ライフサイエンスコーパス: choroidal

1 luating patient response to therapy, because choroidal activity seems to mimic systemic activity.

2 gnificant difference between the retinal and choroidal anatomical OCT outcomes, rates of polyp closur

3 Concomitant less than 3 mm choroidal and any prelaminar/laminar optic nerve invasio

4 ilateral optic disc edema, globe flattening, choroidal and retinal folds, hyperopic refractive error

5 ) can be an effective method to evaluate the choroidal and retinal microvasculature.

6 Here, we attempt to explore choroidal and retinal vascular compliance in patients wi

7 These findings suggest choroidal angioarchitecture declines from the 4th decade

8 hown to be sensitive in detecting changes in choroidal angioarchitecture in a range of ocular disease

9 Models of retinal and choroidal angiogenesis, including oxygen-induced retinop

10 aled subtly changed, decreased FAF while the choroidal architecture recovered completely as demonstra

11 (SFCT), total choroidal area (TCA), luminal choroidal area (LCA), and stromal choroidal area (SCA) w

12 ), luminal choroidal area (LCA), and stromal choroidal area (SCA) were analyzed.

13 Total choroidal area (TCA), luminal area (LA), and the choroid

14 subfoveal choroidal thickness (SFCT), total choroidal area (TCA), luminal choroidal area (LCA), and

15 Total choroidal area, luminal area and stromal area were all s

16 e posterior cerebral artery and the anterior choroidal artery or a single supply by the posterior cer

17 40 (95% CI: 1.50-41.10; P = .031); and inner choroidal attenuation had an HR of 13.20 (95% CI: 1.07-3

18 interval [CI]: 1.10-37.24; P = .040); inner choroidal attenuation had an HR of 9.66 (95% CI: 1.07-22

19 yporeflective flat, irregular PED; and inner choroidal attenuation.

20 ncrease of blood pressure is consistent with choroidal blood flow dysregulation in patients with CSCR

21 cells represent a minority of the total RPE/choroidal cell population but are strongly implicated in

22 xpression profiles specific to RPE and major choroidal cell populations were identified.

23 The SS-OCT was evaluated for choroidal changes.

24 s carried out in 30 eyes of 20 patients with choroidal coloboma.

25 Eyes with choroidal complications diagnosed later than 35 days aft

26 e the risk of potentially vision-threatening choroidal complications.

27 nerve involvement; and 15 had focal (< 3 mm) choroidal concomitant with lamina or prelamina optic ner

28 ing that the pathogenesis of PCV may include choroidal congestion and dilatation.

29 ection was applied on B-scans to enhance the choroidal contrast and facilitate more accurate automati

30 ificant as a range of ocular conditions with choroidal degeneration are associated with aging.

31 (n = 1; 2.3%), wound leak (n = 1; 2.3%), and choroidal detachment (n = 2; 4.5%).

32 t, retinal tear, macular edema, glaucoma, or choroidal detachment-and used adjusted regression models

33 mined that these differences were due to the choroidal disease subgroup, which demonstrates significa

34 ring the number of age-related diseases with choroidal dysfunction, these results provide foundationa

35 dema (10%), corneal decompensation (6%), and choroidal effusion (4%).

36 P increase (n = 9), pupillary block (n = 1), choroidal effusion (n = 2), CME (n = 4), and redislocati

37 Development of myopia, lens thickening, choroidal effusion and retinal striae at the macula with

38 ae may be caused by the volume effect of the choroidal effusion.

39 Spectral-domain OCT findings included choroidal elevation and a double-layer sign (separation

40 As the choroidal elevation decreased on OCT, increased reflecta

41 revealed scleral thickening with peripheral choroidal elevation leading to the diagnosis of UES.

42 eyes with severe inflammation and increased choroidal elevation on OCT, the double-layer sign was ve

43 ist neural sensory retinal angiogenesis from choroidal endothelial cell invasion.

44 igmented epithelial (RPE), and-more recently-choroidal endothelial cells has grown exponentially.

45 As choroidal endothelial cells represent a minority of the

46 e tissue-specific transcriptomic features of choroidal endothelial cells.

