ライフサイエンスコーパス: cigarette smoker

1 R, n = 9) or poor (PR, n = 7; including five cigarette smokers).

2 luated in two prospective cohorts of Chinese cigarette smokers.

3 at high concentrations in the bloodstream of cigarette smokers.

4 on to the increased atherothrombotic risk of cigarette smokers.

5 r genotype increases risk particularly among cigarette smokers.

6 and thereby investigate vascular disease in cigarette smokers.

7 ene influence the pathogenesis of COPD among cigarette smokers.

8 function independent of changes in lipids in cigarette smokers.

9 users and was appreciably less than that of cigarette smokers.

10 Participants were 220 cigarette smokers.

11 n are efficacious in producing abstinence in cigarette smokers.

12 s and in 17% of bronchial brushes from heavy cigarette smokers.

13 bolytic therapy for myocardial infarction in cigarette smokers.

14 dults with invasive pneumococcal disease are cigarette smokers.

15 The participants were 199 cigarette smokers.

16 tients had pulmonary contusion, and 59% were cigarette smokers.

17 effects of intravenous nicotine in 16 active cigarette smokers.

18 mokers, while 88.2% of the HIV subjects were cigarette smokers.

19 xcretion of both isoprostanes is elevated in cigarette smokers.

20 PD probands were found only in current or ex-cigarette smokers.

21 enty-seven percent came from households with cigarette smokers.

22 has been found in type-II alcoholism and in cigarette smokers.

23 that MAO A would be reduced in the brain of cigarette smokers.

24 sferrin differed in upper and lower lobes of cigarette smokers.

25 aspirin and antioxidant vitamins in chronic cigarette smokers.

26 d the mechanisms leading to heart failure in cigarette smokers.

27 Caries was most common among cigarette smokers.

28 ) among Italian non-mentholated 7 mg ISO tar cigarette smokers.

29 as high and sometimes higher than exclusive cigarette smokers.

30 and sometimes higher than those of exclusive cigarette smokers.

31 ignificantly worse drug-related problem than cigarette smokers.

32 nicotine salt were stronger in never vs ever cigarette smokers.

33 learning algorithm on sequential CT scans in cigarette smokers.

34 f not more cigarettes per day than exclusive cigarette smokers.

35 lls present in the bronchoalveolar lavage of cigarette smokers.

36 owing evaluation of associations among never cigarette smokers.

37 g never cigarette smokers but not among ever cigarette smokers.

38 pregulate beta2* nAChRs more than nonmenthol cigarette smokers.

39 similar in menthol compared with nonmenthol cigarette smokers.

40 hewing tobacco separately for never and ever cigarette smokers.

41 s) cigarette smokers, or were high-intensity cigarette smokers.

42 g to susceptibility to infectious disease in cigarette smokers.

43 els of testosterone within the same range as cigarette smokers.

44 d cutpoint recommended for identifying adult cigarette smokers.

45 investigated dopamine synthesis capacity in cigarette smokers.

46 nd pipe smoking were not elevated among ever cigarette smokers.

47 to contribute to the nicotine dependence of cigarette smokers.

48 on profiles found in alveolar macrophages of cigarette smokers.

49 lso shown that these reflexes deteriorate in cigarette smokers.

50 rodigestive reflexes on airway protection in cigarette smokers.

51 emity PAD identified from medical records as cigarette smokers.

52 ere periodontitis (25.7%) compared to former cigarette smokers (20.2%), and non-smokers (13.1%).

