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1 region containing non-B6-derived segments on chromosome 13.
2 gp130 genes on human chromosome 5 and mouse chromosome 13.
3 region that contains other genes from human chromosome 13.
4 osome 14 and the orthologous region of human chromosome 13.
5 essive inheritance of a single gene on mouse chromosome 13.
6 of 11 probes spanning the entire long arm of chromosome 13.
7 rranged in a closely linked direct repeat on chromosome 13.
8 romosome 5p15.3, which is syntenic to murine chromosome 13.
9 gene has been characterized and localized to Chromosome 13.
10 alysed had an allele loss on the long arm of chromosome 13.
11 cus of the mouse mH2A1 gene and mapped it to chromosome 13.
12 is encoded by a novel gene, BRI, located on chromosome 13.
13 The mouse Gng4 gene is mapped to chromosome 13.
14 12, and 15 and at two separate locations on chromosome 13.
15 a candidate amplicon target gene located on chromosome 13.
16 ose from a transgene insertional mutation on chromosome 13.
17 patients with deletions and duplications of chromosome 13.
18 ately 3.6 cM interval in the middle of mouse chromosome 13.
19 The most common region lost was chromosome 13.
20 ependent histone genes found on Mus musculus chromosome 13.
21 or 19p and complete or partial deletions of chromosome 13.
22 present in the histone gene cluster on mouse chromosome 13.
23 romosome 5, with a second GRK6-like locus on chromosome 13.
24 contig of the Lyst critical region on mouse Chromosome 13.
25 maps to human chromosome 5q23-q31 and mouse chromosome 13.
26 s localized Bft to the central part of mouse chromosome 13.
27 ion of GRK6, and localizes this homologue to chromosome 13.
28 itative trait in mice and found it linked to chromosome 13.
29 type revealed the loss of one entire copy of chromosome 13.
30 mology between human chromosome 5q and mouse chromosome 13.
31 a region (q23) known to be syntenic to mouse chromosome 13.
32 parum sodium-hydrogen exchanger (pfnhe-1) on chromosome 13.
33 nd particularly any such disorders linked to chromosome 13.
34 specific backcross analysis to the middle of chromosome 13.
35 to the vicinity of the pearl locus on mouse chromosome 13.
36 s that maps to a 2.2-centimorgan interval on chromosome 13.
37 he plasmid was integrated at the telomere of chromosome 13.
38 hromosome 4, Mf3 to Chromosome 9, and Mf4 to Chromosome 13.
39 e segment of the bg-critical region on mouse chromosome 13.
40 orrelations were discovered, with 11 SNPs on chromosome 13.
41 small disease-associated set of variants on chromosome 13.
42 analysis, we mapped the rd11 locus to mouse chromosome 13.
43 oat pattern locus to a presumptive region on chromosome 13.
44 of individuals with late-onset FCD linked to chromosome 13.
45 enic cluster of microRNAs (miRNA) located on chromosome 13.
46 pression of a cluster of six miRNAs on human chromosome 13.
47 Both genes were genetically mapped to chromosome 13.
48 ode of inheritance was mapped to centromeric chromosome 13.
49 was found 15 kb downstream of Scarf on mouse chromosome 13.
51 s and loss of heterozygosity in a segment of chromosome 13 (13q14) in cases of B-cell chronic lymphoc
52 ion between BRCA2 and RB1 in the q14 band of chromosome 13 (13q14) in human cancers, including prosta
55 int in SST1, a macrosatellite DNA located on chromosomes 13, 14 and 21, which commonly undergo Robert
58 DNA clusters and flanking sequences on human chromosomes 13, 14, 15, 21 and 22 represent large gaps i
61 loidy status indicated an increased level of chromosome 13, 18, 21 and 22 disomy, up to 7 x (2.7 - 15
65 analysis confirmed their locations on mouse chromosomes 13, 4 and 3, respectively, within regions di
66 om the YACs and probes for markers mapped to chromosome 13, 511 were assembled in contigs that were e
67 candidate genes for the mnd locus from human chromosomes 13,8, and 19, and we are mapping these genes
68 hich likely corresponds to portions of human chromosomes 13,8, or 19; we note that the chromosome 13
69 genetic mapping, identified a small locus on chromosome 13 (95% CI 92.22-94.09 Mb) that is significan
70 ing YAC clones containing histone genes from chromosome 13, a contig of approximately 2 Mb has been d
71 e nucleotide transhydrogenase (Nnt) on mouse chromosome 13, a nuclear-encoded mitochondrial protein i
77 ic analysis of these tumors showed recurring chromosome 13 alterations via chromosomal loss or transl
79 mors, we identify recurrent amplification of chromosome 13, an alteration highly restricted to colore
80 lications of partial or complete deletion of chromosome 13 and 11q abnormalities, we now observed tha
82 in regions of strong selection signatures on chromosome 13 and 14, as described in Southeast Asia (SE
84 cant differences in the fetal DNA ratios for chromosome 13 and chromosome 21, indicative of trisomy 2
85 ned to support the physical mapping of human chromosome 13 and expanded to support several gene-ident
86 3 ortholog map to syntenic regions of murine chromosome 13 and human chromosome 9q22, respectively.
