ライフサイエンスコーパス: chronic renal failure

1 curs owing to hyperkalaemia in patients with chronic renal failure).

2 of adipocytes may be a therapeutic target in chronic renal failure.

3 contribute to the common, complex causes of chronic renal failure.

4 infections, and, in some patient subgroups, chronic renal failure.

5 n than hospitalizations for complications of chronic renal failure.

6 d to start dialysis earlier in patients with chronic renal failure.

7 SHPT is a frequent consequence of chronic renal failure.

8 g-associated TTP-HUS and can cause death and chronic renal failure.

9 R) injury is an important cause of acute and chronic renal failure.

10 atients died, and 7 survivors currently have chronic renal failure.

11 tic patients, and patients with diabetes and chronic renal failure.

12 kidney, is a potentially reversible cause of chronic renal failure.

13 led increasing frequencies of relapse and of chronic renal failure.

14 homocysteinemia in patients with predialysis chronic renal failure.

15 iovascular disease (CVD) among patients with chronic renal failure.

16 reatment of secondary hyperparathyroidism in chronic renal failure.

17 d man in Michigan with diabetes mellitus and chronic renal failure.

18 ange in response inhibits the development of chronic renal failure.

19 l sexual function in both men and women with chronic renal failure.

20 a common finding in both men and women with chronic renal failure.

21 n 5 different test days in six patients with chronic renal failure.

22 ism, and metabolic acidosis in patients with chronic renal failure.

23 , are present in the kidney during acute and chronic renal failure.

24 n, heart failure, myocardial infarction, and chronic renal failure.

25 al status and well-being among patients with chronic renal failure.

26 n animal model of hypertension that develops chronic renal failure.

27 es a case of periodontitis in a patient with chronic renal failure.

28 ronic heart failure, cardiac arrhythmias and chronic renal failure.

29 le of candidate genes in the pathogenesis of chronic renal failure.

30 dren under 6 months of age and children with chronic renal failure.

31 parathyroidism is a frequent complication of chronic renal failure.

32 s novel risk factors for ST in patients with chronic renal failure.

33 ent predictor of cardiovascular mortality in chronic renal failure.

34 ltiple cysts in the kidneys that can lead to chronic renal failure.

35 atous infiltration, splenic involvement, and chronic renal failure.

36 on, irreversible tissue and organ damage and chronic renal failure.

37 e heart and possibly the carotid arteries in chronic renal failure.

38 cute renal failure or acute deterioration of chronic renal failure.

39 hazard of diabetes mellitus and possibly of chronic renal failure.

40 ons with vascular disease, hypertension, and chronic renal failure.

41 tes, acute postprocedural renal failure, and chronic renal failure.

42 ing anaemia due to various causes, including chronic renal failure.

43 leading cause of mortality in patients with chronic renal failure.

44 tissue injury and accelerates the process of chronic renal failure.

45 ormone (GH) improves growth in children with chronic renal failure.

46 teinuria is a risk factor for progression of chronic renal failure.

47 reduces the renal dysfunction and injury of chronic renal failure.

48 dysfunction and injury in rats with induced chronic renal failure.

49 thy is an underrecognized cause of acute and chronic renal failure.

50 (more hypertension [90.4% versus 82.5%] and chronic renal failure [14.4% versus 7.6%]).

51 ative microbiology presented more frequently chronic renal failure (15 [18%] vs 11 [6%]; p=0.003), ch

52 .8%), coronary bypass graft surgery (39.2%), chronic renal failure (18.8%), and heart failure (17.3%)

53 pulation attributable fractions were 10% for chronic renal failure, 18% for congestive heart failure,

54 examined, 27 (77%) had acute or progressive chronic renal failure, 29 (83%) had involvement of other

55 cirrhotic patients (47.7%) and patients with chronic renal failure (38.6%) who complained of unsatisf

56 We hypothesized that in patients with chronic renal failure, a condition associated with both

57 se inhibitor, p21(WAF1/CIP1), do not develop chronic renal failure after ablation.

