ライフサイエンスコーパス: chromosome 9

1 e., 68-76 MB in chromosome 7 and 26-36 MB in chromosome 9.

2 ) was a frequent event, especially affecting chromosome 9.

3 that result in the fusion of NUT to BRD3 on chromosome 9.

4 The rc locus was previously mapped to Chromosome 9.

5 valent to a similar stay-green locus on rice chromosome 9.

6 Submergence 1 (Sub1) near the centromere of chromosome 9.

7 hort (p) arm and part of the long (q) arm of chromosome 9.

8 road, gene-rich centromeric region of canine chromosome 9.

9 at had been translocated to the short arm of chromosome 9.

10 of regulatory domains identified a locus on chromosome 9.

11 ransferrin-like gene map close to the QTL on chromosome 9.

12 spots coinciding with GLS resistance QTLs on chromosome 9.

13 of late-onset Alzheimer's disease (LOAD) to chromosome 9.

14 y of Plasmodium falciparum was identified on chromosome 9.

15 ern leaf blight-has been identified on maize chromosome 9.

16 Genetic mapping placed mhl1 within bin 4 on chromosome 9.

17 nkage group marker BNL4053 was reassigned to chromosome 9.

18 ignment of marker BNL4053 to the long arm of chromosome 9.

19 nge of 1.6-2.1 previously reported for maize chromosome 9.

20 ce of a BP QTL in a large 34.2-cM segment of chromosome 9.

21 motion susceptibility locus, Psl1, to distal chromosome 9.

22 d 17, and for triglyceride concentrations on chromosome 9.

23 ked to either the bz or the wx donor loci on chromosome 9.

24 n in neonatal brain and was located on mouse chromosome 9.

25 mapped to human chromosome 15q22.1 and mouse chromosome 9.

26 A0020, a gene of unknown function located on chromosome 9.

27 polymorphism placing the pccb locus on mouse chromosome 9.

28 22.3-q23 and to the syntenic region on mouse chromosome 9.

29 , and NY-BR-1.1 was tentatively localized to chromosome 9.

30 ysis, GBDR1 cDNA identified a single gene on chromosome 9.

31 y to rat type II collagen LOD 5.2) mapped to chromosome 9.

32 ructure, and assigned its location to distal chromosome 9.

33 d all three genes in close vicinity on mouse chromosome 9.

34 ied due to its proximity to the Punc gene on chromosome 9.

35 e MMP gene cluster at the centromeric end of chromosome 9.

36 TB-9Sb is a translocation between the B and chromosome 9.

37 e families is linked to markers near TIE2 on chromosome 9.

38 spans approximately 9 kb pericentromeric on chromosome 9.

39 apolipoprotein (apo) AI-CIII gene cluster on chromosome 9.

40 gene, which maps close to the Waxy locus of chromosome 9.

41 Hnf6 gene is located in the middle of mouse chromosome 9.

42 ll adhesion molecule (Ncam) locus located on chromosome 9.

43 the very terminal region of the short arm of chromosome 9.

44 e major histocompatibility complex (MHC) and chromosome 9.

45 ansion recently found within the gene X25 on chromosome 9.

46 a putative pseudogene of MMAC1 localized on chromosome 9.

47 ects were detected across a large portion of chromosome 9.

48 osome 7 and a deletion of at least 232 kb on chromosome 9.

49 he rapid mapping of the response to SCR74 to chromosome 9.

50 ing cases previously identified as linked to chromosome 9.

51 gene, harlequin (H), previously localized to chromosome 9.

52 was mapped to a position near the bottom of chromosome 9.

53 keratinocyte stem cell locus (Ksc1) on mouse chromosome 9.

54 ion of the chromosome 7 locus and a locus on chromosome 9.

55 pedigrees, we mapped the EIC locus to canine chromosome 9.

56 tic crosses that brought together two broken chromosomes 9.

57 identified five new susceptibility loci, on chromosomes 9, 10 and 11 (P = 4.6 x 10(-7) to P = 3.2 x

58 n FPC, the scenario for building the maps of chromosomes 9, 10 and 13, and the results from the simul

59 800 clones selected for sequencing for human chromosomes 9, 10 and 13.

