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1 ferences in lumbar spinal cord following the chronic constriction injury.
2 to the second phase of the formalin test and chronic constriction injury.
6 g sciatic cryoneurolysis or 6 days following chronic constriction injury as compared with normal, uno
9 nd hyperalgesia, for several weeks in murine chronic constriction injury (CCI) and spared nerve injur
11 using two models of peripheral nerve injury: chronic constriction injury (CCI) and spinal nerve ligat
12 that mechanical hypersensitivity induced by chronic constriction injury (CCI) decreases over time in
13 We recently demonstrated that sciatic nerve chronic constriction injury (CCI) in male and female mic
14 uvant (CFA)-induced thermal hyperalgesia and chronic constriction injury (CCI) induced cold allodynia
15 LRP-alpha into mouse sciatic nerves prior to chronic constriction injury (CCI) inhibited p38 MAPK act
17 cord neuropeptides in the unilateral sciatic chronic constriction injury (CCI) model of Bennett and X
18 of lamina I spinoparabrachial neurons in the chronic constriction injury (CCI) model of neuropathic p
20 TNF in the evolution of hyperalgesia in the chronic constriction injury (CCI) model of neuropathic p
21 The present study utilizes the rat sciatic chronic constriction injury (CCI) model of neuropathic p
22 chemotherapeutic oxaliplatin, but not in the chronic constriction injury (CCI) model of the sciatic n
27 nged reaction of astrocytes were found after chronic constriction injury (CCI) of the rat infraorbita
29 As a model of pathologic pain, we used a chronic constriction injury (CCI) of the sciatic nerve i
30 e TSPO levels in the brain across time after chronic constriction injury (CCI) of the sciatic nerve i
32 h lumbar (L4) spinal nerve ligation (SNL) or chronic constriction injury (CCI) of the sciatic nerve p
36 test these hypotheses, adult rats underwent chronic constriction injury (CCI) of the sciatic nerve.
37 current densities of small DRG neurons after chronic constriction injury (CCI) of the sciatic nerve.
39 ty following intravenous administration in a chronic constriction injury (CCI) rat model of neuropath
41 ix weeks of voluntary wheel running prior to chronic constriction injury (CCI) reduced the terminal c
42 In order to investigate the consequences of chronic constriction injury (CCI) to nerve, we explored
44 ngoing and evoked pain behavior in rats with chronic constriction injury (CCI) to the infraorbital ne
45 er complete sciatic nerve transection (SNT), chronic constriction injury (CCI), or spared nerve injur
46 nd the late phase neuropathic pain following chronic constriction injury (CCI), were reduced in CKO m
47 g well-characterized mouse and rat models of chronic constriction injury (CCI)-induced neuropathic pa
48 methods to investigate the role of MrgprD in chronic constriction injury (CCI)-induced neuropathic pa
54 ACAP receptor antagonist PACAP(6-38) blocked chronic constriction injury-induced behavioral responses
55 I formalin model for persistent pain and the chronic-constriction-injury-induced model for neuropathi
56 c pain: neuropathic (spared nerve injury and chronic constriction injury), inflammatory (Freund's com
60 we have used a rodent partial sciatic nerve chronic constriction injury model (n = 5-8 per group per
61 inhibition, 3 reversed cold allodynia in the chronic constriction injury model of neuropathic pain in
65 in the trigeminal ganglion is reduced after chronic constriction injury of the infraorbital nerve.
67 aperitoneal) suppressed allodynia induced by chronic constriction injury of the sciatic nerve and sup
68 layer 5 (L5) of the ACC of adult mice after chronic constriction injury of the sciatic nerve of the
69 ed mouse model of neuropathic pain following chronic constriction injury of the sciatic nerve was rap
74 f peak behavioral hypersensitivity following chronic constriction injury to the infraorbital nerve (C
75 ial hypersensitivity, we used a rat model of chronic constriction injury to the infraorbital nerve (C
76 o different neuropathic pain models (EAE and chronic constriction injury) was dependent on the gonada
77 the Gel-Cur-M to sciatic nerves of mice with chronic constriction injuries, we found that the bioacti