membrane microdomains play essential ro

187 ex to stabilize the BCR in sphingolipid- and cholesterol-rich membrane microdomains termed lipid raft

188 Lipid rafts are cholesterol-rich membrane microdomains that are thought

189 t well characterized, and the involvement of cholesterol-rich membrane microdomains, or lipid rafts,

190 A subset of BACE1 localizes to cholesterol-rich membrane microdomains, termed lipid raf

191 Disruption of cholesterol-rich membrane microdomains, the localization

192 hannel function by regulating trafficking to cholesterol-rich membrane microdomains.

193 ne-targeted form of the protein to reside in cholesterol-rich membrane microdomains.

194 interactions in vivo with sphingolipid- and cholesterol-rich membrane microdomains.

195 esynaptic termini is dependent on sorting to cholesterol-rich membrane microdomains.

196 mine efflux and enhances DAT localization in cholesterol-rich membrane microdomains.

197 ies have suggested that prestin localizes in cholesterol-rich membrane microdomains.

198 The ordered environment of cholesterol-rich membrane nanodomains is thought to excl

199 endodomain for an efficient interaction with cholesterol-rich membrane patches.

200 and NP have been shown to be concentrated in cholesterol-rich membrane raft domains, whereas M2, alth

201 seudomonas aeruginosa, which is initiated by cholesterol-rich membrane rafts and is dependent on Lyn,

202 ut) p75(NTR) differ in their partitioning in cholesterol-rich membrane regions upon nerve growth fact

203 Here we show that this platform is a cholesterol-rich membrane structure.

204 the interaction of a bacterial toxin with a cholesterol-rich membrane.

205 the situation in the bilayer regions of this cholesterol-rich membrane.

206 ing that oncogenic Akt is overrepresented in cholesterol-rich membranes compared with wild-type Akt.

207 Cs) assemble their giant beta-barrel pore in cholesterol-rich membranes has been the subject of inten

208 nificantly loosens both cholesterol-poor and cholesterol-rich membranes made from DPPC.

209 for membranes in general and preference for cholesterol-rich membranes may account for its great neu

210 hat the translocation of anionic NPs through cholesterol-rich membranes must be accompanied by format

211 corrals up to 48 copies of PLY, targets the cholesterol-rich membranes of liposomes and red blood ce

212 Cholesterol-rich membranes play a pivotal role in cancer

213 essed the role of cPLA2 in the regulation of cholesterol-rich membranes that contain glycosylphosphat

214 Streptococcus intermedius, does not bind to cholesterol-rich membranes unless they contain the human

215 Treatment of cholesterol-rich membranes with a series of beta cyclode

216 ve Rac1 binds preferentially to low-density, cholesterol-rich membranes, and specificity is determine

217 that the MBP preferentially associates with cholesterol-rich membranes, and we find that cholesterol

218 PITP exhibits a preference for CerPCho- and cholesterol-rich membranes, we prepared unilamellar vesi

219 o human CD59 (hCD59) rather than directly to cholesterol-rich membranes.

220 transduction when the protein is present in cholesterol-rich membranes.

221 ng the specific interaction of the CDCs with cholesterol-rich membranes.

222 pendent on Src, reactive oxygen species, and cholesterol-rich membranes.

223 able to induce higher membrane curvature in cholesterol-rich membranes.

224 ts increased affinity for, and retention in, cholesterol-rich membranes.

225 ), and lipid rafts/caveolae (plasma membrane cholesterol-rich microdomain purified by a nondetergent

226 with flexible actin networks or sphingolipid-cholesterol rich microdomains in live cell membranes.

227 ked with anti-IgE, molecules associated with cholesterol-rich microdomains (e.g., saturated lipids (t

228 Whether cholesterol-rich microdomains (lipid rafts/caveolae) are

229 Cholesterol-rich microdomains (or "lipid rafts") within

230 nd tumor Ag were localized in membrane lipid cholesterol-rich microdomains and are thought to belong

231 These data suggest that (1) disruption of cholesterol-rich microdomains and caveolae by MCD leads

232 F-R transactivation by angiotensin II and 2) cholesterol-rich microdomains as well as focal adhesions

233 g hair cells was in keeping with the reduced cholesterol-rich microdomains in matured hair cells.

234 g domains in guinea pig endothelium and that cholesterol-rich microdomains in these cells can respond

235 MOG into TX-100-insoluble glycosphingolipid-cholesterol-rich microdomains initiates specific cellula

236 membrane and that the association with these cholesterol-rich microdomains is important for excitator

237 Alterations in plasma membrane lipids and cholesterol-rich microdomains might also contribute to a

238 entiation, and apoptosis is initiated in the cholesterol-rich microdomains of the plasma membrane kno

239 Additionally, Ply(WT) localized to cholesterol-rich microdomains on the HCEC surface, howev

240 ement, consistent with CFTR recruitment into cholesterol-rich microdomains with dimensions below the

241 e demonstrate that PI(4,5)P2 is localized to cholesterol-rich microdomains, lipid rafts, and the acti

242 aling are coordinated processes that require cholesterol-rich microdomains, whereas IC signaling is a

243 cholesterol content and, potentially, intact cholesterol-rich microdomains.

