ライフサイエンスコーパス: cigarette smoking

1 on employed (pharmacologic administration or cigarette smoking).

2 nongenetic risk factors (eg, hypertension or cigarette smoking).

3 marijuana, particularly in combination with cigarette smoking.

4 eversible airflow obstruction usually due to cigarette smoking.

5 mutagen, specifically ionizing radiation or cigarette smoking.

6 x, current smoking status, and pack-years of cigarette smoking.

7 d initial use of e-cigarettes and subsequent cigarette smoking.

8 on pulmonary function decline as mediated by cigarette smoking.

9 whether e-cigarette use affects the risk of cigarette smoking.

10 OPSCC and a history of </= 10 pack-years of cigarette smoking.

11 previous genome-wide association studies of cigarette smoking.

12 mprecise estimates, with limited control for cigarette smoking.

13 ciated with increased exercise and decreased cigarette smoking.

14 cardiac diseases or cardiac risks, including cigarette smoking.

15 nderstanding the brain mechanisms underlying cigarette smoking.

16 used e-cigarettes (18.9%) progressed toward cigarette smoking.

17 sk for subsequent progression to traditional cigarette smoking.

18 All analyses included fine adjustment for cigarette smoking.

19 both similar to and disparate from those of cigarette smoking.

20 their appeal to youth and curb the onset of cigarette smoking.

21 The main exposure was cigarette smoking.

22 s associated with progression to traditional cigarette smoking.

23 of nicotine, the main addictive substance in cigarette smoking.

24 eir potential to undermine efforts to reduce cigarette smoking.

25 r controlling for age, sex, alcohol use, and cigarette smoking.

26 was largely concentrated in snus use but not cigarette smoking.

27 sing and is now more common among youth than cigarette smoking.

28 .73 m(2), higher most recent pulse rate, and cigarette smoking.

29 a debilitating lung disease associated with cigarette smoking.

30 elopment independent of the other effects of cigarette smoking.

31 This increase is further elevated by cigarette smoking.

32 ronic lung diseases that are associated with cigarette smoking.

33 r combination effects of dietary regimen and cigarette smoking.

34 n after either electrically heated hookah or cigarette smoking.

35 ted hookah, comparable to the decrease after cigarette smoking.

36 cigarette smoking initiation and past 30-day cigarette smoking.

37 otentially offer a lower risk alternative to cigarette smoking.

38 he initiation and progression of combustible cigarette smoking.

39 characterized a novel microRNA signature of cigarette smoking.

40 limit the future population-level burden of cigarette smoking.

41 mmon mental disorders (1.77, 1.08-2.90), and cigarette smoking (2.01, 1.29-3.14).

42 conditions: (1) nicotine patch+denicotinized cigarette smoking, (2) nicotine patch+abstinence from sm

43 She discontinued cigarette smoking 20 years ago and has an occasional gla

44 Keratoconus was associated with regular cigarette smoking (38.5% vs. 14.6%; P = 0.04), but showe

45 Further, employees had a lower prevalence of cigarette smoking (9.7 vs. 17.3% in 2010, P < 0.001) com

46 Furthermore, we showed that cigarette smoking, a globally detrimental environmental

47 emale sex (around 2-fold increased odds) and cigarette smoking (about 5-fold reduced odds) predicted

48 ent cigarette smoking initiation and current cigarette smoking, accounting for established sociodemog

49 Although cigarette smoking acutely impairs endothelial function,

50 Cigarette smoking adds an estimated $100 billion in annu

51 hese data suggest that, after accounting for cigarette smoking, adherence to a set of healthy behavio

52 Our risk model included current cigarette smoking (adjusted odds ratio (OR) = 3.3, 95% c

53 ted odds ratio, 1.35; 95% CI, 1.12 to 1.63), cigarette smoking (adjusted odds ratio, 1.91; 95% CI, 1.

54 3; 95% CI, 1.43-9.76, respectively), current cigarette smoking (adjusted odds ratios: 2.48; 95% CI, 1

55 0.56, respectively), and higher intensity of cigarette smoking (adjusted proportional odds ratios: 2.

