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1 f interferon regulatory factor 1 and the MHC class II transactivator.
2 hal reporter construct and subsequently, the class II transactivator.
3 his in turn was caused by lack of functional class II transactivator.
4 used CRISPR/Cas9 to disrupt the gene for the class II transactivator.
5 nvestigated in cells stably transfected with class II transactivator.
6  is associated with up-regulation of the MHC class II transactivator, a key transcription factor with
7 t ovariectomy up-regulates IFN-gamma-induced class II transactivator, a multitarget immune modulator,
8 prevented transcription of the gene encoding class II transactivator, a transactivator protein requir
9  also induced by HPIV3 in cells defective in class II transactivator, an important intermediate of th
10            These transacting factors are the class II transactivator and 3 subunits of regulatory fac
11 nsfection of small interfering RNA targeting class II transactivator attenuates major histocompatibil
12 ecules by small interfering RNA targeting of class II transactivator can reduce the capacity of human
13           We previously established that the class II transactivator CIITA binds GTP and disruption o
14                          Coexpression of the class II transactivator CIITA in these cells, which stim
15                                          The class II transactivator CIITA is required for transcript
16  factor RFX (RFX-B, RFX5, and RFXAP) and the class II transactivator CIITA.
17                                      The MHC class II transactivator (CIITA) activates the expression
18                                              Class II transactivator (CIITA) activates the expression
19                          The presence of the class II transactivator (CIITA) activates the transcript
20   The major histocompatibility complex (MHC) class II transactivator (CIITA) acts as a master switch
21                                          The class II transactivator (CIITA) and B cell octamer-bindi
22 une evasion strategies such as inhibition of class II transactivator (CIITA) and MHC-II expression, t
23 ractions of chromatin-modifying enzymes with class II transactivator (CIITA) and relevant DNA-binding
24 ty complex (MHC)-II and its master regulator class II transactivator (CIITA) are downregulated in CML
25 a signal transduction (Jak/Stat pathway) and class II transactivator (CIITA) are required components
26 igation has defined major histocompatibility class II transactivator (CIITA) as a key factor in media
27  lines were used to demonstrate that the MHC class II transactivator (CIITA) can induce surface expre
28                   Here, we show that the MHC class II transactivator (CIITA) can suppress this pathwa
29 e-expressed major histocompatibility complex class II transactivator (CIITA) contributes to myeloma-i
30 ells and in a cell line that is defective in class II transactivator (CIITA) demonstrates that NK cel
31 feron-gamma treatment or transduction of the class II transactivator (CIITA) gene induces class II ex
32                                              Class II transactivator (CIITA) has a distinct function
33 t the major histocompatibility complex (MHC) class II transactivator (CIITA) has antiviral activity a
34 anges in cancer immunity mediated by the MHC class II transactivator (CIITA) have not been extensivel
35                          The role of the MHC class II transactivator (CIITA) in Ag presentation by as
36 scriptional regulation, which is governed by class II transactivator (CIITA) in all cells, microbial
37 P-deficient cells, transfection of exogenous class II transactivator (CIITA) into these RFX-B-deficie
38                                      The MHC class II transactivator (CIITA) is a critical regulator
39                                      The MHC class II transactivator (CIITA) is a critical transcript
40                                              Class II transactivator (CIITA) is a global transcriptio
41                                          The class II transactivator (CIITA) is a highly specific tra
42           Although complex, the co-activator class II transactivator (CIITA) is a key regulator of MH
43                                          The class II transactivator (CIITA) is a master regulator of
44                                              Class II transactivator (CIITA) is a master regulator re
45                                          The class II transactivator (CIITA) is a master transcriptio
46                                          The class II transactivator (CIITA) is a master transcriptio
47                                          The class II transactivator (CIITA) is a transcriptional co-
48                                              Class II transactivator (CIITA) is an unusual transcript