60 cardiac modifiers of nmd were identified on chromosomes 9, 10 and 16, which account for over 26% of

61 e loss strategy to individually delete mouse chromosomes 9, 10, 12, or 14 in tetraploid immortalized

62 s (Cia15, Cia16*, Cia17, Cia18* and Cia19 on chromosomes 9, 10, 18 and two on the X chromosome, respe

63 genes among the three cell lines, notably on chromosomes 9, 11, 12, and 14, suggesting their involvem

64 n chromosome 10q22 and suggestive linkage on chromosomes 9, 11, and 13 for major genetic determinants

65 al quantitative trait loci (QTLs) located on chromosomes 9, 12, 14, and 19, whereas maximum disease s

66 ce for linkage of LOFAD age-at-onset loci to chromosomes 9, 12, or 21.

67 age to loci on proximal chromosome 14 and on chromosome 9; 129S6 alleles protect against the lethal e

68 itional areas of interest were identified on chromosomes 9, 14, and 18.

69 less assemblies of chromosome 3 (236 Mb) and chromosome 9 (162 Mb), and 53 Mb of the Ab10 meiotic dri

70 This revealed several loci located on chromosomes #9, #18, and X that were suggestive of linka

71 The suggestive QTL near the proximal end of chromosome 9 (2.4 cM, LOD: 3.12) was replicated at both

72 .1], on chromosome 1 and a suggestive QTL on chromosome 9 (38 cM, LOD 2.3) that influenced plasma glu

73 icant QTL for aortic root lesion size was on chromosome 9 (61 Mb, LOD=6.9), but it was distinct from

74 Deletion of the distal short arm of chromosome 9 (9p) has been reported in a number of cases

75 n showing homology to the short arm of human chromosome 9 (9p13).

76 We have identified a 14 cM interval on chromosome 9 (9q12-9q22.2), flanked by proximal marker D

77 B subunit gene, localized to the long arm of chromosome 9 (9q34) by fluorescence in situ hybridizatio

78 DMT1 maps to the distal short arm of chromosome 9, a location implicated in human XY sex reve

79 r umc 105a near the brown pericarp1 locus on chromosome 9, accounted for 10.8% of the variance.

80 at 30, 40, and 50 krad) of a monosomic maize chromosome 9 addition line produced maize chromosome 9 r

81 lated from a genomic library of an oat-maize chromosome 9 addition line with the help of the 180-bp k

82 ic library made of DNA purified from a maize chromosome 9 addition line.

83 erferon-kappa is located on the short arm of chromosome 9 adjacent to the type I interferon gene clus

84 ve transcription factor ALL1-fused gene from chromosome 9 (Af9) as well as the release of this repres

85 ntification of a previously unknown locus on chromosome 9S affecting flavone level.

86 The applicability of chromosome 9 allelic losses as non-invasive markers of u

87 This area of chromosome 9 also contains a short, direct tandem repeat

88 re located close to the telomeric regions of chromosome 9 and 6, respectively.

89 ficant QTL, named Ath22 (42 cM, LOD 4.1), on chromosome 9 and a suggestive QTL near D11mit236 (20 cM,

90 and two distinct interacting QTLs (AS-f1 on chromosome 9 and AS-f2 on chromosome11, LOD 8.9) in fema

91 , we have now localized the B10.Q/J locus to chromosome 9 and established its identity as Tyk2, a Jan

92 ed the gene encoding alpha-tectorin to mouse chromosome 9 and human chromosome 11 in a known region o

93 p within the region of synteny between mouse chromosome 9 and human chromosome 15.

94 ic single nucleotide polymorphisms (SNPs) on chromosome 9 and identified two SNPs in the death-associ

95 The Fem1b gene localizes to chromosome 9 and is highly expressed in adult testis.

96 5, and the orthologous regions are on human chromosome 9 and mouse chromosome 4.

