ライフサイエンスコーパス: chromosome 8

1 f chromosome 16q, which is syntenic to mouse chromosome 8.

2 deleterious mutations in the RECQL4 gene on chromosome 8.

3 ne is localized in the C3-D1 region of mouse chromosome 8.

4 (HPC), using 24 markers on the short arm of chromosome 8.

5 ined in exon 1, and is located at band E1 on chromosome 8.

6 confirmed the synteny between LG24 and human chromosome 8.

7 ive, demyelinating peripheral neuropathy, to chromosome 8.

8 at or kat(2J), that map to the same locus on chromosome 8.

9 allele on chromosome 19 and an A/J allele on chromosome 8.

10 ene and the mapping of its location to mouse chromosome 8.

11 For radiation, abnormalities were mostly in chromosome 8.

12 The gene is located on mouse chromosome 8.

13 ML1 gene on chromosome 21 is fused to ETO on chromosome 8.

14 ndromes map or have been suggested to map to chromosome 8.

15 proto-oncogene at 67.0 centimorgans (cM) on chromosome 8.

16 Arg polymorphism) spanning a 57-cM region of chromosome 8.

17 1 (CBFA2) on chromosome 21 and ETO (MTG8) on chromosome 8.

18 ue to independent allelic mutations on mouse chromosome 8.

19 hereas the murine homolog gene is located on chromosome 8.

20 lepsy (IGE), for linkage to markers spanning chromosome 8.

21 ty (LOH) for 11 microsatellite loci on mouse chromosome 8.

22 to a noncoding region at 61.0 cM on the same chromosome 8.

23 to correspond to a SRPK2 pseudogene on human chromosome 8.

24 -STAR mapped to the syntenic region on human chromosome 8.

25 hroid ankyrin is encoded by the Ank1 gene on Chromosome 8.

26 isomic segregation relatively more common in chromosome 8.

27 alized to a highly syntenic region of distal Chromosome 8.

28 t 15 microsatellite loci on the short arm of chromosome 8.

29 owed us to map the CK2 alpha' gene to murine Chromosome 8.

30 ogy to other CC chemokines and maps to mouse chromosome 8.

31 homology region is likely to reside on human chromosome 8.

32 vical ITD in two large Mennonite families to chromosome 8.

33 to TrnR1, and is located on the short arm of chromosome 8.

34 on chromosome 21 and the ETO (MTG8) gene on chromosome 8.

35 lele loss with RFLP probes from both arms of chromosome 8.

36 , GRINA, has been previously mapped to human chromosome 8.

37 , was mapped to the proximal region of mouse Chromosome 8.

38 by an interspecies backcross panel to mouse chromosome 8.

39 der using 30 microsatellite polymorphisms on chromosome 8.

40 translocations involving the gene for MYC on chromosome 8.

41 e NODULIN3 (N3) gene family, located on rice chromosome 8.

42 cell line haploid for all chromosomes except chromosome 8.

43 and TG, not fully explained by the locus on chromosome 8.

44 icated an association with rearrangements of chromosome 8.

45 esolution over 8p12 and 1 Mb resolution over chromosome 8.

46 As part of this project, we have focused on chromosome 8.

47 The lop11 locus was mapped to mouse chromosome 8.

48 1) on chromosome 15 and mALT2 gene (gpt2) on chromosome 8.

49 (eight-twenty one, also called MTG8) gene on chromosome 8.

50 tility restorer gene of CMS-C, is located on chromosome 8S.

51 nd are therefore present on both the X and Y chromosomes [8].

52 th significant increases in hyperdiploidy of chromosomes 8 (1.2, 1.5, and 2.4 per 100 metaphases; P <

53 d rice by resequencing 630 gene fragments on chromosomes 8, 10, and 12 from a diverse set of wild and

54 some ploidy of each cell, the centromeres of chromosomes 8, 10, and 17.

55 ere probes and locus-specific arm probes for chromosomes 8, 11, 17, and 18, we demonstrate that chrom

56 they are both associated with polysomies for chromosomes 8, 11, 17, and 20.

