ライフサイエンスコーパス: chromosome 19

1 of linkage of the trait to the LDL receptor (chromosome 19).

2 o close the four remaining gaps on the human chromosome 19.

3 within a 1-cM region near the centromere of chromosome 19.

4 hich are located in a 5.5-megabase region on chromosome 19.

5 e site specifically into the AAVS1 region of chromosome 19.

6 structed a first-generation haplotype map of chromosome 19.

7 gh frequency of genetic alterations on mouse chromosome 19.

8 one village) using 22 polymorphic markers on chromosome 19.

9 ene insertion maps to the proximal region of chromosome 19.

10 c integration of the viral genome into human chromosome 19.

11 n of the entire PLP1 gene to the telomere of chromosome 19.

12 (KIR) genes are a multigene family on human chromosome 19.

13 mes, we sequenced mouse DNA related to human chromosome 19.

14 e-gated P/Q-type Ca(2+) channel (CACNA1A) on chromosome 19.

15 tein MGC2663 by GenBank, is encoded by human chromosome 19.

16 suggestive linkage evidence for a marker on chromosome 19.

17 PCL, was recently described and localized to chromosome 19.

18 7, syntenic with human CEBPA and G genes on chromosome 19.

19 the presence of another unrearranged gene on chromosome 19.

20 uggested the localization of MIP-2A at human chromosome 19.

21 They appear to be located together on chromosome 19.

22 located 16 kb upstream of the CYP4F2 gene on chromosome 19.

23 me order that these genes are found on human chromosome 19.

24 n cytochrome P450 4F genes form a cluster on chromosome 19.

25 s to the murine dn (deafness) locus on mouse chromosome 19.

26 rs187848990 on chromosome 2 and rs8100891 on chromosome 19.

27 ats and the AAVS1 integration locus on human chromosome 19.

28 orresponding human genomic sequence on human chromosome 19.

29 of the mouse tauCstF-64 cDNA, which maps to chromosome 19.

30 by integration into a specific site on human chromosome 19.

31 suggested possible linkage to loci on mouse chromosome 19.

32 n superfamily inhibitory receptor encoded on chromosome 19.

33 ion of the AAV genome into a region of human chromosome 19.

34 aps the murine Hells gene to region C3-D1 on chromosome 19.

35 nsion of a CTG trinucleotide repeat on human chromosome 19.

36 ressor gene, we mapped Lvis1 to distal mouse chromosome 19.

37 a 4-Mb region around the human APOE locus on chromosome 19.

38 ink between familial leukoencephalopathy and chromosome 19.

39 -specifically into a defined region of human chromosome 19.

40 a syntenic region close to the centromere on chromosome 19.

41 cosmids in the metric physical map of human chromosome 19.

42 in a region of conserved synteny with human chromosome 19.

43 s (oc), which is localized to proximal mouse chromosome 19.

44 ed both Gng3 and Gng3lg to the same locus on chromosome 19.

45 s to the murine dn (deafness) locus on mouse chromosome 19.

46 ocus for the brachymorphic mutation on mouse chromosome 19.

47 (AD) is the apolipoprotein E (APOE) locus on chromosome 19.

48 tandem repeats in the DNA sequence of human chromosome 19.

49 ers and 15 Yb8 Alu family members from human chromosome 19.

50 ma cells despite retention of both copies of chromosome 19.

51 phia myotonica-protein kinase gene (DMPK) on chromosome 19.

52 novel gene disrupted by the translocation on chromosome 19.

53 ion of the AAV genome into a region of human chromosome 19.

54 Pitx3 was mapped close to aphakia on mouse chromosome 19.

55 hromosome 10 and in the ERCC1 locus on human chromosome 19.

56 a syntenic region on the proximal region of chromosome 19.

57 a major suppressor locus (Mvb-1) to proximal chromosome 19.

58 novel microsatellite polymorphisms on human chromosome 19.

59 quences within the viral genome and in human chromosome 19.

60 mouse chromosome 7 and the long arm of human chromosome 19.

61 site-specific integration of AAV into human chromosome 19.

62 he functional gene maps to the MANB locus on chromosome 19.

63 marker in all the affected family members on chromosome 19.

64 atic cell hybrids assigned the human gene to chromosome 19.

65 transformed osteosarcoma cells, focusing on chromosome 19.

66 as the result of a ~400-kilobase deletion on chromosome 19.

67 csf-chr12 in chromosome 12 and gcsf-chr19 in chromosome 19.