47 We found that choroidal endothelium adjacent to the RPE expresses high

48 ion size, the presence of satellite lesions, choroidal excavation, and choroidal lacunae (large choro

49 ion size, the presence of satellite lesions, choroidal excavation, and choroidal lacunae can provide

50 ning at baseline (P = 0.005) and showed less choroidal expansion at 1 hour and 3 hours after sildenaf

51 Central macular thickness (CMT) and choroidal features including subfoveal choroidal thickne

52 The CNV area of choroidal flatmounts was evaluated by immunostaining wit

53 ous visual impairment, retinal thinning, and choroidal flow defects.

54 0%), terminal bulbing in 6 (14.6%), abnormal choroidal flush in 3.5 (8.5%), and abnormal vessel strai

55 PRSS56 families were more likely to have choroidal folds than other solved families, while MFRP f

56 tudy were to further define the functions of choroidal gammadelta T cells and to explore the underlyi

57 The diffuse choroidal hemangioma (DCH) was characterized by loss of

58 Management of choroidal hemangioma in the PDT era has allowed for sign

59 Patients with circumscribed choroidal hemangioma treated with PDT were identified, a

60 -nine eyes of 79 patients with circumscribed choroidal hemangioma were treated with PDT.

61 3) of the port wine stain and in the diffuse choroidal hemangioma.

62 effective treatment method for circumscribed choroidal hemangioma.

63 were found in 100% (6 of 6) of the solitary choroidal hemangiomas and (c.626A > C; p.Gln209Pro) in t

64 By contrast, solitary choroidal hemangiomas have mutations in the Q209 codon,

65 An R183 codon is mutant in diffuse choroidal hemangiomas, consistent with other Sturge-Webe

66 th Sturge-Weber syndrome [SWS]) and solitary choroidal hemangiomas.

67 sion (54%), subretinal hemorrhage (51%), and choroidal hemorrhage (30%).

68 ing endophthalmitis, retinal detachment, and choroidal hemorrhage following EK procedures is low.

69 cidence of postoperative endophthalmitis and choroidal hemorrhage following EK was 0.03% and 0.05%, r

70 rative complications (i.e., endophthalmitis, choroidal hemorrhage, infectious keratitis, cystoid macu

71 wever mechanisms that maintain or compromise choroidal homeostasis are obscure.

72 complete retinal reattachment, reduction of choroidal hyperpermeability on ICGA and improvement of v

73 rformed with unremarkable results except for choroidal hyperpermeability on indocyanine green angiogr

74 vely correlated with choroidal thickness and choroidal hyperpermeability, supporting that the pathoge

75 trols with emmetropia in both eyes underwent choroidal imaging using spectral-domain optical coherenc

76 RPE/choroid and uncover a Hedgehog-regulated choroidal immunomodulatory signaling circuit.

77 ,15), venous stasis retinopathy (VSR; 10,2), choroidal infarction (0,1), and branch retinal artery oc

78 ome contributed to both the acute unilateral choroidal infarction and to the chronic development of b

79 ude CNVM, ME, SRF, VSR, chorioretinal folds, choroidal infarction, and BRAO.

80 ion of Igf1r expression, and preservation of choroidal integrity.

81 High-risk eyes demonstrated massive choroidal invasion (4/38) or trans-scleral, extraocular,

82 concomitant greater than 3 mm peripapillary choroidal invasion and 1.5 mm or greater of postlaminar

83 retinal vasculitis (374 of 874 [42.8%]), and choroidal involvement (419 of 651 [64.4%]).

84 , respectively) and uveitis patients without choroidal involvement (785 mum(2); P < .0001; 821 mum(2)

85 Little is known about choroidal involvement in anterior uveitis.

86 provide foundational knowledge to understand choroidal involvement in these diseases.

87 her subdivided by the presence or absence of choroidal involvement, and quantitative metrics were com

88 ics of control subjects and patients without choroidal involvement.

89 marily retinal in location without report of choroidal involvement.

90 Acute choroidal ischemia, defined as any new choroidal ischemia clinically diagnosed within 35 days a

91 Acute choroidal ischemia developed in 35 of 206 included eyes

92 Acute choroidal ischemia, defined as any new choroidal ischemi

93 satellite lesions, choroidal excavation, and choroidal lacunae (large choroidal vessels) on SSOCT dif

94 ), deeply excavated lesions (n = 11/15) with choroidal lacunae (n = 8/15).

95 satellite lesions, choroidal excavation, and choroidal lacunae can provide a clue toward the etiology

96 helium plus drusen (RPE+drusen) complex, and choroidal layers from each sector of the standard macula

97 eased on OCT, increased reflectance from the choroidal layers was evident.