57 Related sequences were mapped on Chromosomes 8, 11, and 2 unlinked loci on Chromosome 2 u

58 the extent of wound closure were detected on chromosomes 8, 12, and 15 and at two separate locations

59 Detailed analysis of aneuploidy for chromosomes 8, 12, and 17 on intact paraffin sections re

60 ple whole-chromosome losses, particularly of chromosomes 8, 12, and 19.

61 The shape of the expression profiles for chromosomes 8, 12, and X correlated well with the numeri

62 However, there was random loss of chromosomes 8, 13, X, Y, and alteration in chromosome 4q

63 uction of Separase resulted in trisomies for chromosomes 8, 15, and 17; monosomy for chromosome 10; a

64 The Cat4 locus has been mapped to chromosome 8, 31 cM from the centromere.

65 o identify the translocation partner gene on chromosome 8, 5' and 3' RACE polymerase chain reactions

66 s of chromosome 4 (87%), chromosome 7 (80%), chromosome 8 (53%), chromosome 10 (33%), and chromosome

67 es coelicolor A3(2) possesses a large linear chromosome (8.67 Mb) with a centrally located origin of

68 Allelic losses on the short arm of chromosome 8 (8p) have been reported as frequent events

69 lular carcinoma (HCC) is of the short arm of chromosome 8 (8p).

70 The short arm of chromosome 8, 8p, is often rearranged in carcinomas, typ

71 ocus and a segment of the short arm of human chromosome 8 (8p21-p11.2).

72 Gains in the long arm of chromosome 8 (8q) are believed to be associated with poo

73 indicated a single integration site on mouse chromosome 8 (8qB2) with ~6 gene copies.

74 possible reciprocal translocation involving chromosomes 8, 9, and 12.

75 tein arginine methyltransferase 7 (Prmt7) on chromosome 8, a protein previously implicated in cellula

76 Only two cases (13%) lacked a chromosome 8 abnormality.

77 could be coamplified with a control gene on chromosome 8 (Acta1) so that the yield of each PCR produ

78 ntify a candidate region, which was found on chromosome 8; all genes within this interval were sequen

79 To investigate the chromosome 8 allele status in Chinese HCCs, a panel of 3

80 st tumors examined, such as RIPK2 and MYC on chromosome 8, also parallel these findings.

81 ots with putative sesquiterpene synthases on chromosome 8, an indication that the genes in Sst1 and S

82 higher pathological stage (P = 0.021) and on chromosome 8 and 16 with a higher Gleason score (P = 0.0

83 COP locus, Mcs4, has also been identified on chromosome 8 and acts in contrast to increase the number

84 Our FISH results indicate that: (a) gain of chromosome 8 and amplification of c-myc are potential ma

85 ated on human chromosome 16q22-q23 and mouse chromosome 8 and are alternatively spliced.

86 es, such as large copy number aberrations in chromosome 8 and complex rearrangement chains.

87 The mouse Nod2 locus is located at chromosome 8 and composed of 12 exons, 11 of which encod

88 The murine flt4-L gene was localized to chromosome 8 and demonstrated to be widely expressed.

89 mapping the gene (Arhgap7) to 20 cM on mouse chromosome 8 and for allelotyping of mouse liver tumor D

90 loss of heterozygosity for the short arm of chromosome 8 and has a complex karyotype.

91 didate genes to validate an eQTL hot spot in chromosome 8 and identified the gene 2310061C15Rik as a

92 Myo9b has been previously mapped to mouse chromosome 8 and is a candidate for the mouse mutations

93 tained pedigrees implicated two loci, one on chromosome 8 and the other on chromosome 15, that influe

94 ene duplicates: two alpha-globin paralogs on chromosome 8 and two beta-globin paralogs on chromosome

95 olved in stress and immune responses; and on chromosomes 8 and 10.

96 DNA amplification, particularly of chromosomes 8 and 11, occurs frequently in breast cancer

97 0; and amplification of the distal region of chromosomes 8 and 11.

