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1 of rhizobacteria on the dissolution rate of chrysotile.
2 their lengths and were prepared from natural chrysotile.
3 nt and pulmonary fibrosis in mice exposed to chrysotile.
4 ontributing to ER stress in cells exposed to chrysotile.
9 nitrite formation, was evident in lungs from chrysotile- and crocidolite-exposed rats at 1 and 6 wk.
10 he UPR was present in macrophages exposed to chrysotile asbestos and if ER stress in macrophages was
13 nique for fluorescent tagging and imaging of chrysotile asbestos fibers and prepared samples with a d
15 at lung fibroblasts (RLFs) after exposure to chrysotile asbestos fibers in vitro, which results in as
17 ties and differences between crocidolite and chrysotile asbestos in terms of their transcriptional ef
18 whether the present findings are specific to chrysotile asbestos or would be observed after inhalatio
25 f L. emarginata had no significant effect on chrysotile dissolution or plant accumulation of Ni in th
28 increased significantly within minutes after chrysotile exposure and remained elevated for a prolonge
30 riments also confirmed that highly elongated chrysotile fibers exhibit anisotropic diffusion at short
31 did not show any increase in sensitivity to chrysotile fibers in short-term (4-h) treatment when com
34 This unique dolomite forming mechanism in chrysotile in subduction slabs may facilitate the transp
36 ced gene alterations were sustained, whereas chrysotile-induced gene alterations returned to backgrou
40 xposed to asbestos (crocidolite, amosite, or chrysotile) or control particles at moderate doses (1-10
44 on of carbonate-bearing serpentine-polymorph chrysotile, with in situ electrical conductivity measure