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1 ic vesicle, and a medium size vesicle in the chromaffin cell.
2 mice, we find that NPY is synthesized by all chromaffin cells.
3 es in a millisecond time resolution in mouse chromaffin cells.
4 successfully from multiple individual living chromaffin cells.
5 AP-25 x synaptotagmin-1 interaction in mouse chromaffin cells.
6 iNW-FET) to detect the K(+)-efflux from live chromaffin cells.
7 holamines released from small populations of chromaffin cells.
8 echolamines, exclusively, from fetal adrenal chromaffin cells.
9 iated fusion pore expansion in mouse adrenal chromaffin cells.
10 ippocampus, dorsal root ganglia, and adrenal chromaffin cells.
11 augmented T-type Ca2+ current in IH-treated chromaffin cells.
12 correlation, confirming similar findings in chromaffin cells.
13 stimulated catecholamine release in cultured chromaffin cells.
14 spatial distribution of calcium channels in chromaffin cells.
15 d released by exocytosis in PC12 and primary chromaffin cells.
16 adapted from a model for fast exocytosis in chromaffin cells.
17 typical transmitter storage and release from chromaffin cells.
18 nd neuronal cells, including sympathoadrenal chromaffin cells.
19 on with a D1-like receptor on bovine adrenal chromaffin cells.
20 omosyn-syntaxin 1A complexes in live adrenal chromaffin cells.
21 nced catecholamine loading of LDCVs in mouse chromaffin cells.
22 type/reporter plasmids were transfected into chromaffin cells.
23 receptors on exocytosis from bovine adrenal chromaffin cells.
24 se frequency of catecholamine in dissociated chromaffin cells.
25 matory effects that depend on NPY(+) adrenal chromaffin cells.
26 ant (>90%) mode of secretion in calf adrenal chromaffin cells.
27 calcium-triggered catecholamine release from chromaffin cells.
28 Ca) was produced by P2Y receptors in adrenal chromaffin cells.
29 motions related to the secretory response in chromaffin cells.
30 e in the Ca2+-cooperativity of exocytosis in chromaffin cells.
31 ficant reduction in DCG formation in adrenal chromaffin cells.
32 human embryonic kidney HEK293-S3 and adrenal chromaffin cells.
33 is a rare, but clinically important tumor of chromaffin cells.
34 18-1/syntaxin1A interaction in HEK293-S3 and chromaffin cells.
35 ive anion equilibrium potential, depolarizes chromaffin cells.
36 coupling after this peptidergic stimulus to chromaffin cells.
37 1-L61) were transiently expressed in adrenal chromaffin cells.
38 secretory response triggered by the toxin in chromaffin cells.
39 athetic neurons appears to represent adrenal chromaffin cells.
40 K"- or "Maxi K"-encoding Slo gene in adrenal chromaffin cells.
41 synapses as well as secretory cells, such as chromaffin cells.
42 d," was confirmed by immunohistochemistry at chromaffin cells.
43 in the exocytotic membrane fusion process in chromaffin cells.
44 ranscriptional activation of the VIP gene in chromaffin cells.
45 wo sequential priming steps in mouse adrenal chromaffin cells.
46 ease and extrusion from vesicles at mast and chromaffin cells.
47 ar calcium release, and elevation of cAMP in chromaffin cells.
48 e nerve and form postsynaptic neuroendocrine chromaffin cells.
49 nergic stimulated catecholamine release from chromaffin cells.
50 uptake at the synaptic terminals and adrenal chromaffin cells.
51 NPY(+) noradrenergic neurons and/or adrenal chromaffin cells.
52 ventional active zones and in neuroendocrine chromaffin cells.
53 le priming and secretory amplitude in living chromaffin cells.
54 ion is severely inhibited in Ophn1 knock-out chromaffin cells.
55 hn1 knock-out mice and OPHN1-silenced bovine chromaffin cells.
56 ipid microdomains at the exocytotic sites in chromaffin cells.
57 he nAChR subtypes expressed by human adrenal chromaffin cells.
58 and is also essential for vesicle docking in chromaffin cells.
59 med during catecholamine exocytosis in mouse chromaffin cells.
60 nk exocytosis to compensatory endocytosis in chromaffin cells.
61 duced bulk endocytosis also occurs in bovine chromaffin cells.
62 tivator (tPA) (over many seconds) in adrenal chromaffin cells.
63 large, dense core vesicles (LDCVs) in mouse chromaffin cells.