98 phy revealed numerous additional subclinical choroidal lesions and were used to track disease respons

99 to demonstrate the nature of the retinal and choroidal lesions in each participant and the extent of

100 All 4 patients demonstrated bilateral choroidal lesions on funduscopy and evidence of osteomye

101 Choroidal lesions were identified and analyzed in four o

102 and RPE cells, implicating insufficiency in choroidal macrophage function as a factor in aging- and

103 These findings suggest that choroidal macrophages play a previously unappreciated tr

104 We discovered that the ablation of choroidal macrophages via CSF1R blockade was associated

105 tients who underwent enucleation surgery for choroidal malignant melanomas.

106 dgehog signaling induced significant loss of choroidal mast cells, as well as an altered inflammatory

107 results demonstrate the fundamental role of choroidal MC involvement in GA disease etiology, and wil

108 phenotypes of GA, were driven by continuous choroidal MC stimulation and activation in a slow releas

109 elated with the ERs (P = .03), whereas other choroidal measurements had no significant correlation wi

110 Choroidal melanocytes (HCMs) are melanin-producing cells

111 le (class 1 or class 2) was performed in 207 choroidal melanocytic tumors < 3.5 mm in thickness.

112 ickness may be helpful for identifying small choroidal melanocytic tumors that are more likely to hav

113 cology services submitted 45 patients with a choroidal melanoma 3 mm or less in thickness and 9 mm or

114 s of 57 patients with a clinically diagnosed choroidal melanoma underwent complete 25-gauge posterior

115 treated with primary proton beam therapy for choroidal melanoma with a minimum follow-up of 24 months

116 hough treatment of small pigmented posterior choroidal melanoma with PDT effectively preserves visual

117 of extrascleral extension in a patient with choroidal melanoma" Jacobsen et al. describe a case in w

118 tic management of extra scleral extension in choroidal melanoma".

119 l melanocytic tumors (iris melanoma, ciliary-choroidal melanoma, and melanocytoma) and retinal pigmen

120 ofrin is recommended in exceptional cases of choroidal melanoma, for which other treatments with grea

121 RPE adenoma/adenocarcinoma can simulate choroidal melanoma.

122 to normal structures in the eye after PT for choroidal melanoma.

123 Choroidal melanomas less than 3.0 mm in LBD are highly u

124 tors of an SFCM are similar to those for all choroidal melanomas of similar size.

125 icities after eye-preserving radiotherapy of choroidal melanomas.

126 ective outpatient method for small to medium choroidal metastatic tumors, providing tumor control in

127 m baseline, (3) time to first grading of the choroidal neovascular lesion as inactive, and (4) maximu

128 onger documented period of inactivity of the choroidal neovascular lesion with no further treatments

129 l folds (30,68), macular exudate (ME; 20,5), choroidal neovascular membrane (CNVM; 10,15), venous sta

130 Choroidal neovascular membrane can also be a late compli

131 in clinical characteristics, for example, in choroidal neovascular membrane development and treatment

132 mic disease, however one of them developed a choroidal neovascular membrane.

133 None of the eyes showed a choroidal neovascular membrane.

134 Hence, perilesional nonexudative choroidal neovascular tissue (presumably present before

135 led in the Inhibition of VEGF in Age-related choroidal Neovascularisation (IVAN) trial; after excludi

136 ), RPE disruption (77% vs 3%, P < .001), and choroidal neovascularization (16% vs 0%, P = .028).

137 Baseline occult-type choroidal neovascularization (CNV) (P = .0156) and retin

138 glia and macrophages play a critical role in choroidal neovascularization (CNV) and may, therefore, b

139 age-related macular degeneration-associated choroidal neovascularization (CNV) and VA between 20/25

140 cholesterolemia, worse visual acuity, larger choroidal neovascularization (CNV) area, retinal angioma

141 The mean VA and the type of the choroidal neovascularization (CNV) at the start of treat

142 Choroidal neovascularization (CNV) can complicate AMD an

143 flammation have been implicated in promoting choroidal neovascularization (CNV) in age-related macula

144 s deciphering the transcriptional profile of choroidal neovascularization (CNV) in body donor eyes wi

145 tients in Proxima A had bilateral GA without choroidal neovascularization (CNV) in either eye (N = 29

146 Choroidal neovascularization (CNV) in pregnancy has rare

147 ified: GA in both eyes (GA:GA); GA in 1 eye, choroidal neovascularization (CNV) in the fellow eye (GA