53 42.4% (95% CI, 39.7% to 45.1%) were current cigarette smokers, 20.3% (CI, 18.6% to 22.1%) were forme

54 At admission, 75% were current and 8% former cigarette smokers, 3% were current cigar or pipe smokers

55 Fourteen cigarette smokers (31+/-4 pack years) and 14 age- and se

56 e median IPM3 level was higher for exclusive cigarette smokers (39.8 mug/g creatinine) than for non-u

57 Among the 27 619 current cigarette smokers, 4224 died during follow-up, with 1130

58 ers and 111 patients (7.5%) who were current cigarette smokers; 67 patients (13.7%) smoked pipes and/

59 to be men (94% versus 67%, P<0.001), current cigarette smokers (68% versus 32%, P<0.001), and obese (

60 be male (52.4% vs. 36.2%; P = 0.0003), to be cigarette smokers (68.5% vs. 55.3%; P = 0.003), and to h

61 Tobacco cigarette smokers (75%), who were not abstaining from ci

62 ely to be male (87% vs 67%, P=0.01), current cigarette smokers (84% vs 32%, P<0.001), younger (44+/-8

63 ge, 44 years), predominantly male (80%), and cigarette smokers (95%).

64 myeloid dendritic cells (mDCs) derived from cigarette smokers activate autoreactive Th1 and Th17 cel

65 ing of an AIDS diagnosis was being a current cigarette smoker (adjusted odds ratio = 2.57, 95% confid

66 Cigarette smokers admitted to a study hospital who recei

67 Among cigarette smokers, African Americans and Native Hawaiian

68 ietary intake of n-3 fatty acids may protect cigarette smokers against COPD.

69 Heavy cigarette smokers also had an increased risk of primary

70 nhibition, to the degree observed in chronic cigarette smokers, also increases low-dose nicotine self

71 rtality for menthol compared with nonmenthol cigarette smokers among 65 600 participants in the South

72 with venous occlusion plethysmography in 12 cigarette smokers and 12 age- and gender-matched nonsmok

73 adult participants (11 moderately dependent cigarette smokers and 13 nonsmokers).

74 Twenty normocholesterolemic cigarette smokers and 20 matched healthy control subject

75 ment and root preparation in 53 patients (15 cigarette smokers and 38 non-smokers).

76 ers have greater pulmonary inflammation than cigarette smokers and controls, with a positive associat

77 of this compound are elevated in urine from cigarette smokers and in humans with hypercholesterolemi

78 us coeruleus (LC) were measured in long-term cigarette smokers and in nonsmokers.

79 Compared to cigarette smokers and ketamine users, individuals with p

80 s and CBL around adjacent implants placed in cigarette smokers and never smokers.

81 le around adjacent dental implants placed in cigarette smokers and never smokers.

82 These results indicate that cigarette smokers and nonsmokers exposed to cigarette sm

83 tor (nAChR) agonists augment cognition among cigarette smokers and nonsmokers, yet the systems-level

84 um endotoxin concentrations are increased in cigarette smokers and that endotoxin concentrations are

85 ticipants aged 16 to 26 years who were never cigarette smokers and were attitudinally nonsusceptible

86 aving ever smoked, being a former or current cigarette smoker, and years since quit smoking with recu

87 3 age- and sex-matched groups: 5 EC users, 5 cigarette smokers, and 5 controls who had never smoked o

88 or gum disease, 18.9% reported being current cigarette smokers, and 6.3% reported having COPD.

89 .4) years; 105 (88.2%) were ever combustible cigarette smokers, and 66 (55.5%) were current e-cigaret

90 h/without a history of smoking), 'exclusive' cigarette smokers, and controls (non-users of any tobacc

91 search bronchoscopies on healthy nonsmokers, cigarette smokers, and e-cigarette users (vapers), and d

92 death rates from prostate cancer in current cigarette smokers, and inconsistent findings in incidenc

93 ylase (HDAC) activity is reduced in cells of cigarette smokers, and low concentrations of theophyllin

94 The lungs of cigarette smokers are known to contain increased concent

95 Cigarette smokers are more susceptible to periodontal di

96 Being current daily cigarette smoker at the time of diagnosis of HNSCC and h

97 US youth who were past-30-day cigarette smokers at any of wave 2 through wave 5, compl

98 s were measured in 38 non-smokers, and in 48 cigarette smokers at baseline and after 14 days of smoki

99 Associations were observed among never cigarette smokers but not among ever cigarette smokers.

100 ches substantially increase quit rates among cigarette smokers, but their safety in patients with myo