49                                          The class II transactivator (CIITA) is essential for the exp
50                                          The class II transactivator (CIITA) is induced by gamma inte
51  complex along with major histocompatibility class II transactivator (CIITA) is induced by interferon
52                                              Class II transactivator (CIITA) is known as a coactivato
53                                              Class II transactivator (CIITA) is necessary for express
54                                              Class II transactivator (CIITA) is required for both con
55   The major histocompatibility complex (MHC) class II transactivator (CIITA) is the 'master coactivat
56                                          The class II transactivator (CIITA) is the key regulator of
57                                          The class II transactivator (CIITA) is the master integrator
58                                              Class II transactivator (CIITA) is the master regulator
59                                              Class II transactivator (CIITA) is the master regulator
60                                          The class II transactivator (CIITA) is the master regulator
61   The major histocompatibility complex (MHC) class II transactivator (CIITA) is the master regulatory
62                                              Class II transactivator (CIITA) is the unique mediator o
63 amma-inducible transcription at multiple MHC class II transactivator (CIITA) promoters and suppressed
64 s II MHC genes requires the induction of the class II transactivator (CIITA) protein, a master regula
65 ctivation of class II MHC genes requires the class II transactivator (CIITA) protein, a regulator tha
66   The major histocompatibility complex (MHC) class II transactivator (CIITA) regulates the expression
67 s strictly dependent upon the binding of the class II transactivator (CIITA) to the highly conserved
68                         The discovery of the class II transactivator (CIITA) transcription factor, an
69  negative trans-acting factors that suppress class II transactivator (CIITA) transcription.
70                                Expression of class II transactivator (CIITA) types I and III, but not
71    Depending on whether MHC-II genes and the class II transactivator (CIITA) were being expressed, tw
72                                              Class II transactivator (CIITA), a coactivator required
73 on of MHC II antigen A alpha (H2-Aalpha) and Class II transactivator (Ciita), a master regulator of M
74                         Examples include MHC class II transactivator (CIITA), a master transcriptiona
75 have now transduced tumor cells with the MHC class II transactivator (CIITA), a regulatory gene that
76              The transcriptional coactivator class II transactivator (CIITA), although predominantly
77 tion analysis demonstrated that induction of class II transactivator (CIITA), and consequently, HLA-D
78         Recently, a non-DNA binding protein, class II transactivator (CIITA), has been shown to be re
79 ter regulator of MHC class II transcription, class II transactivator (CIITA), identified HLA-DOA and
80 re, inhibition of promoter activities of MHC class II transactivator (CIITA), IFN-gamma-activated sit
81 ated inhibition of MNV replication in vitro, class II transactivator (CIITA), interferon regulatory f
82 mary and immortalized astrocytes up-regulate class II transactivator (CIITA), invariant chain (Ii) (p
83                              An NLR protein, class II transactivator (CIITA), is a key regulator of M
84        We show that the promoter IV (pIV) of class II transactivator (CIITA), the master regulator of
85 anism involved interactions between CTCF and class II transactivator (CIITA), the master regulator of
86 uires recruitment of a master regulator, the class II transactivator (CIITA), to the MHC class II pro
87 orrelating to a decrease in the mRNA for the class II transactivator (CIITA), whereas CIITA expressio
88 nodeficiency have mutations in a gene termed class II transactivator (CIITA), which coordinately cont
89  non-DNA-binding master regulator of MHC-II, class II transactivator (CIITA), which is crucial for en
90 t the major histocompatibility complex (MHC) class II transactivator (CIITA), which is required for e
91  of several immune function genes, including class II transactivator (CIITA), which regulates class I
92                               Therefore, MHC class II transactivator (CIITA)-deficient mice that do n
93 in line with the role of CREBBP in promoting class II transactivator (CIITA)-dependent transcriptiona
94 ion of this family of genes requires the MHC class II transactivator (CIITA).
95 I BLS patient is a 24 aa deletion in the MHC class II transactivator (CIITA).
96 ators, such as regulatory factor X (RFX) and class II transactivator (CIITA).