56 rst aim of this study was to clarify whether cigarette smoking affects tear secretion, goblet cell de

57 of this study provide further evidences that cigarette smoking affects the regenerative potentials of

58 With approximately 75% of smokers resuming cigarette smoking after using the Gold Standard Programm

59 ratio [aHR], 2.4; 95% CI, 1.5-3.9), current cigarette smoking (aHR, 1.9; 95% CI, 1.1-3.4), and syste

60 ed non-significant when adjusted for current cigarette smoking, alcohol consumption, and illicit drug

61 ndex, education, family history of diabetes, cigarette smoking, alcohol drinking, physical activity,

62 Besides accelerating adult FEV1 decline, cigarette smoking also modifies how early-life exposures

63 menthol concentration in the brain following cigarette smoking, altered neither the [ACh]-response re

64 ng tendency, parental cigarette smoking, and cigarette smoking among friends.

65 the association between biomarker-confirmed cigarette smoking and acute respiratory distress syndrom

66 n which information on important confounders-cigarette smoking and alcohol consumption-was lacking.

67 , being unmarried, living in the rural area, cigarette smoking and alcohol drinking were associated w

68 We compared changes in self-reported cigarette smoking and alcohol use in the 30 days prior t

69 have been formally established as caused by cigarette smoking and are included in official estimates

70 may be a part of the biological link between cigarette smoking and atherosclerosis.

71 Cigarette smoking and attention-deficit/hyperactivity di

72 essed the relationship between self-reported cigarette smoking and biomarker levels, the validity of

73 ctive pulmonary disease (COPD) is induced by cigarette smoking and characterized by inflammation of a

74 l airway collapse [ECAC]) is associated with cigarette smoking and chronic obstructive pulmonary dise

75 gnificantly modifies the association between cigarette smoking and COPD.

76 of snus independently predicted the onset of cigarette smoking and current cigarette smoking at follo

77 rombotic complications include hypertension, cigarette smoking and diabetes mellitus.

78 ehensively determine the association between cigarette smoking and DNA methylation, we conducted a me

79 Frequency of combustible cigarette smoking and e-cigarette use within the past 30

80 been achieved in reducing the prevalence of cigarette smoking and exposure to secondhand smoke.

81 ding assessment of possible joint effects of cigarette smoking and fine particulate matter (particula

82 e pulmonary disease (COPD) is linked to both cigarette smoking and genetic determinants.

83 The negative health effects of cigarette smoking and HIV infection are synergistic.

84 We investigated associations between cigarette smoking and incidence of basal cell carcinoma

85 The association between cigarette smoking and inflammation is well known.

86 f DNA methylation in the association between cigarette smoking and inflammation.

87 ation in monocytes, a cell type sensitive to cigarette smoking and involved in atherogenesis, may be

88 was to quantify the long-term association of cigarette smoking and its cessation with the incidence o

89 sively compared the long-term association of cigarette smoking and its cessation with the incidence o

90 ty of exposure to 15 case-control studies of cigarette smoking and lung cancer (1985-2009).

91 fication of the association of pack-years of cigarette smoking and lung cancer by intensity of cigare

92 Application of our method in this example of cigarette smoking and lung cancer demonstrated that redu

93 an genome that has been associated with both cigarette smoking and mortality.Objectives: We sought to

94 study criteria, including no current tobacco cigarette smoking and no known health problems or prescr

95 DR was associated with diabetes duration, cigarette smoking and peripheral neuropathy.

96 y have implications for predicting long-term cigarette smoking and problems with other substance abus

97 Except for the association between cigarette smoking and provoked VTE, which is potentially

98 outcome measures were weighted prevalence of cigarette smoking and quit ratio (ratio of former smoker

99 e sought to describe a microRNA signature of cigarette smoking and relate it to smoking-associated cl

100 y was to investigate the association between cigarette smoking and smoking cessation and the prevalen

101 s are crucial for reducing the prevalence of cigarette smoking and smoking-related illnesses.