101 se contributes to endothelial dysfunction in cigarette smokers by administering the inhibitor allopur

102 In cigarette smokers, cariogenic bacteria from genus Strept

103 Among baseline never cigarette smokers, cigarette smoking initiation between

104 Among baseline non-cigarette smokers, cigarette smoking initiation, current

105 Among cigarette smokers, combined use of varenicline and bupro

106 Urinary IPF2alpha-I is elevated in cigarette smokers compared with controls (1525 +/- 180 v

107 5% confidence interval (CI): 1.09, 1.81) for cigarette smokers compared with never smokers.

108 re higher around adjacent implants placed in cigarette smokers compared with never smokers.

109 ain responses to external stimuli in chronic cigarette smokers compared with nonsmokers, only a few s

110 the preceding survey (eg, wave 2 past-30-day cigarette smokers completed wave 1), and indicated wheth

111 by weight reduction, if indicated), and, for cigarette smokers, counseling aimed at cessation.

112 s assessed and compared with the profiles of cigarette smokers (CS), dual users, and non-users.

113 to compare effects across four study groups: cigarette smokers (CS), e-vapor users (EV), former smoke

114 immunological peri-implant parameters among cigarette smokers (CS), individuals vaping e-cigarettes

115 D) and crestal bone loss [CBL]) are worse in cigarette-smokers (CS) and patients with type-2 diabetes

116 and self-perceived oral symptoms (OSs) among cigarette smokers (CSs) (group 1), individuals exclusive

117 etermine differences in breath odour between cigarette smokers (CSs) and e-cigarette consumers (ECs)

118 mmation between water-pipe smokers (WPs) and cigarette smokers (CSs).

119 ) and controls (P = 0.01), whereas BP(ND) in cigarette smokers did not differ from that in controls (

120 s, non-smokers (NS), CS, EC, and dual EC and cigarette smokers (DS).

121 confinement period, 32 healthy adult current cigarette smokers/e-cigarette dual users used a single s

122 Materials and Methods Current and former cigarette smokers enrolled between 2008 and 2011 from th

123 ally exclusive groups at baseline: exclusive cigarette smokers, exclusive e-cigarette users, and dual

124 een craving and regional metabolism in heavy cigarette smokers exposed to cigarette-related cues.

125 Many cigarette smokers express a desire to quit smoking, but

126 alth effects, first detectable in adolescent cigarette smokers, extend into adulthood.

127 Recidivism rates for cigarette smokers following treatment often exceed 80%.

128 for screening high-risk individuals (such as cigarette smokers) for cancer.

129 as validated by comparing blood levels among cigarette smokers from the National Health and Nutrition

130 sed a nationally representative sample of US cigarette smokers from the Population Assessment of Toba

131 Cigarette smokers (group 1) and never smokers (group 2)

132 We found that heavy cigarette smokers (>60 pack-years) have significantly mo

133 Current, former, and non- cigarette smokers had 5.1, 3.9, and 2.8 missing teeth, r

134 Moderate cigarette smokers had a significantly higher risk for mo

135 Cigarette smokers had considerably more cough, phlegm, a

136 Cigarette smokers had reduced odds of having AK, with cu

137 Cigarette smokers had significantly higher breath ethane

138 About a third of current cigarette smokers had started before age 15 years.

139 1, 2013), we determined whether baseline non-cigarette smokers had subsequently tried cigarette smoki

140 Current cigarette smokers had the highest prevalence of moderate

141 Pancreatic carcinomas from cigarette smokers harbor more mutations than do carcinom

142 uced risk for Parkinson's disease (PD) among cigarette smokers has been observed consistently during

143 inal study of over 10,000 current and former cigarette smokers, has been pivotal to these breakthroug

144 ies suggest that children raised in homes of cigarette smokers have a higher incidence of asthma than

145 Neutrophils in peripheral blood of cigarette smokers have been shown to be primed and more

146 Cigarette smokers have enhanced oxidative stress from ci

147 Cigarette smokers have higher rates of in vivo and in vi

148 ide treatment reduces cue-induced craving in cigarette smokers; however, the mechanism by which bupro

149 2.06; 95% CI, 1.53-2.76) than for exclusive cigarette smokers (HR, 1.86; 95% CI, 1.41-2.45).

