戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1  loss of any one of them results in impaired ciliary motility.
2 oregulated gene (PACRG), a protein linked to ciliary motility.
3 romolecular complexes that are essential for ciliary motility.
4 nd are left-biased at the LRO in response to ciliary motility.
5 3 and that RS3 and the CSC are important for ciliary motility.
6 symptoms and reducing the effects of reduced ciliary motility.
7 ositioned to integrate signals that regulate ciliary motility.
8 d SPEF2 have a PCD phenotype with defects in ciliary motility.
9 ivity of specific dynein isoforms to control ciliary motility.
10 (FAP221) complex is essential for control of ciliary motility.
11 begun to elucidate the mechanisms underlying ciliary motility.
12 st the repeated mechanical stress induced by ciliary motility.
13 alus, and defined a unique role for hydin in ciliary motility.
14 erves as a central hub for the regulation of ciliary motility.
15 ized mechanotransduction pathway to regulate ciliary motility.
16 ork to understand their functions in driving ciliary motility.
17 n all lead to defects in dynein assembly and ciliary motility.
18 nvolved in the pathway by which I1 regulates ciliary motility.
19 d with regeneration of ATP and regulation of ciliary motility.
20 ze and architecture during cell division and ciliary motility.
21 ctions that play crucial roles in regulating ciliary motility.
22                 Despite their importance for ciliary motility across eukaryotes, the molecular functi
23 ere disease that is not explained by loss of ciliary motility alone.
24 associated with the protofilament ribbon and ciliary motility, also positively regulates lifespan.
25 rd1 mechanism contributes to deceleration of ciliary motility and age-dependent ventricular enlargeme
26   Outer dynein arms (ODAs) are essential for ciliary motility and are preassembled in the cytoplasm b
27                 Axonemal dynein motors drive ciliary motility and can consist of up to twenty distinc
28 e from the underlying epithelia, to maximize ciliary motility and clearance of bacteria.
29 ctional ion signaling compartment connecting ciliary motility and flow to molecular LR signaling.
30 is caused by a central pair defect impairing ciliary motility and fluid transport in the brain.
31                   Thus, Ofd1 is required for ciliary motility and function in zebrafish, supporting d
32 long which motor proteins transmit force for ciliary motility and intraflagellar transport.
33 ss of the proteins involved causes defective ciliary motility and laterality abnormalities in zebrafi
34 nt of the upper airway respond to defects in ciliary motility and mucociliary clearance.
35 es that have a range of functions, including ciliary motility and mucous secretion, and contain enzym
36 ubpopulations of cells which retained active ciliary motility and others which demonstrated specializ
37 n central pair apparatus (CPA) genes perturb ciliary motility and result in PCD in mouse models.
38                                              Ciliary motility and signaling are essential for human d
39  broken at the left-right organizer (LRO) by ciliary motility and the resultant directional flow of e
40 y dyskinesia (PCD) has relied on analysis of ciliary motility and ultrastructure; however, these test
41 mice, zebrafish and frogs results in loss of ciliary motility and/or reduced length and number of mot
42 n factor that controls centriole docking and ciliary motility, and airways fail to become fully cilia
43 terms of the assembly and stability of DMTs, ciliary motility, and other microtubule systems.
44 inducing gene product whose mutation impairs ciliary motility, and polycystin-2, whose ablation is as
45              Nevertheless, neither cilia nor ciliary motility are absolutely required for otolith tet
46 onal control of primary cilium formation and ciliary motility are beginning to be understood, but lit
47 n mechanism, axonemal integrity and possibly ciliary motility are required for signal amplification o
48 nein, the macromolecular machine that powers ciliary motility, assembles in the cytosol with the help
49 o TTLL6 paralogs caused severe deficiency in ciliary motility associated with abnormal waveform and r
50                         In humans, defective ciliary motility can lead to male infertility and a cong
51 sh likewise disrupts dynein arm assembly and ciliary motility, causing primary ciliary dyskinesia phe
52 n, spontaneous CSF-cN activity decreased and ciliary motility changed, suggesting physical interactio
53 a, and, consequently, this process underlies ciliary motility, cilium-based signaling, and ciliopathi
54 x1c1 in zebrafish also caused laterality and ciliary motility defects.
55                                              Ciliary motility depends on both the precise spatial org
56  nature of olfactory cilia, we observed that ciliary motility genes are repressed in zebrafish, mouse
57 5, DRC4/GAS8) have been linked to defects in ciliary motility in humans and lead to a ciliopathy know
58 roscopy to investigate the role of cilia and ciliary motility in otolith formation.
59 of mutant forms leads to profound defects in ciliary motility, including the failure of the hydrodyna
60                                              Ciliary motility is driven by axonemal dyneins that are
61                                 However, how ciliary motility is perceived and transduced into asymme
62                                              Ciliary motility is regulated by the conserved nexin-dyn
63 yses begin to reveal the mechanisms by which ciliary motility is regulated.
64  to generate sensory potentials and regulate ciliary motility, is normally localized in the cilia, pr
65  and Ncx4a morphants and that the defects in ciliary motility, KV fluid flow and placement of interna
66  In mutants with defective cilia (iguana) or ciliary motility (lrrc50), otoliths are frequently ectop
67 utant mice with impaired ciliogenesis and/or ciliary motility of the node.
68                 A reporter gene required for ciliary motility placed under the control of the HYDA1 p
69 y only one or two cilia per cell, which lack ciliary motility-related proteins (DNAH5; CCDC39) as see
70 constituent of airway lining fluid, enhances ciliary motility, relaxes airway smooth muscle, inhibits
71 al, and biophysical studies, some aspects of ciliary motility remain elusive, such as the regulation
72    Furthermore, our work suggests additional ciliary motility-signaling connections, since EFHC1 (EF-
73      Asymmetric ICOs occur with onset of LRO ciliary motility, thus representing the earliest known L
74 at the LRO that connect ciliary sensation of ciliary motility to downstream left-right signaling.
75 e (MT) biology, the sliding MT hypothesis of ciliary motility was born.
76 , a cation channel, are required for sensing ciliary motility, yet their function and the molecular m