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1 icrotubule nucleation, which is critical for chromosome segregation.
2 eling SUMOylated proteins to ensure faithful chromosome segregation.
3 ggers the termination of the SAC and enables chromosome segregation.
4 result in meiosis I arrest before homologous chromosome segregation.
5 for crossover formation and ensuring proper chromosome segregation.
6 cell division contributes to the fidelity of chromosome segregation.
7 moted by separase, is essential for faithful chromosome segregation.
8 e in fundamental cellular processes, such as chromosome segregation.
9 itional regulation of division completion by chromosome segregation.
10 t it occurs after cytokinetic events such as chromosome segregation.
11 e attachment errors and ensure high-fidelity chromosome segregation.
12 on of the centromere-the locus essential for chromosome segregation.
13 sist microtubule-mediated forces for mitotic chromosome segregation.
14 iotic spindle assembly can lead to erroneous chromosome segregation.
15 cluding instances of whole-genome equational chromosome segregation.
16 replication, DNA damage repair, and faithful chromosome segregation.
17 r chromatid cohesion, which is essential for chromosome segregation.
18 gesting shortened mitosis owing to premature chromosome segregation.
19 lar compartments as well as during bacterial chromosome segregation.
20 ese effects resulted in mitotic exit without chromosome segregation.
21 des SAC silencing, thereby ensuring accurate chromosome segregation.
22 inated activity being essential for accurate chromosome segregation.
23 function that interacts with CpsD and drives chromosome segregation.
24 feature in the establishment of reductional chromosome segregation.
25 naceous complex that is essential for proper chromosome segregation.
26 functional spindle architecture and correct chromosome segregation.
27 rminus in mediating CPC-binding and accurate chromosome segregation.
28 IF18A and KIF15 activity to promote accurate chromosome segregation.
29 enanes, posing topological challenges during chromosome segregation.
30 a conserved function to ensure high-fidelity chromosome segregation.
31 portant pathogen of humans, is essential for chromosome segregation.
32 e-associated Bub1 kinase, a key regulator of chromosome segregation.
33 eres provide a pivotal function for faithful chromosome segregation.
34 es SAC responsiveness for much more accurate chromosome segregation.
35 ividualize chromosomes, thereby facilitating chromosome segregation.
36 egulation of SPICE1 is important for correct chromosome segregation.
37 for accurate chromosome alignment and proper chromosome segregation.
38 ich provide physical links to guide accurate chromosome segregation.
39 to promote mitotic checkpoint signaling and chromosome segregation.
40 plication, transcription, recombination, and chromosome segregation.
41 ate recognition that is essential for proper chromosome segregation.
42 yed spindle assembly and increased errors in chromosome segregation.
43 the SET-Sgo1 binding is required for timely chromosome segregation.
44 nation/decatenation during recombination and chromosome segregation.
45 c5-mediated Cse4 phosphorylation in faithful chromosome segregation.
46 link centromeres to the mitotic spindle for chromosome segregation.
47 pendent of biorientation, and ensures proper chromosome segregation.
48 chromosomes to microtubules and facilitates chromosome segregation.
49 somes where it acts as a platform regulating chromosome segregation.
50 at different locations, cooperatively drive chromosome segregation.
51 e interface and along the spindle to control chromosome segregation.
52 lls coordinate their shape and polarity with chromosome segregation.
53 otubules, helping elucidate the mechanism of chromosome segregation.
54 les spore morphogenesis to the completion of chromosome segregation.
55 unresolved replication intermediates impair chromosome segregation.
56 esolve mitotic interlinks, thus facilitating chromosome segregation.
57 ect improper attachments and ensure faithful chromosome segregation.
58 in gene silencing, chromosome packaging, and chromosome segregation.
59 hout DNA re-replication, leading to aberrant chromosome segregation.
60 ultiple rounds of genome replication without chromosome segregation.
61 ading to defects in kinetochore assembly and chromosome segregation.
62 g the mechanisms underlying its functions in chromosome segregation.
63 ule during mitosis to ensure the fidelity of chromosome segregation.
64 consequences of R-loops at CEN chromatin and chromosome segregation.
65 s on centromere DNA is required for faithful chromosome segregation.
66 y improves the efficiency of cell growth and chromosome segregation.
67 ation, transcription, DNA damage repair, and chromosome segregation.
68 tivity to kinetochores that ensures accurate chromosome segregation.
69 cess known as spreading, to enable efficient chromosome segregation.
70 tin beta and disrupts MI spindle function in chromosome segregation.
71 meres into a structure that is competent for chromosome segregation.
72 to entering mitosis, which ensures accurate chromosome segregation.
73 nvolves cells exiting mitosis without proper chromosome segregation.
74 t are generated by kinetochores (KTs) during chromosome segregation.
75 a signaling network that guards fidelity of chromosome segregation.
76 induces centromere instability and abnormal chromosome segregation.