150 8), healthy nonsmokers (NS; n = 8), or heavy cigarette smokers (HS; n = 8).

151 s (SES) of the mother, and the presence of a cigarette smoker in the household.

152 The risk among cigarette smokers increased with more years of exposure

153 As population sizes grow and number of cigarette smokers increases, it is vital that we underst

154 pendent vasodilation in normocholesterolemic cigarette smokers independent of changes in lipids.

155 Adult daily cigarette smokers initially not using e-cigarettes and w

156 and cigarette smoking cessation among adult cigarette smokers, irrespective of their motivation for

157 we hypothesized that the delayed healing in cigarette smokers is caused by the afflicted regenerativ

158 e specimens could provide a marker to screen cigarette smokers long before any symptoms of bladder ca

159 Some cigarette smokers may not be ready to quit immediately b

160 ce imaging data from 20 moderately dependent cigarette smokers (mean age = 25 years; no history of ne

161 d to quantify how this is altered in chronic cigarette smokers.Methods: Whole right upper lobes from

162 relapse were assessed in daily marijuana and cigarette smokers (n = 15) under two within-subject, cou

163 concentration of ELF GSH in HIV-seropositive cigarette smokers (n = 30) compared with nonsmokers (n =

164 F) and serum from control subjects (n = 10), cigarette smokers (n = 8), and individuals with CBD (n =

165 ive internal control, we studied age-matched cigarette smokers (n=15) who smoked 1 cigarette.

166 Postmortem LCs were obtained from long-term cigarette smokers (n=7) and from nonsmokers (n = 9), all

167 P/HTP, perceptions and attitudes in UK adult cigarette smokers naive to these products.

168 These data suggest that in abstinent cigarette smokers, nicotine augments initial responses t

169 Cigarette smokers not planning to quit are often overloo

170 Among cigarette smokers not willing or able to quit within the

171 was strongly associated with HNC among never cigarette smokers (odds ratio (OR) = 1.71, 95% confidenc

172 es, were far more likely to relapse than non-cigarette smokers (odds ratio: 19, p < .01).

173 n cross-sectional baseline analyses, current cigarette smokers of either sex had significantly more m

174 tal of 1492 observations among 1096 US youth cigarette smokers, of whom 49.4% were female, 67.2% were

175 Restricting analysis to cigarette smokers only provided increased evidence for l

176 an increased risk of lung cancer among male cigarette smokers or asbestos-exposed persons taking bet

177 d IPD incidence rates of men who were either cigarette smokers or nonsmokers at the time of enrollmen

178 sers were less frequently current than never cigarette smokers (OR 0.19; 95% CI: 0.06-0.61) and more

179 g was weakly associated with HNC among never cigarette smokers (OR = 1.20, 95% CI: 0.81, 1.77), analy

180 death were more likely to have been current cigarette smokers (OR = 1.3, 95% CI = 1.0 to 1.8).

181 y and OSCC risk among those who were current cigarette smokers (OR, 4.2; CI, 2.4-7.1) was stronger th

182 garette smoking, were current (past 30 days) cigarette smokers, or were high-intensity cigarette smok

183 displaceable binding potential (BP(ND)) than cigarette smokers (P = 0.03) and controls (P = 0.01), wh

184 Cigarette smokers performed significantly better than th

185 maintenance of strong nicotine dependence in cigarette smokers posit (i) a rapid brain nicotine accum

186 ess tobacco and HNC were observed among ever cigarette smokers, possibly reflecting residual confound

187 ated reflexive pharyngeal swallow in chronic cigarette smokers predispose them to risks of aspiration

