ライフサイエンスコーパス: chronic renal insufficiency

1 tant implications in the clinical setting of chronic renal insufficiency.

2 processes and is a major cause of acute and chronic renal insufficiency.

3 the clinical and public health importance of chronic renal insufficiency.

4 ter contrast administration in patients with chronic renal insufficiency.

5 sed along with the changes that occur during chronic renal insufficiency.

6 ze in patients with renovascular disease and chronic renal insufficiency.

7 ts with obstructive renovascular disease and chronic renal insufficiency.

8 of the renal CaSR is altered in experimental chronic renal insufficiency.

9 etes mellitus (45.5% versus 30.8%; P<0.001), chronic renal insufficiency (19.2% versus 10.7%; P<0.001

10 hypertension (77% versus 65%; P=0.006), and chronic renal insufficiency (34% versus 19%; P<0.0001).

11 51%), diabetes (58%), tobacco use (52%), and chronic renal insufficiency (39%).

12 Chronic renal insufficiency (adjusted hazard ratio [HR]

13 h terms including "chronic kidney disease", "chronic renal insufficiency", "albuminuria", "proteinuri

14 cline in renal function of patients who have chronic renal insufficiency, although these agents do no

15 In patients with chronic renal insufficiency and global obstructive ather

16 dy mass index, 36 kg/m(2)), stage 3 vascular chronic renal insufficiency, and hebephrenic schizophren

17 sity (body mass index, 36), stage 3 vascular chronic renal insufficiency, and hebephrenic schizophren

18 ients with decompensated heart failure, mild chronic renal insufficiency, and renal function that had

19 endovascular repair (EVAR) in patients with chronic renal insufficiency because of the concern that

20 ma proteins among 1067 participants from the Chronic Renal Insufficiency Cohort (CRIC) and 536 indivi

21 oth time periods in 3249 participants in the Chronic Renal Insufficiency Cohort (CRIC) and 703 partic

22 nts with predialysis CKD using data from the Chronic Renal Insufficiency Cohort (CRIC) and CKD Japan

23 l analysis of participants enrolled into the Chronic Renal Insufficiency Cohort (CRIC) and Hispanic-C

24 participants undergoing hemodialysis in the Chronic Renal Insufficiency Cohort (CRIC) and Predictors

25 The Chronic Renal Insufficiency Cohort (CRIC) is a multicent

26 Disease and Hypertension (AASK, n =703), the Chronic Renal Insufficiency Cohort (CRIC) study ( n =319

27 ease in Children (CKiD) cohort II ( n =248), Chronic Renal Insufficiency Cohort (CRIC) study ( n =337

28 Data from 3,939 participants enrolled in the Chronic Renal Insufficiency Cohort (CRIC) Study between

29 ers in the United States who enrolled in the Chronic Renal Insufficiency Cohort (CRIC) Study between

30 The Chronic Renal Insufficiency Cohort (CRIC) study enrolled

31 AKI (ASSESS-AKI) study and the subset of the Chronic Renal Insufficiency Cohort (CRIC) study enrollee

32 We followed 1502 participants of the Chronic Renal Insufficiency Cohort (CRIC) Study for a me

33 tion and quality with CKD progression in 431 Chronic Renal Insufficiency Cohort (CRIC) Study particip

34 Chronic Renal Insufficiency Cohort (CRIC) Study particip

35 ysis, we studied 3483 people with CKD in the Chronic Renal Insufficiency Cohort (CRIC) Study recruite

36 s among 2696 participants in the prospective Chronic Renal Insufficiency Cohort (CRIC) study to ident

37 The Chronic Renal Insufficiency Cohort (CRIC) Study was esta

38 d HF among 3093 participants enrolled in the Chronic Renal Insufficiency Cohort (CRIC) Study who did

39 ine from participants with diabetes from the Chronic Renal Insufficiency Cohort (CRIC) study, the Sin

40 7) selected for metabolomic profiling in the Chronic Renal Insufficiency Cohort (CRIC) study, the sta

41 Using a subset of 1214 participants in the Chronic Renal Insufficiency Cohort (CRIC) Study, we dete

42 In the Chronic Renal Insufficiency Cohort (CRIC) study, we eval

43 Among adult participants in the Chronic Renal Insufficiency Cohort (CRIC) study, we iden

44 WAS of 537 blood metabolites measured in the Chronic Renal Insufficiency Cohort (CRIC) Study, with se

45 s) with mild to moderate CKD enrolled in the Chronic Renal Insufficiency Cohort (CRIC) Study.