102 essment of interactions with patients' heavy cigarette smoking and the relationship of this mechanism

103 Cigarette smoking and tooth loss are seldom considered c

104 n the frequency and intensity of combustible cigarette smoking and vaping.

105 Educational level, cigarettes smoking and physical exercise were not signif

106 rom smoking, (3) placebo patch+denicotinized cigarette smoking, and (4) placebo patch+abstinence from

107 level, sensation-seeking tendency, parental cigarette smoking, and cigarette smoking among friends.

108 fficients for age, sex, waist circumference, cigarette smoking, and family history of colorectal canc

109 s: older age, white race, diabetes mellitus, cigarette smoking, and hypertension.

110 k factors such as hypertension, weight gain, cigarette smoking, and loss of cardiorespiratory fitness

111 been advertised as a healthy alternative to cigarette smoking, and users are under the impression th

112 ears with at least a 30 pack-year history of cigarette smoking, and, if a former smoker, had quit wit

113 spiratory illnesses in smokers and highlight cigarette smoking as a potential driver of virulence in

114 with ever use of e-cigarettes or past 30-day cigarette smoking associated with past 30-day e-cigarett

115 s, and the pooled odds ratio for past 30-day cigarette smoking at follow-up was 4.28 (95% CI, 2.52-7.

116 The pooled probabilities of past 30-day cigarette smoking at follow-up were 21.5% for baseline p

117 e smoking initiation, current (past 30 days) cigarette smoking at follow-up, and the intensity of cig

118 e past 30-day e-cigarette users, past 30-day cigarette smoking at follow-up.

119 snus use were associated with high-intensity cigarette smoking at follow-up.

120 e smoking at follow-up, and the intensity of cigarette smoking at follow-up.

121 d the onset of cigarette smoking and current cigarette smoking at follow-up.

122 Our data indicate that cigarette smoking before first cancer diagnosis increase

123 versive conditioning during sleep will alter cigarette-smoking behavior during ensuing wakefulness.

124 o better understand the biological impact of cigarette smoking behaviours in adolescence.

125 Our findings illustrate the impact of cigarette smoking behaviours on DNA methylation at some

126 ncentrations correlated with age, sex, race, cigarette smoking, body mass index, inheritance of the e

127 practice.A 64-year-old man with a history of cigarette smoking but no significant comorbidities prese

128 as associated with a decreased prevalence of cigarette smoking, but not with lower prevalence of obes

129 on of the excess odds ratio per pack-year of cigarette smoking by time since smoking cessation among

130 ihypertensive medication, diabetes mellitus, cigarette smoking, cardiovascular disease history, atria

131 Cigarette smoking causes emphysema, a fatal disease invo

132 the association between e-cigarette use and cigarette smoking cessation among adult cigarette smoker

133 The primary endpoint was cigarette smoking cessation.

134 Members had a lower prevalence of cigarette smoking compared to nonmembers (overall year-a

135 d with changes in CBF from the denicotinized cigarette smoking conditions to the abstinent conditions

136 Advanced age, male sex, and cigarette smoking contribute to the development of bladd

137 Cigarette smoking contributes to the development of dest

138 ciation between baseline e-cigarette use and cigarette smoking, controlling for sex, age, race/ethnic

139 Cigarette smoking (CS) can exacerbate the incidence and

140 Progression to cigarette smoking, defined using 3 specific states along

141 ess, physical activity, alcohol consumption, cigarette smoking, diabetes risk and primary cardiovascu

142 , use of antihypertensive treatment, current cigarette smoking, diabetes status, body mass index, C-r

143 e increased risk of colorectal cancer due to cigarette smoking differed by anatomical subsite or sex.