97 with this translocation, one on the abnormal chromosome 9 and one on the Philadelphia chromosome (Ph

98 atient's cells enabled screening of the MSSE chromosome 9 and showed no CDC14B deletion or mutation t

99 ly been localized to a small region of mouse chromosome 9 and we have now identified the gene and the

100 The stc1 gene is located on chromosome 9S and does not seem to have a closely relate

101 copy number changes indicate that losses of chromosomes 9 and 10 occur early on in melanoma progress

102 plications of the X chromosome, deletions of chromosomes 9 and 10, and translocations involving chrom

103 ed for genes with trans-eQTLs in hotspots on chromosomes 9 and 10, which also coincided with phenotyp

104 1 onto human chromosomes 9 and 10.

105 r Alzheimer's disease susceptibility loci on chromosomes 9 and 10.

106 Our analysis revealed loci on Chromosomes 9 and 11 that affect prion susceptibility.

107 of the highly similar copies of this gene on chromosomes 9 and 11.

108 ere unique to each population with blocks on chromosomes 9 and 12 in DRH and 3, 4, 6 and 8 in D84.

109 vidence of clustering, particularly on human chromosomes 9 and 12.

110 3q, and 22q are syntenic with loci on mouse chromosomes 9 and 16 that are implicated in a murine tra

111 fically, the frequency of losing portions of chromosomes 9 and 16 was significantly modulated by gene

112 anager QT revealed QTLs linked to markers on chromosomes 9 and 16.

113 duced thymocyte apoptosis were identified on chromosomes 9 and 16.

114 heterozygosity and microsatellite shifts on chromosomes 9 and 17, sequence mutations in CDKN2A/MTS1/

115 distal eQTL hotspots between the crosses on chromosomes 9 and 17.

116 arises by a reciprocal translocation between chromosomes 9 and 22 and harbors the BCR-ABL fusion onco

117 esults from a balanced translocation between chromosomes 9 and 22 and is found in patients with chron

118 formed by a reciprocal translocation between chromosomes 9 and 22 that fuses BCR-encoded sequences up

119 The reciprocal translocation between chromosomes 9 and 22 that fuses coding sequences of the

120 by a reciprocal translocation between human chromosomes 9 and 22.

121 sult from a reciprocal translocation between chromosomes 9 and 22; this translocation leads to a shor

122 Chromosomes 9 and Y can be discriminated and purified by

123 rophic lateral sclerosis have been linked to chromosome 9, and an expansion of the GGGGCC hexanucleot

124 Ruminococcus and rs150018970 near RAPGEF1 on chromosome 9, and between Coprococcus and rs561177583 wi

125 74, PDCD1LG2, and the centromeric portion of chromosome 9, and immunohistochemistry (IHC) using an an

126 distal mouse chromosome 1 and proximal mouse chromosome 9, and the human regions with conserved synte

127 We generate chromosome 9- and Y-specific 'paints' from the sorted fr

128 Linkage analysis mapped rd6 to mouse Chromosome 9 approximately 24 cM from the centromere, su

129 n the empirical distribution of F(ST) across chromosome 9 (approximately 99.5-99.9th centile).

130 Allelic losses in the long arm of chromosome 9 are commonly encountered in many human mali

131 romising pair of genes located 8 MB apart on chromosome 9 are identified based on as demonstrating st

132 Deletions of the long arm of chromosome 9 are the most common genetic alteration in t

133 ification of terpene synthase21 (ZmTps21) on chromosome 9 as a beta-costic acid pathway candidate gen

134 paralogous to the pericentromeric regions of chromosome 9 as well as to 2q13, the site of an ancestra

135 s panel, we place the Adamts8 locus on mouse chromosome 9 at a consensus position of 11 cM and its hu

136 related antimicrobial peptide, was mapped to chromosome 9 at a region of conserved synteny to which g

137 uman LHX3 maps to the subtelomeric region of chromosome 9 at band 9q34.3, within a region noted for c

138 localization analysis assigned Boo to murine chromosome 9 at band d9.

139 The gene for this enzyme maps to chromosome 9 at band p21 where the cyclin-dependent kina

140 ne encoding this chain, LAMC3, which maps to chromosome 9 at q31-34.