68 pecifically into PPP1R12C, a gene located on chromosome 19.

69 he highly repetitive KIR region on the Human chromosome 19.

70 globin transgene in the AAVS1 site on human chromosome 19.

71 ther genes, most frequently the BRD4 gene on chromosome 19.

72 number of differentially methylated loci on chromosome 19.

73 vicinity of interferon (IFN)-lambda genes on chromosome 19.

74 SNP data from the HLA region, the MS32, and chromosome 19.

75 to the region previously localized on baboon chromosome 19.

76 tion hot spots have been identified on mouse chromosome 19.

77 tes its genome into the AAVS1 locus on human chromosome 19.

78 r chromosome Y and segmental duplication for chromosome 19.

79 including two remaining euchromatic gaps on chromosome 19.

80 in two inverted regions of tandem repeats on chromosome 19.

81 out 2/3rds of cases, NUT is fused to BRD4 on chromosome 19.

82 epresents a new, not yet identified locus on chromosome 19.

83 BPA gene coincided with a large-scale UPD on chromosome 19.

84 4S1644 using the same covariate model as for chromosome 19.

85 G-isl 6, and CAG-isl 7, were mapped to human chromosomes 19, 16, 12, and 3, respectively, via somatic

86 en introns and eight exons and is located on chromosome 19 (19C2-3).

87 ecifically integrating its genome into human chromosome 19 (19q13.3-qter) at a locus designated AAVS1

88 ization, the murine Kcnk8 gene was mapped to chromosome 19, 2B, the locus of the murine dancer phenot

89 experiments we mapped the Bam gene to distal chromosome 19, a locus syntenic to human chromosome 10q2

90 r AAV replication, targeted integration into chromosome 19 AAVS1 DNA may involve a replicative type o

91 its genome into a defined sequence on human chromosome 19 (AAVS1) makes it of particular interest fo

92 n a site-specific manner in a locus on human chromosome 19 (AAVS1).

93 On chromosome 19, all exons and the exon-intron borders of

94 On chromosome 19, allele frequency differences localized ne

95 both subfamilies of Alu elements along human chromosome 19 also appears to be random.

96 rearrangements and are frequently located on chromosome 19 (although not at the wildtype AAV integrat

97 eractions occur between a C57BL/6J allele on chromosome 19 and an A/J allele on chromosome 8.

98 Interestingly, the chromosome 19 and chromosome 11 QTLs, but not the chromo

99 y (cotinine/cigarettes per day (CPD)) shared chromosome 19 and chromosome 4 loci with the NMR, and th

100 approximately 5 kb upstream of Fgf8 on mouse Chromosome 19 and consists of six exons spanning 2 kb.

101 ntegration site known as AAVS1 is located in chromosome 19 and contains multiple GCTC repeats that ar

102 lysis between short-tandem repeat markers on chromosome 19 and COPD phenotypes was followed by associ

103 rous clustered ZNF families located on human chromosome 19 and elsewhere in the human and mouse genom

104 56, ISG54, and ISG49, which are clustered on chromosome 19 and encode the corresponding proteins p56,

105 tudy refines the gene order for distal mouse chromosome 19 and expands the comparative map between mo

106 e performed fine mapping of the short arm of chromosome 19 and found that the LKB1/STK11 gene mapped

107 ly within the detailed physical map of human chromosome 19 and genetic data that assign Rfx1 and Rfx2

108 Psl4 maps near D19Mit38 on distal chromosome 19 and inheritance of the dominant C57BL/6 al

109 ne locus mapped to the proximal end of mouse chromosome 19 and is inherited as a fully penetrant auto

110 Human PIASgamma spans approximately 23 kb on chromosome 19 and is organized into ten exons.

111 metalloprotease encoded by a locus on human chromosome 19 and mouse chromosome 17.

112 hundreds of loci spanning megabases on human chromosome 19 and simultaneously track genome organizati

113 We detect significant genomewide linkage on chromosome 19 and suggestive evidence of QTLs on chromos

114 analyses mapped the location of the gene to chromosome 19 and targeted MPS of the linkage region ide

115 ed major histocompatibility complex locus on chromosome 19 and tested functional significance through

116 esents the largest deletion yet described on chromosome 19 and the first report of the involvement of

117 nd expands the comparative map between mouse chromosome 19 and the human chromosome 10q23-q26 homolog

118 2 endogenous loci, the PPP1R12C/p84 gene on chromosome 19 and the IL2Rgamma gene on the X chromosome

119 the third patient had paternal isodisomy for chromosome 19 and was homozygous for p.Met148Thr.