144 Taken together, our results suggest that cigarette smoking disrupts endogenous proresolving pathw

145 race/ethnicity, alcohol drinking intensity, cigarette smoking duration and intensity, and/or family

146 Cigarette smoking during pregnancy is a major public hea

147 Cigarette smoking during pregnancy is a major public hea

148 Maternal cigarette smoking during pregnancy remains one of the mo

149 Smokers were abstinent from cigarette smoking for 12 hours for all sessions.

150 isease (COPD) risk is strongly influenced by cigarette smoking, genetic factors are also important de

151 s based on glycemic status and self-reported cigarette-smoking habit: a) CS with T2DM; b) CS without

152 Cigarette smoking has a broad impact on genome-wide meth

153 Cigarette smoking has been associated with higher risk o

154 umerous DNA methylation (DNAm) biomarkers of cigarette smoking have been identified in peripheral blo

155 omial regression to model the association of cigarette smoking (having ever smoked regularly, cigaret

156 sociated with higher odds of ever or current cigarette smoking, higher odds of established smoking, h

157 National Health Interview Surveys yielded cigarette smoking histories for the US adult population

158 atus (surrogate of human papillomavirus) and cigarette smoking history (pack-years) randomly assigned

159 viduals aged 55-80 years with a 30 pack-year cigarette smoking history and, if they are former smoker

160 disease, two diagnostic breathing tests, and cigarette smoking history.

161 haviors, particularly through differences in cigarette smoking; however, the association with fatal C

162 cigs) provide an alternative to conventional cigarette smoking; however, the evidence base of risks a

163 with adjustment for the depth and amount of cigarette smoking (HR [95% CI]: 1.37 [1.06, 1.77]), whil

164 on of modifiable risk factors: dyslipidemia, cigarette smoking, hypertension, obesity, and hyperglyce

165 treatment, including precipitous declines in cigarette smoking, improvements in hypertension treatmen

166 ion into lung lining fluid was diminished by cigarette smoking in humans and mice.

167 roduced nicotine pharmacokinetics resembling cigarette smoking in humans.

168 A history of cigarette smoking in patients with new-onset adult asthm

169 be a stronger mediator of inflammation than cigarette smoking in patients with T2DM.

170 34,988 to 36,268, depending on the outcome), cigarette smoking in the past 30 days among underreprese

171 is associated with the highest prevalence of cigarette smoking in the United States, and is also a ti

172 Cigarette smoking increases complication risk in surgica

173 These combined negative effects suggest that cigarette smoking increases interleukin-18 levels throug

174 Cigarette smoking increases overall mortality, but it is

175 Cigarette smoking increases risk for multiple diseases.

176 We hypothesized that cigarette smoking induces extensive transcriptomic chang

177 snus use were independently associated with cigarette smoking initiation (adjusted odds ratios: 2.56

178 ine snus use were associated with subsequent cigarette smoking initiation and current cigarette smoki

179 associated with greater risk for subsequent cigarette smoking initiation and past 30-day cigarette s

180 ngitudinal studies reporting odds ratios for cigarette smoking initiation associated with ever use of

181 Among baseline never cigarette smokers, cigarette smoking initiation between baseline and follow

182 were associated with decreased likelihood of cigarette smoking initiation compared with control inter

183 moking, the pooled odds ratio for subsequent cigarette smoking initiation was 3.62 (95% CI, 2.42-5.41

184 The pooled probabilities of cigarette smoking initiation were 30.4% for baseline eve

185 Among baseline non-cigarette smokers, cigarette smoking initiation, current (past 30 days) cig

186 Cigarette smoking is a chronic relapsing brain disorder,

187 Cigarette smoking is a leading cause of preventable mort

188 Cigarette smoking is a leading modifiable cause of death

189 Cigarette smoking is a major factor for the development

190 Cigarette smoking is a major public health danger.

191 Cigarette smoking is a major risk factor for acquisition

192 Cigarette smoking is a major risk factor for chronic obs

193 Cigarette smoking is a major risk factor in the developm

194 Studies have shown that cigarette smoking is a modifiable risk factor for diabet

195 Cigarette smoking is a risk factor for a wide range of h

196 Cigarette smoking is a risk factor for many perioperativ

197 Cigarette smoking is a risk factor for stroke and is lin

198 Cigarette smoking is a significant risk factor for Alzhe

199 While cigarette smoking is a strong risk factor for invasive p

200 Maternal cigarette smoking is a well-established risk factor for

201 Cigarette smoking is an established environmental risk f

202 Cigarette smoking is associated with cognitive decline a

203 his study adds to the existing evidence that cigarette smoking is associated with higher all-cause an

204 Purpose Cigarette smoking is associated with increased incidence

205 Cigarette smoking is associated with increased risk for

206 Cigarette smoking is associated with metabolite abnormal

207 Cigarette smoking is associated with structural changes

208 Cigarette smoking is known to increase the risk of atria

209 Cigarette smoking is linked to important aspects of tube

210 Intriguingly, cigarette smoking is most strongly and robustly associat

211 Cigarette smoking is one such lifestyle factor.

212 rs; and prohibiting ENDS use in places where cigarette smoking is prohibited.