80 is, the genome is restructured to facilitate chromosome segregation, accompanied by dramatic changes
84 a nucleoid protection system ensuring proper chromosome segregation and cell division in coordination
88 ys diverse roles in division site selection, chromosome segregation and controlling peptidoglycan hom
90 ions impairs mitotic chromatin organization, chromosome segregation and cytokinesis, and induces an a
93 gous recombination, which facilitates proper chromosome segregation and enables the reciprocal exchan
94 ly checkpoint (SAC) is critical for accurate chromosome segregation and for cell division in a timely
96 iosis that are required for achieving proper chromosome segregation and highlights how these are both
98 intains correct CENP-C recruitment, faithful chromosome segregation and long-term cell viability.
99 will reveal new insights into mechanisms of chromosome segregation and may expedite the development
100 that the ParA-DivIVA interaction facilitates chromosome segregation and modulates cell elongation.
101 stalk formation in Caulobacter crescentus to chromosome segregation and motility in Myxococcus xanthu
102 e arms in prophase is important for accurate chromosome segregation and normal activation of gene exp
103 being linked to the fundamental processes of chromosome segregation and offspring diversification, me
104 different velocities, as well as asymmetric chromosome segregation and positioning throughout the ce
105 des a unique mechanism by which cells ensure chromosome segregation and preserve genome integrity.
106 bipolar spindles exhibit reduced fidelity of chromosome segregation and promote genetic instability.
107 te meiotic prophase I, resulting in accurate chromosome segregation and providing a mechanism to prev
110 orphology and a maternal-effect on embryonic chromosome segregation and survival, which was completel
111 at Arabidopsis APC/C is required for meiotic chromosome segregation and that APC/C-mediated regulatio
112 centriole duplication is critical for normal chromosome segregation and the maintenance of genomic st
113 ersus canonical H2A controls the fidelity of chromosome segregation and the rate of acquisition of to
115 ndle assembly is a prerequisite for faithful chromosome segregation and unperturbed cell-cycle progre
117 fact, reduced PP1 docking on Spc105 improved chromosome segregation and viability of mutant/stressed
118 sister chromatid cohesion to ensure accurate chromosome segregation, and also influences gene transcr
119 y perturbing centrosome function, preventing chromosome segregation, and attenuating the spindle asse
122 ccumulation at kinetochores, the fidelity of chromosome segregation, and genome stability in larval n
123 ellular processes, including gene silencing, chromosome segregation, and maintenance of genome stabil
125 s contribute to DNA repair, gene expression, chromosome segregation, and potentially other biological
126 misaligned chromosomes, errors of homologous chromosome segregation, and production of aneuploid oocy
127 cked form of DNA involved in gene silencing, chromosome segregation, and protection of genome stabili
128 nisms that ensure high-fidelity replication, chromosome segregation, and repair of germ cell genomes
129 tent with the processes of nuclear division, chromosome segregation, and transition from M to G1 phas
130 e that these key adaptations for reductional chromosome segregation are achieved through separable co
131 However, mutations in genes that control chromosome segregation are rare in human tumors as these
133 olar body and be fertilised, despite chaotic chromosome segregation at the first meiotic division.
135 g the critical differences in the control of chromosome segregation between mitosis and meiosis II.
136 on plane positioning is crucial for faithful chromosome segregation but also influences cell size, po
137 ses demonstrated that Eip1p is important for chromosome segregation but not essential, and modulated
139 ssover recombination is critical for meiotic chromosome segregation, but how mammalian crossing over
140 protection must be disabled to allow timely chromosome segregation, but how this is achieved is not
141 molecular motors required for high-fidelity chromosome segregation, but their specific contributions
142 omplexes at centromeres that ensure accurate chromosome segregation by attaching chromosomes to spind
143 ests that telomere dysfunctions also perturb chromosome segregation by contributing to the formation
144 lti-protein machine, the kinetochore, drives chromosome segregation by coupling the chromosomes to dy
145 assembly checkpoint (SAC) prevents premature chromosome segregation by inactivating the anaphase prom
146 an polo-like kinase PLK1 coordinates mitotic chromosome segregation by phosphorylating multiple chrom
148 s of genes that are not directly involved in chromosome segregation can lead to aneuploidy induction.
149 ficits in cohesin function that do not alter chromosome segregation cause significant birth defects.
150 on indicate that rhizobial genes involved in chromosome segregation, cell division, GABA metabolism,
152 spite seemingly divergent functions, such as chromosome segregation, chromosome maintenance, sister c
153 and meiosis, including functions related to chromosome segregation, cohesin removal, and kinetochore
154 r periphery, and that its depletion leads to chromosome segregation defects and increased levels of e
156 thermore, a dominant-negative Torsin induces chromosome segregation defects in a LAP1-dependent manne
157 cytotoxic analog (SM15) was shown to produce chromosome segregation defects in cancer cells by inhibi
158 nsitive and systematic quantitation of these chromosome segregation defects in cells undergoing mitos
159 cts at the metaphase plate leading to robust chromosome-segregation defects and nonmodal karyotypes.