120 ids coded by five, seven, and eight exons on chromosomes 19 and 1, and they all have two predicted tr

121 were not consistent among chromosomes, with Chromosomes 19 and 22 standing out as different from the

122 n lines, and detected two additional QTLs on chromosomes 19 and 26.

123 The associations in chromosomes 19 and 6 were independent and additive and e

124 e clusters near centromeric regions of human chromosomes 19 and 7 with smaller clusters or isolated c

125 Chromosomes 19 and Y are exceptions, possessing more ele

126 ended existing synteny between LG7 and human chromosome 19, and confirmed the synteny between LG24 an

127 en associated with the DM phenotype: DM1, on chromosome 19, and DM2, on chromosome 3.

128 6,7), which is syntenic to the Dac region on chromosome 19, and may disrupt the orthologue of Dac.

129 Both CNVs span genes encoded on chromosome 19, and quantitative RT-PCR demonstrated that

130 disruption at the AAVS1 integration locus on chromosome 19 ( approximately 100% of infected cells).

131 Many of the loci on chromosome 19 are associated with genes belonging to the

132 In osteoblasts, approximately 81% of chromosome 19 are observed to consist of regions charact

133 We identify rs281379 near FUT2/MAMSTR on chromosome 19 as a novel risk locus (OR = 1.18 (95% CI =

134 ng here an adult AML case with an additional chromosome 19 as the sole cytogenetic abnormality.

135 tory receptors and map to the same region of chromosome 19 as these genes.

136 ene include a transcribed retropseudogene on chromosome 19, as well as expressed genes in mouse, rat,

137 sociation studies have identified a locus on chromosome 19 associated with plasma triglyceride (TG) c

138 mechanism to integrate its genome into human chromosome 19 at 19q13.4 (termed AAVS1), thereby establi

139 t of the human gene; (ii) it is localized on chromosome 19 at bands C2-C3 that is syntenic to human c

140 about 500-kb pairs on the long arm of human chromosome 19 at q13.3.

141 Although ENL and ELL localize to chromosome 19, bands p13.3 and p13.1, respectively, thes

142 int contigs were directly anchored to cattle chromosome 19 [Bos taurus, (BTA) 19].

143 ZNF320 is oriented toward the centromere of chromosome 19, both genes appeared on the same derivativ

144 d box domain-containing zinc finger genes on chromosome 19, both of which are associated with heteroc

145 sociated parvovirus into its target on human chromosome 19, both of which involve stretches of G-G-G-

146 tween human chromosome 17 (HSA17) and bovine chromosome 19 (BTA19), two chromosomes known previously

147 struct a radiation hybrid (RH) map of bovine chromosome 19 (BTA19).

148 ales (n = 352), the top variant was found on chromosome 19 but differed by sex (females: rs11878604,

149 f a PrCa susceptibility locus, rs11672691 on chromosome 19, but also showed an association with aggre

150 ntegrates in a site-specific manner on human chromosome 19, but this has never been demonstrated dire

151 elper virus, the AAV genome is released from chromosome 19 by a process termed rescue, and productive

152 obtained, fine-mapped to the physical map of chromosome 19 by hybridization to a chromosome-specific

153 Limatin was mapped to mouse Chromosome 19 by restriction fragment length polymorphis

154 gene (gene symbols KHSRP and Khsrp) to human chromosome 19 by using radiation hybrid panels and to mo

155 Alu SINEs within primate-specific chromosome 19 (C19MC) and B1 SINEs within rodent-specifi

156 The primate-specific microRNA cluster on chromosome 19 (C19MC) is exclusively expressed in the pl

157 the preferred AAV integration site in human chromosome 19, called AAVS1.

158 AAV integration to a specific site on human chromosome 19, called AAVS1.

159 3' UTR of the gene DMPK at the DM1 locus on chromosome 19 causes myotonic dystrophy, a dominantly in

160 1 (NACC1) gene at c.892C>T (p.Arg298Trp) on chromosome 19 causes severe neurodevelopmental delay.

161 rget viral integration to a specific site on chromosome 19 (ch-19).

162 tively, despite both exhibiting loss of Pten/chromosome 19 (chr19) and PI3K/Akt activation with sub-t

163 This TGT cDNA sequence is encoded in human chromosome 19 clone CTC-539A10 (GenBank accession no. AC

164 expressed in the developing lens and maps to chromosome 19 close to ak in mouse.