98 Moreover, gains of sequences from chromosomes 8 and 13 occurred in most tumors, indicating

99 we evaluated microsatellite markers spanning chromosomes 8 and 13, for linkage to schizophrenia, in 2

100 ovided initial evidence for modifier loci on chromosomes 8 and 15 and suggestive evidence for a locus

101 nd saturation mapping purported intervals on chromosomes 8 and 15, two additional regions were identi

102 Rfx1 and Rfx2 to homologous regions of mouse chromosomes 8 and 17, respectively.

103 tors for PERV-A, encoded by genes located on chromosomes 8 and 17.

104 BIG2 genes were localized, respectively, to chromosomes 8 and 20.

105 -embedded tissue with centromeric probes for chromosomes 8 and 20.

106 Numerical and structural aberrations in chromosomes 8 and 21 are commonly observed in AML.

107 In the present study, we painted chromosomes 8 and 21 in lymphocyte metaphases from 43 he

108 Translocations between chromosomes 8 and 21 were increased up to 15-fold in hig

109 cation of the amplified Myc and HPV genes on chromosomes 8 and 21.

110 und on chromosomes 2 and 16 for liver and on chromosomes 8 and 9 for spleen.

111 nt1 and Ant2) and assigned the loci to mouse chromosomes 8 and X, respectively.

112 ents with t(8;14)(q11;q32) involved CEBPD on chromosome 8, and 9 patients with t(14;19)(q32;q13) invo

113 lines, aberrant gene expression, trisomy of chromosome 8, and abnormal H2A.X deposition were disting

114 to an extra isochromosome of the long arm of chromosome 8, and the near-diploid karyotype appears to

115 PHS1 and NDPS1 map to chromosome 8, and the presence of a segment of chromosom

116 t anomaly in PIN and carcinoma was a gain of chromosome 8, and the presence of this anomaly strongly

117 fied AML1 on chromosome 21 and ETO (MTG8) on chromosome 8, and the resultant chimeric gene product, A

118 The toppler mutation was mapped to mouse chromosome 8, and to assess whether it was novel or a re

119 A male-specific locus on proximal chromosome 8 (Ap8q) affected 3% ethanol preference, but

120 Genomic aberrations on chromosome 8 are common in colon cancer, and are associa

121 mor samples of high-grade chondrosarcoma for chromosome 8 are presented.

122 Deletions on the short arm of chromosome 8 are recurrent in liver, breast, lung, and p

123 Sequences previously localized to chromosome 8 are shown to be a pseudogene, and an additi

124 rder between CFA29 and the long arm of human chromosome 8 argued for homology between the cd locus an

125 romosome 8 polysomy had up to five copies of chromosome 8 as an isolated cytogenetic finding in an ot

126 g whether GRINA mapped to the same region of chromosome 8 as BFNC.

127 of evidence have implicated the short arm of chromosome 8 as harboring genes important in prostate ca

128 ous studies have implicated the short arm of chromosome 8 as the site of one or more tumor suppressor

129 gene encoding cortexin, Ctxn, which maps to chromosome 8, as a candidate for the mouse neurological

130 sruption of a cluster of six genes on murine chromosome 8, as exemplified by the Fused Toes (Ft) muta

131 Os) is a radiation-induced mutation on mouse chromosome 8 associated with early postimplantation leth

132 e we characterized the structural changes of chromosome 8 associated with this mutation.

133 hm of odds [LOD]=4.93, permuted P=0.002) and chromosome 8 at 33 cM (LOD=3.27, permuted P=0.058).

134 ific QTL, which we name Fsia1, is located on chromosome 8 at 52-84 cM from the centromere and account

135 The myd mutation maps to mouse chromosome 8 at approximately 33 centimorgans (cM).

136 zed by fluorescence in situ hybridization on chromosome 8 at bands p21.3-22.

137 ive HPV18 integration sites: three on normal chromosomes 8 at 8q24 and two on derivative chromosomes,

138 syntenic; the hTRF1 gene localized to human chromosome 8 band q13 while the mTRF1 gene localized to

139 ed that the murine FATP gene is localized to chromosome 8, band 8B3.3.