58 unctional and morphologic characteristics of chronic renal failure after partial renal ablation, incl

59 The five-year risk of chronic renal failure after transplantation of a nonrena

60 1.41; 95% CI: 1.23 to 1.62; p < 0.001), and chronic renal failure (aHR: 1.37; 95% CI: 1.23 to 1.53;

61 The occurrence of chronic renal failure among patients with a nonrenal tra

62 s insidious onset, invariable progression to chronic renal failure and a strong association with tran

63 acute kidney injury' have replaced the terms chronic renal failure and acute renal failure, respectiv

64 for the treatment of anemia associated with chronic renal failure and cancer.

65 dividual, a severe multisystem presentation (chronic renal failure and cardiomyopathy) in infancy.

66 Similarly, the prevalence of chronic renal failure and diabetes mellitus were signifi

67 The number of patients with chronic renal failure and end-stage renal disease contin

68 Chronic renal failure and end-stage renal disease have e

69 PI3KC2alpha-deficient mice developed chronic renal failure and exhibited a range of kidney le

70 ontrast-induced nephropathy in patients with chronic renal failure and GFR 30ml-60ml/min/1.73m(2) is

71 LED >/=3, predictors of increased death were chronic renal failure and heart failure (both P<0.05), w

72 is a common and debilitating complication of chronic renal failure and human immunodeficiency virus i

73 BCA MR imaging and NSF were acute and severe chronic renal failure and liver or renal transplantation

74 erum galactomannan levels in the presence of chronic renal failure and ongoing hemodialysis.

75 e include metastatic calcification caused by chronic renal failure and orthotopic liver transplantati

76 ialysis); only 4 of these (4%) progressed to chronic renal failure and permanent dialysis.

77 er and bone mineral density in children with chronic renal failure and Prader-Willi syndrome.

78 the rates were greatest among patients with chronic renal failure and smallest among patients with c

79 ism classically appears during the course of chronic renal failure and sometimes after renal transpla

80 Predictors of major bleeding were chronic renal failure and the use of drug-eluting stents

81 ts were compared with those of subjects with chronic renal failure and those of healthy controls.

82 ere identified who had received dialysis for chronic renal failure and who had sufficient data to cal

83 pertension, chronic cardiac diseases, and/or chronic renal failure), and had more severe PH at their

84 In this case, immunosuppression, chronic renal failure, and a history of diabetes mellitu

85 coinfection with hepatitis C and/or D virus, chronic renal failure, and children.

86 Male sex, chronic renal failure, and dementia were associated with

87 er disease, diabetes with end-organ disease, chronic renal failure, and dementia) diagnosed in 306 ho

88 es is complicated by oxalate accumulation in chronic renal failure, and heightened excretion in the e

89 arathyroidism develops in most patients with chronic renal failure, and is associated with the histol

90 7 deficiency as a pathophysiologic factor in chronic renal failure, and they demonstrate its efficacy

91 lure (aOR 1.26, 95%CI 1.19-1.35, p < 0.001), chronic renal failure (aOR 1.18, 95%CI 1.10-1.27, p < 0.

92 associated with lower rates of BVD, whereas chronic renal failure (aOR: 1.46; 95% CI: 1.03 to 2.08;

93 milar to patients without transplantation or chronic renal failure, approximately 70% at 1 year.

94 regimens appear to be safe and patients with chronic renal failure are able to activate normal compen

95 Patients with chronic renal failure are commonly started on renal repl

96 ein and energy requirements of patients with chronic renal failure are similar to those of healthy su

97 All CKD groups developed a similar degree of chronic renal failure as reflected by high serum creatin

98 tudied among patients who began dialysis for chronic renal failure at the New England Medical Center

99 Nineteen patients had chronic renal failure at the time of GBP, eight had tran

100 us anatomy was performed in 70 patients with chronic renal failure before surgical evaluation.

101 obstructive pulmonary disease, diabetes, and chronic renal failure) between October 1, 1995, and Sept

102 congestive heart failure, angina, diabetes, chronic renal failure, bipolar disorder, and major depre

103 ra of patients with diabetic nephropathy and chronic renal failure, both forms of polysulfides were d

104 ody mass is common in patients with acute or chronic renal failure but the mechanisms causing this lo

105 one patient did not become normal because of chronic renal failure, but she remains healthy.

