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1 ced by formalin, acetic acid, capsaicin, and cinnamaldehyde.
2 mmatory capacity was primarily attributed to cinnamaldehyde.
3 aldehydes, a hindered aromatic aldehyde, and cinnamaldehyde.
4 ctrophilic compounds such as mustard oil and cinnamaldehyde.
5 ingredients such as allyl isothiocyanate and cinnamaldehyde.
6 ctively prevented by higher release of trans-cinnamaldehyde.
7 in a chemospecific anti-aldol reaction with cinnamaldehyde.
8 duct of decarboxylation from such compounds, cinnamaldehydes.
9 for by elevated levels of benzaldehydes and cinnamaldehydes.
11 0.31 mg GAE/g), TFC (0.50 +/- 0.01 mg QE/g), cinnamaldehyde (19.33 +/- 0.002 mg/g), eugenol (10.57 +/
13 bes that contain 2-nitro- (1-3) and 4-fluoro-cinnamaldehyde (4-6) and applied them to the anion recog
14 ve 4-chloro-N-methyl-N-nitrosoaniline (76%), cinnamaldehyde (55%), 3-phenyl-5-hydroxyisoxazoline (26%
15 ensitive, lung-specific neurons responded to cinnamaldehyde, a TRPA1 agonist, with increases in intra
16 of 1,2,4-trimethoxybenzene, indole, and (E)-cinnamaldehyde, all blossom components, is highly attrac
23 75% calcium chloride and combination of 0.1% cinnamaldehyde and 0.75% calcium chloride) were used to
25 he cinnamaldehyde alone and a combination of cinnamaldehyde and calcium chloride treatments yielded b
26 nds with demonstrated halogenation pathways (cinnamaldehyde and citric acid) across guar gels with va
28 , little to mild toxicity was seen for trans-cinnamaldehyde and eugenol, respectively, while carvacro
31 3-phenylpropionate, methyl 2-phenylacetate, cinnamaldehyde and methyl cinnamate were produced during
33 ty problems with the lipophilic vanillin and cinnamaldehyde and neutralizing their volatility, produc
34 king interactions between the phenyl ring of cinnamaldehyde and phenylated SAMs allowed tuning of rea
35 ontrolling the liquid phase hydrogenation of cinnamaldehyde and related benzylic aldehydes over Pt na
36 criteria for the subsurface halogenation of cinnamaldehyde and the broad capacity for THM formation
37 The TRPA1 agonists allyl isothiocyanate and cinnamaldehyde and the TRPV4 agonist GSK1016790A caused
42 emicals such as allyl isothiocyanate (AITC), cinnamaldehyde, and allicin, produce nociceptive sensati
43 amon have a higher concentration of eugenol, cinnamaldehyde, and antioxidant capacity, as well as a l
44 ing nonenolizable aliphatic aldehydes, trans-cinnamaldehyde, and beta-substituted styrenes were also
46 ymethylfurfural, anisaldehyde, benzaldehyde, cinnamaldehyde, and phenylaldehyde are commonly generate
47 rovided good encapsulation and protection of cinnamaldehyde, and the controlled release of cinnamalde
50 (hydroxycinnamoyl-SCoAs) to cinnamaldehydes; cinnamaldehydes are then reduced to cinnamyl alcohols by
51 lic plant compounds, such as mustard oil and cinnamaldehyde, are TRPA1 agonists, it is unknown whethe
56 ral properties on the hydrogenation of trans-cinnamaldehyde by using a cobalt phosphide catalyst.
61 th most other common oral irritants, such as cinnamaldehyde, capsaicin, and alcohol, which irritate m
62 ti-component coating via layer deposition of cinnamaldehyde (CIN)-doped chitosan/poly(vinyl alcohol)/
64 d cinnamic acids (hydroxycinnamoyl-SCoAs) to cinnamaldehydes; cinnamaldehydes are then reduced to cin
65 MS), highlighting the bioactive potential of cinnamaldehyde, cinnamic acid, benzoic acid, coumarin, l
67 ed that 2,3-butanediol, hexanal, hexanol and cinnamaldehyde contributed the most to classification of
71 nylmethyl benzenesulfonamide and beta-chloro-cinnamaldehyde, depending on whether one uses either NaI
73 vation, this study presents the synthesis of cinnamaldehyde derivatives i.e., chalcones and pyrazoles
74 lation of 2-arylimidazo[1,2-a]pyridines with cinnamaldehyde derivatives to construct fused N-heterocy
75 Wadsworth-Emmons reaction, were treated with cinnamaldehyde derivatives, acetic acid, and borohydride
76 ratio when 20 mol % of the chiral imidazole-cinnamaldehyde-derived carbamate was utilized in the rea
78 Here, we investigated the toxicity of trans-cinnamaldehyde, eugenol, and carvacrol after intramuscul
79 ), antioxidant activity, and key bioactives (cinnamaldehyde, eugenol, and cinnamic acid) using two ex
81 ivo vagal innervated mouse lung preparation, cinnamaldehyde evoked action potential discharge in mous
83 imp preservation, where headspace release of cinnamaldehyde from emulsions at non-contact mode was mo
85 ch-turnip peel extract (PS-TPE) and guar gum-cinnamaldehyde (GG-CA) for freshness monitoring and enha
90 anistic insight from kinetic mapping reveals cinnamaldehyde hydrogenation is structure-insensitive ov
91 at halogenated products of additives such as cinnamaldehyde (i.e., a-chlorocinnamaldehyde and a-bromo
92 to selectively orient the reactant molecule cinnamaldehyde in a configuration associated with hydrog
94 he conjugate addition of nitromethane with a cinnamaldehyde in the presence of the Jorgensen-Hayashi
101 rocess evolves through a [3 + 2] 1,3-DC when cinnamaldehyde is used in the presence of an azomethine
107 s did not result in selective degradation of cinnamaldehyde over other compounds (i.e., benzoate and
109 uinone and the electrophilic TRPA1 activator cinnamaldehyde produced antinociception that was lost in
110 innamaldehyde, and the controlled release of cinnamaldehyde promoted sustained antibacterial efficacy
111 alf-life values (DT(50) or t(1/2)) for trans-cinnamaldehyde ranged from 2.02 to 2.49 h, while for lim
113 ty in enhancing C = O hydrogenation (through cinnamaldehyde reorientation), a general phenomenon exte
114 ipeptide Gly-Glu-Glu to the para position of cinnamaldehyde resulted in an inhibitor (Cinn-GEE) of su
115 urons that express TRPA1, a mustard oil- and cinnamaldehyde-sensitive channel, and that these respons
116 inate (CAS) and two natural aldehydes (trans-cinnamaldehyde (TC) and citral) were studied by evaluati
120 al Diabrotica to structural analogues of (E)-cinnamaldehyde, the major attractant for Diabrotica unde
122 ng linoleic acid (LA) to SN38 and JQ-1 via a cinnamaldehyde thioacetal (CT) bond, facilitating co-del
123 d cyclization between beta-keto enamines and cinnamaldehydes to furnish the functionalized biphenyls.
124 were present in 50% (menthol, triacetin, and cinnamaldehyde) to 80% (ethyl maltol) of the samples.
126 in essential oils i.e. carvacrol, thymol and cinnamaldehyde was developed following the Quality by De
130 yed to install a pyridyl to the alkene trans-cinnamaldehyde while Ag(I) ions are used in a second ste
133 ansient receptor potential (TRP) A1 agonist, cinnamaldehyde, with ozone having no effect on cinnamald