165 with >60 mutations within the MHS-1 locus on chromosome 19 coding for ryanodine receptor type 1 (RyR1

166 ean Americans: rs1061477 in the KLK3 gene on chromosome 19 (combined odds ratio = 1.18; 95% CI, 1.10-

167 ound frequent deletion of a small segment of chromosome 19 containing MBD3, also a member of the NuRD

168 We characterized 102 kb of chromosome 19 containing the apolipoprotein (APO) E/C1/C

169 he leukocyte receptor complex (LRC) on human chromosome 19 contains related Ig superfamily killer cel

170 A region on chromosome 19 continues to show evidence for linkage, by

171 istatic effects located at distinct sites on chromosome 19 control susceptibility.

172 ly, amplification of the human RHPN2 gene on chromosome 19 correlates with a dramatic decrease in the

173 and cardiac arrhythmias) but do not have the chromosome-19 D CTG expansion.

174 Detailed microsatellite analysis of chromosome 19 deletions identified three independent reg

175 ntegrate its DNA into a 4-kb region of human chromosome 19, designated AAVS1.

176 dults, can be caused by a mutation on either chromosome 19 (DM1) or 3 (DM2).

177 and Yb8 Alu family members located on human chromosome 19 does not differ from that expected based u

178 e 3' untranslated region of the DMPK gene on chromosome 19 (dystrophia myotonica type 1 [DM1]), or an

179 An additional chromosome 19 element, which is responsible for DNA rear

180 that a cluster of 5 galectin genes on human chromosome 19 emerged during primate evolution as a resu

181 Three closely positioned genes on human chromosome 19 encode distinct but paralogous proteins, w

182 mmunoglobulin-like receptor (KIR) complex on chromosome 19 encodes receptors that modulate the activi

183 modest evidence that MOLF-derived alleles on chromosome 19 enhance development of bilateral TGCTs.

184 ease in this cross was the Fas gene on mouse chromosome 19, exhibiting a lod score of 60.

185 and integrates into a preferred location on chromosome 19, features that have fostered development o

186 ivergent haplotype of the core MHC region on chromosome 19 for six expressed genes not found in the z

187 scription units, juxtaposed to snaR genes on chromosome 19, formed by a promoter-containing left mono

188 is causally related to a previously reported chromosome 19 GWAS signal that was ascribed to the gene

189 constructed pangenome graphs for 1000 human chromosome 19 haplotypes and 2146 Escherichia coli seque

190 th our results, provide strong evidence that chromosome 19 harbors a gene for tumor aggressiveness.

191 Chromosome 19 has the highest gene density of all human

192 Allelic losses of the q13.3 region of chromosome 19 have been documented in malignant gliomas,

193 mon site-specific integration locus on human chromosome 19; however, most AAV vectors do not appear t

194 46-Mb gene-rich euchromatic portion of human chromosome 19 (HSA19) was utilized to generate a sequenc

195 Evidence for linkage to chromosome 19 (IBD6) was observed in Crohn's disease pai

196 gene encoding the ribosomal protein RPS19 on chromosome 19 in 25% of patients with DBA.

197 ch affects fasting insulin levels, to distal chromosome 19 in a leptin-deficient obese F2 intercross

198 rt the existence of a major modifier gene on chromosome 19 in a region previously linked to MI.

199 ting the sequence-based linkage approach for chromosome 19 in CEPH pedigrees.

200 hybridization, to chromosome 7 in mouse and chromosome 19 in human.

201 ated on X and Y chromosomes in humans but on chromosome 19 in mice.

202 x 10-5) located between ZNF788 and ZNF20 on chromosome 19 in the pooled population.

203 d1 and Scd2, respectively, and maps to mouse chromosome 19 in very close proximity to Scd1 and Scd2.

204 t leads to the integration of AAV into human chromosome 19 in vivo.

