ライフサイエンスコーパス: chromate

1 c contaminants, As(V) (arsenate) and Cr(VI) (chromate).

2 cyte MOLT4 cells by treatment with potassium chromate.

3 ghtly coloured paints were pigmented by lead chromate.

4 r stress signaling and survival responses to chromate.

5 lity constraints suggest this is released as chromate.

6 cleotide repeat stability and sensitivity to chromate.

7 micronuclei, and apoptosis in human cells by chromate.

8 g moieties are displayed toward the incoming chromate.

9 endent manner after chronic exposure to lead chromate.

10 CysK, YieF, or KatE) were more sensitive to chromate.

11 zymes responded most strongly to cadmium and chromate.

12 utene, CO(2), and Cr(aq)(3+), in addition to chromate.

13 compared to the sulfur-poor or nondoped lead chromates.

14 th unified parameters successfully predicted chromate adsorption for a range of capacitance values.

15 Chromate adsorption isotherms recorded at pH 7 are sugge

16 tical frequency calculations to characterize chromate adsorption on 2-line ferrihydrite.

17 applied to a large and diverse data set for chromate adsorption on iron (oxy)hydroxides (ferrihydrit

18 chromium, making the method selective to Pb chromate adulteration assuming that this is its dominant

19 [Ni(bpe)(2)(MoO(4))], MOOFOUR-1-Ni, and its chromate analogue, CROFOUR-1-Ni, exhibit high CO(2) affi

20 ture calculations also demonstrate that lead chromate and its coprecipitates are p-type semiconductor

21 h affinity for oxyanions (i.e., arsenate and chromate) and suggests that BIOs may be similarly reacti

22 Selenate, chromate, and arsenate produce transient APX intermediat

23 erienced by Escherichia coli K-12 exposed to chromate, and mechanisms that may enable cells to withst

24 nts (arsenate, arsenite, selenate, selenite, chromate, and perchlorate) were selected for study.

25 ins with different abilities to reduce iron, chromate, and uranium.

26 iment and continuously infused with lactate, chromate, and various native electron acceptors diverged

27 nalized fused quartz/water interfaces toward chromate are consistent with nearly 50% slower transport

28 inherently as oxo-anions (e.g., perchlorate, chromate, arsenate, pertechnetate, etc.) or organic anio

29 sition and the crystalline structure of lead chromate-based pigments influence their stability.

30 multisite complexation (CD-MUSIC) framework, chromate binding constants and the Stern Layer capacitan

31 As bacterial chromate bioremediation is limited by the toxicity of ch

32 ctase that has many applications, such as in chromate bioremediation.

33 s stress will improve their effectiveness in chromate bioremediation.

34 Because chromate causes certain nuclear proteins to form complex

35 cluding a cpxR homolog were perturbed in the chromate-challenged mutant.

36 ient and reflection coefficients of the lead chromates change as a result of the sulfate doping in su

37 Molybdate, selenate, chromate ("chromium VI"), arsenate, tungstate, chlorate,

38 ead adulteration of spices, primarily via Pb chromate compounds, has been documented globally as a gr

39 The effects of pH, aqueous chromate concentration, ionic strength, and deuterium ex

40 rs ago the Chinese Qin developed an advanced chromate conversion coating technology (CCC) to prevent

41 ajor debris component either wooden pallets, chromated copper arsenate (CCA) treated wood, or alkalin

42 PbO), alloys (SnPb, SbPb, SnAg, SnCu, SnZn), chromated copper arsenate-related nanomaterials (CuCrO(2

43 The redox chemistry of chromate (Cr(VI)) and arsenite (As(III)) on the iron oxy

44 Contamination of the environment with Cr as chromate (Cr(VI)) from industrial activities is of signi

45 h the transcript and protein levels to acute chromate [Cr(VI)] challenge.

46 Heavy metal chromate (Cr2O7(2-)) anions consisting of chromium Cr(VI

47 lms to U(VI) (uranyl, UO(2)(2+)) and Cr(VI) (chromate, CrO(4) (2-)) using non-invasive nuclear magnet

48 Higher concentrations of chromate cross-linked many other proteins to DNA.

49 Trend analyses on chromate (decreasing), epoxy resin (increasing) and nick

50 tamination by hexavalent chromium [Cr(VI) or chromate] due to anthropogenic activities has become an

51 ubstantial fraction of all Cr-DNA adducts in chromate-exposed cells are represented by ternary comple

52 Chromate exposure depleted cellular levels of reduced gl

53 Specifically, a 24-hour lead chromate exposure induced no aneugenic effect, whereas a

54 we investigated the effects of chronic lead chromate exposure on centrosomes.