65 e changes in the surface membrane of adrenal chromaffin cells after stimulation of exocytosis with a
66 e via desensitization/down-regulation of the chromaffin cell alpha(2)-adrenergic receptors that norma
67 n of neural crest-derived catecholaminergic (chromaffin) cells already associated with blood vessels
70 alogue enhances exocytosis in neuroendocrine chromaffin cells, altering the quantal release of catech
72 in the purified large dense-core vesicles of chromaffin cells and associated with synaptotagmin-1.
73 yme, phenyl-N-methyl transferase, by adrenal chromaffin cells and changes in cell cycle dynamics.
74 an directly affect the secretory capacity of chromaffin cells and contribute, in part, to elevated ca
77 and vesicular catecholamine transporters of chromaffin cells and facilitates localization of the pri
78 investigated this question in mouse adrenal chromaffin cells and found that SNAP-25 inhibits Ca(2+)
79 nergic receptors are found on bovine adrenal chromaffin cells and have been implicated in the facilit
80 (APWs) using both native channels in adrenal chromaffin cells and heterologously expressed channels i
81 irst study of EP receptor signaling in mouse chromaffin cells and identifies a molecular mechanism fo
83 tory capacity measured amperometrically from chromaffin cells and in the expression of tyrosine hydro
84 nd [Ca2+]i responses in neonatal rat adrenal chromaffin cells and involves reactive oxygen species (R
85 ly, many imprinted genes are up-regulated in chromaffin cells and may play key roles in their develop
88 repinephrine released from adrenal medullary chromaffin cells and norepinephrine released locally fro
89 mega-shaped structure in live neuroendocrine chromaffin cells and pancreatic beta-cells, visualized u
92 ibitor of catecholamine release from adrenal chromaffin cells and postganglionic sympathetic axons.
93 protein released from secretory granules of chromaffin cells and sympathetic nerves, triggers endoth
97 hrough voltage-gated Ca2+ channels in bovine chromaffin cells and the domain of this receptor variant
98 n the development of sympathetic neurons and chromaffin cells and the mechanisms involved in Lin28B-i
99 proximity to the surface of adherent bovine chromaffin cells and to amperometrically record single e
101 at controls epinephrine release from adrenal chromaffin cells and, consequently, hepatic glucose prod
102 that is found in neurons, platelets, adrenal chromaffin cells, and a few other neurosecretory cells.
103 epinephrine is released from adrenomedullary chromaffin cells, and compensatory glucogenesis ensues.
104 ed transcriptional mechanisms in transfected chromaffin cells, and concluded with observations on blo
105 e into melanocytes, parasympathetic neurons, chromaffin cells, and dental mesenchymal populations.
106 n of individual secretory granules in living chromaffin cells, and related their mobilities to postfu
110 onclude that sympathetic neurons and adrenal chromaffin cells are more vulnerable to diabetes than pa
111 findings demonstrate that the fetal adrenal chromaffin cells are the source for acute hypoxaemia-ind
112 y from neural crest precursors while adrenal chromaffin cells arise from neural crest-derived cells t
114 d catestatin is secreted from neuroendocrine chromaffin cells as an autocrine regulator of nicotine-s
115 hromocytoma cells and bovine adrenomedullary chromaffin cells as detected by Northern blotting, Weste
116 acitance measurements to probe exocytosis in chromaffin cells at low concentrations of intracellular
118 ion did not evoke action potential firing in chromaffin cells but did cause a persistent subthreshold
119 brane area through exocytosis does not swell chromaffin cells but may decrease membrane tension.