106 D incidence is increased in individuals with chronic renal failure, but the underlying mechanisms rem

107 ere younger and less likely to have heart or chronic renal failure, but they were more likely to rece

108 Late evaluation of patients with chronic renal failure by a nephrologist is associated wi

109 Chronic renal failure can give rise to a wide spectrum o

110 tes mellitus, osteoporosis, non-AIDS cancer, chronic renal failure, cardio and cerebrovascular diseas

111 tes mellitus, osteoporosis, non-AIDS cancer, chronic renal failure, cardiovascular and cerebrovascula

112 events analyzed included diabetes mellitus, chronic renal failure, cardiovascular events, NLR-NAR ca

113 Growth in children with chronic renal failure caused by polyuric, salt-wasting d

114 Chronic renal failure causes acidosis; therefore, the ro

115 .93; P = 0.024) and decline in the hazard of chronic renal failure close to the threshold of signific

116 sified as high risk: age more than 80 years, chronic renal failure (creatinine > 2.0), compromised ca

117 With far-advanced chronic renal failure (creatinine clearance [CCr] < 15 t

118 Accelerated vascular disease is common in chronic renal failure (CRF) and accounts for significant

119 alnutrition and muscle wasting are common in chronic renal failure (CRF) and adversely affect morbidi

120 esource utilization and associated costs for chronic renal failure (CRF) and ESRD.

121 ere examined in skeletal muscle of rats with chronic renal failure (CRF) and sham operated (SO), pair

122 Rats and humans with chronic renal failure (CRF) are reported to have resista

123 xcretion of oxalate was studied in rats with chronic renal failure (CRF) by measuring the magnitude a

124 ines the incidence and treatment of ESRD and chronic renal failure (CRF) in OLTX patients.

125 isotope-dilution techniques in patients with chronic renal failure (CRF) in the basal state, during h

126 Chronic renal failure (CRF) is a major cause of morbidit

127 Chronic renal failure (CRF) is a major public health pro

128 Chronic renal failure (CRF) is associated with negative

129 Chronic renal failure (CRF) is associated with resistanc

130 binant erythropoietin (EPO) in patients with chronic renal failure (CRF) is frequently complicated by

131 even patients seropositive for anti-HCV with chronic renal failure (CRF) referred to UCLA Medical Cen

132 l and angiographic outcomes of patients with chronic renal failure (CRF) treated with intracoronary r

133 In chronic renal failure (CRF), plasma concentrations of th

134 ycation and relevant glycoxidative damage in chronic renal failure (CRF).

135 o the prevention of ectopic calcification in chronic renal failure (CRF).

136 axonal membrane properties in patients with chronic renal failure (CRF).

137 ion in the brain is elevated in animals with chronic renal failure (CRF).

138 ation transport is impaired in many cells in chronic renal failure (CRF).

139 in the genesis of hypertension in rats with chronic renal failure (CRF).

140 eparin hepatic lipase activity is reduced in chronic renal failure (CRF).

141 knowledge of the oral and dental aspects of chronic renal failure (CRF).

142 itional indices of patients with progressive chronic renal failure declines when they consume unrestr

143 rder to estimate the cumulative incidence of chronic renal failure (defined as a glomerular filtratio

144 were the presence of cardiovascular disease, chronic renal failure, dementia, solid organ malignancy,

145 During a median follow-up of 36 months, chronic renal failure developed in 11,426 patients (16.5

146 one in the mycophenolate mofetil group), and chronic renal failure developed in five (three in the cy

147 nts with HNF1B mutations develop progressive chronic renal failure, diabetes mellitus (40-50%), and l

148 orbid conditions that alter immune function (chronic renal failure, diabetes mellitus, HIV, alcohol a

149 Chronic renal failure directly linked to HIV infection i

150 zophrenia with a history of hypertension and chronic renal failure due to renal artery occlusion was

151 Increased UHP in chronic renal failure enhances the formation of pentosid

152 Patients with chronic renal failure exhibit plasma fatty acid patterns

153 Patients with chronic renal failure frequently develop cardiac hypertr

154 essive renal insufficiency and is a model of chronic renal failure from diverse causes.