148 low eye statuses were analyzed: (1) no GA or choroidal neovascularization (CNV) in the fellow eye, (2

149 al active Rap1a expression and inhibition of choroidal neovascularization (CNV) induced by laser inju

150 Choroidal neovascularization (CNV) is a major cause of v

151 Choroidal neovascularization (CNV) is a prevalent cause

152 Choroidal neovascularization (CNV) is the major cause of

153 Myopic choroidal neovascularization (CNV) is the most common si

154 Choroidal neovascularization (CNV) leads to loss of visi

155 ither pericytes or astrocytes, laser-induced choroidal neovascularization (CNV) was significantly red

156 es are the main infiltrating immune cells in choroidal neovascularization (CNV), a hallmark of the hu

157 ith AMD also lose vision as a consequence of choroidal neovascularization (CNV).

158 outcomes in eyes with treatment-naive myopic choroidal neovascularization (mCNV) in the United States

159 econd eye had angioid streaks complicated by choroidal neovascularization and underwent prior thermal

160 ent-naive patients also showed no detectable choroidal neovascularization at week 12 on fluorescein a

161 Choroidal neovascularization developed in 1 eye in the P

162 Patients (N = 1817) with untreated, active choroidal neovascularization due to age-related macular

163 pressed initiation and growth of spontaneous choroidal neovascularization in a mouse model, and the c

164 ithout evidence of or previous treatment for choroidal neovascularization in either eye and best-corr

165 sunitinib microparticles potently suppresses choroidal neovascularization in mice for six months and

166 lipsoid zone disruption, RPE disruption, and choroidal neovascularization in peripapillary combined h

167 red for endothelial cell growth in vitro and choroidal neovascularization in vivo.

168 ffect on both area and number of spontaneous choroidal neovascularization lesions.

169 in angiograms and OCT images were graded for choroidal neovascularization type.

170 8; P = 0.0005), and more visits at which the choroidal neovascularization was graded as active (OR, 1

171 No signs of choroidal neovascularization were observed.

172 ed injury that was associated with increased choroidal neovascularization, a hallmark of advanced exu

173 inal pigment epithelium (RPE) abnormalities, choroidal neovascularization, acquired vitelliform lesio

174 cted data on VA, OCT changes, development of choroidal neovascularization, and length of follow-up.

175 ng oxygen-induced retinopathy, laser-induced choroidal neovascularization, and transgenic mouse model

176 models with deficient or spontaneous retinal/choroidal neovascularization, as well as models with ind

177 at is commonly misdiagnosed as disc edema or choroidal neovascularization.

178 ng choroidal vessels at the site of baseline choroidal neovascularization.

179 al Best vitelliform dystrophy and unilateral choroidal neovascularization.

180 use of intravitreal anti-VEGF for traumatic choroidal neovascularizations (CNV).

181 features from pathologic RPE detachments and choroidal neovascularizations (CNVs).

182 14 women; mean age, 45.1+/-13.4 years) with choroidal nevi and 14 healthy age-matched volunteers (24

183 nstrated excellent potential for identifying choroidal nevi and was in full agreement with convention

184 Retrospective analysis of choroidal nevi imaged with SS OCT and NIR.

185 Choroidal nevi present distinct patterns according to SS

186 Five distinct EDI-OCT patterns of choroidal nevi seemed flat on ultrasonography, and many

187 Of 104 choroidal nevi, 97 lesions (93.3%) could be classified i

188 structed to evaluate the ability to identify choroidal nevi.

189 reasonable for most patients with suspicious choroidal nevi.

190 nal methods in the evaluation of the area of choroidal nevi.

191 None of the widely used choroidal nevus risk factors for tumor growth, nor docum

192 ngiomas and (c.626A > C; p.Gln209Pro) in the choroidal nevus.

193 Presence of CMvD was evaluated on choroidal OCTA slabs.

194 vestigate the diagnostic usefulness of these choroidal parameters in a myriad of ocular and systemic

195 These choroidal parameters may provide additional quantitative

196 In the fellow eye group, VA, CMT, and all choroidal parameters showed no differences between basel

197 Association of choroidal parameters with AD, MCI, or control subjects w

198 To characterize choroidal thickness and choroidal reflectivity in the eyes of patients with bird

199 covers a significant mechanism for defective choroidal revascularization in OIR, revealing a new role

200 But the mechanism underlying the lack of choroidal revascularization remains unclear and was inve

201 ion, restored Igf1r expression, and elicited choroidal revascularization.