166 As in all domains of life, with key roles in chromosome segregation, DNA repair and regulation of gen
167 ignment of bivalents at metaphase I, unequal chromosome segregation during anaphase II, and subsequen
168 omatin modification, RNAi components promote chromosome segregation during both mitosis and meiosis a
185 suggests that Aurora B kinase ensures proper chromosome segregation during mitosis not only by contro
186 on of the mitotic spindle to ensure faithful chromosome segregation during mitosis, cell polarization
187 heterochromatin formation in interphase and chromosome segregation during mitosis, demonstrating tha
198 moval from mitotic chromatin, accompanied by chromosome segregation errors and changes in post-mitoti
199 , at least in part, from DNA replication and chromosome segregation errors due to cell division durin
203 centriole overduplication and contributes to chromosome segregation errors in breast cancer cells.
204 GADD45 induced significantly higher rates of chromosome segregation errors in cultured cells and supp
205 ilitate understanding of the contribution of chromosome segregation errors to the development of aneu
206 ension may be critical in preventing mitotic chromosome segregation errors, however, the nature of fo
207 nderstand how the mitotic kinase PLK1 drives chromosome segregation errors, with a specific focus on
212 distribution bias was diminished in nonpolar chromosome segregation events observable in dyn1 mutants
213 romosomes do not form crossovers, leading to chromosome segregation failure in ANKRD31-deficient sper
215 the Ctf19 C-terminus whose deletion affected chromosome segregation fidelity in Sli15 wild-type cells
216 eres are key elements for the maintenance of chromosome segregation fidelity via a specialized chroma
218 degradation of Atg14 and observed homologous chromosome segregation followed by sister chromatid sepa
219 ificantly reduced CENP-A levels and perturbs chromosome segregation following the resumption of cell
221 ome-associated protein complex essential for chromosome segregation, gene expression, and repair of D
222 ve chromatin structure that is essential for chromosome segregation, genome stability and regulation
223 on in embryos of a single protein regulating chromosome segregation (GPR-1) provides a germline deriv
224 position as a major determinant of accurate chromosome segregation has not been previously directly
230 omplex and has been implicated in regulating chromosome segregation in both mitosis and meiosis in an
231 The role of the kinetochore during meiotic chromosome segregation in C. elegans oocytes has been a
232 tions of central-spindle microtubules during chromosome segregation in diverse spindles and suggest t
234 ement of central-spindle microtubules during chromosome segregation in human mitotic spindles and Cae
245 endent Aurora B pool that supported faithful chromosome segregation in otherwise unchallenged cells.
246 omes from the NE being required for faithful chromosome segregation in yeast and segregation of tethe
247 eltapkl1Delta spindle is fully competent for chromosome segregation independently of motor activity,
248 lpXP-mediated proteolysis before the time of chromosome segregation, indicating that SpbR turnover is
249 number calls, we reconstructed histories of chromosome segregation, inferring that 55 (74%) embryos
251 nd that APC/C-mediated regulation of meiotic chromosome segregation is a conserved mechanism among eu
258 ter chromatid cohesion essential for mitotic chromosome segregation is thought to involve the co-entr
260 ntromere, as an essential element to mediate chromosome segregation, is epigenetically determined by
262 Despite a conserved requirement in mediating chromosome segregation, kinetochores display remarkable
264 a cell-division-independent function for the chromosome-segregation machinery and define a microtubul
265 volved in key steps of cell division such as chromosome segregation, mitotic duration and cytokinesis
266 ium morphology, nuclear membrane morphology, chromosome segregation, mitotic spindle formation, and c
267 that support key cellular functions such as chromosome segregation, motor-based cargo transport, and
268 oses unique challenges because two rounds of chromosome segregation must be executed without interven
271 ific factors to drive the unique reductional chromosome segregation of meiosis I, which also results
275 that Wapl is required for accurate meiosis I chromosome segregation, predominantly releases Scc1-cohe
276 ties generate mutations, rearrangements, and chromosome segregation problems that cause many human di
277 ns with cse4-9SA exhibit increased errors in chromosome segregation, reduced levels of CEN-associated
287 mic, microtubule-based spindle that mediates chromosome segregation scales to a wide range of cell si
289 s centromeric cohesion protection but delays chromosome segregation, suggesting that the SET-Sgo1 bin
292 nto higher order structures is essential for chromosome segregation, the repair of DNA-damage, and th
293 a demonstrate that Cdc23 is required for the chromosome segregation through regulating the spindle as
295 cket in the human PLK1 PBD regulates mitotic chromosome segregation to preserve genome integrity.
296 ibution of different mechanisms of erroneous chromosome segregation to the production of aneuploid eg
297 eria, DNA replication is quickly followed by chromosome segregation, when one of the newly duplicated
299 absence of Yta7 causes defects in growth and chromosome segregation with mutations in components of t
300 contacts must be broken to allow for proper chromosome segregation; yet how this occurs remains ill-