91 report the mapping of such a region to mouse chromosome 13 and to the critical interval for Lgn1, a m
92 nce of at least one tumor suppressor gene on chromosome 13 and two tumor suppressor genes on chromoso
96 ites confirmed that the copy number of fetal chromosomes 13 and 21 was established correctly for 58 o
101 ues on chromosomes 18 and 20 (18|20 PRT) and chromosomes 13 and X (13|X PRT) were tested using archiv
102 ytes may be the source of the nonrecombinant chromosomes 13 and X suggested, by genetic studies, to b
105 some 18, three primer pairs for sequences on chromosome 13, and one primer pair to identify the sex c
106 h t(4;14), t(14;16), del(17) (p13.1), and/or chromosome 13 anomalies (primarily monosomy 13) had poor
107 16) had poor survival, whereas patients with chromosome 13 anomalies had intermediate survival: inter
110 djacent to the breakpoints on the derivative chromosome 13 are remarkable for their resemblance to re
111 at mutations in the BRI2 isoform, located on chromosome 13, are associated with dementia in humans.
112 ufficiency of RB1 and other genes mapping to chromosome 13, as well as activation of IGF1R, appears t
113 me-wide association study and a major QTL on chromosome 13 associated to different color-traits was d
114 LOD=2.46), chromosome 7 at 29 cM (LOD=2.50), chromosome 13 at 108 cm (LOD=2.10), and chromosome 15 at
117 D = 2.22, P = 0.00070) and a third region on chromosome 13 (at 26 cM, D13S894; LOD = 2.50, P = 0.0003
120 We identified additional copies on human chromosomes 13 (BCL8B), 22 (BCL8C), 2 (BCL8D), and 10 (B
123 r suppressor gene located on the long arm of chromosome 13, between the retinoblastoma (RB) and D13S2
124 eakpoints fell within YAC 899e2 and that the chromosome 13 breakpoints are clustered in a region flan
127 sistance phenotype, Lgn1, has been mapped to chromosome 13, but the responsible gene has not been ide
128 plexes have been linked to a single locus on chromosome 13 (Bxs6) in the BXSB model, to which linkage
129 es have been placed onto the physical map of chromosome 13, by means of FISH mapping with cosmid prob
130 dentified a genome-wide significant locus on chromosome 13-C3 and multiple additional suggestive loci
131 togenetic abnormalities (CAs), especially of chromosome 13 (CA 13), confer a grave prognosis in multi
134 To localize a minimal deleted region of chromosome 13, clonotypic plasma cells from 50 consecuti
138 vals: D13S292-cdx3GA1 and cdx3GA1-D13S289 on chromosome 13, consistent with the results of FISH analy
139 osome 5q11.2-q13.3 and its ortholog on mouse chromosome 13 contain candidate genes for an inherited h
140 ed by LOH studies to have lost the region of chromosome 13 containing BRCA2 were examined for alterat
141 lated with enhanced drug efflux; transfer of chromosome 13 containing the amplified MRP4 gene conferr
143 Two major quantitative trait loci (QTLs) on chromosomes 13 (Crgn1) and 16 (Crgn2) with logarithm of
146 ranslocations such as the t(4;14), t(14;16), chromosome 13 deletion by conventional cytogenetics and
147 ients with cytogenetic abnormalities such as chromosome 13 deletion, advanced bone disease, extramedu
148 with sex, race, performance status, isotype, chromosome 13 deletion, number or type of previous thera
149 or prognostic genetic and clinical subtypes (chromosome 13 deletion, t(4;14), t(14;16) and PCLI > 1%,
150 abnormalities, particularly the presence of chromosome 13 deletion, which is a grave prognostic indi
152 8, structural and numerical abnormalities of chromosome 13, deletion of Y chromosome, and complex cyt
155 ci have been identified to date which map to chromosomes 13 (DFNB1), 11 (DFNB2), 17 (DFNB3), 7 (DFNB4
156 ed Rf5 at an interval of 5.8 cM on sunflower chromosome 13, distal to a rust resistance gene R(11) at