213 Cigarette smoking is significantly associated with new-o

214 Despite well-established evidence that cigarette smoking is the largest modifiable risk factor

215 Cigarette smoking is the leading cause of preventable de

216 Cigarette smoking is the leading cause of preventable mo

217 Cigarette smoking is the leading preventable cause of de

218 Cigarette smoking is the main risk factor for COPD, and

219 he association between psychotic illness and cigarette smoking is well known, the reasons are unclear

220 tes to the lung destruction that accompanies cigarette smoking; it simultaneously inhibits lung tumor

221 We show herein, for the first time, that cigarette smoking leads to an increase in platelet-activ

222 Cigarette smoking leads to upregulation of nicotinic ace

223 a-modulating risk factors as antibiotic use, cigarette smoking, levels of sanitation, and diet in the

224 diometabolic risk factors, including current cigarette smoking, low fruit and vegetable intake, low p

225 Impaired lung function is often caused by cigarette smoking, making it challenging to disentangle

226 jectory included high school heavy drinking, cigarette smoking, marijuana use, poly-prescription drug

227 brain activation, genetic variation, and/or cigarette smoking may account for this mixed response pr

228 These findings suggest that active cigarette smoking may increase susceptibility to acute r

229 Therefore, we hypothesized that cigarette smoking may influence stroke, at least in part

230 se pathways that are activated by asthma and cigarette smoking may optimize therapeutic responses.

231 Cigarette smoking measured both by history and biomarker

232 One woman had AC. Cigarette smoking more than doubled HSIL risk.

233 Cigarette smoking more than doubled HSIL risk.

234 Infections, cigarette-smoking, mucosal dysbiosis, host genetics and

235 Cigarette smoking (odds ratio [OR] 1.89, 95% CI 1.26-2.8

236 at mLOY is a major mediator of the effect of cigarette smoking on cancer risk, as mLOY was observed i

237 nd meta-analysis was to assess the impact of cigarette smoking on clinical outcomes following periodo

238 ative effect of self-reported tooth loss and cigarette smoking on COPD among United States adults age

239 vides potential insights into the effects of cigarette smoking on gene expression in whole blood and

240 ustrate, we consider the possible effects of cigarette smoking on homocysteine levels, with self-repo

241 was studied along with aggravative effect of cigarette smoking on liver status in mice.

242 dearth of studies regarding the influence of cigarette smoking on periodontal inflammatory conditions

243 ed to ascertain (1) site-specific effects of cigarette smoking on the periodontium compared to never-

244 55-65 years, with at least 20 pack-years of cigarette smoking or current smokers.

245 proximate steady-state concentration between cigarette smoking or vaping, or during nicotine patch us

246 [OR] = 6.31; 95% CI, 5.39-7.39) and current cigarette smoking (OR = 5.96; 95% CI, 5.67-6.27).

247 s (OR = 7.42; 95% CI, 5.63-9.79) and current cigarette smoking (OR = 7.88; 95% CI, 6.01-10.32).

248 d understanding of causal mechanisms linking cigarette smoking, oral health, and COPD, particularly t

249 der (ADHD) are at increased risk for adverse cigarette smoking outcomes, and little is known about fa

250 evalence of the five major HRBs-alcohol use; cigarette smoking, physical inactivity, unhealthy diet,

251 Two-sided z tests comparing cigarette smoking prevalence revealed no significant dif

252 roviders should counsel their patients about cigarette smoking, preventive dental care, and COPD risk

253 Cigarette smoking promotes body weight reduction in huma

254 vy drinking, treatment attendance, drug use, cigarette smoking, psychiatric symptoms, and HIV-risk be

255 in the late luteal phase, whereas aging and cigarette smoking reduced (11)C-vorozole uptake.