120 nt from mature secretory vesicles in adrenal chromaffin cells, but localizes to a compartment near th
121 ls (a rat insulinoma cell line) and cultured chromaffin cells, but not in AtT-20 cells (derived from
122 ediating SNARE-dependent exocytosis in mouse chromaffin cells, but the role of a closely related calc
123 or monitoring single vesicle exocytosis from chromaffin cells by constant potential amperometry as we
124 alin in secretory vesicles of neuroendocrine chromaffin cells by immunofluorescent confocal and immun
125 sponse element motif, an effect confirmed in chromaffin cells by site-directed mutagenesis on the tra
129 e was measured from bovine adrenal medullary chromaffin cell (CC) cultures maintained over a period o
132 o shrink the Omega-profile in neuroendocrine chromaffin cells containing approximately 300 nm vesicle
133 are increased by Sp and that elevate 5-HT in chromaffin cell cultures, suggesting direct metabolic si
134 tailed structure-function analysis in murine chromaffin cells demonstrates that linker motifs play a
135 describes the primary culture of individual chromaffin cells derived by enzymatic digestion from the
138 l sympathetic neuroblast hyperplasia, blocks chromaffin cell differentiation, and ultimately triggers
139 evoked secretion simultaneously from several chromaffin cells directly cultured on the device surface
140 owever, unlike hypothalamic nerve terminals, chromaffin cells do not display syntilla activation by d
141 synapses lacking Munc13s, the corresponding chromaffin cells do not exhibit a vesicle docking defect
143 techolamine biosynthetic capacity of adrenal chromaffin cells during periods of sustained catecholami
145 Application of etomidate directly to the chromaffin cells elicited robust catecholamine secretion
147 e) Ca2+ and voltage-dependent K+ channels in chromaffin cells exhibit an inactivation that probably a
149 ously reported that N-type current in bovine chromaffin cells exhibits very little voltage-dependent
150 evious studies have shown that naive adrenal chromaffin cells express a nominal Ca(v)3.2-dependent co
151 d secretion from Munc18-1-null mouse adrenal chromaffin cells expressing Munc18-1 mutants designed to
152 residues, single exocytotic events in bovine chromaffin cells expressing R198Q, R198E, K201Q, or K201
154 arge dense core vesicle (LDCV) exocytosis in chromaffin cells follows a well characterized process co
157 ally occurring splice variants of CAPS2 into chromaffin cells from CAPS1/CAPS2 double-deficient mice
158 A30P or wild-type (WT) alpha-syn, as well as chromaffin cells from control and alpha-syn null mice, t
161 e have examined the kinetics of secretion in chromaffin cells from mice lacking phosphatidylinositol
163 SCR-1 calcium-insensitive mutant or by using chromaffin cells from PLSCR-1(-)/(-) mice prevents outwa
166 s highly expressed in bovine adrenomedullary chromaffin cells, human pheochromocytoma tissue, PC12 ph
169 ime-lapse imaging of Lifeact-GFP-transfected chromaffin cells in combination with fluorescent 70 kDa
173 es and other hormones, released from adrenal chromaffin cells in response to Ca(2+) influx through vo
174 to the extent of catecholamine release from chromaffin cells in response to stimulation by carbachol
176 umor entity originating from adrenomedullary chromaffin cells in the adrenal medulla or in sympatheti
179 0 were colocalized to the Golgi apparatus of chromaffin cells in vivo and shared localization with CH
180 We hypothesize that Slo splicing in adrenal chromaffin cells in vivo is differentially regulated by
181 ociated with distinctive membrane changes of chromaffin cells including increased electron density, a
182 evealed decreased LDCV size in noradrenergic chromaffin cells, increased adrenal norepinephrine and e
183 ated exocytic function in Mecp2(-/y) adrenal chromaffin cells, indicating that the Mecp2 null mutatio
184 ially regulate fusion pore dynamics in mouse chromaffin cells, indicating TMD flexibility as a mechan
185 pression of Ca(v)3.2 channels in MPC 9/3L-AH chromaffin cells induced low-threshold secretion that co
188 ominant calcium signal regulating release in chromaffin cells is generated by the cooperative action
189 Transient stimulation of secretion in calf chromaffin cells is invariably followed by rapid endocyt
191 heteromeric nAChR expressed by human adrenal chromaffin cells is the alpha3beta4* subtype (asterisk i
196 equently, the absence of Snapin in embryonic chromaffin cells leads to a significant reduction of cal
198 These findings suggest one way in which chromaffin cells may regulate cargo release is via diffe
202 SDH deficiency based on epithelial cells, a chromaffin cell model retains aspects of metabolic "heal
203 letion of a gene highly expressed by adrenal chromaffin cells, NIK-related kinase, a gene on the X-ch
204 e docking, neither synchronized secretion in chromaffin cells nor Ca(2+)-triggered SUV-GUV fusion was
205 exhibiting differential mobility shifting to chromaffin cell nuclear proteins during EMSA, binding of
206 own to accumulate mainly in association with chromaffin cells, occasional nerve endings and macrophag
207 also expressed in neonatal adrenal medullary chromaffin cells of rats and mice whose hypoxia-evoked c
212 c neurons are severely depleted in CIPA, but chromaffin cells of the adrenal medulla are spared.