140 ST7 is located on human chromosome 8, band q22.2-23.1, the same locus as the gen

141 itu hybridization localized the ERK8 gene to chromosome 8, band q24.3.

142 for insulin response to glucose was found on chromosome 8 between D8S1130 and D8S1106, near the lipop

143 We mapped this new gene to chromosome 8, between markers D8S256 and D8S284 on a rad

144 gene is expressed in leaf tissue and maps to chromosome 8 (bin 8.05), with a duplicate locus on chrom

145 At the chromosome 8 breakpoint we identify a novel gene, MOZ, w

146 bridization (FISH) analysis to show that the chromosome 8 breakpoints fell within YAC 899e2 and that

147 s further localized to the central region of chromosome 8 by interspecific backcross mapping; a relat

148 ditional Southern and Northern analysis, and chromosome 8 cDNA microarray expression profiling.

149 The centromere of rice Chromosome 8 (Cen8) has an unusually low abundance of hi

150 creases in c-myc copy number relative to the chromosome 8 centromere), which was detected in 0, 8, an

151 ncrease in c-myc copy number relative to the chromosome 8 centromere), which was identified in 42, 25

152 ncrease in c-myc copy number relative to the chromosome 8 centromere, which was found in 8, 11, and 2

153 , perhaps as a result of a deletion near the chromosome 8 centromere.

154 alterations including chromosome 8p loss and chromosome 8 centromeric gain.

155 to have 8p LOH using a chromosome 8 paint, a chromosome 8 centromeric probe, and a series of single-c

156 two QTLs with significant linkage for TC: on chromosome 8, chr8-60.4 Mbp (LOD 3.8), and chromosome 2,

157 s 88% +/- 3%; Plog rank = .0008) and gain of chromosome 8 (CIR/NR, 16% +/- 7% vs 3% +/- 2%, PGray = .

158 The yellowbeak mutation mapped to chromosome 8, close to a cluster of cytochrome P450 loci

159 Slug maps to the long arm of chromosome 8, closely linked to D8S2090 between D8S519 a

160 F344(Cia4) (chromosome 7) and DA.F344(Cia6) (chromosome 8) congenics had significantly lower exudate

161 for relative water content were detected on chromosome 8, congruent with an osmotic adjustment QTL i

162 ithin mouse chromosome 8 was confirmed using chromosome 8 consomic mice.

163 etastasis suppressor gene, a truncated human chromosome 8 containing this region was transferred into

164 Interestingly, this region of chromosome 8 contains a cluster of three other genes imp

165 tients with MYC:CEP8 ratio < 1.3 with normal chromosome 8 copy number (n = 141; 18%) and >/= 1.3 or <

166 tential associations between alternative MYC/chromosome 8 copy number alterations and differential be

167 A single marker on chromosome 8 (D8S593) and two adjacent markers on chromo

168 Mutations of the MD-2 associated gene on chromosome 8 degrade a human's innate responses.

169 s of DNA from cell lines that contain either chromosome 8 deletions or duplications further localized

170 romosome 8, and the presence of a segment of chromosome 8 derived from Solanum pennellii LA0716 cause

171 Here we report that substitution of the chromosome 8-derived ETO protein for the multifunctional

172 ed, including three new loci in intervals on chromosomes 8 (eae14), 10 (eae17), and 18 (eae18).

173 significant QTL found was for CD4 levels on chromosome 8 (empirical P=.00005).

174 show that AML+8 blasts overexpress genes on chromosome 8, estimated at 32% on average, suggesting ge

175 Unique loci were identified on chromosome 8 for orchitis, chromosome 16 for epididymiti

176 neity within IGE and provide no evidence, on chromosome 8, for a gene common to all IGEs.

