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1 intubated patients with sepsis-related acute circulatory failure.
2 er dysfunction with vascular leakage-induced circulatory failure.
3 ogressive, often dilated, cardiomyopathy and circulatory failure.
4 in postnatal death, most probably because of circulatory failure.
5 ns suggest that TF(-/-) embryos are dying of circulatory failure.
6  exceptional during the early phase of acute circulatory failure.
7 f Ramsay sedation scale >4]) and, in case of circulatory failure, a second set of measurements after
8 patients in whom VE was indicated because of circulatory failure and clinical indices.
9 n = 90) presenting with sepsis-induced acute circulatory failure and considered for volume expansion.
10 cuspid regurgitation (TR) is associated with circulatory failure and death.
11 tion of iNOS activity with L-NIL reduced the circulatory failure and liver injury, while selective in
12 -permeable radical scavenger (tempol) on the circulatory failure and MODS (kidney, liver, lung) cause
13 le(4,3-alpha)quinoxaline-1-one (ODQ), on the circulatory failure and multiple organ dysfunction syndr
14 , life-threatening condition associated with circulatory failure and multiple organ dysfunctions.
15 sms by which calpain inhibitor I reduces the circulatory failure as well as the organ injury and dysf
16 c shock currently refers to a state of acute circulatory failure associated with infection.
17 fail to form a ventral heart tube and die of circulatory failure at embryonic day (E) 8.5.
18                    In severe cases, profound circulatory failure can result.
19 f endogenous nitric oxide contributes to the circulatory failure caused by endotoxin (lipopolysacchar
20 survival in a cohort of patients with severe circulatory failure correlated with their relative frequ
21  of norepinephrine tartrate) with persistent circulatory failure (defined by lactate > 2 mmol/L, olig
22  was significant liver damage resulting from circulatory failure during cardiopulmonary arrest before
23 e Pco2 reflects metabolic alterations due to circulatory failure during circulatory shock.
24  might participate in the pathophysiology of circulatory failure during sepsis, and represent a poten
25 e heart failure and impending respiratory or circulatory failure especially in the presence of a diag
26  managing critically ill patients with acute circulatory failure, especially in the absence of cardia
27                           It predicts 90% of circulatory-failure events in the test set, with 82% ide
28                                       Fontan circulatory failure (FCF) is a chronic state in palliate
29 y-five mechanically ventilated patients with circulatory failure for whom the decision to give fluid
30                    Adult patients with acute circulatory failure, having continuous cardiac output mo
31 ight ventricular assistance (and right-sided circulatory failure), hemodynamic indexes, percent trans
32 sferase and aspartate aminotransferase), and circulatory failure (hypotension) as well as the increas
33 or risk of progression to severe sepsis with circulatory failure in a multicenter setting.
34 eatment of anticipated and established acute circulatory failure is growing.
35  physiological change determines whether CSF circulatory failure manifests as Alzheimer's disease (AD
36 types in ARDS that more closely aligned with circulatory failure mechanisms and mortality than curren
37                                 During acute circulatory failure (n = 83), arm noninvasive blood pres
38 responsiveness in ventilated patients with a circulatory failure of any cause.
39  and caspase-8 decreased heat stress-induced circulatory failure, organ injury, and lethality.
40 evious G1 ACLF, with liver, coagulation, and circulatory failure posing the highest increased risk.
41 utilized in the treatment of respiratory and circulatory failure refractory to conventional managemen
42                                       During circulatory failure, the ultimate goal of treatments tha
43               Therefore, we believe that CSF circulatory failure, ultimately resulting in reduced neu
44                                              Circulatory failure, ventricular dysfunction, atrioventr
45  of the acute heart failure patient, such as circulatory failure, volume overload, renal insufficienc
46 early identification of patients at risk for circulatory failure with a much lower false-alarm rate t
47 We postulate a new nosological entity of CSF circulatory failure, with features of AD and NPH.