244 and Smoothened might also be found in these cholesterol-rich microdomains.

245 ns known to associate with sphingolipid- and cholesterol-rich microdomains.

246 1 cells reside in the low-density lipid (ie, cholesterol-rich) microdomains (lipid rafts).

247 Nanometer-scale domains in cholesterol-rich model membranes emulate lipid rafts in

248 Further, incubation in cholesterol-rich mouse serum resulted in the formation o

249 sclerosis (MS) necessitates the clearance of cholesterol-rich myelin debris by microglia/macrophages

250 Engulfment of cholesterol-rich myelin debris endows subsets of TAMs to

251 like transition, wherein macrophages feed on cholesterol-rich myelin debris to provide lipids to mese

252 form supramolecular assemblies that exclude cholesterol-rich nanodomains, increase membrane fluidity

253 t strikingly different binding properties to cholesterol-rich natural and synthetic membranes.

254 er some conditions, a sphingomyelin (SM) and cholesterol-rich ordered domain in one leaflet induces o

255 other GPI-anchored proteins can be found in cholesterol-rich ordered domains within the plasma membr

256 n added to zwitterionic membranes containing cholesterol-rich ordered domains, PrP(106-126) oligomers

257 The discovery that macrophages release cholesterol-rich particles during cellular locomotion is

258 nts was hampered, leading to accumulation of cholesterol-rich particles in the circulation.

259 macrophages release large numbers of ~30-nm cholesterol-rich particles.

260 parameters, although the composition of the cholesterol-rich phase varies as a function of the lipid

261 e identified as a phospholipid-rich phase, a cholesterol-rich phase, and a condensed complex-rich pha

262 Lipid-lipid interactions across cholesterol-rich phospholipid bilayers were investigated

263 ncipal protein component of triglyceride and cholesterol-rich plasma lipoproteins.

264 Furthermore, 121 proteins from cholesterol-rich plasma membrane domains (caveolar and l

265 Recently, sphingolipid- and cholesterol-rich plasma membrane lipid microdomains, ter

266 In this report, we investigate the role of cholesterol-rich plasma membrane microdomains (caveolae

267 ted PP2A, and PP2A/Balpha are co-enriched in cholesterol-rich plasma membrane microdomains/rafts puri

268 Because cholesterol-rich, plasma membrane rafts serve as platfor

269 ffect of flow was inhibited by disruption of cholesterol-rich plasmalemma domains and deletion of PEC

270 tially partition into sphingomyelin-rich and cholesterol-rich plasmalemmal microdomains, thereby acqu

271 Thus CD47-alpha(v)beta(3) complexes in cholesterol-rich raft domains appear to engage in G(i)-d

272 imulations in the presence of a pure POPC or cholesterol-rich raft model membrane.

273 22, but not a demyristoylated form, binds to cholesterol-rich raft-like domains in planar-supported m

274 sing PrP-sen reconstituted into sphingolipid-cholesterol-rich raft-like liposomes (SCRLs).

275 nraft) domains and excludes DHA from SM-rich/cholesterol-rich (raft) domains.

276 sion molecule of Entamoeba, were enriched in cholesterol-rich (raft-like) fractions, whereas EhCP5, a

277 Sphingolipid/cholesterol-rich rafts are membrane domains thought to e

278 Both CD47 and IgV-GPI were found in cholesterol-rich rafts prepared in the absence of deterg

279 Signaling cascades that localize to cholesterol-rich regions of the plasma membrane include

280 ysical aspects of the membrane, particularly cholesterol-rich regions, in spike-mediated fusion, whic

281 of lipids in the outer leaf increases in the cholesterol-rich regions, so the areal density of lipids

282 that NAP-22 binding may be employed to image cholesterol-rich regions, such as caveolae/rafts, on the

283 ficient mice resulted in the accumulation of cholesterol-rich remnant lipoproteins in the circulation

284 addressed by showing that sphingomyelin and cholesterol-rich (SCOR) lipid mixtures with phosphatidyl

285 myelin induction by stimulating formation of cholesterol-rich signaling domains between oligodendrocy

286 endothelial P2Y receptors are organized into cholesterol-rich signaling domains, such as caveolae and

287 suggest that the functional organization of cholesterol-rich signaling microdomains allows agonist-s

288 sterol, a role of potential significance for cholesterol-rich tissues with high oxidative stress.

289 The balance of cholesterol-rich to local hexagonal order is proposed to

290 Sphingolipid and cholesterol-rich Triton X-100-insoluble membrane fragmen

291 suggests that impaired hepatic clearance of cholesterol-rich TRL remnants leads to their accumulatio

292 ially associated with actin and localized in cholesterol-rich vesicles.

293 2 orders of magnitude, to <10(3) s(-1), in a cholesterol-rich virus envelope-mimetic membrane ("viral

294 ses a high-curvature isotropic phase to both cholesterol-rich virus-mimetic membranes and 1,2-dimyris

295 atial contacts within ~2 nm, are observed in cholesterol-rich virus-mimetic membranes but are suppres

296 nse oligonucleotides to Apoe(-/-) mice fed a cholesterol-rich (Western) diet for 8 weeks.

297 or anionic bacterial membranes as opposed to cholesterol-rich zwitterionic mammalian membranes.