188 A total of 17 cigarette smokers rated the pleasantness of cigarette pi

189 atigue in n = 18 healthy, briefly abstinent, cigarette smokers scanned repeatedly in a placebo-contro

190 Current cigarette smokers should also be given sufficient inform

191 Therefore, cigarette smokers should be: 1) encouraged to abstain fr

192 A total of 606 cigarette smokers started open-label nicotine patch trea

193 reptococcus mutans and Veillonella dispar in cigarette smokers, Streptococcus sanguinis and Tannerell

194 Cigarette smokers tend to drink more alcohol than their

195 l aspects of nicotine addiction, and menthol cigarette smokers tend to upregulate beta2* nAChRs more

196 ary 8-iso-PGF2alpha was 7.1-fold higher in a cigarette smoker than respective control subjects.

197 of lower acoustic neuroma risk among current cigarette smokers than among never smokers.

198 that the risk of tooth loss is greater among cigarette smokers than among non-smokers.

199 cancer (1.4 [1.2-1.7]) that was stronger for cigarette smokers than for never smokers (p for interact

200 capacity was elevated in a larger sample of cigarette smokers that included females.

201 cigarettes or vaping products, and exclusive cigarette smokers that was initiated in 2015.

202 tional imaging to examine neural activity in cigarette smokers, the most prevalent substance-dependen

203 Among never cigarette smokers, the odds ratio for ever cigar smoking

204 Among cigarette smokers, there was some evidence of a dose-res

205 ogistic regression models among baseline non-cigarette smokers to assess whether baseline water pipe

206 RTs) are intended for short-term use to help cigarette smokers to quit.

207 Nine were former cigarette smokers, two were current smokers, and one rep

208 d with smoking and nicotine dependence among cigarette smokers, using 1990-1991 data from the Coronar

209 f IMs was 36% to 56% greater in the lungs of cigarette smokers versus nonsmokers.Conclusions: The pre

210 Eighty-one tobacco-dependent cigarette smokers (volunteer sample) underwent positron

211 tional magnetic resonance imaging in chronic cigarette smokers, we measured neural and behavioral res

212 For biologic and behavioral reasons, cigarette smokers weigh less than nonsmokers.

213 Compared with never-smokers, current cigarette smokers were 1.7 times as likely to have root

214 Respondents who were ever cigarette smokers were asked when they began smoking reg

215 m 8072 adults who were currently established cigarette smokers were collected from December 2018 to N

216 crease in binding to MAO-B was observed when cigarette smokers were compared to nonsmoking subjects.

217 In study 1, 18 healthy volunteer cigarette smokers were exposed to three conditions in a

218 Additionally, when EC users and cigarette smokers were pooled together, number of vaping

219 Five hundred six adult (>/=18 years) cigarette smokers were randomly assigned and 315 (62%) c

220 AGE-apolipoprotein B and serum AGE levels in cigarette smokers were significantly higher than those i

221 Current cigarette smokers were those who had smoked > or =100 ci

222 Among patients with sepsis and ARDS, active cigarette smokers were younger and had lower severity of

223 e likely to be female, and less likely to be cigarette smokers when compared with other patients with

224 Furthermore, cigarette smokers who additionally used other tobacco pr

225 Furthermore, cigarette smokers who additionally used other tobacco pr

226 Cigarette smokers who additionally used other tobacco pr

227 treatment trial, the authors identified 222 cigarette smokers who failed to show a reduction of more

228 In 2011, current cigarette smokers who had ever used e-cigarettes were mo

229 history of smoking cigarettes, we identified cigarette smokers who quit, decreased, maintained, or in

230 The weighted population of adult daily cigarette smokers who were not using e-cigarettes and ha

231 The 1510 participants were cigarette smokers who were not willing or able to quit s

232 Among baseline non-past 30-day cigarette smokers who were past 30-day e-cigarette users

233 Most cigarette smokers who wish to quit too often relapse wit

234 Materials and Methods Cigarette smokers with and those without COPD participat

235 ma in participants who are current or former cigarette smokers with and without chronic obstructive p