141 fluorescence in situ hybridization to mouse chromosome 9 at region 9E3-F1 and to human chromosome 3

142 uman type I interferon (IFN) gene complex on chromosome 9, band p21-22, to examine the overall struct

143 entified by linkage analysis to distal mouse chromosome 9 between D9Mit154 and D9Mit330, closely link

144 etween D3S3053 and D3S2427 (LOD = 1.36), and chromosome 9 between D9S1120 and D9S910 (LOD = 1.46).

145 .2 gene and genetically mapped the marker on chromosome 9 between markers D9S1825 and D9S290 with odd

146 he original collection sites was observed on chromosome 9 but not on chromosome 10 or 19.

147 was localized to the distal region of mouse chromosome 9 by interspecific backcross mapping.

148 enes, and localizes to the central region of chromosome 9 by single-strand polymorphism analysis.

149 The recent discovery that ORF 72 on chromosome 9 (C9orf72), the gene most commonly mutated i

150 al that genetic information, such as loss of chromosome 9, can be obtained from widely available tech

151 patient had segmental uniparental disomy of chromosome 9, carrying 2 copies of the maternal CARD9 mu

152 erations to the locus of E-NTPDase2 on human chromosome 9 cause severe head and eye defects, includin

153 etained heterozygosity of the TSC1 region on chromosome 9 caused an apparently TSC2- and mTOR-indepen

154 gest that genetic variation at this locus on chromosome 9 causes sporadic ALS and familial ALS-fronto

155 9/CNND1), as well as homozygous deletions in chromosome 9 (CDKN2A).

156 nomic copies of CNTNAP3 on both sides of the chromosome 9 centromere flanking the polymorphic heteroc

157 prcd close to the centromeric end of canine chromosome 9 (CFA9), and excludes RARA as a candidate ge

158 pid valve malformation (CTVM) maps to canine chromosome 9 (CFA9), in a region syntenic with gene-dens

159 ve retinal degeneration of dogs that maps to chromosome 9 (CFA9).

160 Other significant loci were on chromosome 9 close to ASTN2, chromosome 6 between FILIP1

161 The rc locus was mapped at 32.0 cM on chromosome 9, close to the loxl gene.

162 Scn11a is located on mouse chromosome 9, close to the two other TTX-R sodium channe

163 The finished euchromatic sequence of chromosome 9 comprises 109,044,351 base pairs and repres

164 have recently identified a portion of mouse chromosome 9 containing a paternally methylated region a

165 apping of the H locus to a 3.27 Mb region of chromosome 9 containing approximately 20 genes.

166 egion to an approximately 105 kb interval on chromosome 9 containing six genes.

167 l genome-wide significant locus with FEV1 on chromosome 9 [DBH] in a meta-analysis of three study pop

168 ogene highly homologous to ESO3 was found on chromosome 9, designated psiESO3.A search of the rodent

169 u, which is encoded by a cluster of genes on chromosome 9, differs between the sexes in cattle, as as

170 Chromosome 9 displayed the highest levels of AI and comp

171 ssing over and pericentric inversions during chromosome 9 evolution.

172 ocus, in PTPN22, and in a SNP (rs3761847) on chromosome 9 for all samples tested, the latter with an

173 By separating chromosome 9 from chromosomes 10-12 on the basis of poly

174 low DNA damage (low genomic scar score with chromosome 9 gain) and a superior outcome (100% overall

175 associations (P values to ) with a region on chromosome 9 harboring cinnamoyl-CoA reductase, a key en

176 e also identified a novel significant QTL on chromosome 9 (Hfhl3) with moderate effects on 2f1-f2 emi

177 n mapped to the centromeric region of canine chromosome 9, homologous to human 17q, which contains th

178 panel, mouse Treh was shown to be located on Chromosome 9 in a broad region that is orthologous with

179 titative trait loci with additive effects on chromosome 9 in a region likely to have been inherited f

180 ouse lysyl oxidase-like gene (Loxl) to mouse Chromosome 9 in a region that shares linkage conservatio

181 acterized the RNAs encoded by this region of chromosome 9 in cell lines derived from individuals homo

182 nine genetically mapped single-copy genes on chromosome 9 in one FISH experiment.