177 L1 gene on chromosome 21, to the ETO gene on chromosome 8, forming the AML1/ETO fusion gene.

178 ML1 gene on chromosome 21 to the ETO gene on chromosome 8 fuses the NH2-terminal portion of AML1 to n

179 ency, distribution, proliferative state, and chromosome 8 gain of benign prostate, SA, PAH, HGPIN, an

180 ingle region-data from 64 MSI GOAs show that chromosome 8 gains are early genetic events and that the

181 tify TRC8 (translocation in renal carcinoma, chromosome 8 gene), an ER-resident E3 ligase previously

182 on chromosome 21 and the ETO (MTG8) gene on chromosome 8, generates AML1-ETO fusion proteins.

183 L1 gene on chromosome 21 and the ETO gene on chromosome 8, generates an AML1-ETO fusion transcription

184 osome 21 and the ETO (MTG8, RUNX1T1) gene on chromosome 8 generating the AML1-ETO fusion proteins.

185 er variation in beta-defensin genes on human chromosome 8 has been proposed to underlie susceptibilit

186 We demonstrate that the Os chromosome 8 has suffered two breaks that are 5 cM ( app

187 orphism of the beta-defensin region on human chromosome 8 in 179 Dutch individuals with psoriasis and

188 high-resolution map of genomic imbalances of chromosome 8 in 51 primary colon carcinomas using a cust

189 have investigated interstitial deletions of chromosome 8 in 70 colorectal carcinomas and 11 colonic

190 how that its murine homologue maps to murine chromosome 8 in a region syntenic with the human WRN gen

191 We found evidence for linkage to chromosome 8 in adolescent-onset IGE families in which J

192 separate, two-exon genes that are located on chromosome 8 in both mice and humans.

193 ore was associated with the SNP rs2645424 on chromosome 8 in farnesyl diphosphate farnesyl transferas

194 onfirmed loss of CDKN2A and amplification of chromosome 8 in IM but not in a nearby NGM biopsy.

195 exon genes that map to homologous regions of chromosome 8 in mice and humans, and the differential ex

196 involvement of a tumor suppressor gene(s) on chromosome 8 in prostatic neoplasms, we performed a comp

197 Another three lipoblastomas had polysomy for chromosome 8 in the absence of PLAG1 rearrangement.

198 Smpd3 locus as part of a 980-kb deletion on chromosome 8 in the fro/fro mutant, generated by chemica

199 lines, we previously identified a region of chromosome 8 in the Solanum pennellii LA0716 genome (IL8

200 translocation breakpoint on the short arm of chromosome 8 in this patient.

201 ternal meiosis, we investigated the maternal chromosomes 8 in eight mothers of subjects with inv dup(

202 ce, Lmp10 is a single-copy gene localized to chromosome 8, in a region of conserved synteny with huma

203 The Zfp319 gene maps to chromosome 8, in a region of conserved synteny with the

204 me 6 for JME (lod score 2.5/4.2), a locus on chromosome 8 influencing non-JME forms of IGE (lod score

205 nature of divergent selection in a region of chromosome 8 introgressed from maize and encompassing ZC

206 ersion (p31q12), and 3,168 were analyzed for chromosome 8 inversion (p23q22).

207 ecombination frequency of 13.1% recorded for chromosome 8 inversion was similar to the frequency of 1

208 This region of chromosome 8 is a gene desert, far from any recognized g

209 e have discovered that the Ft locus on mouse chromosome 8 is associated with cell cycle deficits with

210 henotype linked to the pericentric region of chromosome 8 is associated with mutations in a gene desi

211 he region of linkage to DTNBP1 expression on chromosome 8 is contiguous with linkage findings based u

212 content and fraction segmentally duplicated, chromosome 8 is distinctly typical in character, being v

213 The short arm of chromosome 8 is frequently lost in epithelial cancers, a

214 The CP on chromosome 8 is predicted to have a CPA-like specificity

215 The gene on chromosome 8 is the fibroblast growth factor receptor 1

216 Gain of chromosome 8 is the most common chromosomal gain in huma

217 The beta3 adrenergic receptor, located on chromosome 8, is a regulator of energy expenditure and l

218 which is a 278 amino acid protein encoded on chromosome 8, known to be synthesized in the liver.

