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1 allergic rhinitis, nonallergic rhinitis, and chronic rhinosinusitis.
2 participants who underwent ESS for acute or chronic rhinosinusitis.
3 in nasal epithelial cells from patients with chronic rhinosinusitis.
4 ) and TGFbeta in pulmonary fibrosis (PF) and chronic rhinosinusitis.
5 , asthma, chronic spontaneous urticaria, and chronic rhinosinusitis.
6 stic sinuses may complicate and develop into chronic rhinosinusitis.
7 he understanding of the cognitive effects of chronic rhinosinusitis.
8 rrected for sex and the Lund-Mackay score of chronic rhinosinusitis.
9 nce to support this practice, especially for chronic rhinosinusitis.
10 review of the current literature relevant to chronic rhinosinusitis.
11 airway diseases, such as cystic fibrosis and chronic rhinosinusitis.
12 ave recently been associated with asthma and chronic rhinosinusitis.
13 llergy, in particular those of patients with chronic rhinosinusitis.
17 omorbidities of asthma, such as eosinophilic chronic rhinosinusitis, allergic rhinitis, and atopic de
19 of severe asthma complicated by eosinophilic chronic rhinosinusitis and eosinophilic otitis media, re
20 y disease (N-ERD) asthma is characterized by chronic rhinosinusitis and intolerance of aspirin and ot
21 cated in nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role
22 cated in nasal polyps (NPs) of patients with chronic rhinosinusitis and might play a significant role
24 ver, the exact role of microbial biofilms in chronic rhinosinusitis and orbital cellulitis were not e
25 roducing ability of the clinical isolates in chronic rhinosinusitis and orbital cellulitis, and to lo
28 osinophilic oesophagitis, allergic rhinitis, chronic rhinosinusitis, and asthma, are complex and infl
32 atory tract diseases including otitis media, chronic rhinosinusitis, and exacerbations of both cystic
33 ne-mediated disease characterized by asthma, chronic rhinosinusitis, and hypersensitivity to cyclooxy
34 mucosal tissue homogenates in patients with chronic rhinosinusitis, and this effect was most promine
35 These features mimic essential aspects of chronic rhinosinusitis-associated olfactory loss, and il
36 These observations establish a mechanism of chronic rhinosinusitis-associated olfactory loss, caused
38 widespread prevalence of allergic, viral and chronic rhinosinusitis, but how the brain encodes and ma
39 mucin can mediate corticosteroid efficacy in chronic rhinosinusitis, but the role of MUC1 in uncontro
40 more willing to travel longer distances for chronic rhinosinusitis care, results suggest that distan
41 urgical specimens derived from patients with chronic rhinosinusitis compared to control patients.
50 chanisms and immune pathways associated with chronic rhinosinusitis (CRS) are not fully understood.
52 of antibiotic therapy is often initiated for chronic rhinosinusitis (CRS) based on symptomatology.
56 lines provide composite criteria to evaluate chronic rhinosinusitis (CRS) control, taking into consid
64 however, the association between smoking and chronic rhinosinusitis (CRS) has not been well character
65 LP protein and its function in patients with chronic rhinosinusitis (CRS) have not been fully explore
66 revalence of asthma and its association with chronic rhinosinusitis (CRS) have not been widely studie
68 s (EPOS) criteria to study the prevalence of chronic rhinosinusitis (CRS) in a general-population sam
69 ds (INCs) remain the first-line treatment of chronic rhinosinusitis (CRS) in both adults and children
70 host-microbial interactions in patients with chronic rhinosinusitis (CRS) in hopes of elucidating mec
102 Our understanding of the pathophysiology of chronic rhinosinusitis (CRS) is continuously evolving.
103 o the paranasal sinuses for diseases such as chronic rhinosinusitis (CRS) is particularly challenging
110 Studies of the underlying cause or causes of chronic rhinosinusitis (CRS) over the past 20 or more ye
112 Recently, ILC2s enrichment was noted in chronic rhinosinusitis (CRS) patients; however, the role
115 ecretions, were collected from patients with chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP)
119 y Diseases (EUFOREA) treatment algorithm for chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP),
120 tissue was obtained from subjects with AERD, chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP),
121 g hypersensitivity to NSAIDs, asthma, and/or chronic rhinosinusitis (CRS) with nasal polyps (CRSwNP).
123 patients with or without allergic rhinitis, chronic rhinosinusitis (CRS) with or without nasal polyp
124 is study aimed to explore the association of chronic rhinosinusitis (CRS) with prior chronic periodon
125 following endoscopic sinus surgery (ESS) for chronic rhinosinusitis (CRS) without nasal polyposis, th
128 luding eosinophilia, which is in contrast to chronic rhinosinusitis (CRS) without nasal polyps (NPs).