46 elf-reported cardiovascular disease from the Chronic Renal Insufficiency Cohort (CRIC) study.

47 ive impairment and stroke in the prospective Chronic Renal Insufficiency Cohort (CRIC) Study.

48 severe CKD participating in the multicenter Chronic Renal Insufficiency Cohort (CRIC) Study.

49 valuated 3416 participants with CKD from the Chronic Renal Insufficiency Cohort (CRIC) study.

50 ns) was analysed in 2906 participants of the Chronic Renal Insufficiency Cohort (CRIC) with validatio

51 redictors of incident hospitalized HF in the Chronic Renal Insufficiency Cohort (CRIC), a multiethnic

52 ney disease (ARIC) and a population with CKD Chronic Renal Insufficiency Cohort (CRIC).

53 in Communities (ARIC) study with CKD and the Chronic Renal Insufficiency Cohort (CRIC).

54 rom the Cure Glomerulopathy Network and 3939 Chronic Renal Insufficiency Cohort adult participants.

55 n and African ancestry participants from the Chronic Renal Insufficiency Cohort and Atherosclerosis R

56 sease stages 2 through 4 who enrolled in the Chronic Renal Insufficiency Cohort between June 2003 and

57 n analysis in 500 subjects with DKD from the Chronic Renal Insufficiency Cohort for DKD phenotypes, i

58 The Chronic Renal Insufficiency Cohort internal validation s

59 tamin K status is low, were measured in 3066 Chronic Renal Insufficiency Cohort participants (median

60 eparation by CKD-EPI estimating equations in Chronic Renal Insufficiency Cohort participants, on the

61 Using 2003-2013 data from the Chronic Renal Insufficiency Cohort Study (3,597 particip

62 Using 2003-2013 data from the Chronic Renal Insufficiency Cohort Study (3,597 particip

63 of 4091 proteins and 630 metabolites in the Chronic Renal Insufficiency Cohort study (N=1708; averag

64 cations in the United States enrolled in the Chronic Renal Insufficiency Cohort Study and followed up

65 with mild to severe CKD who enrolled in the Chronic Renal Insufficiency Cohort Study between June 19

66 In our case-cohort study of 894 Chronic Renal Insufficiency Cohort Study participants wi

67 The prospective Chronic Renal Insufficiency Cohort study recruited adult

68 a subset of 942 participants with CKD in the Chronic Renal Insufficiency Cohort Study who had at leas

69 Among 5599 adult participants in the Chronic Renal Insufficiency Cohort study with serum crea

70 asma proteins among 3235 participants of the Chronic Renal Insufficiency Cohort Study with the primar

71 ts (1778 women and 2161 men) enrolled in the Chronic Renal Insufficiency Cohort Study, a large, diver

72 In the Chronic Renal Insufficiency Cohort study, genomic disord

73 In 3655 participants of the Chronic Renal Insufficiency Cohort study, we evaluated t

74 pective analysis of 3747 participants in the Chronic Renal Insufficiency Cohort Study, we investigate

75 In 1431 participants from the Chronic Renal Insufficiency Cohort study, we measured 25

76 y (CKD-EPI) equation race coefficient in the Chronic Renal Insufficiency Cohort study.

77 -4 enrolled in the prospective observational Chronic Renal Insufficiency Cohort study.

78 We studied 3,052 adults with CKD in the Chronic Renal Insufficiency Cohort Study.

79 association study among participants in the Chronic Renal Insufficiency Cohort Study.

80 ortality among 3939 patients with CKD in the Chronic Renal Insufficiency Cohort Study.

81 associations among 3487 participants of the Chronic Renal Insufficiency Cohort Study.

82 rum phosphate among 2879 participants in the Chronic Renal Insufficiency Cohort Study.

83 olymorphisms [SNPs]) of CAC within the CRIC (Chronic Renal Insufficiency Cohort) study (N = 1,509; 57

84 The prospective CRIC (Chronic Renal Insufficiency Cohort) study measured the e

85 th chronic kidney disease in the CRIC study (Chronic Renal Insufficiency Cohort) who were free of HF

86 prospective cohort study used data from the Chronic Renal Insufficiency Cohort, a multicenter cohort

87 rogression in four CKD cohorts (N=5654): the Chronic Renal Insufficiency Cohort, the African American

88 In a prospective case-cohort study of the Chronic Renal Insufficiency Cohort, we measured FGF23 at

89 ovascular risk in 2182 participants from the Chronic Renal Insufficiency Cohort.