219 ) identified one novel genomic risk locus on chromosome 8 (lead SNP rs17052966, p = 4.53 x 10(-9), od

220 Abnormalities in chromosome 8, leading to rearrangements of the PLAG1 gen

221 clone spanning the OVE459 insertion locus on chromosome 8 led to the identification of two novel cand

222 hese analyses suggest that the action of the chromosome 8 locus is specific to unesterified cholester

223 Linkage to the chromosome 8 locus was excluded in this family.

224 OD = 1.54) for moderate airflow obstruction, chromosomes 8 (LOD = 1.36) and 19 (LOD = 1.09) for mild

225 d non-drug-induced locomotor activity (e.g., chromosome 8; LOD = 18.9).

226 Chromosome 8 loss of heterozygosity (LOH) in cancer of t

227 nd a cytological marker on the centromere of chromosome 8 made it possible to discriminate between ea

228 No loss of heterozygosity of p53 or for chromosome 8 markers was found.

229 tations, loss of heterozygosity for TP53 and chromosome 8 markers, and ras mutations.

230 r Ets-1, located within one of these loci on chromosome 8, mediates glomerular injury in SS hypertens

231 Mf1 was assigned to mouse Chromosome 8, Mf2 to Chromosome 4, Mf3 to Chromosome 9,

232 el or a recurrence of a previously described chromosome 8 mouse mutant, toppler mice were crossed wit

233 d mapping localized this gene to a region of chromosome 8 near several other defensins, MBD-2, MBD-3,

234 s a single copy and located at the middle of Chromosome 8 near the mutations for myodystrophy (myd) a

235 The SNPs were rs4355801, on chromosome 8, near to the TNFRSF11B (osteoprotegerin) ge

236 roups: (a) those with simple gain of a whole chromosome 8 (no increase in c-myc copy number relative

237 Both genes lie on chromosome 8, not previously suspected to contain imprin

238 ing peptide receptor (GRPR) gene and that on chromosome 8 occurred approximately 30 kb distal to the

239 otein lipase (LPL) locus on the short arm of chromosome 8 (P = 0.002, n = 125 sib-pairs, for the hapl

240 t evidence for association was at a locus on chromosome 8 (P=1.5 x 10(-15)) spanning 4 Mb and contain

241 unts (P = .007), and more often trisomies of chromosomes 8 (P = .01) and 21 (P < .001) and less often

242 Loss of the p arms of chromosome 8 (p10>pter) and chromosome 10 (p10>pter) and

243 sma fatty acids identified a 20-cM region on chromosome 8 (p12-p21) with a quantitative trait locus f

244 cell lines predicted to have 8p LOH using a chromosome 8 paint, a chromosome 8 centromeric probe, an

245 Notably, the lipoblastomas with chromosome 8 polysomy had up to five copies of chromosom

246 nhardtii, which we mapped to the long arm of chromosome 8, provides a good experimental system for th

247 osome 10 (p10>pter) and gain of the q arm of chromosome 8 (q10>qter) were only observed in the tumor

248 ing 26 microsatellite markers, we mapped the chromosome 8 regions frequently deleted in lung cancer i

249 Two chromosome 8 regions were associated: p = 4.45 x 10(-8)

250 The region of human chromosome 8 retained in each microcell hybrid was deter

251 s of a major resistance QTL in RIL LA3952 on chromosome 8 revealed that the presence of Lso is requir

252 significant association was revealed for the chromosome 8 rs6996198 with HIV-1 acquisition and was re

253 Two YAC probes targeted chromosome 8 short arm regions known to be deleted frequ

254 several chromosome regions, particularly the chromosome 8 short arm.

255 The data from chromosome 8 should allow a better understanding of both

256 Alterations in chromosome 8 show unique correlation with melanocytosis.

257 on chromosomes 2, 4, and 8, with the QTL on chromosome 8 showing the highest significance, as well a

258 contain the GPT sequence were derived from a chromosome 8-specific library.

