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1 accompanied by a rapid loss of viability and chromosome breakage.
2 e competition among gametes with and without chromosome breakage.
3 omosome condensation, with or without actual chromosome breakage.
4  the liver is correlated with aneuploidy and chromosome breakage.
5 ks (SSBs) disrupt DNA replication and induce chromosome breakage.
6 s, only the latter trigger fork collapse and chromosome breakage.
7 log of 53BP1, rescued repeat instability and chromosome breakage.
8  in direct or reverse orientation can induce chromosome breakage.
9 CRs), indicating a possible role for LCRs in chromosome breakage.
10  replication forks collapse and give rise to chromosome breakage.
11 xpansion causes several genetic diseases and chromosome breakage.
12 lting in dicentric chromosomes), followed by chromosome breakage.
13  quantitative differences in their degree of chromosome breakage.
14 eads to genomewide fork stalling followed by chromosome breakage.
15 e 1 (SOD1), in a transgenic mouse, increases chromosome breakage.
16 ate in the nucleus and are unable to prevent chromosome breakage.
17 G and F) is essential for protection against chromosome breakage.
18  function in suppressing GCR formation after chromosome breakage.
19 esult in a G(2)/M arrest, mitotic arrest, or chromosome breakage.
20 y an important role in a common mechanism of chromosome breakage.
21 NA interstrand crosslink repair resulting in chromosome breakage(1-3).
22 ely to further investigate the mechanisms of chromosome breakage, a 40-Mb YAC contig covering the dis
23 d (RH) mapping is based on radiation-induced chromosome breakage and analysis of chromosome segment r
24 through S phase was required to trigger both chromosome breakage and arrest in the next cell cycle.
25 cells, WRN degradation caused G(2)/M arrest, chromosome breakage and ATM kinase activation.
26                                              Chromosome breakage and break-fusion-bridge (BFB) cycles
27 ns caused by the depletion of Mcm10 leads to chromosome breakage and cell cycle checkpoint activation
28 lived or dead-end product that can result in chromosome breakage and cell death.
29 morphology, but their cells had the expected chromosome breakage and DNA cross-linker sensitivity.
30 netic reorganization including site-specific chromosome breakage and DNA deletion.
31 lpha interaction is important for preventing chromosome breakage and elucidate a DNA repair mechanism
32 epeats, which are preferentially involved in chromosome breakage and exchange, rendering it an intere
33  and inter- and intragenomic rearrangements, chromosome breakage and fusion, rDNA changes, and loss o
34 ssive disorders characterized by spontaneous chromosome breakage and hematological cancers.
35 sition disorder characterized by spontaneous chromosome breakage and high cellular sensitivity to gen
36  chromothripsis-like pattern generated after chromosome breakage and illegitimate rejoining.
37 the Fancg(-/-) mice demonstrated spontaneous chromosome breakage and increased sensitivity to mitomyc
38          Diminished folate status results in chromosome breakage and is associated with several disea
39 demonstrating that FRA3B does play a role in chromosome breakage and loss in RCC.
40 ous mitotic recombinations, a translocation, chromosome breakage and loss, chromosomal fusion or telo
41 RCA1-BARD1 accumulate catastrophic levels of chromosome breakage and micronucleation, leading to cell
42 treatment with these agents, have pronounced chromosome breakage and other cytogenetic abnormalities.
43 duced DNA damage and ameliorated spontaneous chromosome breakage and radials in human FA patient-deri
44 eir dysfunction could precipitate widespread chromosome breakage and rearrangement in the course of m
45 11-dependent manner, followed by spontaneous chromosome breakage and segmental aneuploidies.
46 stigated the relationship between programmed chromosome breakage and telomere formation in Tetrahymen
47 ts suggest a strong mechanistic link between chromosome breakage and telomere formation.