183 ntified deletions near the Atm gene on mouse chromosome 9 in radiation-induced lymphomas from p53 het

184 r tumor suppressor genes on the short arm of chromosome 9 in SCLC, we tested 46 primary SCLCs by micr

185 The arm ratio (long/short) for maize chromosome 9 in the addition line was 1.7, comparable to

186 e for chagun is recessive and maps to distal chromosome 9, in a region homologous to human chromosome

187 ines possessing different fragments of maize chromosome 9 including intergenomic translocations and m

188 The evolution of alterations on chromosome 9, including the putative tumor suppressor ge

189 of linkage to renal disease, and a locus on chromosome 9 influenced serum cholesterol but not nephro

190 ssing the R low-blood-pressure QTL allele on chromosome 9 into the S strain was constructed.

191 The homologous location on mouse chromosome 9 is also the site of the mouse neurologic mu

192 mon genetic variant at the TRAF1-C5 locus on chromosome 9 is associated with an increased risk of ant

193 Chromosome 9 is highly structurally polymorphic.

194 rs indicating that loss of heterozygosity on chromosome 9 is not driven by Atm, but by an alternative

195 OS case, WGS revealed paternal isodisomy for chromosome 9, leading to identification of the causal ho

196 eat expansion in C9orf72 was responsible for chromosome 9-linked amyotrophic lateral sclerosis and fr

197 xpansion in the C9ORF72 gene as the cause of chromosome 9-linked frontotemporal dementia and motor ne

198 mice, we validate that both the MHC and the chromosome 9 loci influence the proportion and absolute

199 Our findings indicate that a chromosome 9 locus harboring tightly linked variants in

200 (LOD 9.7) for systolic blood pressure and on chromosome 9 (LOD 8.7) for aortic diameter.

201 both persistent and recovered stuttering) on chromosome 9 (LOD = 2.3 at 60 cM) and of the narrower di

202 143 cM of chromosome 5 (lod = 2.0), 7 cM of chromosome 9 (lod = 2.8), 12 cM of chromosome 11 (lod =

203 age was observed at the telomere (159 cM) of chromosome 9 (lod = 4.5) in all pedigrees.

204 6 cM on chromosome 5; lod = 3.5 at 160 cM on chromosome 9; lod = 2.5 at 7 cM on chromosome 12; and lo

205 A suggestive linkage was also found on mouse Chromosome 9 (logarithm of odds ratio score 2.0) around

206 bility to whipworm infection, one located on chromosome 9 (logarithm of the odds ratio [LOD] score, 3

207 sity (LOH) and reduced genome copy number on chromosomes 9 (LOH9) and 16 (LOH16), aberrations which w

208 ified another trichome-expressed ASH gene on chromosome 9 (M82, Solyc09g075710; LA0716, Sopen09g03052

209 The novel region q34.11 on chromosome 9 (max NPL= 2.07 at rs913275) was identified.

210 One significant QTL on chromosome 9, named Bglu17 [26.4 cM, logarithm of odds r

211 stributions for 12 markers spanning 20 cM on chromosome 9 narrowed the possible location of an DAT su

212 n the discovery meta-analysis, rs10757269 on chromosome 9 near CDKN2B had the strongest association w

213 th the strongest evidence of linkage were on chromosome 9 near D9S301 and on 10 near D10S1230, with p

214 Deletions were found on chromosome 9 near the Atm locus in radiation-induced tum

215 dehydrin-like protein maps to a new locus on chromosome 9S near wx1, which we have named dhn2.

216 mallest P-value was observed for a marker on chromosome 9 (near PTPRD, P=4.13 x 10(-)(7)).

217 3, near D3S3053 (multipoint LOD = 1.48); on chromosome 9, near D9S910 (multipoint LOD = 1.12) and D9

218 Deletions involving chromosome 9 occur in more than 50% of human bladder can

219 nit length was isolated from the knob DNA of chromosome 9 of Zea mays L.