205 d localized a gene causing EEC to a locus on chromosome 19, in a region defined by D19S894 and D19S41

206 ansport functions and maps to proximal mouse chromosome 19, in a region to which the oc mutation has

207 ated Ucp1 expression, the C57BL/6J allele on chromosome 19 increased Ucp1 mRNA to levels higher than

208 s in the AAV2 ITR, the AAV5 ITR, and the AAV chromosome 19 integration locus identified some conserve

209 Introgression of a 7-Mb segment of the B6 chromosome 19 into the BTBR background (strain 1339A) re

210 another 25% of tumors harbored deletions in chromosome 19 involving Pten, implicating the loss of ot

211 The single copy gene that was mapped to chromosome 19 is intronless and encodes a 92-kDa protein

212 This region of chromosome 19 is known to contain genes involved in vari

213 e have demonstrated that the gene-rich human chromosome 19 is located in a more central position in t

214 vestigation of these associations outside of chromosome 19 is required, as they did not replicate.

215 urth, whereas the Siglec-11 gene is on human chromosome 19, it lies outside the previously described

216 Chromosome 19, known to have a high gene density, contai

217 n recently described near the MFSD12 gene on chromosome 19 (lead variant rs112332856).

218 ation of Casp7 to the distal region of mouse chromosome 19, linked to Mxi1, Adra2a, and Aop1.

219 A variant in the chromosome 19 locus (rs838145) was associated with highe

220 males (n = 541), one significant (P < 5e-8) chromosome 19 locus was found (top variant: rs56113850,

221 three most plausible candidate genes for the chromosome 19 locus, our only genome-wide significant th

222 ide significant variants, and replicated the chromosome 19 locus.

223 linkage was also detected at two regions on chromosome 19 (LOD = 2.84 and 2.54).

224 LOD = 1.09) for mild airflow obstruction and chromosomes 19 (LOD = 1.21) and 22 (LOD = 1.37) for chro

225 ree of the identified markers are located on chromosome 19 (meta-analysis: full sample P = 6.94E - 81

226 cause of MHL; these alterations activate the chromosome 19 microRNA cluster (C19MC).

227 r early endogenous ligand (ELABELA), and the chromosome 19 microRNA cluster, and increased mRNA expre

228 tocellular carcinoma (HCC) overexpresses the chromosome 19 miRNA cluster (C19MC) and is associated wi

229 cies include miRNA from the primate-specific chromosome 19 miRNA cluster (C19MC), which is expressed

230 onging to the primate- and placenta-specific chromosome 19 miRNA cluster (C19MC)-were significantly d

231 rate a unique oncogenic amplification of the chromosome 19 miRNA cluster, C19MC.

232 We show that miRNA members of the chromosome 19 miRNA cluster, which are almost exclusivel

233 tion of seven new meiotic hot spots on mouse chromosome 19, more than doubling the number of currentl

234 nce-tagged site mapping places cPLA2gamma on chromosome 19 near calmodulin.

235 strain (CSS; or 'consomic strain') in which chromosome 19 of 129/Sv+/+ was replaced by its MOLF-deri

236 . latifolia X chromosome, was applied to the chromosome 19 of Populus trichocarpa, an incipient sex c

237 VI, fatty acid 2-hydroxylase, ceruloplasmin, chromosome 19 open reading frame 12 and ATPase type 13A2

238 current changes detected by CGH were gain of chromosome 19 or 19p and complete or partial deletions o

239 hort arm of chromosome 1 and the long arm of chromosome 19, or 1p19q codeletion; and (c) methylguanin

240 tein-coding genes and on the gene-rich human chromosome 19 ortholog, suggesting that the Cat Ba langu

241 inding in monocytes was located in ZNF823 on chromosome 19 (p = 1.38 x 10(-10)) previously associated

242 ) or balanced for at least one participating chromosome (19 paired breakpoints).

243 emphasis on genes also included in the human chromosome 19 physical map.

244 the rumen microbiota in multiple ways; some (chromosome 19; position 3.0-4.0 Mb) are associated with

245 y low retention frequency of markers on this chromosome (19%) prevented unambiguous ordering of the o

246 to the high-resolution physical map of human chromosome 19 provides a framework for isolation of dise

247 of schizophrenia, the identification of the chromosome 19 quantitative trait locus is a novel findin

248 2 million bp of raw sequence data from human chromosome 19 region q13.

249 A type 5 locus on chromosome 11; and 11) the chromosome 19 region that contains several ion channels

250 The chromosome 19 region was identified in our data despite

251 carry a deletion of approximately 400 kb in chromosome 19, resulting in a fusion of the genes for th

252 Fine-mapping of chromosome 19 revealed 13 putatively causal variants, wi

253 o an atherosclerosis susceptibility locus on chromosome 19 revealed in an intercross between atherosc

254 nt locus was also found in or near ZNF626 on chromosome 19 (rs11085374; OR, 0.77; 95% CI, 0.70-0.85;

255 we generated congenic strains harboring MOLF chromosome 19 segments on 129 strain background and moni

256 The AML1 gene is fused out of frame to chromosome 19 sequences, resulting in a truncated AML pr

257 CblR is located at p13.2 on the short arm of chromosome 19, spans a length of 6.224 kb, and is compos

258 discovery project focused on characterizing chromosome 19-specific cDNAs.