48 uces centrosome overduplication, aneuploidy, chromosome breakage and the formation of micronuclei by
49       Stalled replication forks are sites of chromosome breakage and the formation of toxic recombina
50                                     In turn, chromosome breakage and translocations arise specificall
51 e modifications predispose genome regions to chromosome breakage and translocations.
52 fic histone modifications as facilitators of chromosome breakage and translocations.
53 ternative transposition reactions leading to chromosome breakage and various types of stable chromoso
54 of genome instability leading to aneuploidy, chromosome breakage, and chromosome rearrangements that
55 promote: transposition, insertion, excision, chromosome breakage, and ectopic recombination.
56                         Sites of spontaneous chromosome breakage are concordant with sites of DNA syn
57 gurations of Ac/Ds elements that can lead to chromosome breakage are not completely defined.
58  and/or 5-azadeoxycytidine treatment reduced chromosome breakage at CFSs.
59 licative stress treatment known to result in chromosome breakage at common fragile sites.
60 nt inactivation via a mec1 mutation leads to chromosome breakage at replication forks initiated from
61  source of genome instability resulting from chromosome breakage at selective sites.
62     Specifically, while MEC1 cells exhibited chromosome breakage at stress-response transcription fac
63 n factors, mec1 cells predominantly suffered chromosome breakage at transporter genes, many of which
64 mays) Ac/Ds transposable elements can induce chromosome breakage, but the precise configurations of A
65 is, they form anaphase bridges, resulting in chromosome breakage by an unknown mechanism.
66 t the FRA16D Flex1 sequence causes increased chromosome breakage by forming secondary structures that
67                  The region is more prone to chromosome breakage by gametocidal gene action than gene
68 rich DNA cruciforms, preventing catastrophic chromosome breakage by the ERCC1-ERCC4 complex.
69           Collectively, these data implicate chromosome breakage by TOP2 as an endogenous threat to g
70                                 The elevated chromosome breakage correlates histologically with a sig
71 in chromosome maintenance are linked to rare chromosome breakage disorders.
72 nce of chromatin bridges, thereby preventing chromosome breakage during cytokinesis.
73 retained in the macronucleus does not induce chromosome breakage during vegetative growth and that ex
74 nferred nine-chromosome ancestral state: (a) chromosome breakage followed by loss of centromere DNA a
75 ying the SCKL1 mutation would show increased chromosome breakage following replication stress.
76       The distributions of radiation-induced chromosome breakage for the GB4 and the G3 radiation hyb
77                                          The chromosome breakage-fusion-bridge (BFB) cycle is a mutat
78 274 (948h1) and D9S285 (767f2), suggesting a chromosome-breakage hotspot.
79 aics were created using gamma-rays to induce chromosome breakage in a cr4/Cr4+ heterozygote.
80                 Deletion of the RFB inhibits chromosome breakage in a sub-population of developing ma
81               These results demonstrate that chromosome breakage in evolution is nonrandom and that H
82 ent advances in chromatin changes induced by chromosome breakage in mammalian cells and their implica
83 tocidal (Gc) factors carried in 4S(L) induce chromosome breakage in meiospores not containing them, e
84 es and 4Ssh of Ae. sharonensis induce severe chromosome breakage in pollen lacking them.
85  hypomethylating agent 5-azacytidine induces chromosome breakage in root tips.
86 sical assays to be length-dependent sites of chromosome breakage in Saccharomyces cerevisiae.
87 is in cin8Delta kip3Delta cells, we detected chromosome breakage in the meiosis II cells.
88 tantly, these regions were not only prone to chromosome breakage in the presence of exogenous stress
89 radiation and show an increased frequency of chromosome breakage in the subsequent anaphase.
90 functionally distinct from ends generated by chromosome breakage, in that telomeres, unlike double-st
91                               We explore how chromosome breakage is integrated with meiotic progressi
92                              By contrast, no chromosome breakage is observed with alleles containing
93 s the construction of a library enriched for chromosome breakage junctions and the development of a s
94 genetic characterization of these functional chromosome breakage junctions is reported in the accompa
95 nd genetic characterization of 30 functional chromosome breakage junctions.
