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1 tic Schwann-cell disorder featuring a 1.4 Mb chromosomal duplication.
2 ed and was demonstrated in 83% of cases with chromosomal duplication.
3 echanisms might lead to allelic deletion and chromosomal duplication.
4 ts that the clusters may have arisen through chromosomal duplication.
5 he fitness cost associated with this ancient chromosomal duplication.
6 d be the result of subsequent, smaller-scale chromosomal duplications.
7 mya5 and Fsd2 appear to have originated by a chromosomal duplication and are found within evolutionar
8 and AtMTM2 have originated from a segmental chromosomal duplication and encode catalytically active
9 less likely for these paralogs to show inter-chromosomal duplication and testis-dominant transcriptio
10 ve general implications for trans effects of chromosomal duplications and aneuploidies on epigenetic
13 nal priming process is associated with inter-chromosomal duplications and the insertion of filler DNA
14 that could select for different patterns of chromosomal duplications and/or losses, and their nearly
15 , the lac operon was inserted within a small chromosomal duplication, and selection stimulated RecA-d
17 reducens genome was found to contain a 10-kb chromosomal duplication consisting of two tandem three-g
18 e also found that three telomerically placed chromosomal duplications, containing approximately 700 o
21 loci detected by 357 probes suggest ancient chromosomal duplication followed by extensive rearrangem
22 ved in our laboratory evolution experiments; chromosomal duplications gained under stress were elimin
23 In contrast, amplifications caused by whole chromosomal duplication have little to no impact on fitn
24 the relative impacts of retrotransposons and chromosomal duplication in plant genome evolution, and t
26 termine whether allelic loss correlates with chromosomal duplication in the same tumor cell populatio
27 lled by NOTCH1 that is targeted by recurrent chromosomal duplications in human T cell acute lymphobla
32 -seq of brain DNA from mouse models with sub-chromosomal duplications mimicking DS reveals partial bu
33 identified an XY intersex patient carrying a chromosomal duplication of the WNT4 locus and proposed t
35 er eukaryotes, owing to their propensity for chromosomal duplication or even triplication in a few ca
36 easure frequencies of sperm carrying partial chromosomal duplications or deletions of 1cen or 1p36.3
37 to the propensity of angiosperms to undergo chromosomal duplication ('polyploidization') and subsequ
38 tive fiber-FISH mapping results support that chromosomal duplications, rather than regional expansion
39 scinating case study for the coordination of chromosomal duplication, repair, and segregation with ea
40 due to heterozygous inheritance of a partial chromosomal duplication resulting in over-expression of
42 nclude that LOH may occur in the presence of chromosomal duplication, suggesting that the duplicated
43 ty was assessed in Ts65Dn mice that harbor a chromosomal duplication syntenic to human chromosome 21q
44 mutation and, conversely, is suppressed by a chromosomal duplication that contains the wild type Smrt
47 utative tumor suppressor sites subsequent to chromosomal duplication; these losses do not necessarily
49 integrate a phylogenetic approach (relating chromosomal duplications to the tree of life) with a gen
51 ar time and structural variants within early chromosomal duplications, we indeed identified pregain d