217 ak amplitude of nicotine-induced currents in chromaffin cells or in human embryonic kidney cells ecto
218 cement of exocytosis by PMA in either bovine chromaffin cells or the INS-1 insulin-secreting cell lin
222 suggest that PROG inhibits CA secretion from chromaffin cells predominantly by rapidly inhibiting nAC
227 est that a spontaneous syntilla, at least in chromaffin cells, releases Ca2+ into a cytosolic microdo
228 wise, inactivation of the V0 a1-I subunit in chromaffin cells resulted in a decreased frequency and p
229 comparable with those of bPAC1hop in bovine chromaffin cells resulted in acquisition by PC12-G cells
230 hat overexpression of neuronal AP-3 in mouse chromaffin cells results in a striking decrease in the n
231 rometric measurements of exocytosis in mouse chromaffin cells revealed that syb2 TMD mutations altere
234 CG10 by synthetic siRNAs virtually abolished chromaffin cell secretion of a transfected CHGA-EAP chim
236 extraordinary accumulation of solutes inside chromaffin cell secretory vesicles, although this has ye
237 r with time, that vesicles in bovine adrenal chromaffin cells segregate into distinct populations, ba
239 l firing rates, set by the sympathetic tone, chromaffin cells selectively release catecholamines at a
241 of this phenomenon in neuroendocrine adrenal chromaffin cells show that a single 2-ns, 16 MV/m unipol
243 e mice, expressing Cre recombinase under the chromaffin cell-specific phenylethanolamine N-methyltran
246 gnals was severalfold faster than in adrenal chromaffin cells, suggesting profound differences in the
251 medulla gland and of cultured human adrenal chromaffin cells that demonstrated prominent expression
253 Src family kinases (SFKs) are abundant in chromaffin cells that reside in the adrenal medulla and
256 , high-affinity nAChRs expressed in cultured chromaffin cells, they do not appear to be involved in f
257 hape of the bipolar pulse, Ca(2+) entry into chromaffin cells through electropores could be attenuate
260 tentials delivered at 0.5 Hz) causes adrenal chromaffin cells to selectively release catecholamines t
261 hetic tone, basal synaptic excitation drives chromaffin cells to selectively secrete modest levels of
263 CPE and PC activity in extracts of cultured chromaffin cells; total protein levels were unaltered fo
265 ies have demonstrated that adrenal medullary chromaffin cells transplanted into the spinal subarachno
266 ection of catecholamine release from adrenal chromaffin cells trapped in a microfluidic network.
267 ificity for the diagnosis of adrenomedullary chromaffin cell tumors can be jeopardized by physiologic
269 We studied insertion of granules in bovine chromaffin cells using capacitance as a measure of plasm
270 ne release in individual quantal events from chromaffin cells using cell-attached patch amperometry.
271 s of exocytosis from populations of mast and chromaffin cells using chemoreceptive neuron MOS (CnuMOS
273 Experiments on diI-stained bovine adrenal chromaffin cells using polarized TIRFM demonstrate rapid
274 strate that CIH increases the RRP in adrenal chromaffin cells via ROS-mediated activation of PKC and
275 punctate staining of norepinephrine-enriched chromaffin cells visualized using confocal microscopy in
276 Secretion of catestatin intermediates from chromaffin cells was accompanied by the cosecretion of c
277 histamine in H(1) receptor/TRPC4-expressing chromaffin cells was comparable with that triggered by a
278 ing of individual exocytotic fusion pores in chromaffin cells was imaged electrochemically with high
279 eover, regulated secretion of endopin 2 from chromaffin cells was induced by nicotine and KCl depolar
280 lamines, measured by amperometry in cultured chromaffin cells, was found to be increased either by pr
283 ophysiological measurements in mouse adrenal chromaffin cells, we show that PI(4,5)P2 uncaging potent
285 these mutations on the exocytotic process in chromaffin cells were assessed using capacitance measure
288 egulates stimulus-secretion coupling, bovine chromaffin cells were infected with Semliki Forest virus
292 red how alpha-syn overexpression in PC12 and chromaffin cells, which exhibit low endogenous alpha-syn
293 s to hypoxia by the carotid body and adrenal chromaffin cells, which regulate cardio-respiratory func
295 y monitoring CME of single vesicles in mouse chromaffin cells with cell-attached capacitance measurem
297 s and whole-cell recordings from rat adrenal chromaffin cells with parallel experiments on inactivati
299 monstrate that electrostimulation of adrenal chromaffin cells with ultrashort pulses can be modulated
300 foundly impairs priming of granules in mouse chromaffin cells without altering catecholamine release