259 We have screened a human chromosome 19-specific cosmid library for the presence o

260 eotide repeat polymorphisms were detected in chromosome 19-specific cosmids that were physically mapp

261 All of the chromosome 19-specific Ya5/8 and Yb8 Alu family members

262 together in the homologous region of murine chromosome 19, suggesting that one of these loci might b

263 C-G was mapped to the distal region of mouse chromosome 19 (syntenic with human chromosome 10q), but

264 gene identified as responsible for the mouse chromosome 19 T2dm2 quantitative trait locus for fasting

265 nic cycle, AAV integrates at a site on human chromosome 19 termed AAVS1.

266 specifically into a defined region of human chromosome 19 (termed AAVS1).

267 A somatic, 400 Kb deletion on chromosome 19 that fuses part of the DnaJ heat shock pro

268 report we present a gene map of a section of chromosome 19 that has been derived by combining the use

269 gain (duplication) of the genomic region on chromosome 19 that includes PRKACA.

270 e localized (MMU)Minpp1 to a region of mouse chromosome 19 that includes the murine homolog of Pten.

271 The Krd mouse has a deletion in chromosome 19 that includes the Pax2 gene locus.

272 carries a 7-cM transgene-induced deletion on chromosome 19 that includes the Pax2 locus.

273 Ldb1 maps to the distal region of mouse chromosome 19 that is syntenic with human chromosome 10q

274 as mapped by linkage disequilibrium to mouse chromosome 19, the same site to which several mouse muta

275 dergoes site-specific integration into human chromosome 19 through a deletion-substitution mechanism

276 rosses across the proximal 10 cM of proximal Chromosome 19 to identify haplotype blocks that segregat

277 this locus interacted with BALB/c alleles on chromosome 19 to increase the severity of infection.

278 eal a novel oncogenic mechanism in which the chromosome 19 translocation breakpoint interrupts the co

279 The chromosome 19 translocation breakpoint targets the BRD4

280 The human ELL gene on chromosome 19 undergoes frequent translocation with the

281 The human ELL gene on chromosome 19 undergoes frequent translocations with the

282 We mapped the NRAP gene to mouse chromosome 19 using interspecific crosses and to human c

283 nd the four-exon cathelicidin genes to sheep chromosome 19 using sheep-hamster somatic cell hybrids i

284 An amplicon on chromosome 19 was detected in 26% of ABC DLBCLs but in o

285 mutation at the pale ear (ep) locus on mouse chromosome 19 was found to be the homologue of human Her

286 lagen-binding inhibitory receptor encoded on chromosome 19, was inserted between the V and DJ segment

287 L on chromosome 5 and rs2302188/NM_033543 on chromosome 19), we provide strong evidence for associati

288 e high-resolution cosmid contig map of human chromosome 19, we found that the previous report was inc

289 d rs4251805 within the promoter) of PLAUR on chromosome 19 were each independently associated with su

290 romosomes 15 and 16 and one affecting LDL on chromosome 19 were identified.

291 osomes 2, 3, and 8 and one C57BL/6J locus on chromosome 19 were linked to Ucp1 induction in retroperi

292 Rep-mediated site-specific integration into chromosome 19 when present in inverted terminal repeat-c

293 ved in the apolipoprotein E (APOE) region on chromosome 19 where several SNPs surpassed the conservat

294 in smokers only, the maximum LOD was 1.64 at chromosome 19, whereas for chronic bronchitis in smokers

295 Additionally, chromosome 19, which contains Pten, was also selectively

296 o the Siglec-3-related gene cluster on human chromosome 19, which, in contrast, contains seven Siglec

297 The localization of MLIV to chromosome 19 will permit genetic prenatal diagnosis in

298 marker D19S433 at 51.88 centimorgans (cM) on chromosome 19 with 36 families (75 ARPs) when including

299 Human IGFL genes are clustered together on chromosome 19 within a 35-kb interval.

300 significant associations were identified on chromosome 19 within APOE (apolipoprotein E) (rs429358,