90 investigated among 3680 participants in the Chronic Renal Insufficiency Cohort.

91 ere replicated in an independent cohort, the Chronic Renal Insufficiency Cohort.

92 s], CHS [Cardiovascular Health Study], CRIC [Chronic Renal Insufficiency Cohort]).

93 nt deployment was performed in patients with chronic renal insufficiency (creatinine >1.5 mg. dL(-1))

94 his is caused by the increased prevalence of chronic renal insufficiency (CRI) among blacks or by the

95 mance, and LV contractility in children with chronic renal insufficiency (CRI) and children undergoin

96 ease and total mortality among patients with chronic renal insufficiency (CRI) and end-stage renal di

97 ody mass are commonly found in patients with chronic renal insufficiency (CRI) and especially in dial

98 but less is known about the epidemiology of chronic renal insufficiency (CRI) and its relationship t

99 tion develop at the time of mild to moderate chronic renal insufficiency (CRI) and progress as renal

100 Comorbid conditions that develop during chronic renal insufficiency (CRI) contribute to the high

101 sive growth retardation is a complication of chronic renal insufficiency (CRI) in children and often

102 tension frequently complicates the course of chronic renal insufficiency (CRI) in children.

103 Chronic renal insufficiency (CRI) is a predictor of stro

104 Anemia associated with chronic renal insufficiency (CRI) may have substantial c

105 Patients with chronic renal insufficiency (CRI) or the nephrotic syndr

106 t there are few studies of iron status among chronic renal insufficiency (CRI) subjects not yet requi

107 owever, SHPT begins during earlier stages of chronic renal insufficiency (CRI), and little is known a

108 nd (2) to compare health of patients who had chronic renal insufficiency (CRI), were on dialysis, and

109 nty-four children were treated with polyuric chronic renal insufficiency (CRI; creatinine clearance <

110 n incident ESRD outpaced growth in prevalent chronic renal insufficiency, demonstrating that the ESRD

111 , cardiomegaly, peripheral vascular disease, chronic renal insufficiency, diabetes mellitus, low body

112 Prevalent chronic renal insufficiency (estimated glomerular filtra

113 , 2.2 [CI, 1.5 to 3.3]; P < 0.001), and mild chronic renal insufficiency (hazard ratio, 2.4 [CI, 1.7

114 Treating the acidosis of chronic renal insufficiency improves both bone and muscl

115 For every 1000 adults with chronic renal insufficiency in 1978, 9 new cases of ESRD

116 eveloped in 1983, but every 1000 adults with chronic renal insufficiency in 1991 produced 16 new case

117 the number of adults age 20 to 74 years with chronic renal insufficiency increased from 2.6 to 3.9 mi

118 Chronic renal insufficiency inexorably progresses in pat

119 Chronic renal insufficiency is associated with elevated

120 Chronic renal insufficiency leads to muscle wasting, whi

121 ted with CAD score were age, hyperlipidemia, chronic renal insufficiency, left ventricular function,

122 11 million people in the United States have chronic renal insufficiency, little is known about ethni

123 uggest that reduced renal CaSR expression in chronic renal insufficiency may play a role in disordere

124 The remaining 17 patients all have developed chronic renal insufficiency (mean serum creatinine, 2.4

125 Chronic renal insufficiency, once established, tends to

126 Among those with chronic renal insufficiency or end-stage renal disease,

127 In 34 patients with chronic renal insufficiency or failure, 43 small-bore ce

128 who smoked less than 5 cigarettes/day, with chronic renal insufficiency or prior cancer, were exclud

129 ge, sex, baseline blood pressure, history of chronic renal insufficiency, presence of diabetes mellit

130 In chronic renal insufficiency, the 2 degrees HPT may resul

131 Four weeks after chronic renal insufficiency was induced by 5/6 nephrecto

132 ntified in plasma samples from patients with chronic renal insufficiency were also found in the plasm

133 Patients with chronic renal insufficiency were further stratified into

134 tion in patients with glomerular disease and chronic renal insufficiency, which might inhibit or pote