155 ovel guanosine triphosphate-binding protein, chronic renal failure gene (CRFG), was discovered by dif

156 servational study included 112 patients with chronic renal failure (glomerular filtration rate (GFR)

157 ndrome, intrauterine growth restriction, and chronic renal failure has changed the practice of endocr

158 In chronic renal failure, higher circulating AGE levels res

159 ere age (hazard ratio [HR], 1.05; P<0.0001), chronic renal failure (HR, 2.41; P=0.004), inactivity (H

160 asive mechanical ventilation, heart failure, chronic renal failure, hypertension, high RASS or schedu

161 Glomerulosclerosis is a common cause of chronic renal failure in aging.

162 al recessive cystic kidney disease, leads to chronic renal failure in children.

163 They are a relevant cause of chronic renal failure in children.

164 (NPHP) is the most frequent genetic cause of chronic renal failure in children.

165 The factors that initiate chronic renal failure in patients with hypertension, dia

166 us, B6 mice model the increased frequency of chronic renal failure in postmenopausal women and provid

167 sease, is the most frequent genetic cause of chronic renal failure in the first 3 decades of life.

168 ations constitute the most frequent cause of chronic renal failure in the first two decades of life.

169 e variables except for a higher frequency of chronic renal failure in the MAB-T88 group (4.4% vs. 1.3

170 cation of diabetes and is a leading cause of chronic renal failure in the Western world.

171 0 patients (50%), alcoholism in three (15%), chronic renal failure in two (10%), and drug abuse in tw

172 Age, chronic renal failure, inactivity, and aortic valve velo

173 The frequency of chronic renal failure increases with age, especially in

174 Care for chronic renal failure involves management of complicatio

175 Nonregenerative anemia due to chronic renal failure is a common problem in domestic ca

176 Chronic renal failure is complicated by high cardiovascu

177 -Calve-Perthes disease, AVN in children with chronic renal failure is frequently asymptomatic and has

178 the severity of renal injury associated with chronic renal failure is not known.

179 In conclusion, chronic renal failure leads to alterations in cardiac ge

180 Chronic renal failure leads to amenorrhea, and successfu

181 for hepatitis C virus (HCV) in patients with chronic renal failure may be inadequate to detect infect

182 In a chronic renal failure model, Phd(2/3)hKO mice maintained

183 in HIV infection (n = 768), 25.3 to 30.6% in chronic renal failure (n = 270), 25.0% to 37.2% in rheum

184 is a frequent complication in children with chronic renal failure, occurring in approximately 7% of

185 ded in the final multivariable analysis were chronic renal failure (odds ratio (OR) = 2.6, 95% confid

186 ence interval [CI], 1.78-5.13; P<0.001), and chronic renal failure (odds ratio, 2.73; 95% CI, 1.60-4.

187 of development of renal fibrosis, which, in chronic renal failure of any origin, represents the path

188 Thirty morbidly obese patients with chronic renal failure or transplantation underwent gastr

189 Patients with chronic renal failure or weight <3 kg were excluded.

190 R = 9.648, 95% CI: 1.729-53.817, p = 0.010), chronic renal failure (OR = 15.683, 95% CI: 2.727-90.203

191 , those with flash pulmonary edema, advanced chronic renal failure, or ESRD (who have much to gain),

192 bid conditions that frequently accompany the chronic renal failure patient.

193 dicates that both nephrotic and nonnephrotic chronic renal failure patients can activate normal homeo

194 ds of the PLA2R, is more highly expressed in chronic renal failure patients than in controls.