157 bled a high-resolution physical map of human chromosome 13 DNA (approximately 114 Mb) from hybridizat
158 The BRCA2 gene, which is located on human chromosome 13, encodes a very large protein of only poor
159 reviously mapped to an interval on sunflower chromosome 13 encompassing three candidate genes annotat
160 esolution genetic map of the pearl region of chromosome 13 establishes the order of multiple markers,
162 as measured by insulitis), 3) a new locus on chromosome 13 for disease progression in response to env
163 6J mice with a QTL from the distal region of chromosome 13 from DBA/2J were able to survive for as lo
164 y for erythrocyte invasion was identified on chromosome 13 from the Plasmodium falciparum genome sequ
165 ssed genes mapping to localized amplicons on chromosome 13, gains of which occur often in colorectal
166 ex families with PD, one marker (D13S779) on chromosome 13 gave a logarithm of odds score of more tha
167 contributed by a novel and widely expressed chromosome-13 gene (ZNF198), are fused to the FGFR1 tyro
168 r sequencing, and assist in the discovery of chromosome 13 genes associated with hereditary diseases.
169 enetic analyses suggest that the q21 band of chromosome 13 harbors a tumor suppressor gene(s) involve
170 A recessive lethal insertional mutation on chromosome 13 has been identified in a transgenic mouse
172 terozygosity (LOH) involving the long arm of chromosome 13 has been reported to occur in as many as o
173 ng a defined set of rearrangements involving chromosome 13 has been used to assign 35 novel gene tran
176 Aberrations in the structure or number of chromosome 13 have been identified in a variety of human
177 HG context, especially at a narrow region of chromosome 13, hinting at an as-of-yet unsuspected role
178 zygous or homozygous deletions at band 14 on chromosome 13 in a variety of neoplasms suggests the pre
179 terozygosity and subchromosomal copy loss on chromosome 13 in DNA from fluorescence-activated cell so
180 e results confirm the importance of Crgn1 on chromosome 13 in EAG susceptibility, mediated partly thr
181 al analysis shows that NRP/B is localized to chromosome 13 in mouse and chromosome 5q12-13 in human.S
182 tissue mass, and bone mineral content and on chromosome 13 in the same region as lean tissue mass, bo
184 ndicate that the GRK6-hybridizing species on chromosome 13 is a transcriptionally inactive processed
185 ing results indicate that the q arm of human chromosome 13 is almost entirely conserved in bovine chr
187 though cytogenetic and molecular deletion of chromosome 13 is associated with poor prognosis, a MM tu
189 s indicated that the bg gene region of mouse chromosome 13 is likely homologous to the distal portion
190 of alleles of an autosomal recessive gene on chromosome 13 is necessary and sufficient to cause such
192 ne spans 13 kilobase pairs and is located on chromosome 13 just 2.8 kilobase pairs 5' to the Factor X
193 in a kelch protein encoded on P. falciparum chromosome 13 (K13) have been associated with resistance
194 n of the P. falciparum kelch protein gene on chromosome 13 (kelch13), were detected throughout mainla
195 esent in three copies in the mouse genome on chromosomes 13 (KIFC4A), 10 (KIFC4B), and 17 (KIFC4C).
197 nesis of HNSCCs and that the observed LOH of chromosome 13 loci is due to other, as yet, unidentified
199 major quantitative trait locus (QTL) on rat chromosome 13 (LOD = 3.9) linked to the percentage of gl
200 d genomewide significance, including QTLs on chromosomes 13 (logarithm of the odds ratio [LOD] score,
202 ecifically undertake mutation studies of the chromosome 13 member of this family and have identified
203 uding regions of multiple supporting SNPs on chromosomes 13 (minimum p = 7.5 x 10(-7)) and 14 (p = 4.