220 Hexanucleotide repeat expansions in chromosome 9 open reading frame 72 (C9ORF72) are the mos

221 Expanded hexanucleotide repeats in the chromosome 9 open reading frame 72 (C9orf72) gene are th

222 The identification of the chromosome 9 open reading frame 72 (c9orf72) gene hexanu

223 CC) expansion in the noncoding region of the chromosome 9 open reading frame 72 (C9orf72) gene is the

224 Hexanucleotide repeat expansions in the chromosome 9 open reading frame 72 (C9orf72) gene underl

225 Here, two patients with bvFTD-SP with chromosome 9 open reading frame 72 (C9ORF72) hexanucleot

226 w is to describe disease mechanisms by which chromosome 9 open reading frame 72 (C9ORF72) repeat expa

227 ogy (p = 0.02), including but not limited to chromosome 9 open reading frame 72 (C9orf72) repeat expa

228 carriers (n=185) of a pathogenic mutation in chromosome 9 open reading frame 72 (C9orf72), progranuli

229 The molecular mechanisms that underlie chromosome 9 open reading frame 72 (C9orf72)-associated

230 icrotubule-associated protein tau (MAPT), or chromosome 9 open reading frame 72 (C9orf72).

231 repeat expansion in the noncoding region of chromosome 9 open reading frame 72 (C9ORF72).

232 ucleotide repeat in the non-coding region of chromosome 9 open reading frame 72 has recently been ide

233 es including the recently described c9orf72 (chromosome 9 open reading frame 72) gene, suggesting an

234 otide repeat in the non-coding region of the Chromosome 9 open-reading frame 72 (C9orf72) gene is the

235 G4C2) hexanucleotide repeat expansion in the chromosome 9 ORF 72 (C9ORF72) gene is a common cause of

236 sted, with the exclusion of samples carrying chromosome 9 ORF 72 (C9orf72) repeat expansions.

237 ne (P = 4.19 x 10(-8) for rs6445055), two on chromosome 9 (P = 2.80 x 10(-11) for rs2472493 near ABCA

238 Fluorescence in situ hybridization with a chromosome 9 paint revealed that some of the Ncam deleti

239 it was associated with concomitant increased chromosome 9 polysomy (P = .002); and with a younger age

240 or CD1 genotype, CD1 protein expression, and chromosome 9 polysomy (representing genomic instability)

241 e evidence for linkage conditional on either chromosome 9 (positive) or chromosome 7 (negative); this

242 ysical map with the cytological structure of chromosome 9 provides a comprehensive cross-referenced c

243 hromosomes, although most (34%) were on rice chromosome 9 (R9).

244 ze chromosome 9 addition line produced maize chromosome 9 radiation hybrids (M9RHs)-oat lines possess

245 hermore, we mapped an overlapping homozygous chromosome 9 region containing PSAT1 in four consanguine

246 mia patients harboring a partial deletion of chromosome 9 revealed a significant decrease in HNRNPK e

247 One SNP located near DBH on chromosome 9 (rs3025343[G], OR = 1.12, 95% Cl 1.08-1.18,

248 Finally, we identified a locus on chromosome 9 (rs755383, OR=1.37, P=1.12x10(-23)), contai

249 a second, epistatic dwarfing QTL on sorghum chromosome 9 (Sb-HT9.1).

250 o be 9S.79 (at position 79% of the length of chromosome 9 short arm) for sCBM9.1, 9S.65 for sCBM9.2,

251 One locus, at ALAD on chromosome 9, showed consistent association with blood l

252 ction effects of chromosome 7 SNP rs6960920, chromosome 9 SNP rs4744411, and NLRP1 SNP rs6502867 on b

253 umors selected for loss of heterozygosity on chromosome 9, some of which were present in the germ lin

254 Evidence for linkage on chromosome 9 stemmed primarily from excess homozygosity

255 entify a homozygous loss on the short arm of chromosome 9 suggesting the presence of a novel TSG locu

256 nterestingly, mapping of the mouse Cox7al to chromosome 9 suggests a new syntenic region between the

257 We identified a strong QTL on mouse chromosome 9 that differentially affected macrophage iro

258 e identified a human genomic DNA sequence on chromosome 9 that encodes human inositol 1,3,4,5,6-penta

259 identified including an intergenic region on chromosome 9 that has previously been associated with ne

260 d genome-wide search, identifying a locus on chromosome 9 that influences the severity and progressio

261 and Sheepdog breed and discovered a locus on chromosome 9 that is associated with a dental abnormalit

262 Moreover, we identify a locus on mouse chromosome 9 that is strongly linked to the proportion a

263 ping, Folbp3 was mapped to a region on mouse chromosome 9 that is syntenic to human chromosome 19p13.