96  D-Titin cause chromosome undercondensation, chromosome breakage, loss of diploidy, and premature sis
97 mena suggest a novel mechanism that requires chromosome breakage next to short inverted repeats.
98                                   Programmed chromosome breakage occurs at 50-200 specific sites in t
99                        Extensive, programmed chromosome breakage occurs during formation of the somat
100                  The majority of spontaneous chromosome breakage occurs during the process of DNA rep
101                                   Programmed chromosome breakage occurs in many ciliated protozoa and
102 omain from BLM fails to correct the elevated chromosome breakage of transfected BLM-deficient cells.
103 nducing agent mitomycin C but do not exhibit chromosome breakage or cell cycle arrest after diepoxybu
104  cross-links (ICLs), mammalian cells display chromosome breakage or cell cycle delay with a 4N DNA co
105 ource of genome instability that may lead to chromosome breakage or nondisjunction during mitosis.
106 r complex (CPC) delays abscission to prevent chromosome breakage or tetraploidization.
107  delays completion of cytokinesis to prevent chromosome breakage or tetraploidization.
108       Here, we determined the structures and chromosome breakage properties of 15 maize p1 alleles: e
109 indicates that chromosome 11 is a target for chromosome breakage, rearrangement, and loss during the
110                     Suppression of dicentric chromosome breakage reflects loss of kinetochore functio
111 on is site specific, directed by a conserved chromosome breakage sequence (Cbs element).
112 e specific and directed by a conserved 15-bp chromosome breakage sequence (Cbs element).
113 ar chromosome 1 by site-specific cleavage at chromosome breakage sequence (Cbs) elements, rearranged
114 ve identified a 15 bp sequence, the Cbs (for chromosome breakage sequence), that is necessary and suf
115 ts of a 15-bp DNA sequence known as Cbs, for chromosome breakage sequence.
116                                              Chromosome breakage sequences (Cbs) that determine the s
117   Two distinct previously unreported variant chromosome breakage sequences were found, each in two or
118 f internal eliminated sequences, excision of chromosome breakage sequences, and endoreplication of th
119 ces, while fragmentation occurs at conserved chromosome breakage sequences, generating macronuclear c
120 cubation with CldU in these cells results in chromosome breakage, sister chromatid exchange, and cyto
121 ons in the Cbs on its ability to specify the chromosome breakage site and promote new telomere format
122 ide cloning and identification of functional chromosome breakage sites.
123 more, ultraviolet exposure results in marked chromosome breakage, suggesting that replication gaps cr
124                   Here, we have identified a chromosome breakage syndrome associated with severe lung
125 ations in NSMCE3 with an autosomal recessive chromosome breakage syndrome that leads to defective T a
126 or how the lung disease immunodeficiency and chromosome breakage syndrome-causing mutations could dis
127                                              Chromosome breakage syndromes are defined by chromosome
128 d how mutations in these regions may lead to chromosome breakage syndromes in humans.
129 th the emerging molecular information on the chromosome breakage syndromes.
130 g chromosome loss, micronuclei formation and chromosome breakage that are further elevated by replica
131 artificial chromomosme (YAC)-based assay for chromosome breakage to analyze fragility of CAG/CTG trac
132 sed sequence targeting and telomere-directed chromosome breakage to dissect the sequence requirements
133 formed a mosaic analysis using X-ray induced chromosome breakage to generate wild-type (gn1+/-) secto
134                                   Ds-induced chromosome breakage was used to generate Dek1 loss-of-fu
135                 However, whether SSBs induce chromosome breakage when present behind replication fork
136  into the mechanism of transposition-induced chromosome breakage, which is one outcome of the chromos
137 tosis with incomplete replication results in chromosome breakage, whole and segmental chromosome erro

 
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