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1 l biology, cancer biology, neuroscience, and clinical pathology.
2 at holds promise for both basic research and clinical pathology.
3  dPGMs allows us to correlate structure with clinical pathology.
4 ential for a meaningful application of DL in clinical pathology.
5 o become an indispensable diagnostic tool in clinical pathology.
6  morphologic similarity to colorectal cancer clinical pathology.
7 ssue turnover even in the absence of evident clinical pathology.
8  variants, which segregate with the observed clinical pathology.
9 , with accuracy assessed in correlation with clinical pathology.
10 ludes cataract development among its diverse clinical pathologies.
11 ders (EDs) and have been implicated in their clinical pathologies.
12  functions that could contribute to COVID-19 clinical pathologies.
13 sregulation has been associated with diverse clinical pathologies.
14 d on a thorough investigative toxicology and clinical pathology analysis, anticoagulation effects in
15 ariants (SNVs) in key T cell genes can drive clinical pathologies and could be repurposed to improve
16 cimen, and it can find broad applications in clinical pathology and disease mechanistic studies.
17 asic biochemical research to applications in clinical pathology and intraoperative surgery.
18 oncology, medical oncology, medical imaging, clinical pathology and lab medicine, social work, nutrit
19 nd pathologists were blinded to preoperative clinical, pathology, and imaging data.
20             The model is useful for managing clinical, pathology, and molecular data on >1300 prostat
21  human diseases that accurately recapitulate clinical pathology are indispensable for understanding m
22 can Pathologists (CAP), American Society for Clinical Pathology (ASCP), and the American Society of C
23             Methods The American Society for Clinical Pathology (ASCP), College of American Pathologi
24 (NHP) using survivability, biotelemetry, and clinical pathology assessments.
25 ischemia-reperfusion (IR) injury is a common clinical pathology associated with high mortality.
26 xpression is associated with cancer, but the clinical pathology associated with survivin downregulati
27 on in serum viral titer, and improvements in clinical pathology biomarker levels and lung histology c
28 terozygous parents do not show any conserved clinical pathology but report multiple incidences of int
29 DPR bind to ligands that are associated with clinical pathology, cADPR and CD38 represent novel drug
30 he difference in charges, most of which were clinical pathology charges (54.2%), persisted into the s
31 ising and accelerating the interpretation of clinical pathology data, thereby improving the diagnosti
32 ry diagnostic testing for 2 years, including clinical pathology, diagnostic imaging, and special proc
33 nomic profiles do not always match consensus clinical pathology dysplasia grades.
34 g leveraged for chemical biology studies and clinical pathology, enabled by a diverse toolkit of new
35 tify their nodal levels and sent for routine clinical pathology evaluation.
36 l rich RNA biology in FFPE tissues to aid in clinical pathology evaluation.
37 ding insights into celiac pathogenesis using clinical pathology FFPE samples, and can stimulate new a
38                                              Clinical pathology findings included coagulopathy, leuko
39 aves the way for a human-DL collaboration in clinical pathology for a better translation of DL techni
40 he lifespan, and impairments associated with clinical pathology from a variety of disorders.
41 sed in the context of emerging findings from clinical pathology, functional neuroimaging and other ap
42 precede MSI and may have utility in defining clinical pathology in the absence of features of maligna
43 for a better translation of DL techniques in clinical pathology in the near future.
44 IM technology could be further translated to clinical pathologies including but not limited to trauma
45 olic events were the likely cause of various clinical pathologies, including heart failure, renal and
46 iectasia (A-T) patients can develop multiple clinical pathologies, including neuronal degeneration, a
47                              ETBF can induce clinical pathology, including inflammatory diarrhea, alt
48 RNA sequencing, we identify how age, sex and clinical pathology influence microglia gene expression a
49 tance of Wnt signaling in development and in clinical pathologies is underscored by the large number
50 Historically, the trend toward automation in clinical pathology laboratories has largely bypassed the
51   AnimalGAN, a GAN method to simulate 38 rat clinical pathology measures, was developed with signific
52                                    The major clinical pathologies of this disease include splitting o
53                                              Clinical pathology of IBD is complicated; however, hyper
54                           Differences in the clinical pathology of mammalian prion diseases reflect d
55 ion between TAM-associated spheroids and the clinical pathology of ovarian cancer.
56 ections can induce damage that resembles the clinical pathology of rejection, and this complicates ac
57 ation of articular cartilage underscores the clinical pathology of temporomandibular joint osteoarthr
58 ating the exocrine dysfunction aspect of the clinical pathology of the autoimmune disease.
59                                          The clinical pathology of this complex entity is reviewed an
60 e millimeters) in the jejunum and ileum; and clinical pathology parameters of dehydration, electrolyt
61                                              Clinical pathology parameters were monitored to determin
62 uating terminal body weights, organ weights, clinical pathology parameters, PIXImus mouse densitometr
63                   For each case, we reviewed clinical, pathology, radiology, and endoscopy findings.
64 ical Pathology, and the American Society for Clinical Pathology recommend cessation of cervical cance
65 virus (HCMV) strains sequenced directly from clinical pathology samples were investigated, focusing o
66 nese (DHRD/ML), a rare blinding disease with clinical pathology similar to age-related macular degene
67 introduced during routine tissue fixation of clinical pathology specimens severely hampers chromatin
68 combinations with slice electrophysiology or clinical pathology specimens.
69 niques have enabled deep characterization of clinical pathologies that remain poorly understood, prov
70 n in a wide range of cognitive processes and clinical pathologies, the mechanisms underlying alpha ge
71  28 days after inoculation to track multiple clinical, pathology, virology, and immunology outcomes.
72                 Idua(-/-) mice share similar clinical pathology with patients, including the accumula