97 tch for the expression of these genes is the class II transactivator (CIITA).
98  (HLA-II)-inducing transcription coordinator class II transactivator (CIITA).
99 gene transcription through regulation of the class II transactivator (CIITA).
100 atory factor for X-box 5 (RFX5) complex with class II transactivator (CIITA).
101 xpression is negatively regulated by the MHC class II transactivator (CIITA).
102 factor for X-box 5 (RFX5) complex as well as class II transactivator (CIITA).
103 o involve a defect in IFN-gamma induction of class II transactivator (CIITA).
104  due to differences in the expression of the class II transactivator (CIITA).
105 led by the master transcriptional regulator, class II transactivator (CIITA).
106  a specific transcriptional coactivator, the class II transactivator (CIITA).
107 ications and gene expression mediated by the class II transactivator (CIITA).
108                          One of these is the class II transactivator (CIITA).
109 s due to a decrease in the expression of the class II transactivator (CIITA).IMPORTANCE Human cytomeg
110 of the key mechanisms includes repression of class-II transactivator (CIITA) and MHC-II expression in
111 ages is mediated via epigenetic silencing of class II transactivator, CIITA, the master regulator of
112 y the major histocompatibility complex (MHC) class II transactivator, CIITA, which binds to myogenin
113          This expression is initiated by the class II transactivator, CIITA.
114 g the major histocompatibility complex (MHC) class II transactivator, CIITA.
115 3 that inhibits promoter function of the HLA class II transactivator, decreasing expression of genes
116                                     However, class II transactivator-deficient mice can prime a subop
117 tion and targeted IFN response factor 1- and class II transactivator-dependent and independent promot
118 pathways including Stat-3, beta-catenin, and class II transactivator-dependent antigen presentation.
119                             Up-regulation of class II transactivator derives from increased productio
120            Thus, increased IFN-gamma-induced class II transactivator expression and the resulting enh
121 henotype can be rescued by reintroduction of class II transactivator expression.
122          STAT1 is an activator of CIITA, the class II transactivator for MHC II expression; CIITA exp
123                                      The MHC class II transactivator gene (CIITA) coordinately contro
124                                      The MHC class II transactivator gene (CIITA) is an important tra
125 was accompanied by activation of a repressed class II transactivator gene in a plasma cell tumor but,
126 t from a pig transgenic for a mutant (human) class II transactivator gene, resulting in down-regulati
127 in reaction analysis for gene transcripts of class II transactivator, HLA-DRagr;, and HLA-DRbeta1 sho
128 nducible genes-MHC class II gene E beta; MHC class II transactivator; IFN regulatory factor-1; and Mg
129                            Re-expressing the class II transactivator in the PELs increased expression
130                     HLA-II downregulation by class II transactivator knockout did not affect the exte
131          Mounting evidence suggests that the class II transactivator modulates promoter accessibility
132 demonstrate that NOD mice lacking the CIITA (class II transactivator) molecule, and hence deficient i
133 tion of the IFN gamma-responsive chromosomal class II transactivator promoter revealed that MCMV infe
134                                  Because the class II transactivator protein (CIITA) induces Ia and I
135 by either IFN-alpha/beta (Mx1) or IFN-gamma (class II transactivator protein [CIITA] and inducible ni
136 gen-activated protein kinase, NF-kappaB, the class II transactivator, RFX5, and the IFN regulatory fa
137 ion arises from biallelic gene disruption in class II transactivator that leaves other essential prop
138              One such NLR protein is the MHC class II transactivator, the master regulator of MHC cla
139 ested that NLRC5 acts in a manner similar to class II transactivator to drive MHC expression and reve
140 ss II was induced in the apparent absence of class II transactivator transcripts.
141 e selective: while transcription of CD64 and class II transactivator were decreased, certain other IF
142          C57BL/6 (B6) mice deficient for the class II transactivator, which have defects in MHC class
143 d proteins form a platform that attracts the class II transactivator, which initiates and elongates M

 
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