202 Fifty-four patients with unilateral choroidal rupture after ocular trauma diagnosed between

203 ase series of patients with CNV secondary to choroidal rupture after ocular trauma receiving intravit

204 Eleven patients with CNV secondary to choroidal rupture were identified.

205 e safe and effective for secondary CNV after choroidal rupture.

206 ckness (MCT), choroidal vessel volume (CVV), choroidal stroma volume (CSV), choroid vascularity index

207 V), choroid vascularity index (CVI), and the choroidal stroma-to-vessel volume ratio (CSVR).

208 The choroidal thickening appeared to be primarily attributed

209 To identify changes in choroidal thickness (CT) and all retinal layers of diabe

210 T), outer retinal thickness (ORT), subfoveal choroidal thickness (CT), temporal and nasal CT, foveal

211 NFL), the ganglion cell layer (GCL), and the choroidal thickness (CT).

212 riocapillaris (CC) flow deficits (FDs), mean choroidal thickness (MCT), and choroidal vascularity ind

213 arameters that we investigated included mean choroidal thickness (MCT), choroidal vessel volume (CVV)

214 en subtypes, fibrosis, atrophy and subfoveal choroidal thickness (SFCT) of both eyes in patients with

215 One week after surgery, the subfoveolar choroidal thickness (SFCT) significantly increased (372

216 Subfoveal choroidal thickness (SFCT) was measured by 2 masked grad

217 ) and choroidal features including subfoveal choroidal thickness (SFCT), total choroidal area (TCA),

218 ly significant except for the association of choroidal thickness and "peripheral vision." The stronge

219 CVD was positively correlated with choroidal thickness and choroidal hyperpermeability, sup

220 To characterize choroidal thickness and choroidal reflectivity in the ey

221 Negative correlations between age and both choroidal thickness and choroidal volume were detected i

222 While choroidal thickness and choroidal volume, which includes

223 of this study was to evaluate the changes in choroidal thickness and lamina cribrosa position after n

224 choroidal vascular pattern, increase in the choroidal thickness and loss of visualization of the scl

225 aim of our study was to evaluate changes in choroidal thickness and volume in eyes with acute anteri

226 Subfoveal choroidal thickness at 11 years of age did not predict i

227 The choroidal thickness at the foveal center was measured.

228 There was no statistical difference in the choroidal thickness between the different time points (b

229 etinal pigment epithelium (RPE) changes, and choroidal thickness in both eyes.

230 fluid is associated with increased subfoveal choroidal thickness in surgical and fellow eyes and with

231 After sildenafil, choroidal thickness increased by 6.0% to 9.0% at 1 and 3

232 e logistic regression analysis revealed that choroidal thickness of 280 mum or more was a significant

233 No statistically significant differences in choroidal thickness or choroidal volume were detected be

234 The choroidal thickness thinned at a rate of -1.09 mum/y dur

235 Baseline choroidal thickness was 172.1 +/- 60.0 mum in non-exudat

236 measured using an interferometric device and choroidal thickness was measured by spectral-domain opti

237 or lamina cribrosa surface depth (ALCSD) and choroidal thickness were by automated segmentation of sp

238 ayer thickness, in addition to peripapillary choroidal thickness were measured.

239 Choroidal thickness, choroidal vascularity, and retinal

240 wide range of individual rates of change of choroidal thickness, from -20.00 to 17.09 mum/year (mean

241 depth imaging (OCT-EDI) was used to measure choroidal thickness, prelaminar tissue thickness and lam

242 ad "hyperreflectivity confined within normal choroidal thickness," 16.0% had characteristic anteriorl

243 cluding age, intraocular pressure (IOP), and choroidal thickness.

244 The mean choroidal thicknesses were not significantly different a

245 yes from older subjects were associated with choroidal thinning at baseline (P = 0.005) and showed le

246 Recently, choroidal thinning has also been detected in children fo

247 Our study suggests that choroidal thinning in eyes with high myopia is associate

248 erly diagnosed with ROP; a similar sustained choroidal thinning is observed in ROP models.