195 with treating thyroid cancer with iodine in chronic renal failure patients who require hemodialysis

196 patients is a challenging task because most chronic renal failure patients with malnutrition are ano

197 Chronic renal failure, pelvic surgery, and lifestyle det

198 ease, chronic obstructive pulmonary disease, chronic renal failure, previous invasive coronary strate

199 igh LBP:BPI ratios observed in patients with chronic renal failure probably imparts an increased susc

200 cohol, steroid use, coronary artery disease, chronic renal failure, pulmonary disease)] and preoperat

201 In rats with chronic renal failure, quantitative cardiac morphology r

202 Anemic cats with chronic renal failure represent a potential in vivo appl

203 isease, atrial fibrillation), genitourinary (chronic renal failure), respiratory (respiratory failure

204 munocompromised patients with HIV infection, chronic renal failure, rheumatoid arthritis, solid-organ

205 cohort analysis to evaluate the incidence of chronic renal failure, risk factors for it, and the asso

206 At a median follow-up of 4.9 years, chronic renal failure, RRT, all fractures, hip fractures

207 ulting in variable hydroureteronephrosis and chronic renal failure secondary to obstructive uropathy.

208 Most patients with chronic renal failure show evidence of secondary hyperpa

209 The occurrence of chronic renal failure significantly increased the risk o

210 f AC-treated patients (n = 159) had previous chronic renal failure than AG-treated patients (n = 87)

211 rus infection, diabetes, obesity, burns, and chronic renal failure than white patients and had a smal

212 status in adults, infants, and children with chronic renal failure that have been tried with varying

213 ment, including the postnatal progression of chronic renal failure that results from the development

214 e the protein anabolic actions of insulin in chronic renal failure, the authors measured four sets of

215 In the treatment of anemia of chronic renal failure, the most common cause of recombin

216 d, placebo-controlled study of patients with chronic renal failure treated by peritoneal dialysis.

217 A total of 126 ambulatory patients with chronic renal failure treated with long-term hemodialysi

218 I) in asymptomatic, ambulatory patients with chronic renal failure treated with long-term hemodialysi

219 tal admissions in asymptomatic patients with chronic renal failure treated with long-term hemodialysi

220 ent papers describing outcome in patients in chronic renal failure undergoing cardiac surgery.

221 f subsequent cardiac events in patients with chronic renal failure undergoing dialysis.

222 ted in EDTA from 28 undialyzed patients with chronic renal failure (undialyzed CRF), 36 patients on c

223 However, in contrast to chronic renal failure, unsatisfactory sleep in cirrhosis

224 Chronic renal failure, use of anticoagulant, and lesion

225 between candidate genes in the RAA axis and chronic renal failure, using 85 African-American sibling

226 The five-year risk of chronic renal failure varied according to the type of or

227 analysis indicated that an increased risk of chronic renal failure was associated with increasing age

228 -up of 8 (range, 1-41) years in 77 patients, chronic renal failure was detected in 19 patients (25%):

229 rate for the composite end point of death or chronic renal failure was higher in the mycophenolate mo

230 (mean age 62 years, 67 men) with predialysis chronic renal failure were randomized to 5 mg folic acid

231 years; mean age, 6 years +/- 3.5 [SD]) with chronic renal failure were reviewed.

232 en and TGF-beta immunostaining, increased in chronic renal failure, were also reduced by GH, suggesti

233 thy are observed in vitamin D deficiency and chronic renal failure, where concentrations of the activ

234 protein-energy malnutrition in patients with chronic renal failure who are undergoing maintenance dia

235 scribed phenomenon in patients with advanced chronic renal failure who are undergoing maintenance hem

236 otein decreases in patients with progressive chronic renal failure who consume unrestricted diets sho

237 ic care facility except for one patient with chronic renal failure who died 2 mo after admission.

238 beyond this point, adults with all types of chronic renal failure who met these criteria on presenta

239 cardiomyopathy is a classic complication of chronic renal failure whose cause is unclear and treatme

240 Early referral of the patient with chronic renal failure will give the nephrologist time to

241 dney disease was diagnosed as suffering from chronic renal failure with end-stage renal disease.

242 Cirrhotic subjects and patients with chronic renal failure with unsatisfactory sleep showed h

243 ding cardiomyopathy, ischemic heart disease; chronic renal failure, with and without dialysis; hepati

244 2 diabetic (Z score, 0.71; P < 0.0002), and chronic renal failure (Z score, 0.65; P < 0.01) patients