264 Apc(Min/+) mice has identified a modifier on chromosome 9 that significantly affects tumor multiplici

265 tified two exon-trapping products from human chromosome 9 that were highly homologous to hamster LMX1

266 polymorphic sites covering a 5-kb region of chromosome 9 (the msp1 gene) have been typed in 547 isol

267 xanucleotide expansion in C9orf72 (ORF 72 on chromosome 9) the two most frequent causes of ALS.

268 In two large unrelated kindreds mapping to chromosome 9, the identical R849W missense mutation was

269 kage to markers in a single region of vervet chromosome 9; this observation suggests the possibility

270 rthologs map to a homologous region on mouse chromosome 9; TMOD4 maps to the telomeric end of 1q12 an

271 We identified susceptibility loci on chromosomes 9 (Tnbs1) and 11 (Tnbs2).

272 interacts with the loci on chromosome 4 and chromosome 9 to affect tumor number.

273 of nondisjunction causing the translocation chromosome 9 to be differentially distributed to the two

274 is in a region of conserved synteny with pig chromosome 9, to which the porcine gene was subsequently

275 vestigated 13 families with IBMPFD linked to chromosome 9 using a candidate-gene approach.

276 ive trait locus was found (LOD = 5.0) on rat chromosome 9 using a large F2 population (N = 233) deriv

277 rait locus (QTL) was previously found on rat chromosome 9 using Dahl salt-sensitive (S) and Dahl salt

278 e estimate (24.2 Mb) of the disease locus on chromosome 9 using exon array capture identified indepen

279 We tested our predictions for chromosome 9 using seven genetically mapped, single-copy

280 29 affected sib pairs, the linkage signal on chromosome 9 vanished, while the chromosome 10 signal de

281 as a single case in which one entire copy of chromosome 9 was deleted.

282 Loss of chromosome 9 was detected in 81% of the tumors, most com

283 ed from the B chromosome to the short arm of chromosome 9 was recently found to be epigenetically sil

284 One marker on the telomeric end of chromosome 9 was suggestive of linkage.

285 On chromosome 9, we found a novel association in the prosta

286 g of the ky locus to a small region of mouse chromosome 9, we have now undertaken a positional clonin

287 n a novel autoreactivity-associated locus on chromosome 9, we identify a putative modifier, TLR9.

288 us localization of the taiep mutation to rat chromosome 9, we tested whether the mutation resided wit

289 Allelic losses of chromosome 9 were associated with expansion of the abnor

290 cohol preference quantitative trait locus on chromosome 9 were identified: Arhgef12, Carm1, Cryab, Co

291 This region is syntenic with human chromosome 9 where the gene encoding IFN-kappa and the t

292 k) x B10.BR) mice and reveal that a locus on chromosome 9, which coincides with Idd2, is linked to th

293 a GAA repeat in intron 1 of the FRDA gene on chromosome 9, which encodes a 210 amino acid protein cal

294 Chromosome 9, which is often partially or fully reduced

295 me 3 with a LOD score of 9.3 and a second on chromosome 9 with a LOD score of 14.0.

296 previously unannotated 693-bp transcript on chromosome 9 with a potential functional role in melanom

297 related to metabolic activities, a region of chromosome 9 with several growth factor proteins, and re

298 on of two single nucleotide polymorphisms on chromosome 9 with sporadic ALS, in line with findings fr

299 The MNAB gene is on human chromosome 9 with wide expression in normal tissues and

300 f repeated in the heterochromatic regions of chromosome 9, Y and 1.