249 thelium degeneration followed by retinal and choroidal thinning, characteristic phenotypes of GA, wer

250 Multivariable mixed modeling showed that choroidal thinning, lower IOP change, and lower corneal

251 hages via CSF1R blockade was associated with choroidal vascular atrophy and retinal pigment epithelia

252 l imaging findings in AMN, with attention to choroidal vascular changes.

253 These findings support the concept that choroidal vascular degeneration, predominantly in the mi

254 Choroidal vascular density (CVD) of PCV eyes was higher

255 capillaris response to complement injury and choroidal vascular disease in age-related macular degene

256 dal choroidal vasculopathy (PCV) is a common choroidal vascular disease particularly in Asians.

257 ers may be useful to quantitatively evaluate choroidal vascular dysfunction in A-AION, serving as a n

258 gioma (DCH) was characterized by loss of the choroidal vascular pattern, increase in the choroidal th

259 ntified gene expression signatures along the choroidal vascular tree, classifying the transcriptome o

260 The choroidal vascularity index (CVI) has been shown to be s

261 Choroidal vascularity index (CVI) was calculated in all

262 oidal area (TCA), luminal area (LA), and the choroidal vascularity index (CVI) were calculated after

263 s (FDs), mean choroidal thickness (MCT), and choroidal vascularity index (CVI) were investigated usin

264 Choroidal thickness, choroidal vascularity, and retinal vessel density were m

265 changes at the level of the optic nerve and choroidal vascularization during acute IOP changes.

266 ly unappreciated trophic role in maintaining choroidal vasculature and RPE cells, implicating insuffi

267 ully elucidated, and the correlation between choroidal vasculature and treatment response of PCV are

268 rognosis of PCV by quantitatively evaluating choroidal vasculature from the entire fundus using ultra

269 angiography revealed dilated hyperpermeable choroidal vasculature on the nasal side of the fovea in

270 determine the subtypes according polypoidal choroidal vasculopathy (PCV) and non-PCV subtypes.

271 Polypoidal choroidal vasculopathy (PCV) is a common choroidal vascu

272 Polypoidal choroidal vasculopathy (PCV) is a variant of neovascular

273 both typical neovascular AMD and polypoidal choroidal vasculopathy (PCV) over 12-months.

274 l ranibizumab in the treatment of polypoidal choroidal vasculopathy (PCV).

275 Polypoidal choroidal vasculopathy is more prevalent in anti-VEGF-re

276 Polypoidal choroidal vasculopathy is more prevalent in Asian patien

277 Polypoidal choroidal vasculopathy was diagnosed in 50% (60/120 eyes

278 Polypoidal choroidal vasculopathy was diagnosed using indocyanine g

279 Polypoidal choroidal vasculopathy was noted in 51.6% (81/157) of ey

280 Polypoidal choroidal vasculopathy was noted to have a prevalence of

281 automatic segmentation of the 3-dimensional choroidal vessel and stroma.

282 Choroidal vessel density may be affected early in the co

283 Correlations between MCT and choroidal vessel metrics of CVV, CSV, CVI, and CSVR were

284 fibrosis, shape, retinal vessel pattern, and choroidal vessel visibility did not vary significantly.

285 ted included mean choroidal thickness (MCT), choroidal vessel volume (CVV), choroidal stroma volume (

286 Choroidal vessel volume and CSV showed significant corre

287 tool to detect the reduced perfusion of the choroidal vessels and for monitoring the disease course.

288 ss and choroidal volume, which includes both choroidal vessels and stroma, decrease with age (all P <

289 opigmentation without exposure of underlying choroidal vessels at the site of baseline choroidal neov

290 athologies such as impaired perfusion of the choroidal vessels can recover spontaneously over a perio

291 The image signal from the retinal and choroidal vessels in living rabbits was enhanced by up t

292 r also caused displacement of the deep large choroidal vessels over the superior macular area even af

293 l MA (>=175 mum greatest linear dimension of choroidal vessels seen on FA and/or color, aided by OCT)

294 dal excavation, and choroidal lacunae (large choroidal vessels) on SSOCT differed significantly among

295 l irregularity with narrowing of retinal and choroidal vessels.

296 ry, which showed early visualization of deep choroidal vessels.

297 ficant differences in choroidal thickness or choroidal volume were detected between AAU eyes at basel

298 between age and both choroidal thickness and choroidal volume were detected in AAU eyes at baseline a

299 Subfoveal thickness and choroidal volume were measured with EDI-OCT in nine Earl

300 While choroidal thickness and choroidal volume, which includes both choroidal vessels