129 en postbronchodilator lung function, asthma, chronic rhinosinusitis (CRS), and atopy with age using a
130 in many mucosal diseases, including asthma, chronic rhinosinusitis (CRS), and eosinophilic esophagit
131 ophilia is a prominent feature of asthma and chronic rhinosinusitis (CRS), and the endothelium plays
132 F-related upper airway disease, specifically chronic rhinosinusitis (CRS), are not characterized.
133 eased in nasal polyps (NPs) of patients with chronic rhinosinusitis (CRS), as well as in bronchoalveo
134 ent of a range of nasal conditions including chronic rhinosinusitis (CRS), caused by an infection and
135 rarily classified as diffuse type 2 dominant chronic rhinosinusitis (CRS), is characterized by eosino
136 Despite the high prevalence and morbidity of chronic rhinosinusitis (CRS), little is known about the
137 nnaire items, we identified respondents with chronic rhinosinusitis (CRS), migraine headache, and fat
138 t-reported outcomes during the management of chronic rhinosinusitis (CRS), PROMs will play an essenti
151 oke (CS) plays a role in the exacerbation of chronic rhinosinusitis (CRS); however, the mechanism for
155 dermatitis, food allergy, allergic rhinitis, chronic rhinosinusitis, eosinophilic esophagitis, and as
156 lops in affected organs in asthma, rhinitis, chronic rhinosinusitis, eosinophilic esophagitis, food a
157 G, AND PARTICIPANTS: This subanalysis of the Chronic Rhinosinusitis Epidemiology Study (CRES), a pros
158 s are involved in asthma, allergic rhinitis, chronic rhinosinusitis, food allergy, and eosinophilic e
159 lated comorbidities are discussed: rhinitis, chronic rhinosinusitis, gastroesophageal reflux, obstruc
162 e relationship between allergic rhinitis and chronic rhinosinusitis has been assessed in a number of
163 ic sinus surgery and antibiotics in managing chronic rhinosinusitis has contributed to a five-times v
165 of asthma (HR 0.678, 95% CI 0.636-0.722) and chronic rhinosinusitis (HR 0.799, 95% CI 0.752-0.850).
166 rhinitis (HR 1.272, 95% CI 1.214-1.333), and chronic rhinosinusitis (HR 1.744, 95% CI 1.671-1.821).
167 spite considerable research, the etiology of chronic rhinosinusitis, including the pathogenic role of
168 lly transmitted infections, cystic fibrosis, chronic rhinosinusitis, inflammatory bowel disease, and
174 es of use included acute inflammation (n=6), chronic rhinosinusitis (n=2), and allergic rhinitis (n=2
175 ory secretions in pathologic states, such as chronic rhinosinusitis or hyperglycemia, promotes tonic
176 underlying disease (asthma, nasal polyps or chronic rhinosinusitis, or both), as well as on the meth
177 substances in randomized clinical trials of chronic rhinosinusitis outcomes suggests that the use of
178 d on the 22-Item Sinonasal Outcomes Test and Chronic Rhinosinusitis Patient-Reported Outcomes Measure
181 n the identification and characterization of chronic rhinosinusitis phenotypes, endotypes, and biomar
182 dings suggest that improved understanding of chronic rhinosinusitis phenotypic and endotypic heteroge
184 has clinical effectiveness in patients with chronic rhinosinusitis, providing significantly improved
186 tures have traditionally guided treatment in chronic rhinosinusitis, recent research has favored cate
189 ce of symptoms of asthma, allergic rhinitis, chronic rhinosinusitis, smoking status, and history of N
190 ns to NSAIDs was higher in participants with chronic rhinosinusitis symptoms (Odds Ratio 2.12; 95%CI
192 polyp specimens from patients with AERD and chronic rhinosinusitis were analyzed by using quantitati
193 rty (240) patients with clinical features of chronic rhinosinusitis were examined; patients with firs
194 D PARTICIPANTS: A total of 505 patients with chronic rhinosinusitis were prospectively enrolled in a
196 rationales for and against the treatment of chronic rhinosinusitis with antibiotics, based on curren
197 s (AFRS) patients with samples from non-AFRS chronic rhinosinusitis with nasal polyp (CRSwNP) patient
205 inosinusitis (AFRS) is a severe phenotype of chronic rhinosinusitis with nasal polyposis (CRSwNP), ch
207 re asthma, but also in such comorbidities as chronic rhinosinusitis with nasal polyposis (CRSwNP).
208 desensitization (ATAD) for the management of chronic rhinosinusitis with nasal polyposis (CRSwNP).