55 Within 3 h of chromate exposure, the latter ceased growth and exhibite

56 ent mutant S. oneidensis under conditions of chromate exposure.

57 endent manner with 0.5 and 1 microg/cm2 lead chromate for 120 hours, inducing aberrant centrosomes in

58 ability of the 6 and 90 nm particles to sorb chromate from solution, despite the greater surface area

59 8 h), nor did the organics interact with the chromate in solution.

60 0-hour exposure to 0.5 and 1 microg/cm2 lead chromate induced 55% and 60% aneuploid metaphases, respe

61 suggest that one possible mechanism for lead chromate-induced carcinogenesis is through centrosome dy

62 Fe homeostasis and the cellular response to chromate-induced stress in S. oneidensis.

63 t exposure of mouse hepatoma Hepa-1 cells to chromate inhibits the induction of the Cyp1a1 and Nqo1 g

64 The color of the pigment, in which the chromate ion acts as a chromophore, is related to its ch

65 calculations on permanganate ion as well as chromate ion and iron tetraoxide.

66 The reduction site of the chromate ion in D. acetoxidans is occupied by a sulfate

67 atisfactory results for permanganate ion and chromate ion.

68 ellow pigments, caused by a reduction of the chromate ions to Cr(III) compounds, is known to affect t

69 ir maximum adsorption potential for lead and chromate ions.

70 contaminated by oxidized pollutants such as chromate is a common and difficult challenge.

71 Chromate is retained on the anion-exchange resin from wa

72 bioremediation is limited by the toxicity of chromate, minimizing oxidative stress during bacterial c

73 Chromate mobility, reactivity, and bioavailability in so

74 istant to effects of sulphate-mimetics (like chromate or molybdate) on sulphate transport.

75 Elevated chromate or sulfate anion concentrations can interfere w

76 ulates may provide more chronic exposures to chromate over time.

77 other anionic pollutants from water such as chromate, pertechnetate, or arsenate may be possible by

78 hanges in each of these amino acids enhanced chromate reductase activity of the enzyme, showing that

79 The specific activity of chromate reductase, NfoR, from Staphylococcus aureus sp.

80 hat NfoR resembles the structure of class II chromate reductase.

81 enzyme that has also been characterized as a chromate reductase; yet we propose that it is the quinon

82 minimizing oxidative stress during bacterial chromate reduction and bolstering the capacity of these

83 g that this network is centrally involved in chromate reduction.

84 t this is the oligomeric form that catalyzes chromate reduction.

85 able production of H(2)O(2) that accompanies chromate reduction.

86 ier to be overcome was the separation of the chromates reduction carried out by ethylene from the sub

87 Thus, enhancing the activity of ChrR in a chromate-remediating bacterial strain may not only incre

88 The path leading to chromate resembles the CH(3)C(O)OO(*) reaction.

89 ical environments can significantly increase chromate residence times.

90 have important implications for the fate of chromate, selenate, and sulfate in subsurface environmen

91 e characterized the adsorption mechanisms of chromate, selenate, and sulfate on Al-substituted ferrih

92 affinity (approximately 5 microM), sulfate, chromate, selenate, phosphate, and chlorate did not bind

93 by an in-frame deletion resulted in enhanced chromate sensitivity and a reduced capacity to remove ex

94 Magnetite-chromate sorption experiments were conducted with approx

95 g/L) of organics had no noticeable impact on chromate sorption, whereas concentrations of 50 mg/L or

96 g/L or more resulted in decreased amounts of chromate sorption.

97 ch diffuse over the support and stabilize as chromate species or as Cr(5+) upon reduction by oxygen v

98 impact the redox chemistry of the magnetite-chromate system over the duration of the experiments (8

99 rove the higher tendency of sulfur-rich lead chromates to darken.

100 Thus, oxidative stress plays a major role in chromate toxicity in vivo, and cellular defense against

101 ial strain may not only increase the rate of chromate transformation, it may also augment the capacit

102 se were also found to cross-link with DNA by chromate treatment.

103 imarily complexed to DNA following 25 microM chromate treatment.

104 Chromate was used as a chemical probe to investigate the

105 apted by overnight growth in the presence of chromate were less stressed than nonadapted controls.

106 In the absence of scavengers, nitrite and chromate were produced.

107 istic effects dominating the interactions of chromate with surface-bound amido acids indicates that c

108 rrant mitosis after chronic exposure to lead chromate with the emergence of disorganized anaphase and

109 ound to be mechanism-specific in the case of chromate, with bidentate complexes disproportionately su

110 ancers with microsatellite instability among chromate workers.

111 The darkening of lead chromate yellow pigments, caused by a reduction of the c