259 an integrated map for a 35-cM area of human chromosome 8 surrounding the Langer-Giedion syndrome del

260 herin-11, was mapped to a specific region of chromosome 8 that also includes cadherins-1, -3, and -5.

261 ited Htn1 to a 131.7-kb physical interval on chromosome 8 that contained three candidate genes encodi

262 to detect a modifier locus, Modpkdr1, on rat chromosome 8 that controls PKD severity, kidney mass and

263 CKbeta4GT-II maps to a region of chicken chromosome 8 that exhibits conserved synteny with human

264 ial tumors show deletion of the short arm of chromosome 8 that is related to aggressive disease or ad

265 lar dystrophy and maps to a segment of mouse chromosome 8 that is syntenic with human 4q31-4q35.

266 is part of a defensin beta (DEFB) cluster on chromosome 8 that is variable in copy number.

267 a high-resolution map of a segment of mouse chromosome 8 that places the nr locus in a genomic segme

268 ned within a "half-YAC" from the long arm of chromosome 8, that is, a YAC containing the 8q telomere.

269 At a second locus, Sst2, on chromosome 8, the L. hirsutum allele specified accumulat

270 gene is localized at the proximal portion of chromosome 8, the site where genes for mouse alpha- and

271 ene was localized at the proximal portion of chromosome 8, the site where mouse alpha-defensins are f

272 ine chromosome 16 and mXCP4 is positioned on chromosome 8; the hXCP1 and hXCP2 genes are located at h

273 ntains six exons and is located on zebrafish chromosome 8; the structure of the gene is highly homolo

274 que to delete in mice the syntenic region on chromosome 8 to create a Dnajb1-Prkaca fusion and monito

275 Allele loss from the long arm of chromosome 8 was also observed with 30.0% (6/20) and 33.

276 A locus within mouse chromosome 8 was confirmed using chromosome 8 consomic m

277 tandem 12 kb repeats which spanned a gap on chromosome 8 was found to be 600 kb to 1.7 Mbp in size,

278 The EXT1 gene on chromosome 8 was recently identified and characterized.

279 Maize chromosome 8 was recovered as a monosomic addition (2n =

280 ingle-nucleotide polymorphism (rs4445257) on chromosome 8 was strongly associated with the normal gro

281 In addition, a QTL on chromosome 8 was suggestive for linkage.

282 ies varied between the chromosomes examined; chromosome 8 was the least affected, and 17p loss was fo

283 The mouse Aprt locus on chromosome 8 was used as the selectable target for the s

284 tion to the previously identified linkage to chromosome 8, we find that a locus on chromosome 17, whi

285 e short arm and one locus on the long arm of chromosome 8 were used for PCR-based LOH analysis on mat

286 y at a pseudo attP sequence located on human chromosome 8, when measured in the native genomic enviro

287 e mapped this mutation to the same region of chromosome 8 where Os has been assigned.

288 ng evidence for selection was found on maize chromosome 8, where several putatively selected loci col

289 4 as well as at distinctly different loci on chromosome 8 which were undetected through CIM.

290 murine mTll gene (Tll) is mapped to central chromosome 8, which is a different chromosomal location

291 t except for the quantitative trait locus on chromosome 8, which is derived from C57BL/6.

292 mnd maps to the centromeric region of mouse chromosome 8, which likely corresponds to portions of hu

293 nd 338-kb deletions in a noncoding region on chromosome 8 with an approximately 200-kb overlapping se

294 slocation, which fuses the ETO gene on human chromosome 8 with the AML1 gene on chromosome 21 (AML1-E

295 Results link the JDCM locus to canine chromosome 8 with two-point and multipoint lod scores of

296 ere not found by the microsatellite markers: chromosome 8, with a maximum model-free LOD score of 2.2

297 hromosome 6, and seven contiguous markers on chromosome 8, with empirical P values of .00001.

298 arkers, the rocker locus was mapped to mouse chromosome 8 within 2 centimorgans of the calcium channe

299 On pericentromeric chromosome 8 within our previously reported linkage peak

300 These SNPs were mapped on Chromosome 8 within the Linkage Group 8 previously ident