204 tions involving immunoglobulin (Ig) loci and chromosome 13 monosomy (Delta 13) are frequent cytogenet
206 cology Group [ECOG]) for IgH translocations, chromosome 13 monosomy/deletions (Delta13), and ploidy b
207 nsposed into the centromeric region of the Y chromosome >13 MYA, providing a unique opportunity to co
209 ies, and glomerulonephritis; an NZB locus on chromosome 13 (Nba6; peak at 28 cM), linked to IgG anti-
211 Three SNPs in high linkage disequilibrium on chromosome 13 near relaxin family peptide receptor 2 (RX
212 The 26.4-Mb disease interval contains the chromosome 13 nucleolus organizer (RNR1), the centromere
214 use variant suggested that jal maps to mouse Chromosome 13, our preliminary mapping analysis did not
215 spaced OL enhancers, which are 3 Kb apart on chromosome 13, overlap two MS SNPs in linkage disequilib
216 most prominent copy number gain occurred on chromosome 13, overlapping key oncogenes such as MYC and
219 e identify a plant height regulating gene on chromosome 13 (PH13) encoding a WD40 protein with three
220 an chromosomes 13,8, or 19; we note that the chromosome 13 portion maps close to a region thought to
222 tate adenocarcinoma displayed instability of chromosome 13, proliferated abnormally in Matrigel, and
225 Genome-wide analysis of SNPs identified a chromosome 13 region that associates with raised piperaq
226 l cloning of the translocation breakpoint on chromosome 13 resulted in the mapping of the breakpoint
227 genome-wide significant locus within FLT3 on chromosome 13, rs2504235, although this association was
228 ulin-like family peptide receptor 2 ( RXP2) (chromosome 13, rs60249166, odds ratio [OR] = 0.65, P = 3
229 ciation with an in-frame deletion in NUDT15 (chromosome 13, rs746071566) and TIM (5.8% [19/328] affec
232 of 10,789 cosmids initially selected from a chromosome 13-specific cosmid library (16,896 colonies)
233 ween the cosmids, which were selected from a chromosome 13-specific cosmid library using inter-Alu PC
234 ffecting growth rate to 30 kg was located on chromosome 13 such that the Large White allele increased
235 ne commonly deleted region was identified on chromosome 13 that centred around 13q13, and two commonl
236 is region is homologous to the area of mouse chromosome 13 that encodes the sodium-dependent amino ac
239 us on chromosome 2 and a suggestive locus on chromosome 13 that increases photoreceptor loss were ide
240 new gene locus, hdf (heart defect), on mouse chromosome 13 that may be required for mechanisms that i
241 m Thailand, identifying a selective sweep on chromosome 13 that shows strong association (P = 10(-6)
242 Exon trapping of a PAC that spanned the chromosome 13 translocation breakpoints led to the ident
243 ar CCR was 52% among 112 CR patients without chromosome 13 (triangle up13) abnormalities and with bet
244 ities and/or complete or partial deletion of chromosome 13 ("unfavorable karyotype") became a dominan
245 nd QTL_13_2) for aphid resistance on soybean chromosome 13 using 184 recombinant inbred lines from a
246 ybrid analysis mapped the GPC6 gene to human chromosome 13 very near the GPC5 gene, a member of the g
248 or gene-poor regions, such as those found in chromosome 13 was observed for full-length insertions.
250 opy of the patched gene (Ptc), which lies on chromosome 13, was deleted in all tested XRCC4/p53-defic
251 with hemizygous deletions of the long arm of chromosome 13, we have defined a discrete region in band
252 ts in the vicinity of the renin locus on rat chromosome 13, we transferred this chromosome segment fr
253 sinin resistance, and extended haplotypes on chromosome 13 were examined to determine whether mutatio
254 chromosome 3 (Bxs5), and another interval on chromosome 13 which were associated with various aspects
255 ysis of 95.5 megabases (Mb) of sequence from chromosome 13, which contains 633 genes and 296 pseudoge
256 lanocytes acquired a substantial deletion in chromosome 13, which encodes the Rb1 tumor suppressor ge
257 Cs, 52% were located in one 15.5-Mb locus on chromosome 13, which encompassed the Bhmt gene and defin
258 inkage analysis uncovered a B10 locus on mid-chromosome 13, which enhanced nephritis and was strongly
259 eficient MBs harbor clonal translocations of chromosome 13, which frequently involve chromosome 6 as
260 ate a molecular genetic linkage map of mouse chromosome 13 with an emphasis on the proximal region in
261 rmal/tumor B-CLL samples for allelic loss on chromosome 13 with nine microsatellite markers located b
262 wo genes (one on chromosome 1 and another on chromosome 13) with clear, significant effects on suscep
263 n toward locations on the distal long arm of chromosome 13, with no localizations noted in the 13q22-
264 within the subtelomeric region of one end of chromosome 13, would result in a preferential switch in