256 We tested the hypothesis that cigarette smoking-relevant nicotine inhalation during pr

257 Cigarette smoking remains one of the leading public heal

258 Substantial progress has been made, but cigarette smoking remains one of the most pressing globa

259 Cigarette smoking remains the leading avoidable cause of

260 rovide some mechanistic explanations whereby cigarette smoking renders a patient susceptible to tuber

261 s coexpressed with the microRNA signature of cigarette smoking revealed enrichment for immune-related

262 Passive cigarette smoking showed very little, if any, increased

263 Cigarette smoking, specifically current smoking, remains

264 Associations were strongest for parental cigarette smoking status; compared to children of non-sm

265 tooth loss (0, 1 to 5, 6 to 31, or all) and cigarettes smoking status (never, former, or current) wi

266 etic resonance imaging data from healthy and cigarette-smoking subjects performing the Iowa Gambling

267 We hypothesized that HIV infection and cigarette smoking synergize to alter the function of alv

268 smokers have a higher risk of initiation of cigarette smoking than never smokers.

269 Unlike cigarette smoking, the effects of e-cigarettes and their

270 sychosocial, and behavioral risk factors for cigarette smoking, the pooled odds ratio for subsequent

271 and the private sector to reduce the toll of cigarette smoking through reduced initiation and increas

272 uding diabetes mellitus, hyperlipidemia, and cigarette smoking to cases.

273 andenburg, Germany, who had complete data on cigarette smoking, tooth loss, and covariates.

274 ry notable for hypertension, hyperlipidemia, cigarette smoking, type II diabetes mellitus, and end-st

275 ry notable for hypertension, hyperlipidemia, cigarette smoking, type II diabetes mellitus, and end-st

276 S) of complex behavioural phenotypes such as cigarette smoking typically employ self-report phenotype

277 he second leading cause of lung cancer after cigarette smoking via inhalation; however, exposure thro

278 In group 1, the mean duration of cigarette smoking was 22.3 +/- 1.6 pack years.

279 The report concluded that cigarette smoking was a cause of lung cancer in men and

280 Conclusion Cigarette smoking was associated with a reduction in sur

281 Cigarette smoking was associated with higher prevalence

282 Cigarette smoking was associated with lower concentratio

283 The question of whether cigarette smoking was associated with lung cancer was ce

284 t finding from VTE subtype analyses was that cigarette smoking was associated with provoked but not u

285 udy (2012-2014), a study of 332 adults whose cigarette smoking was measured in a naturalistic environ

286 A Cox model, adjusting for pack-years of cigarette smoking, was used to calculate hazard ratios a

287 ng 2620 CpG sites previously associated with cigarette smoking, we investigated DNA methylation chang

288 okers, both pharmacologic administration and cigarette smoking were associated with activity decrease

289 e, lower socioeconomic status, diabetes, and cigarette smoking were consistently associated with wors

290 Individual-level data on parental cigarette smoking were obtained from 12 case-control stu

291 hypertension, high blood sugar, and regular cigarette smoking were rare.

292 ver time (a measure similar to pack-years of cigarette smoking), were calculated using predicted BMIs

293 non-cigarette smokers had subsequently tried cigarette smoking, were current (past 30 days) cigarette

294 in general that is not driven exclusively by cigarette smoking, which is the main risk factor for chr

295 Although reductions in cigarette smoking will achieve the greatest impact on lu

296 y population, we examined the association of cigarette smoking with DNA methylation using single nucl

297 pidemiologic evidence clearly links maternal cigarette smoking with FGR, insight into the molecular m

298 rrent study, we assessed the relationship of cigarette smoking with gray matter (GM) and white matter

299 ociations of SNPs, age, body mass index, and cigarette smoking with the rate of progression and relat

300 ly, these effects are modified by history of cigarette smoking, with a strong protective effect media