209 ry disease (AERD) is a condition composed of chronic rhinosinusitis with nasal polyposis and asthma t
210 ab has been observed in patients with severe chronic rhinosinusitis with nasal polyposis and chronic
211 exacerbations, history of late-onset asthma, chronic rhinosinusitis with nasal polyposis, low oral co
213 iomarkers in atopic dermatitis (AD), asthma, chronic rhinosinusitis with nasal polyps (CRSwNP) and eo
215 d with uncinate tissue (UT) of patients with chronic rhinosinusitis with nasal polyps (CRSwNP) and th
216 ma with an eosinophilic phenotype (SAEP) and chronic rhinosinusitis with nasal polyps (CRSwNP) are pr
223 p recurrence (PR) can occur in patients with chronic rhinosinusitis with nasal polyps (CRSwNP) follow
230 composition of patients with diffuse type 2 chronic rhinosinusitis with nasal polyps (CRSwNP) is alt
241 n of PD-1 and its ligands PD-L1 and PD-L2 in chronic rhinosinusitis with nasal polyps (CRSwNP) is poo
244 osinusitis without nasal polyps (CRSsNP) and chronic rhinosinusitis with nasal polyps (CRSwNP) using
245 l stem/progenitor cells (BSCs) obtained from chronic rhinosinusitis with nasal polyps (CRSwNP) when d
246 nts across multiple outcomes of treatment of chronic rhinosinusitis with nasal polyps (CRSwNP) with d
247 y disease (AERD) is characterized by asthma, chronic rhinosinusitis with nasal polyps (CRSwNP), and a
248 hinosinusitis without nasal polyps (CRSsNP), chronic rhinosinusitis with nasal polyps (CRSwNP), and a
249 matory airway diseases, including asthma and chronic rhinosinusitis with nasal polyps (CRSwNP), and i
250 ease (AERD) is characterized by the triad of chronic rhinosinusitis with nasal polyps (CRSwNP), asthm
251 oscopy is not only used for the diagnosis of chronic rhinosinusitis with nasal polyps (CRSwNP), but a
252 is evidence of pathophysiologic diversity in chronic rhinosinusitis with nasal polyps (CRSwNP), but d
261 ssed in the setting of allergic rhinitis and chronic rhinosinusitis with nasal polyps (CRSwNP); howev
262 turbinate mucosa obtained from patients with chronic rhinosinusitis with nasal polyps (n = 7) and pat
263 (181 [35%] female and 333 [65%] male), with chronic rhinosinusitis with nasal polyps (n=410) or chro
265 ve in treating patients with type-2 dominant chronic rhinosinusitis with nasal polyps (T2-CRSwNP).
266 ronic prurigo, chronic urticaria, asthma and chronic rhinosinusitis with nasal polyps - with a high n
267 hronic airway inflammatory diseases, such as chronic rhinosinusitis with nasal polyps and asthma, sho
268 aureus in the initiation and persistence of chronic rhinosinusitis with nasal polyps and asthma.
270 piratory disease compared with patients with chronic rhinosinusitis with nasal polyps and/or asthma a
271 to occur in as many as 15% of patients with chronic rhinosinusitis with nasal polyps and/or asthma.
272 Following the trend in asthma, endotypes for chronic rhinosinusitis with nasal polyps have been estab
273 ated respiratory disease is a severe form of chronic rhinosinusitis with nasal polyps in which nearly
277 d safety in adults with severe, uncontrolled chronic rhinosinusitis with nasal polyps is unknown.
278 evated C3/C3a in patients with asthma and/or chronic rhinosinusitis with nasal polyps may account for
279 n atopic diseases such as atopic dermatitis, chronic rhinosinusitis with nasal polyps, and asthma.
280 wn to include chronic spontaneous urticaria, chronic rhinosinusitis with nasal polyps, and food aller
281 D) is characterized by the clinical triad of chronic rhinosinusitis with nasal polyps, asthma, and a
282 D) is characterized by the clinical triad of chronic rhinosinusitis with nasal polyps, asthma, and an
283 severe, uncontrolled asthma and a history of chronic rhinosinusitis with nasal polyps, but its effica
284 ic spontaneous urticaria, atopic dermatitis, chronic rhinosinusitis with nasal polyps, food allergies
285 the treatment of asthma, atopic dermatitis, chronic rhinosinusitis with nasal polyps, or chronic spo
293 ongside intranasal medication in adults with chronic rhinosinusitis with or without nasal polyps.
294 f asthma (n = 71,481), CRSwNP (n = 9626) and chronic rhinosinusitis without nasal polyposis (CRSsNP,
295 a range of premorbid medical conditions for chronic rhinosinusitis without nasal polyps (CRSsNP) and
296 of IL-19, at lower extent, in patients with chronic rhinosinusitis without nasal polyps (CRSsNP) in
297 luids (NLFs) from controls and patients with chronic rhinosinusitis without nasal polyps (CRSsNP), ch
298 is with nasal polyps (CRSwNP) and those with chronic rhinosinusitis without nasal polyps (CRSsNP; P <
299 rhinosinusitis with nasal polyps (n=410) or chronic rhinosinusitis without nasal polyps (n=104), wer
300 surgery from control subjects, patients with chronic rhinosinusitis without nasal polyps, and patient